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Artigo de periodico
Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis (2023)
Souza, Flávio Falcão Lima de; Schneider, Ayda Henriques ; Machado, Caio Cavalcante ; Almeida, Sérgio Couto Luna de; Silva, Tarcília Aparecida da; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Clinical and Experimental Rheumatology. Unidade: FMRP
Subjects: BIOMARCADORES, ARTRITE REUMATOIDE, OSTEOARTRITE, ESTUDOS TRANSVERSAIS
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ABNT
SOUZA, Flávio Falcão Lima de et al. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, v. 41, n. 7, p. 1473-1479, 2023Tradução . . Disponível em: https://doi.org/10.55563/clinexprheumatol/flzpgb. Acesso em: 19 nov. 2024.
APA
Souza, F. F. L. de, Schneider, A. H., Machado, C. C., Almeida, S. C. L. de, Silva, T. A. da, Cunha, F. de Q., et al. (2023). Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, 41( 7), 1473-1479. doi:10.55563/clinexprheumatol/flzpgb
NLM
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 nov. 19 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Vancouver
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 nov. 19 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Artigo de periodico
Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 (2023)
Formiga, Rodrigo de Oliveira; Amaral, Flávia C.; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza ; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Cunha, Fernando de Queiroz
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: COVID-19, LIPOPOLISSACARÍDEOS, METALOPROTEINASES, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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ABNT
FORMIGA, Rodrigo de Oliveira et al. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, v. 180, n. 11, p. 1460-1481, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16013. Acesso em: 19 nov. 2024.
APA
Formiga, R. de O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2023). Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, 180( 11), 1460-1481. doi:10.1111/bph.16013
NLM
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/bph.16013
Vancouver
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1111/bph.16013
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 19 nov. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis (2023)
Barreto, Ester A.; Cruz, Amanda S.; Veras, Flávio Protásio ; Martins, Ronaldo Bragança ; Bernardelli, Rafaella S.; Paiva, Isadora Marques ; Lima, Thais Melquiades de; Singh, Youvika ; Guimaraes, Raphael C.; Damasceno, Samara; Pereira, Nayara; Alves, João Manoel; Gonçalves, Tiago T.; Forato, Julia; Muraro, Stéfanie P.; Souza, Gabriela F.; Batah, Sabrina Setembre ; Proença-Modena, José L.; Mori, Marcelo A.; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Cunha, Thiago Mattar ; Nakaya, Helder Takashi Imoto ; Fabro, Alexandre Todorovic ; Oliveira, Renê Donizeti Ribeiro de; Arruda, Eurico ; Rea, Rosângela; Réa Neto, Álvaro; Silva, Miguel M Fernandes da; Leiria, Luiz Osório Silveira
Source: Proceedings of the National Academy of Sciences. Unidades: FMRP, FCF
Subjects: GLICOSE, COVID-19, GLICOGENOSE, HIPERGLICEMIA, ESTUDOS RETROSPECTIVOS
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ABNT
BARRETO, Ester A. et al. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, v. 120, n. 21, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2217119120. Acesso em: 19 nov. 2024.
APA
Barreto, E. A., Cruz, A. S., Veras, F. P., Martins, R. B., Bernardelli, R. S., Paiva, I. M., et al. (2023). COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, 120( 21), 1-10. doi:10.1073/pnas.2217119120
NLM
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1073/pnas.2217119120
Vancouver
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1073/pnas.2217119120
Artigo de periodico
Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production (2023)
Córneo, Emily S.; Veras, Flávio Protásio ; Gomes, Giovanni F.; Schneider, Ayda Henriques ; Silva, Bruna Manuella Souza; Almeida, Cícero José Luíz dos Ramos; Silva, Camila M.; Martins, Ronaldo B.; Batah, Sabrina Setembre ; Simon, Carla S.; Prestes, Gabriele da S.; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Fabro, Alexandre Todorovic ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Dal-Pizzol, Felipe
Source: Clinical Immunology. Unidades: FMRP, ESALQ
Subjects: COVID-19, NEUTRÓFILOS, CORONAVIRUS
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ABNT
CÓRNEO, Emily S. et al. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, v. 257, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2023.109836. Acesso em: 19 nov. 2024.
APA
Córneo, E. S., Veras, F. P., Gomes, G. F., Schneider, A. H., Silva, B. M. S., Almeida, C. J. L. dos R., et al. (2023). Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, 257, 1-12. doi:10.1016/j.clim.2023.109836
NLM
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Vancouver
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 19 nov. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex (2022)
Katayama, Pedro L.; Leirão, Isabela P.; Kanashiro, Alexandre; Luiz, João Paulo Mesquita ; Cunha, Fernando de Queiroz ; Navegantes, Luiz Carlos Carvalho ; Menani, Jose Vanderlei; Zoccal, Daniel Breseghello; Colombari, Debora Simoes de Almeida ; Colombari, Eduardo
Source: Brain, Behavior, and Immunity. Unidade: FMRP
Subjects: SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, ANTI-INFLAMATÓRIOS
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ABNT
KATAYAMA, Pedro L. et al. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, v. 102, p. 370-386, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2022.03.014. Acesso em: 19 nov. 2024.
APA
Katayama, P. L., Leirão, I. P., Kanashiro, A., Luiz, J. P. M., Cunha, F. de Q., Navegantes, L. C. C., et al. (2022). The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, 102, 370-386. doi:10.1016/j.bbi.2022.03.014
NLM
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Vancouver
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Artigo de periodico
Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation (2022)
Borghi, Sergio M.; Bussulo, Sylvia K. D.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Carvalho, Thacyana T.; Rasquel-Oliveira, Fernanda S.; Zaninelli, Tiago H.; Ferraz, Camila R.; Casella, Antônio M. B.; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar ; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: EXERCÍCIOS DE RESISTÊNCIA MUSCULAR, MEDULA ESPINHAL, INFLAMAÇÃO, DOR
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ABNT
BORGHI, Sergio M. et al. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, v. 12, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphar.2021.734091. Acesso em: 19 nov. 2024.
APA
Borghi, S. M., Bussulo, S. K. D., Pinho-Ribeiro, F. A., Fattori, V., Carvalho, T. T., Rasquel-Oliveira, F. S., et al. (2022). Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, 12, 1-17. doi:10.3389/fphar.2021.734091
NLM
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Vancouver
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Artigo de periodico
Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice (2022)
Castro, Paula A. T. S.; Machanocker, Dafiner H.; Luna, Genoveva F.; Barbosa, Germanna M.; Cunha, Jonathan E.; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Russo, Thiago L.; Salvini, Tania F.
Source: BioMed Research International. Unidade: FMRP
Subjects: CRIOTERAPIA, INFLAMAÇÃO, ARTRITE, JOELHO
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ABNT
CASTRO, Paula A. T. S. et al. Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice. BioMed Research International, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1155/2022/7442289. Acesso em: 19 nov. 2024.
APA
Castro, P. A. T. S., Machanocker, D. H., Luna, G. F., Barbosa, G. M., Cunha, J. E., Cunha, T. M., et al. (2022). Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice. BioMed Research International, 1-9. doi:10.1155/2022/7442289
NLM
Castro PATS, Machanocker DH, Luna GF, Barbosa GM, Cunha JE, Cunha TM, Cunha F de Q, Russo TL, Salvini TF. Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice [Internet]. BioMed Research International. 2022 ; 1-9.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2022/7442289
Vancouver
Castro PATS, Machanocker DH, Luna GF, Barbosa GM, Cunha JE, Cunha TM, Cunha F de Q, Russo TL, Salvini TF. Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice [Internet]. BioMed Research International. 2022 ; 1-9.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2022/7442289
Artigo de periodico
Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice (2022)
Maganin, Alexandre Gomes de Macedo ; Souza, Guilherme Rabelo de; Fonseca, Miriam D.; Lopes, Alexandre H.; Guimarães, Rafaela Mano ; Dagostin, André; Cecílio, Nerry T.; Mendes, Atlante Silva ; Gonçalves, William Antônio; Silva, Conceição Elidianne Aníbal; Gomes, Francisco Isaac Fernandes ; Marques, Lucas Maciel Mauriz ; Silva, Rangel L.; Arruda, Leticia M.; Santana, Denis A.; Lemos, Henrique; Huang, Lei; Ferreira, Marcela Davoli; Santana-Coelho, Danielle; Sant’Anna, Morena Brasil Martins; Kusuda, Ricardo; Talbot, Jhimmy; Pacholczyk, Gabriela; Buqui, Gabriela A.; Lopes, Norberto Peporine ; Alves Filho, José Carlos Farias ; Leão, Ricardo Maurício Xavier; O’Connor, Jason C.; Cunha, Fernando de Queiroz ; Mellor, Andrew; Cunha, Thiago Mattar
Source: Journal of Clinical Investigation. Unidades: FCFRP, FMRP, FCF
Subjects: CÉLULAS DENDRÍTICAS, METABÓLITOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, NERVOS PERIFÉRICOS, DOR CRÔNICA
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ABNT
MAGANIN, Alexandre Gomes de Macedo et al. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, v. 132, n. 23, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1172/JCI153805. Acesso em: 19 nov. 2024.
APA
Maganin, A. G. de M., Souza, G. R. de, Fonseca, M. D., Lopes, A. H., Guimarães, R. M., Dagostin, A., et al. (2022). Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, 132( 23), 1-17. doi:10.1172/JCI153805
NLM
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1172/JCI153805
Vancouver
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1172/JCI153805
Artigo de periodico
Targeting PARP1 to enhance anticancer checkpoint immunotherapy response: rationale and clinical implications (2022)
Wanderley, Carlos Wagner de Souza ; Correa, Tatiana Strava; Scaranti, Mariana; Cunha, Fernando de Queiroz ; Barroso-Sousa, Romualdo
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: REPARAÇÃO DE DNA, IMUNOTERAPIA, NEOPLASIAS, FARMACOTERAPIA
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ABNT
WANDERLEY, Carlos Wagner de Souza et al. Targeting PARP1 to enhance anticancer checkpoint immunotherapy response: rationale and clinical implications. Frontiers in Immunology, v. 13, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.816642. Acesso em: 19 nov. 2024.
APA
Wanderley, C. W. de S., Correa, T. S., Scaranti, M., Cunha, F. de Q., & Barroso-Sousa, R. (2022). Targeting PARP1 to enhance anticancer checkpoint immunotherapy response: rationale and clinical implications. Frontiers in Immunology, 13, 1-10. doi:10.3389/fimmu.2022.816642
NLM
Wanderley CW de S, Correa TS, Scaranti M, Cunha F de Q, Barroso-Sousa R. Targeting PARP1 to enhance anticancer checkpoint immunotherapy response: rationale and clinical implications [Internet]. Frontiers in Immunology. 2022 ; 13 1-10.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fimmu.2022.816642
Vancouver
Wanderley CW de S, Correa TS, Scaranti M, Cunha F de Q, Barroso-Sousa R. Targeting PARP1 to enhance anticancer checkpoint immunotherapy response: rationale and clinical implications [Internet]. Frontiers in Immunology. 2022 ; 13 1-10.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3389/fimmu.2022.816642
Artigo de periodico
Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels (2022)
Freitas, Daniela Fernanda de; morelo, David Fernando Colon ; Silva, Rangel Leal; Santos, Eloá Mangabeira; Guimarães, Victor Hugo Dantas; Ribeiro, Guilherme Henrique Mendes; Paula, Alfredo Maurício Batista de; Guimarães, André Luiz Sena; Reis, Sidnei Tavares dos; Cunha, Fernando de Queiroz ; Antunes, Maísa Mota; Menezes, Gustavo Batista ; Santos, Sérgio Henrique Souza
Source: Molecular Biology Reports. Unidade: FMRP
Subjects: TECIDO ADIPOSO, INFLAMAÇÃO, NEUTRÓFILOS, OBESIDADE
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ABNT
FREITAS, Daniela Fernanda de et al. Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels. Molecular Biology Reports, v. 49, n. 4, p. 3225-3236, 2022Tradução . . Disponível em: https://doi.org/10.1007/s11033-022-07157-y. Acesso em: 19 nov. 2024.
APA
Freitas, D. F. de, morelo, D. F. C., Silva, R. L., Santos, E. M., Guimarães, V. H. D., Ribeiro, G. H. M., et al. (2022). Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels. Molecular Biology Reports, 49( 4), 3225-3236. doi:10.1007/s11033-022-07157-y
NLM
Freitas DF de, morelo DFC, Silva RL, Santos EM, Guimarães VHD, Ribeiro GHM, Paula AMB de, Guimarães ALS, Reis ST dos, Cunha F de Q, Antunes MM, Menezes GB, Santos SHS. Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels [Internet]. Molecular Biology Reports. 2022 ; 49( 4): 3225-3236.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s11033-022-07157-y
Vancouver
Freitas DF de, morelo DFC, Silva RL, Santos EM, Guimarães VHD, Ribeiro GHM, Paula AMB de, Guimarães ALS, Reis ST dos, Cunha F de Q, Antunes MM, Menezes GB, Santos SHS. Neutrophil extracellular traps (NETs) modulate inflammatory profile in obese humans and mice: adipose tissue role on NETs levels [Internet]. Molecular Biology Reports. 2022 ; 49( 4): 3225-3236.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1007/s11033-022-07157-y
Artigo de periodico
Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition (2022)
Franchin, Marcelo ; Rosalen, Pedro Luiz ; Prado, Douglas da Silva ; Paraluppi, Murilo César; Silva, Rangel Leal ; Damasceno, Luis Eduardo Alves ; morelo, David Fernando Colon ; Napimoga, Marcelo Henrique; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar
Source: European Journal of Pharmacology. Unidades: CENA, FMRP
Subjects: DIFERENCIAÇÃO CELULAR, ENCEFALOMIELITE AGUDA DISSEMINADA, DOENÇAS AUTOIMUNES, INFLAMAÇÃO, FARMACOTERAPIA, INTERLEUCINAS, PRÓPOLIS
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ABNT
FRANCHIN, Marcelo et al. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition. European Journal of Pharmacology, v. 929, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2022.175127. Acesso em: 19 nov. 2024.
APA
Franchin, M., Rosalen, P. L., Prado, D. da S., Paraluppi, M. C., Silva, R. L., Damasceno, L. E. A., et al. (2022). Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition. European Journal of Pharmacology, 929, 1-8. doi:10.1016/j.ejphar.2022.175127
NLM
Franchin M, Rosalen PL, Prado D da S, Paraluppi MC, Silva RL, Damasceno LEA, morelo DFC, Napimoga MH, Cunha F de Q, Alves Filho JCF, Cunha TM. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition [Internet]. European Journal of Pharmacology. 2022 ; 929 1-8.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2022.175127
Vancouver
Franchin M, Rosalen PL, Prado D da S, Paraluppi MC, Silva RL, Damasceno LEA, morelo DFC, Napimoga MH, Cunha F de Q, Alves Filho JCF, Cunha TM. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition [Internet]. European Journal of Pharmacology. 2022 ; 929 1-8.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2022.175127
Artigo de periodico
Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis (2022)
Vale, Aline Halfeld Fernandes ; Nascimento, Daniele Carvalho ; Alvarez, Annie Rocío Piñeros ; Ferreira, Raphael G. ; Santos, Jeimison Duarte ; Aragon, Davi Casale ; Cunha, Fernando de Queiroz ; Ramalho, Fernando Silva ; Alves Filho, José Carlos Farias ; Carlotti, Ana Paula de Carvalho Panzeri
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS, ANTIOXIDANTES, INFERÊNCIA BAYESIANA, QUIMIOCINAS, CREATINA, FARMACOTERAPIA
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ABNT
VALE, Aline Halfeld Fernandes et al. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis. Brazilian Journal of Medical and Biological Research, v. 55, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1590/1414-431X2022e12107. Acesso em: 19 nov. 2024.
APA
Vale, A. H. F., Nascimento, D. C., Alvarez, A. R. P., Ferreira, R. G., Santos, J. D., Aragon, D. C., et al. (2022). Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis. Brazilian Journal of Medical and Biological Research, 55, 1-8. doi:10.1590/1414-431X2022e12107
NLM
Vale AHF, Nascimento DC, Alvarez ARP, Ferreira RG, Santos JD, Aragon DC, Cunha F de Q, Ramalho FS, Alves Filho JCF, Carlotti AP de CP. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis [Internet]. Brazilian Journal of Medical and Biological Research. 2022 ; 55 1-8.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1590/1414-431X2022e12107
Vancouver
Vale AHF, Nascimento DC, Alvarez ARP, Ferreira RG, Santos JD, Aragon DC, Cunha F de Q, Ramalho FS, Alves Filho JCF, Carlotti AP de CP. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis [Internet]. Brazilian Journal of Medical and Biological Research. 2022 ; 55 1-8.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1590/1414-431X2022e12107
Artigo de periodico
Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients (2021)
Rodrigues, Tamara Silva ; Sá, Keyla Santos Guedes de ; Ishimoto, Adriene Yumi; Becerra, Amanda; Oliveira, Samuel; Almeida, Leticia; Gonçalves, Augusto V.; Perucello, Debora B.; Andrade, Warrison A. ; Castro, Ricardo; Veras, Flavio P.; Toller-Kawahisa, Juliana E.; Nascimento, Daniele C.; Lima, Mikhael H. F. de; Silva, Camila M. S.; Caetite, Diego B.; Martins, Ronaldo Bragança ; Castro, Italo A.; Pontelli, Marjorie C.; Barros, Fabio C. de; Amaral, Natália B. do; Giannini, Marcela C.; Bonjorno, Letícia P.; Lopes, Maria Isabel F.; Santana, Rodrigo de Carvalho ; Vilar, Fernando C.; Auxiliadora-Martins, Maria ; Luppino-Assad, Rodrigo; Almeida, Sergio C. L. de; Oliveira, Fabiola R. de; Batah, Sabrina Setembre ; Siyuan, Li; Benatti, Maira N.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz ; Cunha, Larissa D.; Frantz, Fabiani Gai ; Kohlsdorf, Tiana ; Fabro, Alexandre Todorovic ; Arruda, Eurico ; Oliveira, Renê D. R. de; Louzada Júnior, Paulo ; Zamboni, Dario Simões
Source: Journal of Experimental Medicine. Unidades: FMRP, FCFRP, FFCLRP
Subjects: COVID-19, INFLAMAÇÃO
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ABNT
RODRIGUES, Tamara Silva et al. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, v. 218, n. 3, 2021Tradução . . Disponível em: https://doi.org/10.1084/jem.20201707. Acesso em: 19 nov. 2024.
APA
Rodrigues, T. S., Sá, K. S. G. de, Ishimoto, A. Y., Becerra, A., Oliveira, S., Almeida, L., et al. (2021). Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, 218( 3). doi:10.1084/jem.20201707
NLM
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2024 nov. 19 ] Available from: https://doi.org/10.1084/jem.20201707
Vancouver
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2024 nov. 19 ] Available from: https://doi.org/10.1084/jem.20201707
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 19 nov. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β (2018)
Ruiz-Miyazawa, Kenji W.; Borghi, Sergio M.; Ribeiro, Felipe A. Pinho; Ferrari, Larissa Staurengo; Fattori, Victor; Fernandes, Glaura S. A.; Casella, Antonio M.; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Casagrande, Rubia; Verri, Waldiceu A.
Source: Journal of Functional Foods. Unidade: FMRP
Subjects: ARTRITE, DOR, INFLAMAÇÃO, RATOS, MACRÓFAGOS
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ABNT
RUIZ-MIYAZAWA, Kenji W. et al. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, v. 48, p. 106-116, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jff.2018.06.025. Acesso em: 19 nov. 2024.
APA
Ruiz-Miyazawa, K. W., Borghi, S. M., Ribeiro, F. A. P., Ferrari, L. S., Fattori, V., Fernandes, G. S. A., et al. (2018). The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, 48, 106-116. doi:10.1016/j.jff.2018.06.025
NLM
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Vancouver
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Artigo de periodico
Effect of gedunin on acute articular inflammation and hypernociception in mice (2015)
Conte, Fernando P; Ferraris, Fausto K; Costa, Thadeu E. M. M; Pacheco, Patricia; Seito, Leonardo N; Verri Jr, Waldiceu A; Cunha, Fernando de Queiroz; Penido, Carmen; Henriques, Maria G
Source: Molecules. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS NÃO ESTEROIDES (FARMACOLOGIA), CARTILAGEM ARTICULAR, ENDOTELINAS (METABOLISMO), ARTICULAÇÃO DO JOELHO, OSTEOCONDRITE ANIMAL
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ABNT
CONTE, Fernando P et al. Effect of gedunin on acute articular inflammation and hypernociception in mice. Molecules, v. 20, n. 2, p. 2636-2657, 2015Tradução . . Disponível em: https://doi.org/10.3390/molecules20022636. Acesso em: 19 nov. 2024.
APA
Conte, F. P., Ferraris, F. K., Costa, T. E. M. M., Pacheco, P., Seito, L. N., Verri Jr, W. A., et al. (2015). Effect of gedunin on acute articular inflammation and hypernociception in mice. Molecules, 20( 2), 2636-2657. doi:10.3390/molecules20022636
NLM
Conte FP, Ferraris FK, Costa TEMM, Pacheco P, Seito LN, Verri Jr WA, Cunha F de Q, Penido C, Henriques MG. Effect of gedunin on acute articular inflammation and hypernociception in mice [Internet]. Molecules. 2015 ; 20( 2): 2636-2657.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3390/molecules20022636
Vancouver
Conte FP, Ferraris FK, Costa TEMM, Pacheco P, Seito LN, Verri Jr WA, Cunha F de Q, Penido C, Henriques MG. Effect of gedunin on acute articular inflammation and hypernociception in mice [Internet]. Molecules. 2015 ; 20( 2): 2636-2657.[citado 2024 nov. 19 ] Available from: https://doi.org/10.3390/molecules20022636
Artigo de periodico
Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection (2014)
Gutierrez, Fredy R. S; Pavanelli, Wander R; Medina, Tiago S; Silva, Grace K; Mariano, Flávia S; Guedes, Paulo M. M.; Mineo, Tiago W. P.; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: Microbes and Infection. Unidade: FMRP
Subjects: TRYPANOSOMA CRUZI, OXIGENASE, LINFÓCITOS T
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ABNT
GUTIERREZ, Fredy R. S et al. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 1, p. 28-39, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2013.10.007. Acesso em: 19 nov. 2024.
APA
Gutierrez, F. R. S., Pavanelli, W. R., Medina, T. S., Silva, G. K., Mariano, F. S., Guedes, P. M. M., et al. (2014). Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, 16( 1), 28-39. doi:10.1016/j.micinf.2013.10.007
NLM
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Vancouver
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Artigo de periodico
Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents (2013)
Luca, Bianca de; Xavier-Elsas, Pedro; Barradas, Mônica; Luz, Ricardo A.; Queto, Túlio; Jones, Carla; Arruda, Maria Augusta; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Gaspar-Elsas, Maria Ignez
Source: The Scientific World Journal. Unidade: FMRP
Subjects: FARMACOLOGIA, IMUNOLOGIA, ÓXIDO NÍTRICO
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ABNT
LUCA, Bianca de et al. Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents. The Scientific World Journal, v. 2013, 2013Tradução . . Disponível em: https://doi.org/10.1155/2013/208705. Acesso em: 19 nov. 2024.
APA
Luca, B. de, Xavier-Elsas, P., Barradas, M., Luz, R. A., Queto, T., Jones, C., et al. (2013). Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents. The Scientific World Journal, 2013. doi:10.1155/2013/208705
NLM
Luca B de, Xavier-Elsas P, Barradas M, Luz RA, Queto T, Jones C, Arruda MA, Cunha TM, Cunha F de Q, Gaspar-Elsas MI. Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents [Internet]. The Scientific World Journal. 2013 ; 2013[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2013/208705
Vancouver
Luca B de, Xavier-Elsas P, Barradas M, Luz RA, Queto T, Jones C, Arruda MA, Cunha TM, Cunha F de Q, Gaspar-Elsas MI. Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents [Internet]. The Scientific World Journal. 2013 ; 2013[citado 2024 nov. 19 ] Available from: https://doi.org/10.1155/2013/208705

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