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Artigo de periodico
The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism (2023)
Britto Júnior, José ; Prado, Gustavo L. Pereira do ; Cunha, Fernando ; Moraes, Manoel Odorico ; Moraes, Maria Elisabete A. ; Monica, Fabiola Z. ; Antunes, Edson ; Chiavegatto, Silvana ; De Nucci, Gilberto
Source: Nitric Oxide: Biology and Chemistry. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, ÁTRIOS DO CORAÇÃO, VENTRÍCULO CARDÍACO DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, ÓXIDO NÍTRICO, ESPECTROMETRIA DE MASSAS
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ABNT
BRITTO JÚNIOR, José et al. The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism. Nitric Oxide: Biology and Chemistry, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2023.06.001. Acesso em: 18 nov. 2024.
APA
Britto Júnior, J., Prado, G. L. P. do, Cunha, F., Moraes, M. O., Moraes, M. E. A., Monica, F. Z., et al. (2023). The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism. Nitric Oxide: Biology and Chemistry. doi:10.1016/j.niox.2023.06.001
NLM
Britto Júnior J, Prado GLP do, Cunha F, Moraes MO, Moraes MEA, Monica FZ, Antunes E, Chiavegatto S, De Nucci G. The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism [Internet]. Nitric Oxide: Biology and Chemistry. 2023 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2023.06.001
Vancouver
Britto Júnior J, Prado GLP do, Cunha F, Moraes MO, Moraes MEA, Monica FZ, Antunes E, Chiavegatto S, De Nucci G. The importance of the endothelial nitric oxide synthase on the release of 6-nitrodopamine from mouse isolated atria and ventricles and their role on chronotropism [Internet]. Nitric Oxide: Biology and Chemistry. 2023 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2023.06.001
Artigo de periodico
Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection (2022)
Almeida, Leticia de; Silva, Alexandre Luiz Neves; Rodrigues, Tamara Silva; Oliveira, Samuel; Ishimoto, Adriene Yumi; Seribelli, Amanda Aparecida; Becerra, Amanda; Andrade, Warrison A; Ataíde, Marco Antônio; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de; Pelisson, Natália; Martins, Ronaldo Bragança; Souza, Juliano de Paula; Arruda, Eurico; Batah, Sabrina Setembre; Castro, Ricardo; Frantz, Fabiani Gai; Cunha, Fernando Queiróz; Cunha, Thiago Mattar; Fabro, Alexandre Todorovic; Cunha, Larissa Dias; Louzada-Junior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões
Source: Science Advances. Unidades: FMRP, FCFRP
Subjects: INFECÇÕES RESPIRATÓRIAS, COVID-19, ADJUVANTES IMUNOLÓGICOS, INFLAMAÇÃO
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ALMEIDA, Leticia de et al. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, v. 8, n. 37, 2022Tradução . . Disponível em: https://doi.org/10.1126/sciadv.abo5400. Acesso em: 18 nov. 2024.
APA
Almeida, L. de, Silva, A. L. N., Rodrigues, T. S., Oliveira, S., Ishimoto, A. Y., Seribelli, A. A., et al. (2022). Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, 8( 37). doi:10.1126/sciadv.abo5400
NLM
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Vancouver
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Artigo de periodico
SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients (2022)
Pontelli, Marjorie C.; Castro, Ítalo A.; Martins, Ronaldo B; La Serra, Leonardo; Veras, Flávio P.; Nascimento, Daniele C; Silva, Camila M.; Cardoso, Ricardo S; Rosales, Roberta Ribeiro Costa; Gomes, Rogério; Lima, Thais M.; Souza, Juliano P.; Vitti, Brenda C.; Caetité, Diego B.; Lima, Mikhael Haruo Fernandes de; Stumpf, Spencer D; Thompson, Cassandra E; Bloyet, Louis-Marie; Toller-Kawahisa, Juliana E.; Giannini, Marcela C.; Bonjorno, Letícia P; Lopes, Maria Isabel Fernandes; Batah, Sabrina Setembre; Siyuan, Li; Luppino-Assad, Rodrigo; Almeida, Sergio C L; Oliveira, Fabiola R; Benatti, Maíra N; Pontes, Lorena Lobo de Figueiredo; Santana, Rodrigo Carvalho; Vilar, Fernando C; Auxiliadora-Martins, Maria; Shi, Pei-Yong; Cunha, Thiago Mattar; Calado, Rodrigo Tocantins; Alves-Filho, José Carlos; Zamboni, Dario Simões; Fabro, Alexandre Todorovic; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro; Whelan, Sean P J; Cunha, Fernando Queiróz; Arruda, Eurico
Source: Journal of Molecular Cell Biology. Unidades: FCFRP, FMRP
Subjects: COVID-19, LINFÓCITOS, MONÓCITOS, APOPTOSE
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ABNT
PONTELLI, Marjorie C. et al. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, v. 14, n. 4, 2022Tradução . . Disponível em: https://doi.org/10.1093/jmcb/mjac021. Acesso em: 18 nov. 2024.
APA
Pontelli, M. C., Castro, Í. A., Martins, R. B., La Serra, L., Veras, F. P., Nascimento, D. C., et al. (2022). SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, 14( 4). doi:10.1093/jmcb/mjac021
NLM
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Vancouver
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Artigo de periodico
Gasdermin-D activation by SARS-CoV-2 triggers NET and mediate COVID-19 immunopathology (2022)
Silva, Camila Meirelles S.; Wanderley, Carlos Wagner S.; Veras, Flavio Protasio; Gonçalves, Augusto Velozo; Lima, Mikhael Haruo Fernandes; Toller-Kawahisa, Juliana Escher; Gomes, Giovanni Freitas; Nascimento, Daniele Carvalho; Monteiro, Valter V. Silva; Paiva, Isadora Marques; Almeida, Cícero José Luíz Ramos; Caetité, Diego Brito; Silva, Juliana Costa; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Giannini, Marcela Cavichioli; Amaral, Natalia Brasil; Benatti, Maíra Nilson; Santana, Rodrigo Carvalho; Damasceno, Luis Eduardo Alves; Silva, Bruna Manuella Souza; Schneider, Ayda Henriques; Castro, Icaro Maia Santos; Silva, Juan Carlo Santos; Vasconcelos, Amanda Pereira; Gonçalves, Tiago Tomazini; Batah, Sabrina Setembre; Rodrigues, Tamara Silva; Costa, Victor Ferreira; Pontelli, Marjorie Cornejo; Martins, Ronaldo B.; Martins, Timna Varela; Espósito, Danillo Lucas Alves; Cebinelli, Guilherme Cesar Martelossi; Fonseca, Benedito Antônio Lopes da; Leiria, Luiz Osório Silveira; Cunha, Larissa Dias; Arruda, Eurico; Nakaia, Helder I.; Fabro, Alexandre Todorovic; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Louzada-Junior, Paulo; Cunha, Thiago Mattar; Alves-Filho, José Carlos Farias; Cunha, Fernando Queiroz
Source: Critical Care. Unidades: FCFRP, FMRP, BIOINFORMÁTICA, FCF
Subjects: NEUTRÓFILOS, IMUNIDADE, COVID-19
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ABNT
SILVA, Camila Meirelles S. et al. Gasdermin-D activation by SARS-CoV-2 triggers NET and mediate COVID-19 immunopathology. Critical Care, v. 26, n. 1, 2022Tradução . . Disponível em: https://doi.org/10.1186/s13054-022-04062-5. Acesso em: 18 nov. 2024.
APA
Silva, C. M. S., Wanderley, C. W. S., Veras, F. P., Gonçalves, A. V., Lima, M. H. F., Toller-Kawahisa, J. E., et al. (2022). Gasdermin-D activation by SARS-CoV-2 triggers NET and mediate COVID-19 immunopathology. Critical Care, 26( 1). doi:10.1186/s13054-022-04062-5
NLM
Silva CMS, Wanderley CWS, Veras FP, Gonçalves AV, Lima MHF, Toller-Kawahisa JE, Gomes GF, Nascimento DC, Monteiro VVS, Paiva IM, Almeida CJLR, Caetité DB, Silva JC, Lopes MIF, Bonjorno LP, Giannini MC, Amaral NB, Benatti MN, Santana RC, Damasceno LEA, Silva BMS, Schneider AH, Castro IMS, Silva JCS, Vasconcelos AP, Gonçalves TT, Batah SS, Rodrigues TS, Costa VF, Pontelli MC, Martins RB, Martins TV, Espósito DLA, Cebinelli GCM, Fonseca BAL da, Leiria LOS, Cunha LD, Arruda E, Nakaia HI, Fabro AT, Oliveira RDR de, Zamboni DS, Louzada-Junior P, Cunha TM, Alves-Filho JCF, Cunha FQ. Gasdermin-D activation by SARS-CoV-2 triggers NET and mediate COVID-19 immunopathology [Internet]. Critical Care. 2022 ; 26( 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s13054-022-04062-5
Vancouver
Silva CMS, Wanderley CWS, Veras FP, Gonçalves AV, Lima MHF, Toller-Kawahisa JE, Gomes GF, Nascimento DC, Monteiro VVS, Paiva IM, Almeida CJLR, Caetité DB, Silva JC, Lopes MIF, Bonjorno LP, Giannini MC, Amaral NB, Benatti MN, Santana RC, Damasceno LEA, Silva BMS, Schneider AH, Castro IMS, Silva JCS, Vasconcelos AP, Gonçalves TT, Batah SS, Rodrigues TS, Costa VF, Pontelli MC, Martins RB, Martins TV, Espósito DLA, Cebinelli GCM, Fonseca BAL da, Leiria LOS, Cunha LD, Arruda E, Nakaia HI, Fabro AT, Oliveira RDR de, Zamboni DS, Louzada-Junior P, Cunha TM, Alves-Filho JCF, Cunha FQ. Gasdermin-D activation by SARS-CoV-2 triggers NET and mediate COVID-19 immunopathology [Internet]. Critical Care. 2022 ; 26( 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s13054-022-04062-5
Artigo de periodico
Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota (2022)
Arruda, José Alcides Almeida de ; Corrêa, Jôice Dias ; Singh, Youvika ; Oliveira, Sicília Rezende ; Machado, Caio Cavalcante ; Schneider, Ayda Henriques ; Medeiros, Julliane Dutra ; Fernandes, Gabriel R; Macari, Soraia ; Barrioni, Breno Rocha ; Santos, Mariana de Souza; Rodrigues, Letícia Fernanda Duffles ; Nakaya, Helder Takashi Imoto ; Fukada, Sandra Yasuyo ; Graves, Dana T ; Cunha, Fernando Queiroz ; Silva, Tarcília Aparecida
Source: Anaerobe. Unidades: FMRP, FCFRP, FCF, FORP
Subjects: ARTRITE REUMATOIDE, FARMACOTERAPIA, METOTREXATO
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ABNT
ARRUDA, José Alcides Almeida de et al. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota. Anaerobe, v. 75, p. 1-11 art. 102577, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.anaerobe.2022.102577. Acesso em: 18 nov. 2024.
APA
Arruda, J. A. A. de, Corrêa, J. D., Singh, Y., Oliveira, S. R., Machado, C. C., Schneider, A. H., et al. (2022). Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota. Anaerobe, 75, 1-11 art. 102577. doi:10.1016/j.anaerobe.2022.102577
NLM
Arruda JAA de, Corrêa JD, Singh Y, Oliveira SR, Machado CC, Schneider AH, Medeiros JD, Fernandes GR, Macari S, Barrioni BR, Santos M de S, Rodrigues LFD, Nakaya HTI, Fukada SY, Graves DT, Cunha FQ, Silva TA. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota [Internet]. Anaerobe. 2022 ; 75 1-11 art. 102577.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.anaerobe.2022.102577
Vancouver
Arruda JAA de, Corrêa JD, Singh Y, Oliveira SR, Machado CC, Schneider AH, Medeiros JD, Fernandes GR, Macari S, Barrioni BR, Santos M de S, Rodrigues LFD, Nakaya HTI, Fukada SY, Graves DT, Cunha FQ, Silva TA. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota [Internet]. Anaerobe. 2022 ; 75 1-11 art. 102577.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.anaerobe.2022.102577
Artigo de periodico
Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis (2021)
Saraiva, André Lopes; Peres, Raphael Sanches; Veras, Flávio Protasio; Talbot, Jhimmy; Lima, Kalil Alves de; Luiz, João Paulo Mesquita; Cunha, Thiago Mattar; Louzada-Junior, Paulo; Cunha, Fernando Queiróz; Alves-Filho, José Carlos
Source: International Immunopharmacology. Unidades: FCFRP, FMRP
Subjects: ARTRITE REUMATOIDE, PEPTÍDEOS, ARTICULAÇÕES, IMUNOLOGIA MÉDICA
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ABNT
SARAIVA, André Lopes et al. Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis. International Immunopharmacology, v. 101, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2021.108363. Acesso em: 18 nov. 2024.
APA
Saraiva, A. L., Peres, R. S., Veras, F. P., Talbot, J., Lima, K. A. de, Luiz, J. P. M., et al. (2021). Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis. International Immunopharmacology, 101. doi:10.1016/j.intimp.2021.108363
NLM
Saraiva AL, Peres RS, Veras FP, Talbot J, Lima KA de, Luiz JPM, Cunha TM, Louzada-Junior P, Cunha FQ, Alves-Filho JC. Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis [Internet]. International Immunopharmacology. 2021 ; 101[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.intimp.2021.108363
Vancouver
Saraiva AL, Peres RS, Veras FP, Talbot J, Lima KA de, Luiz JPM, Cunha TM, Louzada-Junior P, Cunha FQ, Alves-Filho JC. Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis [Internet]. International Immunopharmacology. 2021 ; 101[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.intimp.2021.108363
Artigo de periodico
Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4 (2021)
Trevelin, Silvia Cellone ; Sag, Can Martin; Zhang, Min; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Santos, Célio Xavier dos; Sawyer, Greta ; Murray, Thomas; Brewer, Alison ; Laurindo, Francisco Rafael Martins; Protti, Andrea ; Lopes, Lucia Rossetti ; Ivetic, Aleksandar ; Cunha, Fernando Queiroz ; Shah, Ajay M.
Source: Shock. Unidades: FMRP, FM, ICB
Subjects: FARMACOLOGIA, LIPOPOLISSACARÍDEOS, CARBOIDRATOS, LEUCÓCITOS, CÉLULAS ENDOTELIAIS, CAMUNDONGOS, INFLAMAÇÃO, NEUTRÓFILOS
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ABNT
TREVELIN, Silvia Cellone et al. Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4. Shock, v. 56, n. 2, p. 268–277, 2021Tradução . . Disponível em: https://doi.org/10.1097/shk.0000000000001706. Acesso em: 18 nov. 2024.
APA
Trevelin, S. C., Sag, C. M., Zhang, M., Alves Filho, J. C. F., Cunha, T. M., Santos, C. X. dos, et al. (2021). Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4. Shock, 56( 2), 268–277. doi:10.1097/shk.0000000000001706
NLM
Trevelin SC, Sag CM, Zhang M, Alves Filho JCF, Cunha TM, Santos CX dos, Sawyer G, Murray T, Brewer A, Laurindo FRM, Protti A, Lopes LR, Ivetic A, Cunha FQ, Shah AM. Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4 [Internet]. Shock. 2021 ; 56( 2): 268–277.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1097/shk.0000000000001706
Vancouver
Trevelin SC, Sag CM, Zhang M, Alves Filho JCF, Cunha TM, Santos CX dos, Sawyer G, Murray T, Brewer A, Laurindo FRM, Protti A, Lopes LR, Ivetic A, Cunha FQ, Shah AM. Endothelial Nox2 limits systemic inflammation and hypotension in endotoxemia by controlling expression of Toll-Like Receptor 4 [Internet]. Shock. 2021 ; 56( 2): 268–277.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1097/shk.0000000000001706
Artigo de periodico
Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity (2021)
Nascimento, Daniele Carvalho ; Viacava, Paula Ramos; Ferreira, Raphael Gomes; Damaceno, Marina Alves; Piñeros, Annie Rocío Alvarez ; Melo, Paulo Henrique de; Donate, Paula Barbim ; Toller-Kawahisa, Juliana Escher; Zoppi, Daniel ; Veras, Flávio Protásio ; Peres, Raphael S. ; Silva, Luisa Menezes ; Caetité, Diego ; Oliveira, Antonio Edson Rocha; Castro, Icaro Maia Santos ; Kauffenstein, Gilles ; Nakaya, Helder Takashi Imoto ; Borges, Marcos de Carvalho ; Zamboni, Dario Simões ; Fonseca, Denise Morais da ; Paschoal, Jonas Augusto Rizzato ; Cunha, Thiago Mattar ; Quesniaux, Valerie ; Linden, Joel ; Cunha, Fernando Queiróz ; Ryffe, Bernhard; Alves-Filho, José Carlos
Source: Immunity. Unidades: FMRP, FCFRP, FCF, ICB, BIOINFORMÁTICA
Subjects: ADENOSINA, FÁRMACOS IMUNOSSUPRESSORES, SEPSE, CAMUNDONGOS, INTERLEUCINA 10, MACRÓFAGOS
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ABNT
NASCIMENTO, Daniele Carvalho et al. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, v. 54, n. 9, p. 2024-2041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.immuni.2021.08.005. Acesso em: 18 nov. 2024.
APA
Nascimento, D. C., Viacava, P. R., Ferreira, R. G., Damaceno, M. A., Piñeros, A. R. A., Melo, P. H. de, et al. (2021). Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, 54( 9), 2024-2041. doi:10.1016/j.immuni.2021.08.005
NLM
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Vancouver
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Artigo de periodico
NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation (2020)
Prado, Douglas Silva ; Veras, Flavio Protasio; Ferreira, Raphael Gomes; Damasceno, Luis Eduardo Alves; Melo, Paulo Henrique; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Cunha, Fernando Queiróz ; Alves Filho, José Carlos Farias
Source: The FASEB Journal. Unidades: FMRP, FCFRP
Subjects: ARTRITE REUMATOIDE, DIFERENCIAÇÃO CELULAR, INFLAMAÇÃO, INTERLEUCINAS, ARTICULAÇÕES, NEUTRÓFILOS, FATORES DE TRANSCRIÇÃO
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ABNT
PRADO, Douglas Silva et al. NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation. The FASEB Journal, v. 34, n. 8, p. 10907-10919, 2020Tradução . . Disponível em: https://doi.org/10.1096/fj.202000795R. Acesso em: 18 nov. 2024.
APA
Prado, D. S., Veras, F. P., Ferreira, R. G., Damasceno, L. E. A., Melo, P. H., Zamboni, D. S., et al. (2020). NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation. The FASEB Journal, 34( 8), 10907-10919. doi:10.1096/fj.202000795R
NLM
Prado DS, Veras FP, Ferreira RG, Damasceno LEA, Melo PH, Zamboni DS, Cunha TM, Cunha FQ, Alves Filho JCF. NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation [Internet]. The FASEB Journal. 2020 ; 34( 8): 10907-10919.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1096/fj.202000795R
Vancouver
Prado DS, Veras FP, Ferreira RG, Damasceno LEA, Melo PH, Zamboni DS, Cunha TM, Cunha FQ, Alves Filho JCF. NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation [Internet]. The FASEB Journal. 2020 ; 34( 8): 10907-10919.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1096/fj.202000795R
Artigo de periodico
Paradoxical interaction between cancer and long-term postsepsis disorder: impairment of de novo carcinogenesis versus favoring the growth of established tumors (2020)
Leite, Caio Abner; Mota, Jose Mauricio; Lima, Kalil William Alves de; Wanderley, Carlos Wagner ; Nascimento, Leticia Alves do; Ferreira, Marcela Davoli; Silva, Camila Meirelles Souza; Colon, David Fernando; Sakita, Juliana Yumi; Kannen, Vinicius ; Viacava, Paula Ramos; Begnami, María Dirlei; Lima Júnior, Roberto Cesar Pereira; Lima, Vladmir Cláudio Cordeiro de; Alves Filho, José Carlos Farias ; Cunha, Fernando Queiróz ; Ribeiro, Ronaldo Albuquerque
Source: Journal for ImmunoTherapy of Cancer. Unidade: FMRP
Subjects: COLITE, IMUNOLOGIA, NEOPLASIAS DO COLON, CITOCINAS, INFLAMAÇÃO, SEPSE, TRANSDUÇÃO DE SINAL CELULAR, LINFÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Caio Abner et al. Paradoxical interaction between cancer and long-term postsepsis disorder: impairment of de novo carcinogenesis versus favoring the growth of established tumors. Journal for ImmunoTherapy of Cancer, v. 8, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1136/jitc-2019-000129. Acesso em: 18 nov. 2024.
APA
Leite, C. A., Mota, J. M., Lima, K. W. A. de, Wanderley, C. W., Nascimento, L. A. do, Ferreira, M. D., et al. (2020). Paradoxical interaction between cancer and long-term postsepsis disorder: impairment of de novo carcinogenesis versus favoring the growth of established tumors. Journal for ImmunoTherapy of Cancer, 8( 1). doi:10.1136/jitc-2019-000129
NLM
Leite CA, Mota JM, Lima KWA de, Wanderley CW, Nascimento LA do, Ferreira MD, Silva CMS, Colon DF, Sakita JY, Kannen V, Viacava PR, Begnami MD, Lima Júnior RCP, Lima VCC de, Alves Filho JCF, Cunha FQ, Ribeiro RA. Paradoxical interaction between cancer and long-term postsepsis disorder: impairment of de novo carcinogenesis versus favoring the growth of established tumors [Internet]. Journal for ImmunoTherapy of Cancer. 2020 ; 8( 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1136/jitc-2019-000129
Vancouver
Leite CA, Mota JM, Lima KWA de, Wanderley CW, Nascimento LA do, Ferreira MD, Silva CMS, Colon DF, Sakita JY, Kannen V, Viacava PR, Begnami MD, Lima Júnior RCP, Lima VCC de, Alves Filho JCF, Cunha FQ, Ribeiro RA. Paradoxical interaction between cancer and long-term postsepsis disorder: impairment of de novo carcinogenesis versus favoring the growth of established tumors [Internet]. Journal for ImmunoTherapy of Cancer. 2020 ; 8( 1):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1136/jitc-2019-000129
Artigo de periodico
MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression (2020)
Luiz, João Paulo Mesquita ; Toller‐Kawahisa, Juliana E.; Viacava, Paula Ramos; Nascimento, Daniele C.; Pereira, Priscilla T.; Saraiva, André L.; Prado, Douglas da Silva ; Le Bert, Marc; Giurisato, Emanuele; Tournier, Cathy; Cunha, Thiago Mattar ; Cunha, Fernando Queiróz ; Quesniaux, Valerie; Ryffel, Bernhard; Alves Filho, José Carlos Farias
Source: Journal of Leukocyte Biology. Unidade: FMRP
Subjects: DIFERENCIAÇÃO CELULAR, ANTÍGENOS, IMUNOLOGIA, REGULAÇÃO GÊNICA, EXPRESSÃO GÊNICA, INTERLEUCINA 4, MACRÓFAGOS, PROTEÍNAS PROTO-ONCOGÊNICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUIZ, João Paulo Mesquita et al. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression. Journal of Leukocyte Biology, v. 108, p. 1215-1223, 2020Tradução . . Disponível em: https://doi.org/10.1002/JLB.1MA0520-016R. Acesso em: 18 nov. 2024.
APA
Luiz, J. P. M., Toller‐Kawahisa, J. E., Viacava, P. R., Nascimento, D. C., Pereira, P. T., Saraiva, A. L., et al. (2020). MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression. Journal of Leukocyte Biology, 108, 1215-1223. doi:10.1002/JLB.1MA0520-016R
NLM
Luiz JPM, Toller‐Kawahisa JE, Viacava PR, Nascimento DC, Pereira PT, Saraiva AL, Prado D da S, Le Bert M, Giurisato E, Tournier C, Cunha TM, Cunha FQ, Quesniaux V, Ryffel B, Alves Filho JCF. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression [Internet]. Journal of Leukocyte Biology. 2020 ; 108 1215-1223.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/JLB.1MA0520-016R
Vancouver
Luiz JPM, Toller‐Kawahisa JE, Viacava PR, Nascimento DC, Pereira PT, Saraiva AL, Prado D da S, Le Bert M, Giurisato E, Tournier C, Cunha TM, Cunha FQ, Quesniaux V, Ryffel B, Alves Filho JCF. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression [Internet]. Journal of Leukocyte Biology. 2020 ; 108 1215-1223.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/JLB.1MA0520-016R
Artigo de periodico
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis (2018)
Peres, Raphael Sanches; Donate, Paula Barbim; Talbot, Jhimmy; Cecílio, Nerry Tatiana; Lobo, Patricia Rolim Mendonça; Machado, Caio Cavalcante; Lima, Kalil William Alves de; Oliveira, Renê Donizete Ribeiro de; Carregaro, Vanessa; Nakaya, Helder Takashi Imoto ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Liew, Foo Yew; Louzada Júnior, Paulo; Cunha, Fernando Queiróz
Source: Journal of Autoimmunity. Unidades: FMRP, FCF
Subjects: ARTRITE REUMATOIDE, METOTREXATO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERES, Raphael Sanches et al. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, v. 90, p. 49-58, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jaut.2018.01.004. Acesso em: 18 nov. 2024.
APA
Peres, R. S., Donate, P. B., Talbot, J., Cecílio, N. T., Lobo, P. R. M., Machado, C. C., et al. (2018). TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, 90, 49-58. doi:10.1016/j.jaut.2018.01.004
NLM
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Vancouver
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Artigo de periodico
Hepatic osteodystrophy: the mechanism of bone loss in hepatocellular disease and the effects of pamidronate treatment (2017)
Spirlandeli, Adriano Levi; Dick-de-Paula, Ingrid; Zamarioli, Ariane; Jorgetti, Vanda; Ramalho, Leandra Naíra Zambelli; Nogueira-Barbosa, Marcello Henrique; Volpon, José Batista; Jordão Júnior, Alceu Afonso; Cunha, Fernando de Queiróz; Fukada, Sandra Yasuyo; Paula, Francisco Jose Albuquerque de
Source: Clinics. Unidades: FMRP, FCFRP
Subjects: HEPATOPATIAS, OSTEOPOROSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SPIRLANDELI, Adriano Levi et al. Hepatic osteodystrophy: the mechanism of bone loss in hepatocellular disease and the effects of pamidronate treatment. Clinics, v. 72, n. 4, p. 231-237, 2017Tradução . . Disponível em: https://doi.org/10.6061/clinics/2017(04)07. Acesso em: 18 nov. 2024.
APA
Spirlandeli, A. L., Dick-de-Paula, I., Zamarioli, A., Jorgetti, V., Ramalho, L. N. Z., Nogueira-Barbosa, M. H., et al. (2017). Hepatic osteodystrophy: the mechanism of bone loss in hepatocellular disease and the effects of pamidronate treatment. Clinics, 72( 4), 231-237. doi:10.6061/clinics/2017(04)07
NLM
Spirlandeli AL, Dick-de-Paula I, Zamarioli A, Jorgetti V, Ramalho LNZ, Nogueira-Barbosa MH, Volpon JB, Jordão Júnior AA, Cunha F de Q, Fukada SY, Paula FJA de. Hepatic osteodystrophy: the mechanism of bone loss in hepatocellular disease and the effects of pamidronate treatment [Internet]. Clinics. 2017 ; 72( 4): 231-237.[citado 2024 nov. 18 ] Available from: https://doi.org/10.6061/clinics/2017(04)07
Vancouver
Spirlandeli AL, Dick-de-Paula I, Zamarioli A, Jorgetti V, Ramalho LNZ, Nogueira-Barbosa MH, Volpon JB, Jordão Júnior AA, Cunha F de Q, Fukada SY, Paula FJA de. Hepatic osteodystrophy: the mechanism of bone loss in hepatocellular disease and the effects of pamidronate treatment [Internet]. Clinics. 2017 ; 72( 4): 231-237.[citado 2024 nov. 18 ] Available from: https://doi.org/10.6061/clinics/2017(04)07
Artigo de periodico
Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis (2017)
Souza, Maria C.; Fonseca, Denise Morais da ; Kanashiro, Alexandre; Benevides, Luciana; Medina, Tiago S.; Dias, Murilo S.; Andrade, Warrison A.; Bonfá, Giuliano; Silva, Marcondes A. B.; Gozzi, Aline; Borges, Marcos de Carvalho; Gazzinelli, Ricardo T.; Alves Filho, José Carlos Farias; Cunha, Fernando Queiróz; Silva, João Santana da
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: MODELOS ANIMAIS DE DOENÇAS, INTERFERONS, ÓXIDO NÍTRICO, SEPSE, TOXOPLASMOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Maria C. et al. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, v. 7, 2017Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2017.00116. Acesso em: 18 nov. 2024.
APA
Souza, M. C., Fonseca, D. M. da, Kanashiro, A., Benevides, L., Medina, T. S., Dias, M. S., et al. (2017). Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, 7. doi:10.3389/fcimb.2017.00116
NLM
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Vancouver
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Artigo de periodico
Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection (2017)
Sacramento, Laís A.; Costa, Jéssica L. da; Lima, Mikhael H. F. de; Sampaio, Pedro A.; Almeida, Roque P.; Cunha, Fernando Queiróz; Silva, João Santana da; Carregaro, Vanessa
Source: Frontiers in Microbiology. Unidade: FMRP
Subjects: LEISHMANIA INFANTUM, CÉLULAS DENDRÍTICAS, LEISHMANIOSE VISCERAL, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SACRAMENTO, Laís A. et al. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.00262. Acesso em: 18 nov. 2024.
APA
Sacramento, L. A., Costa, J. L. da, Lima, M. H. F. de, Sampaio, P. A., Almeida, R. P., Cunha, F. Q., et al. (2017). Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, 8. doi:10.3389/fmicb.2017.00262
NLM
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Vancouver
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Artigo de periodico
The Sesquiterpene Lactone, Budlein A, Inhibits Antigen-Induced Arthritis in Mice: Role of NF-κB and Cytokines (2017)
Zarpelon, Ana C.; Fattori, Victor; Pinto, Larissa G.; Pinho-Ribeiro, Felipe A.; Ruiz-Miyazawa, Kenji W.; Turato, Walter Miguel; Cunha, Thiago Mattar; Costa, Fernando Batista da; Cunha, Fernando de Queiróz; Casagrande, Rubia; Arakawa, Nilton S.; Verri Junior, Waldiceu A.
Source: Inflammation. Unidades: FCF, EESC, FCFRP, FMRP
Subjects: ANTÍGENOS, ARTRITE, CITOCINAS, INFLAMAÇÃO, SESQUITERPENOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZARPELON, Ana C. et al. The Sesquiterpene Lactone, Budlein A, Inhibits Antigen-Induced Arthritis in Mice: Role of NF-κB and Cytokines. Inflammation, v. 40, n. 6, p. 2020-2032, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10753-017-0642-1. Acesso em: 18 nov. 2024.
APA
Zarpelon, A. C., Fattori, V., Pinto, L. G., Pinho-Ribeiro, F. A., Ruiz-Miyazawa, K. W., Turato, W. M., et al. (2017). The Sesquiterpene Lactone, Budlein A, Inhibits Antigen-Induced Arthritis in Mice: Role of NF-κB and Cytokines. Inflammation, 40( 6), 2020-2032. doi:10.1007/s10753-017-0642-1
NLM
Zarpelon AC, Fattori V, Pinto LG, Pinho-Ribeiro FA, Ruiz-Miyazawa KW, Turato WM, Cunha TM, Costa FB da, Cunha F de Q, Casagrande R, Arakawa NS, Verri Junior WA. The Sesquiterpene Lactone, Budlein A, Inhibits Antigen-Induced Arthritis in Mice: Role of NF-κB and Cytokines [Internet]. Inflammation. 2017 ; 40( 6): 2020-2032.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10753-017-0642-1
Vancouver
Zarpelon AC, Fattori V, Pinto LG, Pinho-Ribeiro FA, Ruiz-Miyazawa KW, Turato WM, Cunha TM, Costa FB da, Cunha F de Q, Casagrande R, Arakawa NS, Verri Junior WA. The Sesquiterpene Lactone, Budlein A, Inhibits Antigen-Induced Arthritis in Mice: Role of NF-κB and Cytokines [Internet]. Inflammation. 2017 ; 40( 6): 2020-2032.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10753-017-0642-1
Artigo de periodico
Neuroimmune–glia interactions in the sensory ganglia account for the development of acute herpetic neuralgia (2017)
Silva, Jaqueline Raymondi; Lopes, Alexandre H.; Talbot, Jhimmy; Cecílio, Nerry Tatiana; Rossato, Mateus F.; Silva, Rangel Leal; Souza, Guilherme Rabelo de; Silva, Cássia R.; Lucas, Guilherme de Araujo; Fonseca, Benedito Antonio Lopes da; Arruda, Eurico; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiróz; Cunha, Thiago Mattar
Source: The Journal of Neuroscience. Unidade: FMRP
Subjects: NEURÓGLIA, HERPES ZOSTER, DOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Jaqueline Raymondi et al. Neuroimmune–glia interactions in the sensory ganglia account for the development of acute herpetic neuralgia. The Journal of Neuroscience, v. 37, n. 27, p. 6408-6422, 2017Tradução . . Disponível em: https://doi.org/10.1523/jneurosci.2233-16.2017. Acesso em: 18 nov. 2024.
APA
Silva, J. R., Lopes, A. H., Talbot, J., Cecílio, N. T., Rossato, M. F., Silva, R. L., et al. (2017). Neuroimmune–glia interactions in the sensory ganglia account for the development of acute herpetic neuralgia. The Journal of Neuroscience, 37( 27), 6408-6422. doi:10.1523/jneurosci.2233-16.2017
NLM
Silva JR, Lopes AH, Talbot J, Cecílio NT, Rossato MF, Silva RL, Souza GR de, Silva CR, Lucas G de A, Fonseca BAL da, Arruda E, Alves Filho JCF, Cunha F de Q, Cunha TM. Neuroimmune–glia interactions in the sensory ganglia account for the development of acute herpetic neuralgia [Internet]. The Journal of Neuroscience. 2017 ; 37( 27): 6408-6422.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1523/jneurosci.2233-16.2017
Vancouver
Silva JR, Lopes AH, Talbot J, Cecílio NT, Rossato MF, Silva RL, Souza GR de, Silva CR, Lucas G de A, Fonseca BAL da, Arruda E, Alves Filho JCF, Cunha F de Q, Cunha TM. Neuroimmune–glia interactions in the sensory ganglia account for the development of acute herpetic neuralgia [Internet]. The Journal of Neuroscience. 2017 ; 37( 27): 6408-6422.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1523/jneurosci.2233-16.2017
Artigo de periodico
Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1 (2016)
Pinke, Karen Henriette; Lima, Heliton Gustavo de; Cunha, Fernando Queiróz; Lara, Vanessa Soares
Source: Immunobiology. Unidades: FOB, FMRP
Subjects: MASTÓCITOS, CANDIDA ALBICANS, FAGOCITOSE, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINKE, Karen Henriette et al. Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1. Immunobiology, v. 221, n. 2, p. 220-227, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2015.09.004. Acesso em: 18 nov. 2024.
APA
Pinke, K. H., Lima, H. G. de, Cunha, F. Q., & Lara, V. S. (2016). Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1. Immunobiology, 221( 2), 220-227. doi:10.1016/j.imbio.2015.09.004
NLM
Pinke KH, Lima HG de, Cunha FQ, Lara VS. Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1 [Internet]. Immunobiology. 2016 ; 221( 2): 220-227.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.imbio.2015.09.004
Vancouver
Pinke KH, Lima HG de, Cunha FQ, Lara VS. Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1 [Internet]. Immunobiology. 2016 ; 221( 2): 220-227.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.imbio.2015.09.004
Trabalho de evento-resumo periodico
Mast cells produce nitric oxide along with phagocytosis of C. albicans (2016)
Macedo, Beatriz Gomes; Pinke, Karen Henriette; Lima, Heliton Gustavo de; Cunha, Fernando Queiroz; Lara, Vanessa Soares
Source: Journal of Applied Oral Science. Conference titles: COB - Congresso Odontológico de Bauru "Prof. Dr. Aquira Ishikiriama". Unidades: FMRP, FOB
Subjects: CANDIDA ALBICANS, ÓXIDO NÍTRICO, FAGOCITOSE, MASTÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACEDO, Beatriz Gomes et al. Mast cells produce nitric oxide along with phagocytosis of C. albicans. Journal of Applied Oral Science. Bauru: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 18 nov. 2024. , 2016
APA
Macedo, B. G., Pinke, K. H., Lima, H. G. de, Cunha, F. Q., & Lara, V. S. (2016). Mast cells produce nitric oxide along with phagocytosis of C. albicans. Journal of Applied Oral Science. Bauru: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Macedo BG, Pinke KH, Lima HG de, Cunha FQ, Lara VS. Mast cells produce nitric oxide along with phagocytosis of C. albicans. Journal of Applied Oral Science. 2016 ; 24 205.[citado 2024 nov. 18 ]
Vancouver
Macedo BG, Pinke KH, Lima HG de, Cunha FQ, Lara VS. Mast cells produce nitric oxide along with phagocytosis of C. albicans. Journal of Applied Oral Science. 2016 ; 24 205.[citado 2024 nov. 18 ]
Trabalho de evento-resumo periodico
Macrophages from sepsis surviving mice present differential gene expression associated with an immunotolerant profile (2015)
Mota, J. M.; Wagatsuma, V.; Temporal, J.; Cunha, Fernando de Queiróz; Noushmehr, Houtan; Rego, Eduardo Magalhães
Source: Haematologica. Conference titles: Congress of the European Hematology Association. Unidade: FMRP
Subjects: MACRÓFAGOS, EXPRESSÃO GÊNICA, TOLERÂNCIA IMUNOLÓGICA, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOTA, J. M. et al. Macrophages from sepsis surviving mice present differential gene expression associated with an immunotolerant profile. Haematologica. Pavia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: http://www.haematologica.org/content/100/supplement_1/1.full-text.pdf+html. Acesso em: 18 nov. 2024. , 2015
APA
Mota, J. M., Wagatsuma, V., Temporal, J., Cunha, F. de Q., Noushmehr, H., & Rego, E. M. (2015). Macrophages from sepsis surviving mice present differential gene expression associated with an immunotolerant profile. Haematologica. Pavia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de http://www.haematologica.org/content/100/supplement_1/1.full-text.pdf+html
NLM
Mota JM, Wagatsuma V, Temporal J, Cunha F de Q, Noushmehr H, Rego EM. Macrophages from sepsis surviving mice present differential gene expression associated with an immunotolerant profile [Internet]. Haematologica. 2015 ; 100 467.[citado 2024 nov. 18 ] Available from: http://www.haematologica.org/content/100/supplement_1/1.full-text.pdf+html
Vancouver
Mota JM, Wagatsuma V, Temporal J, Cunha F de Q, Noushmehr H, Rego EM. Macrophages from sepsis surviving mice present differential gene expression associated with an immunotolerant profile [Internet]. Haematologica. 2015 ; 100 467.[citado 2024 nov. 18 ] Available from: http://www.haematologica.org/content/100/supplement_1/1.full-text.pdf+html

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