BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients (2011)
- Authors:
- Carvalho, Daniel Diniz de - Universidade de São Paulo (USP)
- Binato, R.
- Pereira, Welbert de Oliveira - Universidade de São Paulo (USP)
- Leroy, Janine Marie Gisele - Universidade de São Paulo (USP)
- Colassanti, M. D.
- Proto-Siqueira, R.
- Silva, Ana Elisa Barreiros Bueno da - Universidade de São Paulo (USP)
- Zago, Marco Antonio
- Zanichelli, M. A.
- Abdelhay, E.
- Castro, Fabíola Attié de
- Jacysyn, J. F.
- Amarante-Mendes, João Gustavo Pessini
- USP affiliated authors: ZAGO, MARCO ANTONIO - FMRP ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB ; CASTRO, FABÍOLA ATTIÉ DE - FCFRP
- Unidades: FMRP; ICB; FCFRP
- DOI: 10.1038/onc.2010.409
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Basingstoke
- Date published: 2011
- Source:
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CARVALHO, Daniel Diniz de et al. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, v. 30, n. 2, p. 223-233, 2011Tradução . . Disponível em: https://doi.org/10.1038/onc.2010.409. Acesso em: 28 set. 2024. -
APA
Carvalho, D. D. de, Binato, R., Pereira, W. de O., Leroy, J. M. G., Colassanti, M. D., Proto-Siqueira, R., et al. (2011). BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, 30( 2), 223-233. doi:10.1038/onc.2010.409 -
NLM
Carvalho DD de, Binato R, Pereira W de O, Leroy JMG, Colassanti MD, Proto-Siqueira R, Silva AEBB da, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 set. 28 ] Available from: https://doi.org/10.1038/onc.2010.409 -
Vancouver
Carvalho DD de, Binato R, Pereira W de O, Leroy JMG, Colassanti MD, Proto-Siqueira R, Silva AEBB da, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 set. 28 ] Available from: https://doi.org/10.1038/onc.2010.409 - Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression
- Conversion of CD95 (Fas) type II into type I signaling by sub-lethal doses of cycloheximide
- T lymphoma murine models to study the impact of ectopic expression of anti-apoptotic oncogenes
- Cosmomycin D, an anthracycline more potent than Doxorubicin for induction of tumor cell death, synergize with imatinib mesilate to induce apoptosis in Bcr-Abl-positive cells
- Study of pro and anti-apoptotic genes to understand the resistance of apoptosis mediated by Bcr-Abl
- BH3 mimetics and TKI combined therapy for Chronic Myeloid Leukemia
- PRAME expression is associated with down-regulation of trail in chronic myeloid leukemia
- Prame as a secondary target for BCR-ABL-positive leukemias
- Prame as a secondary target for Bcr-Abl-positive leukemia
- BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients
Informações sobre o DOI: 10.1038/onc.2010.409 (Fonte: oaDOI API)
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