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Artigo de periodico
The PI3Kγ/AKT signaling pathway mediates peripheral antinociceptive action of dipyrone (2021)
Cecílio, Nerry Tatiana; Souza, Guilherme Rabelo de; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar
Source: Fundamental & Clinical Pharmacology. Unidade: FMRP
Subjects: ANALGÉSICOS, DOR, INFLAMAÇÃO, ÓXIDO NÍTRICO
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ABNT
CECÍLIO, Nerry Tatiana et al. The PI3Kγ/AKT signaling pathway mediates peripheral antinociceptive action of dipyrone. Fundamental & Clinical Pharmacology, v. 35, n. 2, p. 364-370, 2021Tradução . . Disponível em: https://doi.org/10.1111/fcp.12606. Acesso em: 19 nov. 2024.
APA
Cecílio, N. T., Souza, G. R. de, Alves Filho, J. C. F., Cunha, F. de Q., & Cunha, T. M. (2021). The PI3Kγ/AKT signaling pathway mediates peripheral antinociceptive action of dipyrone. Fundamental & Clinical Pharmacology, 35( 2), 364-370. doi:10.1111/fcp.12606
NLM
Cecílio NT, Souza GR de, Alves Filho JCF, Cunha F de Q, Cunha TM. The PI3Kγ/AKT signaling pathway mediates peripheral antinociceptive action of dipyrone [Internet]. Fundamental & Clinical Pharmacology. 2021 ; 35( 2): 364-370.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/fcp.12606
Vancouver
Cecílio NT, Souza GR de, Alves Filho JCF, Cunha F de Q, Cunha TM. The PI3Kγ/AKT signaling pathway mediates peripheral antinociceptive action of dipyrone [Internet]. Fundamental & Clinical Pharmacology. 2021 ; 35( 2): 364-370.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/fcp.12606
Artigo de periodico
Choline attenuates inflammatory hyperalgesia activating nitric oxide/cGMP/ATP-sensitive potassium channels pathway (2020)
Kusuda, Ricardo; Carreira, Eleonora Uchôa; Ulloa, Luis; Cunha, Fernando de Queiroz ; Kanashiro, Alexandre; Cunha, Thiago Mattar
Source: Brain Research. Unidade: FMRP
Subjects: CANAIS DE POTÁSSIO, ÓXIDO NÍTRICO, NEUROIMUNOMODULAÇÃO, INFLAMAÇÃO, NEURÔNIOS, NOCICEPTIVIDADE
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ABNT
KUSUDA, Ricardo et al. Choline attenuates inflammatory hyperalgesia activating nitric oxide/cGMP/ATP-sensitive potassium channels pathway. Brain Research, v. 1727, p. 1-8, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2019.146567. Acesso em: 19 nov. 2024.
APA
Kusuda, R., Carreira, E. U., Ulloa, L., Cunha, F. de Q., Kanashiro, A., & Cunha, T. M. (2020). Choline attenuates inflammatory hyperalgesia activating nitric oxide/cGMP/ATP-sensitive potassium channels pathway. Brain Research, 1727, 1-8. doi:10.1016/j.brainres.2019.146567
NLM
Kusuda R, Carreira EU, Ulloa L, Cunha F de Q, Kanashiro A, Cunha TM. Choline attenuates inflammatory hyperalgesia activating nitric oxide/cGMP/ATP-sensitive potassium channels pathway [Internet]. Brain Research. 2020 ; 1727 1-8.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.brainres.2019.146567
Vancouver
Kusuda R, Carreira EU, Ulloa L, Cunha F de Q, Kanashiro A, Cunha TM. Choline attenuates inflammatory hyperalgesia activating nitric oxide/cGMP/ATP-sensitive potassium channels pathway [Internet]. Brain Research. 2020 ; 1727 1-8.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.brainres.2019.146567
Artigo de periodico
Peripheral nitric oxide signaling directly blocks inflammatory pain (2020)
Gomes, Francisco Isaac Fernandes; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, ANALGESIA, DOR, INFLAMAÇÃO, NOCICEPTIVIDADE
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ABNT
GOMES, Francisco Isaac Fernandes e CUNHA, Fernando de Queiroz e CUNHA, Thiago Mattar. Peripheral nitric oxide signaling directly blocks inflammatory pain. Biochemical Pharmacology, v. 176, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2020.113862. Acesso em: 19 nov. 2024.
APA
Gomes, F. I. F., Cunha, F. de Q., & Cunha, T. M. (2020). Peripheral nitric oxide signaling directly blocks inflammatory pain. Biochemical Pharmacology, 176. doi:10.1016/j.bcp.2020.113862
NLM
Gomes FIF, Cunha F de Q, Cunha TM. Peripheral nitric oxide signaling directly blocks inflammatory pain [Internet]. Biochemical Pharmacology. 2020 ; 176[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.bcp.2020.113862
Vancouver
Gomes FIF, Cunha F de Q, Cunha TM. Peripheral nitric oxide signaling directly blocks inflammatory pain [Internet]. Biochemical Pharmacology. 2020 ; 176[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.bcp.2020.113862
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 19 nov. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Targeting nitric oxide as a key modulator of sepsis, arthritis and pain (2019)
Spiller, Fernando; Formiga, Rodrigo Oliveira; Coimbra, Jonathan Fernandes da Silva; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz
Source: Nitric Oxide. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO, DOR, SEPSE, ARTRITE
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ABNT
SPILLER, Fernando et al. Targeting nitric oxide as a key modulator of sepsis, arthritis and pain. Nitric Oxide, v. 89, p. 32-40, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.04.011. Acesso em: 19 nov. 2024.
APA
Spiller, F., Formiga, R. O., Coimbra, J. F. da S., Alves Filho, J. C. F., Cunha, T. M., & Cunha, F. de Q. (2019). Targeting nitric oxide as a key modulator of sepsis, arthritis and pain. Nitric Oxide, 89, 32-40. doi:10.1016/j.niox.2019.04.011
NLM
Spiller F, Formiga RO, Coimbra JF da S, Alves Filho JCF, Cunha TM, Cunha F de Q. Targeting nitric oxide as a key modulator of sepsis, arthritis and pain [Internet]. Nitric Oxide. 2019 ; 89 32-40.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.niox.2019.04.011
Vancouver
Spiller F, Formiga RO, Coimbra JF da S, Alves Filho JCF, Cunha TM, Cunha F de Q. Targeting nitric oxide as a key modulator of sepsis, arthritis and pain [Internet]. Nitric Oxide. 2019 ; 89 32-40.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.niox.2019.04.011
Artigo de periodico
Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis (2018)
Staurengo-Ferrari, Larissa; Trevelin, Silvia C.; Fattori, Victor; Nascimento, Daniele C.; Lima, Kalil A. de; Pelayo, Jacinta S.; Figueiredo, Florêncio; Casagrande, Rubia; Fukada, Sandra Yasuyo; Teixeira, Mauro M.; Cunha, Thiago Mattar; Liew, Foo Y.; Oliveira, Rene D.; Louzada Júnior, Paulo; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Verri, Waldiceu A.
Source: Frontiers in Immunology. Unidades: FCFRP, FMRP
Subjects: MACRÓFAGOS, STAPHYLOCOCCUS, INTERLEUCINAS, ÓXIDO NÍTRICO, ARTRITE
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ABNT
STAURENGO-FERRARI, Larissa et al. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis. Frontiers in Immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00962. Acesso em: 19 nov. 2024.
APA
Staurengo-Ferrari, L., Trevelin, S. C., Fattori, V., Nascimento, D. C., Lima, K. A. de, Pelayo, J. S., et al. (2018). Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis. Frontiers in Immunology, 9. doi:10.3389/fimmu.2018.00962
NLM
Staurengo-Ferrari L, Trevelin SC, Fattori V, Nascimento DC, Lima KA de, Pelayo JS, Figueiredo F, Casagrande R, Fukada SY, Teixeira MM, Cunha TM, Liew FY, Oliveira RD, Louzada Júnior P, Cunha F de Q, Alves Filho JCF, Verri WA. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fimmu.2018.00962
Vancouver
Staurengo-Ferrari L, Trevelin SC, Fattori V, Nascimento DC, Lima KA de, Pelayo JS, Figueiredo F, Casagrande R, Fukada SY, Teixeira MM, Cunha TM, Liew FY, Oliveira RD, Louzada Júnior P, Cunha F de Q, Alves Filho JCF, Verri WA. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fimmu.2018.00962
Artigo de periodico
Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase (2017)
Dal Secco, Daniela; DalBó, Silvia; Lautherbach, Natalia Ennes da Silva; Gava, Fábio N.; Celes, Mara Rúbia Nunes; Benedet, Patricia de OLiveira; Souza, Adriana Helena de; Akinaga, Juliana; Lima, Vanessa; Silva, Katiussia P.; Kiguti, Luiz Ricardo A.; Rossi, Marcos Antonio; Kettelhut, Isis do Carmo; Pupo, André Sampaio; Cunha, Fernando de Queiroz; Assreuy, Jamil
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidade: FMRP
Subjects: ATIVAÇÃO ENZIMÁTICA, INSUFICIÊNCIA CARDÍACA, ÓXIDO NÍTRICO, SEPSE, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
DAL SECCO, Daniela et al. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, v. 313, n. 1, p. H149-H163, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00052.2016. Acesso em: 19 nov. 2024.
APA
Dal Secco, D., DalBó, S., Lautherbach, N. E. da S., Gava, F. N., Celes, M. R. N., Benedet, P. de O. L., et al. (2017). Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, 313( 1), H149-H163. doi:10.1152/ajpheart.00052.2016
NLM
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016
Vancouver
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016
Artigo de periodico
Nitric oxide and hydrogen sulfide interact when modulating gastric physiological functions in rodents (2017)
Lucetti, Larisse Tavares; Silva, Renan Oliveira; Santana, Ana Paula Macedo; Tavares, Bruno de Melo; Vale, Mariana Lima; Soares, Pedro Marcos Gomes; Lima Júnior, Francisco José Batista de; Magalhães, Pedro Jorge Caldas; Cunha, Fernando de Queiroz; Ribeiro, Ronaldo de Albuquerque; Medeiros, Jand-Venes Rolim; Souza, Marcellus Henrique Loiola Ponte
Source: Digestive Diseases and Sciences. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, DEPRESSORES, SISTEMA GASTROINTESTINAL, ESTÔMAGO
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ABNT
LUCETTI, Larisse Tavares et al. Nitric oxide and hydrogen sulfide interact when modulating gastric physiological functions in rodents. Digestive Diseases and Sciences, v. 62, n. 1, p. 93-104, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10620-016-4377-x. Acesso em: 19 nov. 2024.
APA
Lucetti, L. T., Silva, R. O., Santana, A. P. M., Tavares, B. de M., Vale, M. L., Soares, P. M. G., et al. (2017). Nitric oxide and hydrogen sulfide interact when modulating gastric physiological functions in rodents. Digestive Diseases and Sciences, 62( 1), 93-104. doi:10.1007/s10620-016-4377-x
NLM
Lucetti LT, Silva RO, Santana APM, Tavares B de M, Vale ML, Soares PMG, Lima Júnior FJB de, Magalhães PJC, Cunha F de Q, Ribeiro R de A, Medeiros J-VR, Souza MHLP. Nitric oxide and hydrogen sulfide interact when modulating gastric physiological functions in rodents [Internet]. Digestive Diseases and Sciences. 2017 ; 62( 1): 93-104.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s10620-016-4377-x
Vancouver
Lucetti LT, Silva RO, Santana APM, Tavares B de M, Vale ML, Soares PMG, Lima Júnior FJB de, Magalhães PJC, Cunha F de Q, Ribeiro R de A, Medeiros J-VR, Souza MHLP. Nitric oxide and hydrogen sulfide interact when modulating gastric physiological functions in rodents [Internet]. Digestive Diseases and Sciences. 2017 ; 62( 1): 93-104.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s10620-016-4377-x
Artigo de periodico
Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB (2016)
Franchin, Marcelo; Rosalen, Pedro Luiz; Cunha, Marcos Guilherme da; Silva, Rangel Leal; Colón, David F.; Bassi, Gabriel Shimizu; Alencar, Severino Matias de; Ikegaki, Masaharu; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Beutler, John A.; Cunha, Thiago Mattar
Source: Journal of Natural Products. Unidades: FMRP, ESALQ
Subjects: ANTI-INFLAMATÓRIOS, LIPOPOLISSACARÍDEOS, ÓXIDO NÍTRICO, MACRÓFAGOS, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
FRANCHIN, Marcelo et al. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB. Journal of Natural Products, v. 79, n. 7, p. 1828-1833, 2016Tradução . . Disponível em: https://doi.org/10.1021/acs.jnatprod.6b00263. Acesso em: 19 nov. 2024.
APA
Franchin, M., Rosalen, P. L., Cunha, M. G. da, Silva, R. L., Colón, D. F., Bassi, G. S., et al. (2016). Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB. Journal of Natural Products, 79( 7), 1828-1833. doi:10.1021/acs.jnatprod.6b00263
NLM
Franchin M, Rosalen PL, Cunha MG da, Silva RL, Colón DF, Bassi GS, Alencar SM de, Ikegaki M, Alves Filho JCF, Cunha F de Q, Beutler JA, Cunha TM. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB [Internet]. Journal of Natural Products. 2016 ; 79( 7): 1828-1833.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1021/acs.jnatprod.6b00263
Vancouver
Franchin M, Rosalen PL, Cunha MG da, Silva RL, Colón DF, Bassi GS, Alencar SM de, Ikegaki M, Alves Filho JCF, Cunha F de Q, Beutler JA, Cunha TM. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB [Internet]. Journal of Natural Products. 2016 ; 79( 7): 1828-1833.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1021/acs.jnatprod.6b00263
Artigo de periodico
IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system (2016)
França, Rafael Freitas de Oliveira; Costa, Renata Sesti; Silva, Jaqueline Raymondi; Peres, Raphael S.; Mendonça, Leila Rodrigues de; Colón, David F.; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Source: Journal of Neuroinflammation. Unidade: FMRP
Subjects: FLAVIVIRUS, ÓXIDO NÍTRICO, INTERLEUCINAS
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ABNT
FRANÇA, Rafael Freitas de Oliveira et al. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system. Journal of Neuroinflammation, v. 13, n. 1, 2016Tradução . . Disponível em: https://doi.org/10.1186/s12974-016-0628-1. Acesso em: 19 nov. 2024.
APA
França, R. F. de O., Costa, R. S., Silva, J. R., Peres, R. S., Mendonça, L. R. de, Colón, D. F., et al. (2016). IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system. Journal of Neuroinflammation, 13( 1). doi:10.1186/s12974-016-0628-1
NLM
França RF de O, Costa RS, Silva JR, Peres RS, Mendonça LR de, Colón DF, Alves Filho JCF, Cunha F de Q. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system [Internet]. Journal of Neuroinflammation. 2016 ; 13( 1):[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/s12974-016-0628-1
Vancouver
França RF de O, Costa RS, Silva JR, Peres RS, Mendonça LR de, Colón DF, Alves Filho JCF, Cunha F de Q. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system [Internet]. Journal of Neuroinflammation. 2016 ; 13( 1):[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/s12974-016-0628-1
Artigo de periodico
Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines (2016)
Santos, Sidnéia S.; Carmo, Amanda M.; Brunialti, Milena K. C.; Machado, Flavia R.; Azevedo, Luciano C.; Assunção, Murillo; Trevelin, Silvia C.; Cunha, Fernando de Queiroz; Salomão, Reinaldo
Source: Intensive Care Medicine Experimental. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, CITOCINAS, INFLAMAÇÃO
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ABNT
SANTOS, Sidnéia S. et al. Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines. Intensive Care Medicine Experimental, v. 4, 2016Tradução . . Disponível em: https://doi.org/10.1186/s40635-016-0078-1. Acesso em: 19 nov. 2024.
APA
Santos, S. S., Carmo, A. M., Brunialti, M. K. C., Machado, F. R., Azevedo, L. C., Assunção, M., et al. (2016). Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines. Intensive Care Medicine Experimental, 4. doi:10.1186/s40635-016-0078-1
NLM
Santos SS, Carmo AM, Brunialti MKC, Machado FR, Azevedo LC, Assunção M, Trevelin SC, Cunha F de Q, Salomão R. Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines [Internet]. Intensive Care Medicine Experimental. 2016 ; 4[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/s40635-016-0078-1
Vancouver
Santos SS, Carmo AM, Brunialti MKC, Machado FR, Azevedo LC, Assunção M, Trevelin SC, Cunha F de Q, Salomão R. Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines [Internet]. Intensive Care Medicine Experimental. 2016 ; 4[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/s40635-016-0078-1
Artigo de periodico
α-Galactosylceramide suppresses murine eosinophil production through interferon-γ-dependent induction of NO synthase and CD95 (2015)
Gaspar-Elsas, Maria Ignez; Queto, Túlio; Masid-de-Brito, Daniela; Vieira, Bruno Marques; Luca, Bianca de; Cunha, Fernando de Queiroz; Xavier-Elsas, Pedro
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: ADJUVANTES IMUNOLÓGICOS, ÓXIDO NÍTRICO, MEDULA ÓSSEA
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ABNT
GASPAR-ELSAS, Maria Ignez et al. α-Galactosylceramide suppresses murine eosinophil production through interferon-γ-dependent induction of NO synthase and CD95. British Journal of Pharmacology, v. 172, n. 13, p. 3313-3325, 2015Tradução . . Disponível em: https://doi.org/10.1111/bph.13126. Acesso em: 19 nov. 2024.
APA
Gaspar-Elsas, M. I., Queto, T., Masid-de-Brito, D., Vieira, B. M., Luca, B. de, Cunha, F. de Q., & Xavier-Elsas, P. (2015). α-Galactosylceramide suppresses murine eosinophil production through interferon-γ-dependent induction of NO synthase and CD95. British Journal of Pharmacology, 172( 13), 3313-3325. doi:10.1111/bph.13126
NLM
Gaspar-Elsas MI, Queto T, Masid-de-Brito D, Vieira BM, Luca B de, Cunha F de Q, Xavier-Elsas P. α-Galactosylceramide suppresses murine eosinophil production through interferon-γ-dependent induction of NO synthase and CD95 [Internet]. British Journal of Pharmacology. 2015 ; 172( 13): 3313-3325.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/bph.13126
Vancouver
Gaspar-Elsas MI, Queto T, Masid-de-Brito D, Vieira BM, Luca B de, Cunha F de Q, Xavier-Elsas P. α-Galactosylceramide suppresses murine eosinophil production through interferon-γ-dependent induction of NO synthase and CD95 [Internet]. British Journal of Pharmacology. 2015 ; 172( 13): 3313-3325.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/bph.13126
Artigo de periodico
The quassinoid isobrucein B reduces inflammatory hyperalgesia and cytokine production by post-transcriptional modulation (2015)
Silva, Rangel L.; Lopes, Alexandre H.; França, Rafael O.; Vieira, Sílvio M.; Silva, Ellen C. C.; Amorim, Rodrigo C. N.; Cunha, Fernando de Queiroz; Pohlit, Adrian M.; Cunha, Thiago Mattar
Source: Journal of Natural Products. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS (FARMACOLOGIA), INFLAMAÇÃO, INTERLEUCINA 1 (METABOLISMO), PROTEÍNAS QUINASES, ÓXIDO NÍTRICO, LIPOPOLISSACARÍDEOS
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ABNT
SILVA, Rangel L. et al. The quassinoid isobrucein B reduces inflammatory hyperalgesia and cytokine production by post-transcriptional modulation. Journal of Natural Products, v. 78, n. 2, p. 241-249, 2015Tradução . . Disponível em: https://doi.org/10.1021/np500796f. Acesso em: 19 nov. 2024.
APA
Silva, R. L., Lopes, A. H., França, R. O., Vieira, S. M., Silva, E. C. C., Amorim, R. C. N., et al. (2015). The quassinoid isobrucein B reduces inflammatory hyperalgesia and cytokine production by post-transcriptional modulation. Journal of Natural Products, 78( 2), 241-249. doi:10.1021/np500796f
NLM
Silva RL, Lopes AH, França RO, Vieira SM, Silva ECC, Amorim RCN, Cunha F de Q, Pohlit AM, Cunha TM. The quassinoid isobrucein B reduces inflammatory hyperalgesia and cytokine production by post-transcriptional modulation [Internet]. Journal of Natural Products. 2015 ; 78( 2): 241-249.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1021/np500796f
Vancouver
Silva RL, Lopes AH, França RO, Vieira SM, Silva ECC, Amorim RCN, Cunha F de Q, Pohlit AM, Cunha TM. The quassinoid isobrucein B reduces inflammatory hyperalgesia and cytokine production by post-transcriptional modulation [Internet]. Journal of Natural Products. 2015 ; 78( 2): 241-249.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1021/np500796f
Artigo de periodico
Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy (2014)
Mendonça, Maria Angélica Oliveira; Souto, Fabrício O.; Micheli, Douglas C.; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Murta, Eddie Fernando C.; Tavares-Murta, Beatriz M.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: NEOPLASIAS MAMÁRIAS, QUIMIOTERAPIA, CITOCINAS, ÓXIDO NÍTRICO, RECEPTORES CELULARES
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ABNT
MENDONÇA, Maria Angélica Oliveira et al. Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy. Cancer Chemotherapy and Pharmacology, v. 73, n. 2, p. 317-324, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00280-013-2348-x. Acesso em: 19 nov. 2024.
APA
Mendonça, M. A. O., Souto, F. O., Micheli, D. C., Alves-Filho, J. C., Cunha, F. de Q., Murta, E. F. C., & Tavares-Murta, B. M. (2014). Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy. Cancer Chemotherapy and Pharmacology, 73( 2), 317-324. doi:10.1007/s00280-013-2348-x
NLM
Mendonça MAO, Souto FO, Micheli DC, Alves-Filho JC, Cunha F de Q, Murta EFC, Tavares-Murta BM. Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy [Internet]. Cancer Chemotherapy and Pharmacology. 2014 ; 73( 2): 317-324.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00280-013-2348-x
Vancouver
Mendonça MAO, Souto FO, Micheli DC, Alves-Filho JC, Cunha F de Q, Murta EFC, Tavares-Murta BM. Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy [Internet]. Cancer Chemotherapy and Pharmacology. 2014 ; 73( 2): 317-324.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00280-013-2348-x
Artigo de periodico
Novel bisabolane derivative from "arnica-da-serra" (Vernonieae: Asteraceae) reduces pro-nociceptive cytokines levels in LPS-stimulated rat macrophages (2013)
Pavarini, Daniel Petinatti; Nogueira, Elídia Fernandes; Callejon, Daniel Roberto; Soares, Denis Melo; Souza, Glória Emília Petto de; Cunha, Fernando de Queiroz; Lopes, Joao Luis Callegari; Lopes, Norberto Peporine
Source: Journal of Ethnopharmacology. Unidades: FCFRP, FMRP
Subjects: COMPOSITAE, ANTI-INFLAMATÓRIOS, INTERLEUCINAS, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAVARINI, Daniel Petinatti et al. Novel bisabolane derivative from "arnica-da-serra" (Vernonieae: Asteraceae) reduces pro-nociceptive cytokines levels in LPS-stimulated rat macrophages. Journal of Ethnopharmacology, v. 148, n. 3, p. 993-998, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.jep.2013.05.003. Acesso em: 19 nov. 2024.
APA
Pavarini, D. P., Nogueira, E. F., Callejon, D. R., Soares, D. M., Souza, G. E. P. de, Cunha, F. de Q., et al. (2013). Novel bisabolane derivative from "arnica-da-serra" (Vernonieae: Asteraceae) reduces pro-nociceptive cytokines levels in LPS-stimulated rat macrophages. Journal of Ethnopharmacology, 148( 3), 993-998. doi:10.1016/j.jep.2013.05.003
NLM
Pavarini DP, Nogueira EF, Callejon DR, Soares DM, Souza GEP de, Cunha F de Q, Lopes JLC, Lopes NP. Novel bisabolane derivative from "arnica-da-serra" (Vernonieae: Asteraceae) reduces pro-nociceptive cytokines levels in LPS-stimulated rat macrophages [Internet]. Journal of Ethnopharmacology. 2013 ; 148( 3): 993-998.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.jep.2013.05.003
Vancouver
Pavarini DP, Nogueira EF, Callejon DR, Soares DM, Souza GEP de, Cunha F de Q, Lopes JLC, Lopes NP. Novel bisabolane derivative from "arnica-da-serra" (Vernonieae: Asteraceae) reduces pro-nociceptive cytokines levels in LPS-stimulated rat macrophages [Internet]. Journal of Ethnopharmacology. 2013 ; 148( 3): 993-998.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.jep.2013.05.003
Artigo de periodico
Skin vasodilation and analgesic effect of a topical nitric oxide-releasing hydrogel (2013)
Vercelino, Rafael; Cunha, Thiago Mattar; Ferreira, Elisa Silva; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique; Oliveira, Marcelo Ganzarolli de
Source: Journal of Materials Science: Materials in Medicine. Unidade: FMRP
Subjects: ANALGÉSICOS, ÓXIDO NÍTRICO, MEDICAMENTO TÓPICO, DOR, FLUXO SANGUÍNEO, INFLAMAÇÃO, PELE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VERCELINO, Rafael et al. Skin vasodilation and analgesic effect of a topical nitric oxide-releasing hydrogel. Journal of Materials Science: Materials in Medicine, v. 24, n. 9, p. 2157-2169, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10856-013-4973-7. Acesso em: 19 nov. 2024.
APA
Vercelino, R., Cunha, T. M., Ferreira, E. S., Cunha, F. de Q., Ferreira, S. H., & Oliveira, M. G. de. (2013). Skin vasodilation and analgesic effect of a topical nitric oxide-releasing hydrogel. Journal of Materials Science: Materials in Medicine, 24( 9), 2157-2169. doi:10.1007/s10856-013-4973-7
NLM
Vercelino R, Cunha TM, Ferreira ES, Cunha F de Q, Ferreira SH, Oliveira MG de. Skin vasodilation and analgesic effect of a topical nitric oxide-releasing hydrogel [Internet]. Journal of Materials Science: Materials in Medicine. 2013 ; 24( 9): 2157-2169.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s10856-013-4973-7
Vancouver
Vercelino R, Cunha TM, Ferreira ES, Cunha F de Q, Ferreira SH, Oliveira MG de. Skin vasodilation and analgesic effect of a topical nitric oxide-releasing hydrogel [Internet]. Journal of Materials Science: Materials in Medicine. 2013 ; 24( 9): 2157-2169.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s10856-013-4973-7
Artigo de periodico
The nitroxyl donor, Angeli's salt, inhibits inflammatory hyperalgesia in rats (2013)
Zarpelon, Ana C.; Souza, Guilherme R.; Cunha, Thiago Mattar; Schivo, Ieda Regina dos Santos; Marchesi, Mario; Casagrande, Rubia; Pinge-Filho, Phileno; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique; Miranda, Katrina M.; Verri Júnior, Waldiceu A.
Source: Neuropharmacology. Unidade: FMRP
Subjects: ANALGÉSICOS (FARMACOLOGIA), ANTI-INFLAMATÓRIOS NÃO ESTEROIDES (ADMINISTRAÇÃO E DOSAGEM), MODELOS ANIMAIS DE DOENÇAS, DOR, NOCICEPTORES, ÓXIDO NÍTRICO
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ABNT
ZARPELON, Ana C. et al. The nitroxyl donor, Angeli's salt, inhibits inflammatory hyperalgesia in rats. Neuropharmacology, v. 71, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.03.009. Acesso em: 19 nov. 2024.
APA
Zarpelon, A. C., Souza, G. R., Cunha, T. M., Schivo, I. R. dos S., Marchesi, M., Casagrande, R., et al. (2013). The nitroxyl donor, Angeli's salt, inhibits inflammatory hyperalgesia in rats. Neuropharmacology, 71, 1-9. doi:10.1016/j.neuropharm.2013.03.009
NLM
Zarpelon AC, Souza GR, Cunha TM, Schivo IR dos S, Marchesi M, Casagrande R, Pinge-Filho P, Cunha F de Q, Ferreira SH, Miranda KM, Verri Júnior WA. The nitroxyl donor, Angeli's salt, inhibits inflammatory hyperalgesia in rats [Internet]. Neuropharmacology. 2013 ; 71 1-9.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.03.009
Vancouver
Zarpelon AC, Souza GR, Cunha TM, Schivo IR dos S, Marchesi M, Casagrande R, Pinge-Filho P, Cunha F de Q, Ferreira SH, Miranda KM, Verri Júnior WA. The nitroxyl donor, Angeli's salt, inhibits inflammatory hyperalgesia in rats [Internet]. Neuropharmacology. 2013 ; 71 1-9.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.03.009
Artigo de periodico
Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents (2013)
Luca, Bianca de; Xavier-Elsas, Pedro; Barradas, Mônica; Luz, Ricardo A.; Queto, Túlio; Jones, Carla; Arruda, Maria Augusta; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Gaspar-Elsas, Maria Ignez
Source: The Scientific World Journal. Unidade: FMRP
Subjects: FARMACOLOGIA, IMUNOLOGIA, ÓXIDO NÍTRICO
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ABNT
LUCA, Bianca de et al. Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents. The Scientific World Journal, v. 2013, 2013Tradução . . Disponível em: https://doi.org/10.1155/2013/208705. Acesso em: 19 nov. 2024.
APA
Luca, B. de, Xavier-Elsas, P., Barradas, M., Luz, R. A., Queto, T., Jones, C., et al. (2013). Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents. The Scientific World Journal, 2013. doi:10.1155/2013/208705
NLM
Luca B de, Xavier-Elsas P, Barradas M, Luz RA, Queto T, Jones C, Arruda MA, Cunha TM, Cunha F de Q, Gaspar-Elsas MI. Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents [Internet]. The Scientific World Journal. 2013 ; 2013[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2013/208705
Vancouver
Luca B de, Xavier-Elsas P, Barradas M, Luz RA, Queto T, Jones C, Arruda MA, Cunha TM, Cunha F de Q, Gaspar-Elsas MI. Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents [Internet]. The Scientific World Journal. 2013 ; 2013[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2013/208705
Artigo de periodico
Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation (2012)
Lima Júnior, Roberto César P.; Figueiredo, Aline A.; Freitas, Helano C.; Melo, Maria Luisa P.; Wong, Deysi Viviana T.; Leite, Caio Abner V. G.; Medeiros, Raul P.; Marques Neto, Raphael D.; Vale, Mariana L.; Brito, Gerly Anne C.; Oriá, Reinaldo B.; Souza, Marcellus H. L. P.; Cunha, Fernando de Queiroz; Ribeiro, Ronaldo A.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS (TOXICIDADE), CITOCINAS (METABOLISMO), MUCOSA INTESTINAL (EFEITOS DE DROGAS), ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA JÚNIOR, Roberto César P. et al. Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation. Cancer Chemotherapy and Pharmacology, v. 69, n. 4, p. 931-942, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00280-011-1780-z. Acesso em: 19 nov. 2024.
APA
Lima Júnior, R. C. P., Figueiredo, A. A., Freitas, H. C., Melo, M. L. P., Wong, D. V. T., Leite, C. A. V. G., et al. (2012). Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation. Cancer Chemotherapy and Pharmacology, 69( 4), 931-942. doi:10.1007/s00280-011-1780-z
NLM
Lima Júnior RCP, Figueiredo AA, Freitas HC, Melo MLP, Wong DVT, Leite CAVG, Medeiros RP, Marques Neto RD, Vale ML, Brito GAC, Oriá RB, Souza MHLP, Cunha F de Q, Ribeiro RA. Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation [Internet]. Cancer Chemotherapy and Pharmacology. 2012 ; 69( 4): 931-942.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00280-011-1780-z
Vancouver
Lima Júnior RCP, Figueiredo AA, Freitas HC, Melo MLP, Wong DVT, Leite CAVG, Medeiros RP, Marques Neto RD, Vale ML, Brito GAC, Oriá RB, Souza MHLP, Cunha F de Q, Ribeiro RA. Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation [Internet]. Cancer Chemotherapy and Pharmacology. 2012 ; 69( 4): 931-942.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00280-011-1780-z
Artigo de periodico
Stimulation of peripheral kappa opioid receptors inhibits inflammatory hyperalgesia via activation of the PI3K'gama'/AKT/nNOS/NO signaling pathway (2012)
Cunha, Thiago Mattar; Souza, Guilherme R.; Domingues, Andressa C.; Carreira, Eleonora U.; Lotufo, Celina M.; Funez, Mani I.; Verri Júnior, Waldiceu Aparecido; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique
Source: Molecular Pain. Unidade: FMRP
Subjects: INFLAMAÇÃO, RECEPTORES CELULARES (METABOLISMO), ÓXIDO NÍTRICO, ENZIMAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, Thiago Mattar et al. Stimulation of peripheral kappa opioid receptors inhibits inflammatory hyperalgesia via activation of the PI3K'gama'/AKT/nNOS/NO signaling pathway. Molecular Pain, 2012Tradução . . Disponível em: https://doi.org/10.1186/1744-8069-8-10. Acesso em: 19 nov. 2024.
APA
Cunha, T. M., Souza, G. R., Domingues, A. C., Carreira, E. U., Lotufo, C. M., Funez, M. I., et al. (2012). Stimulation of peripheral kappa opioid receptors inhibits inflammatory hyperalgesia via activation of the PI3K'gama'/AKT/nNOS/NO signaling pathway. Molecular Pain. doi:10.1186/1744-8069-8-10
NLM
Cunha TM, Souza GR, Domingues AC, Carreira EU, Lotufo CM, Funez MI, Verri Júnior WA, Cunha F de Q, Ferreira SH. Stimulation of peripheral kappa opioid receptors inhibits inflammatory hyperalgesia via activation of the PI3K'gama'/AKT/nNOS/NO signaling pathway [Internet]. Molecular Pain. 2012 ;[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/1744-8069-8-10
Vancouver
Cunha TM, Souza GR, Domingues AC, Carreira EU, Lotufo CM, Funez MI, Verri Júnior WA, Cunha F de Q, Ferreira SH. Stimulation of peripheral kappa opioid receptors inhibits inflammatory hyperalgesia via activation of the PI3K'gama'/AKT/nNOS/NO signaling pathway [Internet]. Molecular Pain. 2012 ;[citado 2024 nov. 19 ] Available from: https://doi.org/10.1186/1744-8069-8-10

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