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Artigo de periodico
Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice (2024)
Franco, Thaina Maquedo ; Tavares, Mariana Rosolen ; Novaes, Leonardo Santana ; Peixoto-Santos, Jose Eduardo ; Araujo, Ronaldo Carvalho ; Bader, Michael ; Wasinski, Frederick ; Munhoz, Carolina Demarchi ; Donato Junior, José
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, BRADICININA, INFLAMAÇÃO, SISTEMA NERVOSO CENTRAL, PRESSÃO SANGUÍNEA, PROTEÍNAS G, RECEPTORES DE CATECOLAMINA, RECEPTORES DE DOPAMINA, CAMUNDONGOS, PESOS E MEDIDAS CORPORAIS, AMINOÁCIDOS
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FRANCO, Thaina Maquedo et al. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, v. 25, n. 3, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25031490. Acesso em: 07 nov. 2024.
APA
Franco, T. M., Tavares, M. R., Novaes, L. S., Peixoto-Santos, J. E., Araujo, R. C., Bader, M., et al. (2024). Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, 25( 3), 14 . doi:10.3390/ijms25031490
NLM
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/ijms25031490
Vancouver
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/ijms25031490
Artigo de periodico
Kinin B1 receptor modulates glucose homeostasis and physical exercise capacity by altering adrenal catecholamine synthesis and secretion (2023)
Gregnani, Marcos Fernandes ; Budu, Alexandre ; Batista, Rogério Oliveira; Ornellas, Fábio Henrique; Estrela, Gabriel Rufino ; Arruda, Adriano Cleis; Lima, Leandro Ceotto Freitas ; Kremer, Jean Lucas ; Mendes, Lys Angela Favaroni ; Casarini, Dulce Elena ; Oyama, Lila Missae ; Bader, Michael ; Araujo, Ronaldo Carvalho ; Lotfi, Claudimara Ferini Pacicco
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: CAMUNDONGOS, EXERCÍCIO FÍSICO, CATECOLAMINAS, GLICOSE, LEPTINA, CININAS
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GREGNANI, Marcos Fernandes et al. Kinin B1 receptor modulates glucose homeostasis and physical exercise capacity by altering adrenal catecholamine synthesis and secretion. Molecular and Cellular Endocrinology, v. 579, p. 16 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2023.112085. Acesso em: 07 nov. 2024.
APA
Gregnani, M. F., Budu, A., Batista, R. O., Ornellas, F. H., Estrela, G. R., Arruda, A. C., et al. (2023). Kinin B1 receptor modulates glucose homeostasis and physical exercise capacity by altering adrenal catecholamine synthesis and secretion. Molecular and Cellular Endocrinology, 579, 16 . doi:10.1016/j.mce.2023.112085
NLM
Gregnani MF, Budu A, Batista RO, Ornellas FH, Estrela GR, Arruda AC, Lima LCF, Kremer JL, Mendes LAF, Casarini DE, Oyama LM, Bader M, Araujo RC, Lotfi CFP. Kinin B1 receptor modulates glucose homeostasis and physical exercise capacity by altering adrenal catecholamine synthesis and secretion [Internet]. Molecular and Cellular Endocrinology. 2023 ; 579 16 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.mce.2023.112085
Vancouver
Gregnani MF, Budu A, Batista RO, Ornellas FH, Estrela GR, Arruda AC, Lima LCF, Kremer JL, Mendes LAF, Casarini DE, Oyama LM, Bader M, Araujo RC, Lotfi CFP. Kinin B1 receptor modulates glucose homeostasis and physical exercise capacity by altering adrenal catecholamine synthesis and secretion [Internet]. Molecular and Cellular Endocrinology. 2023 ; 579 16 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.mce.2023.112085
Artigo de periodico
Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression (2023)
Rubenich, Dominique S. ; Souza, Priscila O. de ; Omizollo, Natalia; Aubin, Mariana R. ; Basso, Paulo J. ; Silva, Luisa M. ; Silva, Eloisa M. ; Teixeira, Fernanda C. ; Gentil, Gabriela F. S.; Domagalski, Jordana ; Cunha, Maico T. ; Gadelha, Kerolainy A. ; Diel, Leonardo F. ; Gelsleichter, Nicolly E. ; Rubenich, Aline S.; Lenz, Gabriela S. ; Abreu, Aline M. ; Kroeff, Giselle M.; Ana H. Paz ; Visioli, Fernanda ; Lamers, Marcelo L. ; Wink, Marcia R. ; Worm, Paulo V. ; Araújo, Anelise B. ; Sévigny, Jean ; Ludwig, Nils ; Braganho, Elizandra ; Câmara, Niels Olsen Saraiva
Source: Frontiers in immunology. Unidade: ICB
Subjects: CAMUNDONGOS, CÉLULAS CULTIVADAS DE TUMOR, NEUTRÓFILOS, GLIOMA, NEOPLASIAS CEREBRAIS, DOENÇAS DO SISTEMA NERVOSO CENTRAL, METABOLISMO ENERGÉTICO, CITOCINAS, FÁRMACOS IMUNOSSUPRESSORES
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RUBENICH, Dominique S. et al. Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression. Frontiers in immunology, v. 14, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1183465. Acesso em: 07 nov. 2024.
APA
Rubenich, D. S., Souza, P. O. de, Omizollo, N., Aubin, M. R., Basso, P. J., Silva, L. M., et al. (2023). Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression. Frontiers in immunology, 14. doi:10.3389/fimmu.2023.1183465
NLM
Rubenich DS, Souza PO de, Omizollo N, Aubin MR, Basso PJ, Silva LM, Silva EM, Teixeira FC, Gentil GFS, Domagalski J, Cunha MT, Gadelha KA, Diel LF, Gelsleichter NE, Rubenich AS, Lenz GS, Abreu AM, Kroeff GM, Ana H. Paz, Visioli F, Lamers ML, Wink MR, Worm PV, Araújo AB, Sévigny J, Ludwig N, Braganho E, Câmara NOS. Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression [Internet]. Frontiers in immunology. 2023 ; 14[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fimmu.2023.1183465
Vancouver
Rubenich DS, Souza PO de, Omizollo N, Aubin MR, Basso PJ, Silva LM, Silva EM, Teixeira FC, Gentil GFS, Domagalski J, Cunha MT, Gadelha KA, Diel LF, Gelsleichter NE, Rubenich AS, Lenz GS, Abreu AM, Kroeff GM, Ana H. Paz, Visioli F, Lamers ML, Wink MR, Worm PV, Araújo AB, Sévigny J, Ludwig N, Braganho E, Câmara NOS. Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression [Internet]. Frontiers in immunology. 2023 ; 14[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fimmu.2023.1183465
Artigo de periodico
Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4 (2023)
Ferreira, Clílton Kraüss de Oliveira ; Campolim, Clara Machado ; Zordão, Olívia Pizetta ; Simabuco, Fernando Moreira ; Anaruma, Chadi Pellegrini ; Pereira, Rodrigo Martins; Boico, Vitor Ferreira ; Salvino, Luiz Guilherme ; Costa, Maira Maftoum ; Ruiz, Nathalia Quintero ; Moura, Leandro Pereira de ; Saad, Mario Jose Abdalla ; Kim, Young-Bum ; Patricia de Oliveira Prada; Costa, Soraia Katia Pereira
Source: Toxicology Reports. Unidade: ICB
Subjects: FARMACOLOGIA, POLUIÇÃO ATMOSFÉRICA, MACRÓFAGOS, TECIDO ADIPOSO, INFLAMAÇÃO, HOMEOSTASE, CAMUNDONGOS, METABOLISMO ENERGÉTICO, GLICEMIA, GLICOSE, RESISTÊNCIA À INSULINA
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FERREIRA, Clílton Kraüss de Oliveira et al. Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4. Toxicology Reports, v. 11, p. 10-22, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.toxrep.2023.06.002. Acesso em: 07 nov. 2024.
APA
Ferreira, C. K. de O., Campolim, C. M., Zordão, O. P., Simabuco, F. M., Anaruma, C. P., Pereira, R. M., et al. (2023). Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4. Toxicology Reports, 11, 10-22. doi:10.1016/j.toxrep.2023.06.002
NLM
Ferreira CK de O, Campolim CM, Zordão OP, Simabuco FM, Anaruma CP, Pereira RM, Boico VF, Salvino LG, Costa MM, Ruiz NQ, Moura LP de, Saad MJA, Kim Y-B, Patricia de Oliveira Prada, Costa SKP. Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4 [Internet]. Toxicology Reports. 2023 ; 11 10-22.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.toxrep.2023.06.002
Vancouver
Ferreira CK de O, Campolim CM, Zordão OP, Simabuco FM, Anaruma CP, Pereira RM, Boico VF, Salvino LG, Costa MM, Ruiz NQ, Moura LP de, Saad MJA, Kim Y-B, Patricia de Oliveira Prada, Costa SKP. Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4 [Internet]. Toxicology Reports. 2023 ; 11 10-22.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.toxrep.2023.06.002
Artigo de periodico
AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity (2023)
Leite, Jefferson Antonio ; Menezes, Luísa ; Martins, Eloisa ; Rodrigues, Tamara Silva; Tavares, Lucas ; Ebering, Anna ; Schelmbauer, Carsten ; Cebinelli, Guilherme Cesar Martelossi ; Zinina, Valeriya ; Golden, Artemy ; Soshnikova, Natalia ; Zamboni, Dario S. ; Cunha, Fernando Q. ; Magdalena Huber ; Waisman, Ari ; Silva, João Santana da ; Carlos, Daniela ; Câmara, Niels Olsen Saraiva
Source: iScience. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, PROTEÍNAS, DIFERENCIAÇÃO CELULAR, LINFÓCITOS T, CÉLULAS SANGUÍNEAS
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LEITE, Jefferson Antonio et al. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity. iScience, v. 26, n. 11, p. art. 108134. 19 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108134. Acesso em: 07 nov. 2024.
APA
Leite, J. A., Menezes, L., Martins, E., Rodrigues, T. S., Tavares, L., Ebering, A., et al. (2023). AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity. iScience, 26( 11), art. 108134. 19 . doi:10.1016/j.isci.2023.108134
NLM
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Carlos D, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Vancouver
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Carlos D, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Artigo de periodico
Single immunizations of self-amplifying or non-replicating mRNA-LNP vaccines control HPV-associated tumors in mice (2023)
Silva, Jamile Ramos da ; Rodrigues, Karine Bitencourt ; Pelegrin, Guilherme Formoso ; Sales, Natiely Silva ; Muramatsu, Hiromi ; Silva, Mariângela de Oliveira ; Porchia, Bruna Felício Milazzotto Maldonado ; Moreno, Ana Carolina Ramos ; Aps, Luana Raposo M. M. ; Carvalho, Aléxia Adrianne Venceslau ; Tombácz, István ; Fotoran, Wesley Luzetti ; Karikó, Katalin ; Lin, Paulo J. C.; Tam, Ying K. ; Diniz, Mariana de Oliveira ; Pardi, Norbert ; Ferreira, Luís Carlos de Souza
Source: Science Translational Medicine. Unidades: ICB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, VACINAS VIRAIS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, PAPILLOMAVIRUS, HPV, INFECÇÕES POR PAPILLOMAVIRUS, NEOPLASIAS DO COLO UTERINO, PROTEÍNAS RECOMBINANTES, NANOPARTÍCULAS, NUCLEOSÍDEOS, GLICOPROTEÍNAS
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SILVA, Jamile Ramos da et al. Single immunizations of self-amplifying or non-replicating mRNA-LNP vaccines control HPV-associated tumors in mice. Science Translational Medicine, v. 15, n. 686, p. 1-18, 2023Tradução . . Disponível em: https://doi.org/10.1126/scitranslmed.abn3464. Acesso em: 07 nov. 2024.
APA
Silva, J. R. da, Rodrigues, K. B., Pelegrin, G. F., Sales, N. S., Muramatsu, H., Silva, M. de O., et al. (2023). Single immunizations of self-amplifying or non-replicating mRNA-LNP vaccines control HPV-associated tumors in mice. Science Translational Medicine, 15( 686), 1-18. doi:10.1126/scitranslmed.abn3464
NLM
Silva JR da, Rodrigues KB, Pelegrin GF, Sales NS, Muramatsu H, Silva M de O, Porchia BFMM, Moreno ACR, Aps LRMM, Carvalho AAV, Tombácz I, Fotoran WL, Karikó K, Lin PJC, Tam YK, Diniz M de O, Pardi N, Ferreira LC de S. Single immunizations of self-amplifying or non-replicating mRNA-LNP vaccines control HPV-associated tumors in mice [Internet]. Science Translational Medicine. 2023 ; 15( 686): 1-18.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1126/scitranslmed.abn3464
Vancouver
Silva JR da, Rodrigues KB, Pelegrin GF, Sales NS, Muramatsu H, Silva M de O, Porchia BFMM, Moreno ACR, Aps LRMM, Carvalho AAV, Tombácz I, Fotoran WL, Karikó K, Lin PJC, Tam YK, Diniz M de O, Pardi N, Ferreira LC de S. Single immunizations of self-amplifying or non-replicating mRNA-LNP vaccines control HPV-associated tumors in mice [Internet]. Science Translational Medicine. 2023 ; 15( 686): 1-18.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1126/scitranslmed.abn3464
Artigo de periodico
Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice (2022)
Kajitani, Gustavo Satoru; Quayle, Carolina de Barros Reis ; Garcia, Camila Carrião Machado ; Fotoran, Wesley Luzetti ; Santos, Juliana F. R. dos; Horst, Gijsbertus T. J. van der ; Hoeijmakers, Jan H. J. ; Menck, Carlos Frederico Martins
Source: Frontiers in Immunology. Unidades: ICB, IB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, RADIAÇÃO ULTRAVIOLETA, REPARAÇÃO DE DNA, CÉLULAS MORTAS, NEOPLASIAS CUTÂNEAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, PROLIFERAÇÃO CELULAR
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KAJITANI, Gustavo Satoru et al. Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice. Frontiers in Immunology, v. 13, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.800606. Acesso em: 07 nov. 2024.
APA
Kajitani, G. S., Quayle, C. de B. R., Garcia, C. C. M., Fotoran, W. L., Santos, J. F. R. dos, Horst, G. T. J. van der, et al. (2022). Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice. Frontiers in Immunology, 13, 1-10. doi:10.3389/fimmu.2022.800606
NLM
Kajitani GS, Quayle C de BR, Garcia CCM, Fotoran WL, Santos JFR dos, Horst GTJ van der, Hoeijmakers JHJ, Menck CFM. Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice [Internet]. Frontiers in Immunology. 2022 ; 13 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fimmu.2022.800606
Vancouver
Kajitani GS, Quayle C de BR, Garcia CCM, Fotoran WL, Santos JFR dos, Horst GTJ van der, Hoeijmakers JHJ, Menck CFM. Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice [Internet]. Frontiers in Immunology. 2022 ; 13 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fimmu.2022.800606
Artigo de periodico
The fibrotic niche impairs satellite cell function and muscle regeneration in mouse models of Marfan syndrome (2022)
Silva, Meiricris Tomaz da ; Santos, Audrei dos Reis ; Koike, Tatiana Emy ; Nascimento, Tabata Leal; Rozanski, Andrei ; Bosnakovski, Darko ; Pereira, Lygia da Veiga ; Kumar, Ashok; Kyba, Michael ; Miyabara, Elen Haruka
Source: Acta Physiologica. Unidades: IB, ICB
Subjects: ANATOMIA, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, MÚSCULO ESQUELÉTICO, ANGIOTENSINAS, CÉLULAS MUSCULARES, FIBRAS MUSCULARES, DOENÇAS GENÉTICAS, DOENÇAS DO TECIDO CONJUNTIVO, MACRÓFAGOS, MIOBLASTOS, NICHO
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SILVA, Meiricris Tomaz da et al. The fibrotic niche impairs satellite cell function and muscle regeneration in mouse models of Marfan syndrome. Acta Physiologica, p. 1-19, 2022Tradução . . Disponível em: https://doi.org/10.1111/apha.13889. Acesso em: 07 nov. 2024.
APA
Silva, M. T. da, Santos, A. dos R., Koike, T. E., Nascimento, T. L., Rozanski, A., Bosnakovski, D., et al. (2022). The fibrotic niche impairs satellite cell function and muscle regeneration in mouse models of Marfan syndrome. Acta Physiologica, 1-19. doi:10.1111/apha.13889
NLM
Silva MT da, Santos A dos R, Koike TE, Nascimento TL, Rozanski A, Bosnakovski D, Pereira L da V, Kumar A, Kyba M, Miyabara EH. The fibrotic niche impairs satellite cell function and muscle regeneration in mouse models of Marfan syndrome [Internet]. Acta Physiologica. 2022 ; 1-19.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/apha.13889
Vancouver
Silva MT da, Santos A dos R, Koike TE, Nascimento TL, Rozanski A, Bosnakovski D, Pereira L da V, Kumar A, Kyba M, Miyabara EH. The fibrotic niche impairs satellite cell function and muscle regeneration in mouse models of Marfan syndrome [Internet]. Acta Physiologica. 2022 ; 1-19.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/apha.13889
Artigo de periodico
Mouse neural stem cell differentiation and human adipose mesenchymal stem cell Transdifferentiation into neuron- and Oligodendrocyte-like cells with myelination potential (2022)
Santos, Anderson Kury ; Gomes, Katia N; Parreira, Ricardo C; Scalzo, Sérgio ; Pinto, Mauro Cunha Xavier ; Santiago, Helton C ; Birbrair, Alexander ; Sack, Ulrich; Resende, Rodrigo Ribeiro ; Ulrich, Henning
Source: Stem Cell Reviews and Reports. Unidade: IQ
Subjects: CÉLULAS-TRONCO, CAMUNDONGOS, PROLIFERAÇÃO CELULAR, NEURÔNIOS
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SANTOS, Anderson Kury et al. Mouse neural stem cell differentiation and human adipose mesenchymal stem cell Transdifferentiation into neuron- and Oligodendrocyte-like cells with myelination potential. Stem Cell Reviews and Reports, v. 18, p. 732–751, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12015-021-10218-7. Acesso em: 07 nov. 2024.
APA
Santos, A. K., Gomes, K. N., Parreira, R. C., Scalzo, S., Pinto, M. C. X., Santiago, H. C., et al. (2022). Mouse neural stem cell differentiation and human adipose mesenchymal stem cell Transdifferentiation into neuron- and Oligodendrocyte-like cells with myelination potential. Stem Cell Reviews and Reports, 18, 732–751. doi:10.1007/s12015-021-10218-7
NLM
Santos AK, Gomes KN, Parreira RC, Scalzo S, Pinto MCX, Santiago HC, Birbrair A, Sack U, Resende RR, Ulrich H. Mouse neural stem cell differentiation and human adipose mesenchymal stem cell Transdifferentiation into neuron- and Oligodendrocyte-like cells with myelination potential [Internet]. Stem Cell Reviews and Reports. 2022 ; 18 732–751.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12015-021-10218-7
Vancouver
Santos AK, Gomes KN, Parreira RC, Scalzo S, Pinto MCX, Santiago HC, Birbrair A, Sack U, Resende RR, Ulrich H. Mouse neural stem cell differentiation and human adipose mesenchymal stem cell Transdifferentiation into neuron- and Oligodendrocyte-like cells with myelination potential [Internet]. Stem Cell Reviews and Reports. 2022 ; 18 732–751.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12015-021-10218-7
Artigo de periodico
Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity (2022)
Monteiro, Lauar de Brito ; Prodonoff, Juliana Silveira ; Aguiar, Cristhiane Favero de ; Silva, Felipe Correa da; Castoldi, Angela ; Bakker, Nikki van Teijlingen ; Davanzo, Gustavo Gastão ; Donato Junior, José ; Câmara, Niels Olsen Saraiva
Source: Diabetes. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO, MACRÓFAGOS, RESISTÊNCIA À INSULINA, CITOCINAS, TECIDO ADIPOSO, INFLAMAÇÃO, OBESIDADE, CAMUNDONGOS
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MONTEIRO, Lauar de Brito et al. Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity. Diabetes, v. 71, n. 7, p. 1546–1561, 2022Tradução . . Disponível em: https://doi.org/10.2337/db21-0842. Acesso em: 07 nov. 2024.
APA
Monteiro, L. de B., Prodonoff, J. S., Aguiar, C. F. de, Silva, F. C. da, Castoldi, A., Bakker, N. van T., et al. (2022). Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity. Diabetes, 71( 7), 1546–1561. doi:10.2337/db21-0842
NLM
Monteiro L de B, Prodonoff JS, Aguiar CF de, Silva FC da, Castoldi A, Bakker N van T, Davanzo GG, Donato Junior J, Câmara NOS. Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity [Internet]. Diabetes. 2022 ; 71( 7): 1546–1561.[citado 2024 nov. 07 ] Available from: https://doi.org/10.2337/db21-0842
Vancouver
Monteiro L de B, Prodonoff JS, Aguiar CF de, Silva FC da, Castoldi A, Bakker N van T, Davanzo GG, Donato Junior J, Câmara NOS. Leptin signaling suppression in macrophages improves immunometabolic outcomes in obesity [Internet]. Diabetes. 2022 ; 71( 7): 1546–1561.[citado 2024 nov. 07 ] Available from: https://doi.org/10.2337/db21-0842
Artigo de periodico
Why do mice squeak? Toward a better understanding of defensive vocalization (2022)
Ruat, Julia ; Genewsky, Andreas J. ; Heinz, Daniel E. ; Kaltwasser, Sebastian F.; Canteras, Newton Sabino ; Czisch, Michael ; Chen, Alon ; Wotjak, Carsten T.
Source: iScience. Unidade: ICB
Subjects: ANATOMIA, RATOS, CAMUNDONGOS, COMPORTAMENTO ANIMAL, ANSIEDADE, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL
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RUAT, Julia et al. Why do mice squeak? Toward a better understanding of defensive vocalization. iScience, v. 25, n. 7, p. 1-21, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2022.104657. Acesso em: 07 nov. 2024.
APA
Ruat, J., Genewsky, A. J., Heinz, D. E., Kaltwasser, S. F., Canteras, N. S., Czisch, M., et al. (2022). Why do mice squeak? Toward a better understanding of defensive vocalization. iScience, 25( 7), 1-21. doi:10.1016/j.isci.2022.104657
NLM
Ruat J, Genewsky AJ, Heinz DE, Kaltwasser SF, Canteras NS, Czisch M, Chen A, Wotjak CT. Why do mice squeak? Toward a better understanding of defensive vocalization [Internet]. iScience. 2022 ; 25( 7): 1-21.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.isci.2022.104657
Vancouver
Ruat J, Genewsky AJ, Heinz DE, Kaltwasser SF, Canteras NS, Czisch M, Chen A, Wotjak CT. Why do mice squeak? Toward a better understanding of defensive vocalization [Internet]. iScience. 2022 ; 25( 7): 1-21.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.isci.2022.104657
Artigo de periodico
Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival (2021)
Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher ; Nalbach, Lisa; Almeida, Davidson Correa de ; Ampofo, Emmanuel ; Carpinelli, Angelo Rafael ; Roma, Leticia Prates ; Ortis, Fernanda
Source: Antioxidants. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, PERÓXIDO DE HIDROGÊNIO, CITOCINAS, DIABETES MELLITUS, CAMUNDONGOS, ILHOTAS DE LANGERHANS, GLICOSE
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, v. 10, n. 8, p. 1-20, 2021Tradução . . Disponível em: https://doi.org/10.3390/antiox10081305. Acesso em: 07 nov. 2024.
APA
Vilas-Boas, E. A., Carlein, C., Nalbach, L., Almeida, D. C. de, Ampofo, E., Carpinelli, A. R., et al. (2021). Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, 10( 8), 1-20. doi:10.3390/antiox10081305
NLM
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/antiox10081305
Vancouver
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/antiox10081305
Artigo de periodico
Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4 (2021)
Trevelin, Silvia Cellone ; Sag, Can Martin; Zhang, Min; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Santos, Célio Xavier dos; Sawyer, Greta ; Murray, Thomas; Brewer, Alison ; Laurindo, Francisco Rafael Martins; Protti, Andrea ; Lopes, Lucia Rossetti ; Ivetic, Aleksandar ; Cunha, Fernando Queiroz ; Shah, Ajay M.
Source: Shock. Unidades: FMRP, FM, ICB
Subjects: FARMACOLOGIA, LIPOPOLISSACARÍDEOS, CARBOIDRATOS, LEUCÓCITOS, CÉLULAS ENDOTELIAIS, CAMUNDONGOS, INFLAMAÇÃO, NEUTRÓFILOS
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ABNT
TREVELIN, Silvia Cellone et al. Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4. Shock, v. 56, n. 2, p. 268–277, 2021Tradução . . Disponível em: https://doi.org/10.1097/shk.0000000000001706. Acesso em: 07 nov. 2024.
APA
Trevelin, S. C., Sag, C. M., Zhang, M., Alves Filho, J. C. F., Cunha, T. M., Santos, C. X. dos, et al. (2021). Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4. Shock, 56( 2), 268–277. doi:10.1097/shk.0000000000001706
NLM
Trevelin SC, Sag CM, Zhang M, Alves Filho JCF, Cunha TM, Santos CX dos, Sawyer G, Murray T, Brewer A, Laurindo FRM, Protti A, Lopes LR, Ivetic A, Cunha FQ, Shah AM. Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4 [Internet]. Shock. 2021 ; 56( 2): 268–277.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1097/shk.0000000000001706
Vancouver
Trevelin SC, Sag CM, Zhang M, Alves Filho JCF, Cunha TM, Santos CX dos, Sawyer G, Murray T, Brewer A, Laurindo FRM, Protti A, Lopes LR, Ivetic A, Cunha FQ, Shah AM. Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4 [Internet]. Shock. 2021 ; 56( 2): 268–277.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1097/shk.0000000000001706
Artigo de periodico
Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets (2021)
Vilas-Boas, Eloisa Aparecida ; Nalbach, Lisa; Ampofo, Emmanuel; Lucena, Camila Ferraz ; Naudet, Léa ; Morgan, Bruce ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Free Radical Biology & Medicine. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, OBESIDADE, DIABETES MELLITUS, HÁBITOS ALIMENTARES, ÁCIDOS GRAXOS, ILHOTAS DE LANGERHANS, ESTRESSE OXIDATIVO, CAMUNDONGOS, INSULINA, HOMEOSTASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VILAS-BOAS, Eloisa Aparecida et al. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, v. 162, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2020.11.023. Acesso em: 07 nov. 2024.
APA
Vilas-Boas, E. A., Nalbach, L., Ampofo, E., Lucena, C. F., Naudet, L., Morgan, B., et al. (2021). Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, 162, 13 . doi:10.1016/j.freeradbiomed.2020.11.023
NLM
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Vancouver
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Artigo de periodico
Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer (2021)
Staquicini, Fernanda I. ; Hajitou, Amin ; Driessen, Wouter HP ; Proneth, Bettina ; Vila, Marina Cardó ; Staquicini, Daniela I.; Markosian, Christopher ; Hoh, Maria ; Cortez, Mauro ; Nehra, Anupama Hooda ; Jaloudi, Mohammed; Silva, Israel T. ; Buttura, Jaqueline; Nunes, Diana; Dias Neto, Emmanuel ; Eckhardt, Bedrich ; Ramírez, Javier Ruiz ; Dogra, Prashant ; Wang, Zhihui; Cristini, Vittorio ; Trepel, Martin; Anderson, Robin ; Sidman, Richard L.; Gelovani, Juri G. ; Cristofanilli, Massimo ; Hortobagy, Gabriel; Bhujwalla, Zaver M. ; Burley, Stephen ; Arap, Wadih ; Pasqualini, Renata
Source: eLife. Unidade: ICB
Subjects: PARASITOLOGIA, QUIMIOTERÁPICOS, ANTINEOPLÁSICOS, NEOPLASIAS MAMÁRIAS, CÉLULAS CULTIVADAS DE TUMOR, MACRÓFAGOS, VITAMINA D, CAMUNDONGOS, CITOCINAS, PEPTÍDEOS, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
STAQUICINI, Fernanda I. et al. Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer. eLife, v. 10, p. 1-24, 2021Tradução . . Disponível em: https://doi.org/10.7554/eLife.65145. Acesso em: 07 nov. 2024.
APA
Staquicini, F. I., Hajitou, A., Driessen, W. H. P., Proneth, B., Vila, M. C., Staquicini, D. I., et al. (2021). Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer. eLife, 10, 1-24. doi:10.7554/eLife.65145
NLM
Staquicini FI, Hajitou A, Driessen WHP, Proneth B, Vila MC, Staquicini DI, Markosian C, Hoh M, Cortez M, Nehra AH, Jaloudi M, Silva IT, Buttura J, Nunes D, Dias Neto E, Eckhardt B, Ramírez JR, Dogra P, Wang Z, Cristini V, Trepel M, Anderson R, Sidman RL, Gelovani JG, Cristofanilli M, Hortobagy G, Bhujwalla ZM, Burley S, Arap W, Pasqualini R. Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer [Internet]. eLife. 2021 ; 10 1-24.[citado 2024 nov. 07 ] Available from: https://doi.org/10.7554/eLife.65145
Vancouver
Staquicini FI, Hajitou A, Driessen WHP, Proneth B, Vila MC, Staquicini DI, Markosian C, Hoh M, Cortez M, Nehra AH, Jaloudi M, Silva IT, Buttura J, Nunes D, Dias Neto E, Eckhardt B, Ramírez JR, Dogra P, Wang Z, Cristini V, Trepel M, Anderson R, Sidman RL, Gelovani JG, Cristofanilli M, Hortobagy G, Bhujwalla ZM, Burley S, Arap W, Pasqualini R. Targeting a cell surface vitamin D receptor on tumor-associated macrophages in triple-negative breast cancer [Internet]. eLife. 2021 ; 10 1-24.[citado 2024 nov. 07 ] Available from: https://doi.org/10.7554/eLife.65145
Artigo de periodico
Cold atmospheric plasma jet as a possible adjuvant therapy for periodontal disease (2021)
Lima, Gabriela de Morais Gouvêa ; Borges, Aline Chiodi ; Nishime, Thalita Mayumi Castaldelli ; Melo, Gabriela de Fatima Santana ; Kostov, Konstantin Georgiev ; Mayer, Marcia Pinto Alves ; Ito, Cristiane Yumi Koga
Source: Molecules. Unidade: ICB
Subjects: MICROBIOLOGIA, DOENÇAS PERIODONTAIS, ANTI-INFLAMATÓRIOS, DOENÇAS PERIODONTAIS, RESISTÊNCIA MICROBIANA ÀS DROGAS, PORPHYROMONAS GINGIVALIS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, STREPTOCOCCUS, QUIMIOTERAPIA ADJUVANTE, PLASMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Gabriela de Morais Gouvêa et al. Cold atmospheric plasma jet as a possible adjuvant therapy for periodontal disease. Molecules, v. 26, n. 18, p. 1-14, 2021Tradução . . Disponível em: https://doi.org/10.3390/molecules26185590. Acesso em: 07 nov. 2024.
APA
Lima, G. de M. G., Borges, A. C., Nishime, T. M. C., Melo, G. de F. S., Kostov, K. G., Mayer, M. P. A., & Ito, C. Y. K. (2021). Cold atmospheric plasma jet as a possible adjuvant therapy for periodontal disease. Molecules, 26( 18), 1-14. doi:10.3390/molecules26185590
NLM
Lima G de MG, Borges AC, Nishime TMC, Melo G de FS, Kostov KG, Mayer MPA, Ito CYK. Cold atmospheric plasma jet as a possible adjuvant therapy for periodontal disease [Internet]. Molecules. 2021 ; 26( 18): 1-14.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/molecules26185590
Vancouver
Lima G de MG, Borges AC, Nishime TMC, Melo G de FS, Kostov KG, Mayer MPA, Ito CYK. Cold atmospheric plasma jet as a possible adjuvant therapy for periodontal disease [Internet]. Molecules. 2021 ; 26( 18): 1-14.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/molecules26185590
Artigo de periodico
Small-Molecule chemical knockdown of MuRF1 in melanoma bearing mice attenuates tumor cachexia associated myopathy (2020)
Adams, Volker; Gußen, Victoria; Zozulya, Sergey ; Oliveira, André Cruz de ; Moriscot, Anselmo Sigari ; Linke, Axel; Labeit, Siegfried
Source: Cells. Unidade: ICB
Subjects: ANATOMIA, MELANOMA ANIMAL, CAQUEXIA, MIOPATIAS MITOCONDRIAIS, MÚSCULO ESQUELÉTICO, ATROFIA MUSCULAR, PERDA DE MASSA, PERDA DE PESO, TECIDO ADIPOSO, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ADAMS, Volker et al. Small-Molecule chemical knockdown of MuRF1 in melanoma bearing mice attenuates tumor cachexia associated myopathy. Cells, v. 9, n. 10, p. 15 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9102272. Acesso em: 07 nov. 2024.
APA
Adams, V., Gußen, V., Zozulya, S., Oliveira, A. C. de, Moriscot, A. S., Linke, A., & Labeit, S. (2020). Small-Molecule chemical knockdown of MuRF1 in melanoma bearing mice attenuates tumor cachexia associated myopathy. Cells, 9( 10), 15 . doi:10.3390/cells9102272
NLM
Adams V, Gußen V, Zozulya S, Oliveira AC de, Moriscot AS, Linke A, Labeit S. Small-Molecule chemical knockdown of MuRF1 in melanoma bearing mice attenuates tumor cachexia associated myopathy [Internet]. Cells. 2020 ; 9( 10): 15 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells9102272
Vancouver
Adams V, Gußen V, Zozulya S, Oliveira AC de, Moriscot AS, Linke A, Labeit S. Small-Molecule chemical knockdown of MuRF1 in melanoma bearing mice attenuates tumor cachexia associated myopathy [Internet]. Cells. 2020 ; 9( 10): 15 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells9102272
Artigo de periodico
The rhythmicity of clock genes is disrupted in the choroid plexus of the APP/PS1 mouse model of Alzheimer’s disease (2020)
Furtado, André ; Astaburuaga, Rosario ; Costa, Ana; Duarte, Ana C.; Gonçalves, Isabel; Lemos, Manuel C.; Carro, Eva ; Relógio, Angela ; Santos, Cecilia R. A.; Quintela, Telma ; Cipolla Neto, José
Source: Journal of Alzheimer's Disease. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, MELATONINA, RITMOS BIOLÓGICOS, PLEXO COROIDE DE ANIMAL, CAMUNDONGOS, BARREIRA HEMATO-ENCEFÁLICA, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FURTADO, André et al. The rhythmicity of clock genes is disrupted in the choroid plexus of the APP/PS1 mouse model of Alzheimer’s disease. Journal of Alzheimer's Disease, v. 77, n. 2, p. 795-806, 2020Tradução . . Disponível em: https://doi.org/10.3233/JAD-200331. Acesso em: 07 nov. 2024.
APA
Furtado, A., Astaburuaga, R., Costa, A., Duarte, A. C., Gonçalves, I., Lemos, M. C., et al. (2020). The rhythmicity of clock genes is disrupted in the choroid plexus of the APP/PS1 mouse model of Alzheimer’s disease. Journal of Alzheimer's Disease, 77( 2), 795-806. doi:10.3233/JAD-200331
NLM
Furtado A, Astaburuaga R, Costa A, Duarte AC, Gonçalves I, Lemos MC, Carro E, Relógio A, Santos CRA, Quintela T, Cipolla Neto J. The rhythmicity of clock genes is disrupted in the choroid plexus of the APP/PS1 mouse model of Alzheimer’s disease [Internet]. Journal of Alzheimer's Disease. 2020 ; 77( 2): 795-806.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3233/JAD-200331
Vancouver
Furtado A, Astaburuaga R, Costa A, Duarte AC, Gonçalves I, Lemos MC, Carro E, Relógio A, Santos CRA, Quintela T, Cipolla Neto J. The rhythmicity of clock genes is disrupted in the choroid plexus of the APP/PS1 mouse model of Alzheimer’s disease [Internet]. Journal of Alzheimer's Disease. 2020 ; 77( 2): 795-806.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3233/JAD-200331
Artigo de periodico
Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA) (2020)
Davanso, Mariana Rodrigues ; Crisma, Amanda Rabello ; Braga, Tarcio Teodoro ; Masi, Laureane Nunes ; Amaral, Catia Lira do ; Leal, Vinicius Nunes Cordeiro ; Lima, Dhêmerson Souza de ; Patente, Thiago Andrade ; Giannella, Maria Lucia Correa ; Lauterbach, Mario ; Kolbe, Carl Christian ; Latz, Eicke ; Barbuto, José Alexandre Marzagão ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Curi, Rui
Source: Clinical Science (Online): an international journal of translational science and medicine. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, DIABETES MELLITUS, ÁCIDOS GRAXOS, MACRÓFAGOS, INFLAMAÇÃO, DOENÇAS AUTOIMUNES, ILHOTAS DE LANGERHANS, MONÓCITOS, HIPERGLICEMIA, CITOCINAS, ÓXIDO NÍTRICO, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAVANSO, Mariana Rodrigues et al. Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA). Clinical Science (Online): an international journal of translational science and medicine, p. [29 ], 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20201348. Acesso em: 07 nov. 2024.
APA
Davanso, M. R., Crisma, A. R., Braga, T. T., Masi, L. N., Amaral, C. L. do, Leal, V. N. C., et al. (2020). Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA). Clinical Science (Online): an international journal of translational science and medicine, [29 ]. doi:10.1042/CS20201348
NLM
Davanso MR, Crisma AR, Braga TT, Masi LN, Amaral CL do, Leal VNC, Lima DS de, Patente TA, Giannella MLC, Lauterbach M, Kolbe CC, Latz E, Barbuto JAM, Câmara NOS, Pontillo A, Curi R. Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA) [Internet]. Clinical Science (Online): an international journal of translational science and medicine. 2020 ;[29 ].[citado 2024 nov. 07 ] Available from: https://doi.org/10.1042/CS20201348
Vancouver
Davanso MR, Crisma AR, Braga TT, Masi LN, Amaral CL do, Leal VNC, Lima DS de, Patente TA, Giannella MLC, Lauterbach M, Kolbe CC, Latz E, Barbuto JAM, Câmara NOS, Pontillo A, Curi R. Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA) [Internet]. Clinical Science (Online): an international journal of translational science and medicine. 2020 ;[29 ].[citado 2024 nov. 07 ] Available from: https://doi.org/10.1042/CS20201348
Artigo de periodico
Paternal exercise protects against liver steatosis in the male offspring of mice submitted to high fat diet (2020)
Batista, Rogério Oliveira; Budu, Alexandre ; Araki, Aline Midori; Gregnani, Marcos Fernandes S. ; Húngaro, Talita G. Rodrigues; Silva, Marina Burgos da ; Wasinski, Frederick ; Lanzoni, Valeria Pereira; Oyama, Lila Missae ; Bader, Michael ; Araujo, Ronaldo Carvalho ; Câmara, Niels Olsen Saraiva
Source: Life Sciences. Unidade: ICB
Subjects: IMUNOLOGIA, EXERCÍCIO FÍSICO, CAMUNDONGOS, DIETA ANIMAL, OBESIDADE, FENÓTIPOS, RESISTÊNCIA À INSULINA, TREINAMENTO FÍSICO, NATAÇÃO, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BATISTA, Rogério Oliveira et al. Paternal exercise protects against liver steatosis in the male offspring of mice submitted to high fat diet. Life Sciences, v. 263, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2020.118583. Acesso em: 07 nov. 2024.
APA
Batista, R. O., Budu, A., Araki, A. M., Gregnani, M. F. S., Húngaro, T. G. R., Silva, M. B. da, et al. (2020). Paternal exercise protects against liver steatosis in the male offspring of mice submitted to high fat diet. Life Sciences, 263, 10 . doi:10.1016/j.lfs.2020.118583
NLM
Batista RO, Budu A, Araki AM, Gregnani MFS, Húngaro TGR, Silva MB da, Wasinski F, Lanzoni VP, Oyama LM, Bader M, Araujo RC, Câmara NOS. Paternal exercise protects against liver steatosis in the male offspring of mice submitted to high fat diet [Internet]. Life Sciences. 2020 ; 263 10 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.lfs.2020.118583
Vancouver
Batista RO, Budu A, Araki AM, Gregnani MFS, Húngaro TGR, Silva MB da, Wasinski F, Lanzoni VP, Oyama LM, Bader M, Araujo RC, Câmara NOS. Paternal exercise protects against liver steatosis in the male offspring of mice submitted to high fat diet [Internet]. Life Sciences. 2020 ; 263 10 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.lfs.2020.118583

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