NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation (2020)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; CUNHA, THIAGO MATTAR - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; VERAS, FLÁVIO PROTÁSIO - FCFRP ; PRADO, DOUGLAS DA SILVA - FMRP
- Unidades: FMRP; FCFRP
- DOI: 10.1096/fj.202000795R
- Subjects: ARTRITE REUMATOIDE; DIFERENCIAÇÃO CELULAR; INFLAMAÇÃO; INTERLEUCINAS; ARTICULAÇÕES; NEUTRÓFILOS; FATORES DE TRANSCRIÇÃO
- Keywords: Autoimmune disease; STAT3; T cell,; Inflammation; NLR
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: The FASEB Journal
- ISSN: 0892-6638
- Volume/Número/Paginação/Ano: v. 34, n. 8, p. 10907-10919, 2020
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
PRADO, Douglas Silva et al. NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation. The FASEB Journal, v. 34, n. 8, p. 10907-10919, 2020Tradução . . Disponível em: https://doi.org/10.1096/fj.202000795R. Acesso em: 27 dez. 2025. -
APA
Prado, D. S., Veras, F. P., Ferreira, R. G., Damasceno, L. E. A., Melo, P. H., Zamboni, D. S., et al. (2020). NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation. The FASEB Journal, 34( 8), 10907-10919. doi:10.1096/fj.202000795R -
NLM
Prado DS, Veras FP, Ferreira RG, Damasceno LEA, Melo PH, Zamboni DS, Cunha TM, Cunha FQ, Alves Filho JCF. NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation [Internet]. The FASEB Journal. 2020 ; 34( 8): 10907-10919.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1096/fj.202000795R -
Vancouver
Prado DS, Veras FP, Ferreira RG, Damasceno LEA, Melo PH, Zamboni DS, Cunha TM, Cunha FQ, Alves Filho JCF. NLRP12 controls arthritis severity by acting as a checkpoint inhibitor of Th17 cell differentiation [Internet]. The FASEB Journal. 2020 ; 34( 8): 10907-10919.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1096/fj.202000795R - MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response
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Informações sobre o DOI: 10.1096/fj.202000795R (Fonte: oaDOI API)
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