Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression (2005)
- Authors:
- USP affiliated authors: MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB ; CASTRO, FABÍOLA ATTIÉ DE - FCFRP
- Unidades: ICB; FCFRP
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Brazilian Society of Immunology
- Publisher place: São Pedro
- Date published: 2005
- Source:
- Título: Resumos
- Conference titles: Meeting of the Brazilian Society of Immunology
-
ABNT
PANTALEÃO, C. et al. Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression. 2005, Anais.. São Pedro: Brazilian Society of Immunology, 2005. . Acesso em: 27 jan. 2026. -
APA
Pantaleão, C., Jacysyn, J. F., Castro, F. A. de, & Amarante-Mendes, J. G. P. (2005). Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression. In Resumos. São Pedro: Brazilian Society of Immunology. -
NLM
Pantaleão C, Jacysyn JF, Castro FA de, Amarante-Mendes JGP. Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression. Resumos. 2005 ;[citado 2026 jan. 27 ] -
Vancouver
Pantaleão C, Jacysyn JF, Castro FA de, Amarante-Mendes JGP. Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression. Resumos. 2005 ;[citado 2026 jan. 27 ] - Conversion of CD95 (Fas) type II into type I signaling by sub-lethal doses of cycloheximide
- Study of pro and anti-apoptotic genes to understand the resistance of apoptosis mediated by Bcr-Abl
- Cosmomycin D, an anthracycline more potent than Doxorubicin for induction of tumor cell death, synergize with imatinib mesilate to induce apoptosis in Bcr-Abl-positive cells
- T lymphoma murine models to study the impact of ectopic expression of anti-apoptotic oncogenes
- BH3 mimetics and TKI combined therapy for Chronic Myeloid Leukemia
- Apoptotic genes expression in head and neck squamous cell carcinoma (HNSCC) patients
- Indoleamine 2,3-dioxygenase expression in chronic myelogenous leukemia patients: association with disease progression
- Down regulation of Hematopoietic SH2 domain containing (HSH2D) in the progression of the chronic myelogenous leukemia
- BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients
- Expression of pro-apoptotic bid and bik genes is decreased in chronic myeloid leukemia and low levels of bik MRNA are associated with lack of response to imatinib treatment
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