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Artigo de periodico
Aldosterone, inflammation, immune system, and hypertension (2021)
Ferreira, Nathanne dos Santos; Tostes, Rita de Cássia Aleixo ; Paradis, Pierre; Schiffrin, Ernesto L.
Source: American Journal of Hypertension. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, SISTEMA IMUNE, LINFÓCITOS, MONÓCITOS, MACRÓFAGOS
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ABNT
FERREIRA, Nathanne dos Santos et al. Aldosterone, inflammation, immune system, and hypertension. American Journal of Hypertension, v. 34, n. 1, p. 15-27, 2021Tradução . . Disponível em: https://doi.org/10.1093/ajh/hpaa137. Acesso em: 10 nov. 2024.
APA
Ferreira, N. dos S., Tostes, R. de C. A., Paradis, P., & Schiffrin, E. L. (2021). Aldosterone, inflammation, immune system, and hypertension. American Journal of Hypertension, 34( 1), 15-27. doi:10.1093/ajh/hpaa137
NLM
Ferreira N dos S, Tostes R de CA, Paradis P, Schiffrin EL. Aldosterone, inflammation, immune system, and hypertension [Internet]. American Journal of Hypertension. 2021 ; 34( 1): 15-27.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/ajh/hpaa137
Vancouver
Ferreira N dos S, Tostes R de CA, Paradis P, Schiffrin EL. Aldosterone, inflammation, immune system, and hypertension [Internet]. American Journal of Hypertension. 2021 ; 34( 1): 15-27.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/ajh/hpaa137
Artigo de periodico
Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium (2021)
Potje, Simone Regina ; Isbatan, Ayman; Tostes, Rita de Cássia Aleixo ; Bendhack, Lusiane Maria ; Dull, Randal O.; Carvalho-de-Souza, João Luis; Chignalia, Andréia Zago
Source: Pharmacological Research. Unidades: FMRP, FCFRP
Subjects: AORTA TORÁCICA, AORTA TORÁCICA, PRESSÃO SANGUÍNEA, CÁLCIO, HIPERTENSÃO, NOREPINEFRINA
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ABNT
POTJE, Simone Regina et al. Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium. Pharmacological Research, v. 172, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2021.105813. Acesso em: 10 nov. 2024.
APA
Potje, S. R., Isbatan, A., Tostes, R. de C. A., Bendhack, L. M., Dull, R. O., Carvalho-de-Souza, J. L., & Chignalia, A. Z. (2021). Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium. Pharmacological Research, 172, 1-11. doi:10.1016/j.phrs.2021.105813
NLM
Potje SR, Isbatan A, Tostes R de CA, Bendhack LM, Dull RO, Carvalho-de-Souza JL, Chignalia AZ. Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium [Internet]. Pharmacological Research. 2021 ; 172 1-11.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.phrs.2021.105813
Vancouver
Potje SR, Isbatan A, Tostes R de CA, Bendhack LM, Dull RO, Carvalho-de-Souza JL, Chignalia AZ. Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium [Internet]. Pharmacological Research. 2021 ; 172 1-11.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.phrs.2021.105813
Artigo de periodico
Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome (2020)
Silva, Josiane Fernandes da; Olivon, Vania Cláudia; Mestriner, Fabiola Leslie Antunes Cardoso; Zanotto, Camila Ziliotto; Ferreira, Raphael Gomes; Ferreira, Nathanne dos Santos; Silva, Carlos Alberto Aguiar da; Luiz, João Paulo Mesquita; Alves, Juliano Vilela; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, INFLAMAÇÃO, SEPSE, CITOCINAS, NEUTRÓFILOS
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ABNT
SILVA, Josiane Fernandes da et al. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome. Frontiers in Physiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.01614. Acesso em: 10 nov. 2024.
APA
Silva, J. F. da, Olivon, V. C., Mestriner, F. L. A. C., Zanotto, C. Z., Ferreira, R. G., Ferreira, N. dos S., et al. (2020). Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome. Frontiers in Physiology, 10. doi:10.3389/fphys.2019.01614
NLM
Silva JF da, Olivon VC, Mestriner FLAC, Zanotto CZ, Ferreira RG, Ferreira N dos S, Silva CAA da, Luiz JPM, Alves JV, Fazan Júnior R, Cunha F de Q, Alves Filho JCF, Tostes R de CA. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fphys.2019.01614
Vancouver
Silva JF da, Olivon VC, Mestriner FLAC, Zanotto CZ, Ferreira RG, Ferreira N dos S, Silva CAA da, Luiz JPM, Alves JV, Fazan Júnior R, Cunha F de Q, Alves Filho JCF, Tostes R de CA. Acute increase in O-GlcNAc improves survival in mice with LPS-induced systemic inflammatory response syndrome [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fphys.2019.01614
Artigo de periodico
Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation (2020)
Amorim, Mateus Ramos; Deus, Júnia Lara de; Pereira, Camila André; Silva, Luiz Eduardo Virgilio da ; Borges, Gabriela Silva; Ferreira, Nathanne dos Santos; Batalhão, Marcelo Eduardo; Antunes-Rodrigues, José; Cárnio, Evelin Capellari ; Tostes, Rita de Cássia Aleixo ; Branco, Luiz Guilherme de Siqueira
Source: Scientific Reports. Unidades: EERP, FMRP, FORP, EEFE
Subjects: BARORREFLEXO, IMUNOLOGIA, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, HEMODINÂMICA, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS
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ABNT
AMORIM, Mateus Ramos et al. Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation. Scientific Reports, v. 10, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-63949-x. Acesso em: 10 nov. 2024.
APA
Amorim, M. R., Deus, J. L. de, Pereira, C. A., Silva, L. E. V. da, Borges, G. S., Ferreira, N. dos S., et al. (2020). Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation. Scientific Reports, 10( 1). doi:10.1038/s41598-020-63949-x
NLM
Amorim MR, Deus JL de, Pereira CA, Silva LEV da, Borges GS, Ferreira N dos S, Batalhão ME, Antunes-Rodrigues J, Cárnio EC, Tostes R de CA, Branco LG de S. Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-020-63949-x
Vancouver
Amorim MR, Deus JL de, Pereira CA, Silva LEV da, Borges GS, Ferreira N dos S, Batalhão ME, Antunes-Rodrigues J, Cárnio EC, Tostes R de CA, Branco LG de S. Baroreceptor denervation reduces inflammatory status but worsens cardiovascular collapse during systemic inflammation [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-020-63949-x
Artigo de periodico
Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats (2019)
Lataro, Renata M.; Silva, Marcondes Alves Barbosa da; Mestriner, Fabiola Leslie Antunes Cardoso; Cáu, Stêfany Bruno de Assis; Tostes, Rita de Cássia Aleixo ; Salgado, Hélio César
Source: American Journal of Hypertension. Unidade: FMRP
Subjects: HIPERTENSÃO, PRESSÃO SANGUÍNEA, RATOS
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ABNT
LATARO, Renata M. et al. Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats. American Journal of Hypertension, v. 32, n. 6, p. 579-587, 2019Tradução . . Disponível em: https://doi.org/10.1093/ajh/hpz036. Acesso em: 10 nov. 2024.
APA
Lataro, R. M., Silva, M. A. B. da, Mestriner, F. L. A. C., Cáu, S. B. de A., Tostes, R. de C. A., & Salgado, H. C. (2019). Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats. American Journal of Hypertension, 32( 6), 579-587. doi:10.1093/ajh/hpz036
NLM
Lataro RM, Silva MAB da, Mestriner FLAC, Cáu SB de A, Tostes R de CA, Salgado HC. Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats [Internet]. American Journal of Hypertension. 2019 ; 32( 6): 579-587.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/ajh/hpz036
Vancouver
Lataro RM, Silva MAB da, Mestriner FLAC, Cáu SB de A, Tostes R de CA, Salgado HC. Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats [Internet]. American Journal of Hypertension. 2019 ; 32( 6): 579-587.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/ajh/hpz036
Trabalho de evento-resumo periodico
Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis (2014)
Olivon, Vânia Cláudia; Ferreira, Raphael G.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Passaglia, Rita de Cassia Aleixo Tostes
Source: Hypertension. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENZIMAS, SEPSE, PRESSÃO SANGUÍNEA
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ABNT
OLIVON, Vânia Cláudia et al. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8. Acesso em: 10 nov. 2024. , 2014
APA
Olivon, V. C., Ferreira, R. G., Mestriner, F. L. A. C., Alves Filho, J. C. F., Cunha, F. de Q., & Passaglia, R. de C. A. T. (2014). Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis. Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
NLM
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis [Internet]. Hypertension. 2014 ; 64[citado 2024 nov. 10 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
Vancouver
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O-GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS-induced sepsis [Internet]. Hypertension. 2014 ; 64[citado 2024 nov. 10 ] Available from: http://hyper.ahajournals.org/content/64/Suppl_1/A336.abstract?sid=845389a1-7442-4900-9bf5-22d8f9c204c8
Trabalho de evento-resumo
Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis (2014)
Olivon, Vânia Cláudia; Ferreira, Raphael G.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Passaglia, Rita de Cassia Aleixo Tostes
Source: Final Program. Conference titles: High Blood Pressure Research Scientific Sessions - HBPR. Unidade: FMRP
Subjects: ENZIMAS, SEPSE, PRESSÃO SANGUÍNEA
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ABNT
OLIVON, Vânia Cláudia et al. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis. 2014, Anais.. San Francisco: AHA, 2014. Disponível em: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf. Acesso em: 10 nov. 2024.
APA
Olivon, V. C., Ferreira, R. G., Mestriner, F. L. A. C., Alves Filho, J. C. F., Cunha, F. de Q., & Passaglia, R. de C. A. T. (2014). Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis. In Final Program. San Francisco: AHA. Recuperado de http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
NLM
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Vancouver
Olivon VC, Ferreira RG, Mestriner FLAC, Alves Filho JCF, Cunha F de Q, Passaglia R de CAT. Acute increase in O‐GlcNAcylation reduces the release of cytokines and attenuates hypotension in mice with LPS‐induced sepsis [Internet]. Final Program. 2014 ;[citado 2024 nov. 10 ] Available from: http://my.americanheart.org/idc/groups/ahamah-public/@wcm/@sop/@scon/documents/downloadable/ucm_468057.pdf
Artigo de periodico
Anti-platelet therapy with clopidogrel prevents endothelial dysfunction and vascular remodeling in aortas from hypertensive rats (2014)
Giachini, Fernanda R.; Leite, Romulo; Osmond, David A.; Lima, Victor V.; Inscho, Edward W.; Webb, R. Clinton; Passaglia, Rita de Cassia Aleixo Tostes
Source: PLoS One. Unidade: FMRP
Subjects: AORTA (EFEITOS DE DROGAS), PRESSÃO SANGUÍNEA, ENDOTÉLIO VASCULAR (FISIOPATOLOGIA), HIPERTENSÃO, MODELOS ANIMAIS DE DOENÇAS
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ABNT
GIACHINI, Fernanda R. et al. Anti-platelet therapy with clopidogrel prevents endothelial dysfunction and vascular remodeling in aortas from hypertensive rats. PLoS One, v. 9, n. 3, p. e91890-1 - e91890-8, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0091890. Acesso em: 10 nov. 2024.
APA
Giachini, F. R., Leite, R., Osmond, D. A., Lima, V. V., Inscho, E. W., Webb, R. C., & Passaglia, R. de C. A. T. (2014). Anti-platelet therapy with clopidogrel prevents endothelial dysfunction and vascular remodeling in aortas from hypertensive rats. PLoS One, 9( 3), e91890-1 - e91890-8. doi:10.1371/journal.pone.0091890
NLM
Giachini FR, Leite R, Osmond DA, Lima VV, Inscho EW, Webb RC, Passaglia R de CAT. Anti-platelet therapy with clopidogrel prevents endothelial dysfunction and vascular remodeling in aortas from hypertensive rats [Internet]. PLoS One. 2014 ; 9( 3): e91890-1 - e91890-8.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0091890
Vancouver
Giachini FR, Leite R, Osmond DA, Lima VV, Inscho EW, Webb RC, Passaglia R de CAT. Anti-platelet therapy with clopidogrel prevents endothelial dysfunction and vascular remodeling in aortas from hypertensive rats [Internet]. PLoS One. 2014 ; 9( 3): e91890-1 - e91890-8.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0091890
Artigo de periodico
Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia (2011)
Yogi, Álvaro; Callera, Gláucia Elena; O'Connor, Sarah E.; He, Ying; Corrêa, José W.; Passaglia, Rita de Cassia Aleixo Tostes; Mazur, Andrzej; Touyz, Rhian M.
Source: Journal of Hypertension. Unidade: FMRP
Subjects: RIM (DANOS), MAGNÉSIO, FÁRMACOS, INFLAMAÇÃO, PRESSÃO SANGUÍNEA
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ABNT
YOGI, Álvaro et al. Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia. Journal of Hypertension, v. 29, n. 7, p. 1400-1410, 2011Tradução . . Disponível em: https://doi.org/10.1097/hjh.0b013e32834786d6. Acesso em: 10 nov. 2024.
APA
Yogi, Á., Callera, G. E., O'Connor, S. E., He, Y., Corrêa, J. W., Passaglia, R. de C. A. T., et al. (2011). Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia. Journal of Hypertension, 29( 7), 1400-1410. doi:10.1097/hjh.0b013e32834786d6
NLM
Yogi Á, Callera GE, O'Connor SE, He Y, Corrêa JW, Passaglia R de CAT, Mazur A, Touyz RM. Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia [Internet]. Journal of Hypertension. 2011 ; 29( 7): 1400-1410.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1097/hjh.0b013e32834786d6
Vancouver
Yogi Á, Callera GE, O'Connor SE, He Y, Corrêa JW, Passaglia R de CAT, Mazur A, Touyz RM. Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia [Internet]. Journal of Hypertension. 2011 ; 29( 7): 1400-1410.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1097/hjh.0b013e32834786d6

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