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Artigo de periodico
Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection (2024)
Mestriner, Fabíola Leslie Antunes Cardoso ; Francisco, Daniely Franco ; Campos, Lígia Cristina Borges ; Couto, Ariel Emiliano Souza do ; Fraga-Silva, Thais Fernanda de Campos ; Dugaich, Vinicius Flora ; D`Avila-Mesquita, Carolina ; Zukowski Kovacs, Henrique ; Vasconcelos, Jociany Lopes de ; Milani, Elizabete Rosa ; Sá, Keyla Santos Guedes de ; Martins, Ronaldo Bragança ; Jordani, Maria Cecília ; Corsi, Carlos Alexandre Curylofo ; Barbosa, Jéssyca Michelon; Vasconcelos, Tauana Fernandes ; Menegueti, Mayra Gonçalves ; Neto, Julio; Costa, Rafael Menezes da ; Evora, Paulo Roberto Barbosa ; Arruda, Eurico ; Tostes, Rita de Cássia Aleixo ; Polonis, Katarzyna ; Bonato, Vânia Luiza Deperon ; Auxiliadora-Martins, Maria ; Ribeiro, Maurício Serra ; Becari, Christiane
Source: Cytokine. Unidades: FMRP, FCFRP, EERP, FOB
Subjects: COVID-19, GLICOPROTEÍNAS, INFECÇÕES POR CORONAVIRUS
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ABNT
MESTRINER, Fabíola Leslie Antunes Cardoso et al. Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection. Cytokine, v. 176, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2024.156503. Acesso em: 04 jul. 2024.
APA
Mestriner, F. L. A. C., Francisco, D. F., Campos, L. C. B., Couto, A. E. S. do, Fraga-Silva, T. F. de C., Dugaich, V. F., et al. (2024). Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection. Cytokine, 176. doi:10.1016/j.cyto.2024.156503
NLM
Mestriner FLAC, Francisco DF, Campos LCB, Couto AES do, Fraga-Silva TF de C, Dugaich VF, D`Avila-Mesquita C, Zukowski Kovacs H, Vasconcelos JL de, Milani ER, Sá KSG de, Martins RB, Jordani MC, Corsi CAC, Barbosa JM, Vasconcelos TF, Menegueti MG, Neto J, Costa RM da, Evora PRB, Arruda E, Tostes R de CA, Polonis K, Bonato VLD, Auxiliadora-Martins M, Ribeiro MS, Becari C. Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection [Internet]. Cytokine. 2024 ; 176[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.cyto.2024.156503
Vancouver
Mestriner FLAC, Francisco DF, Campos LCB, Couto AES do, Fraga-Silva TF de C, Dugaich VF, D`Avila-Mesquita C, Zukowski Kovacs H, Vasconcelos JL de, Milani ER, Sá KSG de, Martins RB, Jordani MC, Corsi CAC, Barbosa JM, Vasconcelos TF, Menegueti MG, Neto J, Costa RM da, Evora PRB, Arruda E, Tostes R de CA, Polonis K, Bonato VLD, Auxiliadora-Martins M, Ribeiro MS, Becari C. Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection [Internet]. Cytokine. 2024 ; 176[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.cyto.2024.156503
Artigo de periodico
Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation (2024)
Costa, Rafael Menezes da ; Dias, Mayara C.; Alves, Juliano Vilela ; Silva, João Lucas M.; Rodrigues, Daniel; Silva, Josiane Fernandes da ; Francescato, Heloisa Della Coletta; Ramalho, Leandra Naira Zambelli ; Coimbra, Terezila Machado ; Passaglia, Rita de Cassia Aleixo Tostes
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: RIM, HIPERGLICEMIA, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
COSTA, Rafael Menezes da et al. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, v. 220, p. 1-10, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2023.115982. Acesso em: 04 jul. 2024.
APA
Costa, R. M. da, Dias, M. C., Alves, J. V., Silva, J. L. M., Rodrigues, D., Silva, J. F. da, et al. (2024). Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, 220, 1-10. doi:10.1016/j.bcp.2023.115982
NLM
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Vancouver
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Artigo de periodico
Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 (2023)
Simplicio, Janaina Aparecida ; Dourado, Thales de Mileto Henrique; Awata, Wanessa Mayumi Carvalho; Vale, Gabriel Tavares do ; Dias, Vinícius Ribeiro; Barros, Paula Rodrigues de ; Martinis, Bruno Spinosa de ; Tostes, Rita de Cássia Aleixo ; Tirapelli, Carlos Renato
Source: Life Sciences. Unidades: FFCLRP, FMRP, EERP
Subjects: ETANOL, INTERLEUCINA 6, TECIDO ADIPOSO, ESTRESSE OXIDATIVO, HIPERTENSÃO
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ABNT
SIMPLICIO, Janaina Aparecida et al. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, v. 319, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121526. Acesso em: 04 jul. 2024.
APA
Simplicio, J. A., Dourado, T. de M. H., Awata, W. M. C., Vale, G. T. do, Dias, V. R., Barros, P. R. de, et al. (2023). Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, 319, 1-10. doi:10.1016/j.lfs.2023.121526
NLM
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Vancouver
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Artigo de periodico
Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 (2023)
Fraga-Silva, Thais Fernanda de Campos ; Rosa, Ualter Guilherme Cipriano; Fumagalli, Marcílio Jorge ; Corrêa, Giseli Furlan ; Fuzo, Carlos Alessandro ; Santos, Douglas dos; Mestriner, Fabíola Leslie Antunes Cardoso ; Ribeiro, Christiane Becari ; Teixeira-Carvalho, Andrea; Coelho-dos-Reis, Jordana; Menegueti, Mayra Gonçalves ; Figueiredo, Luiz Tadeu Moraes ; Cunha, Larissa Dias da; Martins-Filho, Olindo A.; Baruffi, Marcelo Dias ; Martins, Maria Auxiliadora ; Passaglia, Rita de Cassia Aleixo Tostes ; Bonato, Vânia Luiza Deperon
Source: iScience. Unidades: FMRP, EERP, FCFRP
Subjects: SISTEMA IMUNE, VIROLOGIA, MACRÓFAGOS
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ABNT
FRAGA-SILVA, Thais Fernanda de Campos et al. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, v. 26, n. 12, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108366. Acesso em: 04 jul. 2024.
APA
Fraga-Silva, T. F. de C., Rosa, U. G. C., Fumagalli, M. J., Corrêa, G. F., Fuzo, C. A., Santos, D. dos, et al. (2023). Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, 26( 12), 1-19. doi:10.1016/j.isci.2023.108366
NLM
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Vancouver
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2023)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Source: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Subjects: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
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ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 04 jul. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2023). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:https://doi.org/10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor (2023)
Rodrigues, Daniel ; Machado, Mirele Resende ; Alves, Juliano Vilela ; Fraga-Silva, Thais Fernanda de Campos ; Martins, Ronaldo Bragança ; Campos, Lígia Cristina Borges ; Francisco, Daniely Franco ; Couto, Ariel Emiliano Souza do ; Bonato, Vânia Luiza Deperon ; Arruda, Eurico ; Becari, Christiane ; Auxiliadora-Martins, Maria ; Louzada Júnior, Paulo ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FCFRP, FMRP, FOB
Subjects: CÉLULAS ENDOTELIAIS, CITOCINAS, COVID-19, ANORMALIDADES CARDIOVASCULARES
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ABNT
RODRIGUES, Daniel et al. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor. American Journal of Physiology. Heart and Circulatory Physiology, v. 325, n. 2, p. H252–H263 Aug. 2023, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00096.2023. Acesso em: 04 jul. 2024.
APA
Rodrigues, D., Machado, M. R., Alves, J. V., Fraga-Silva, T. F. de C., Martins, R. B., Campos, L. C. B., et al. (2023). Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor. American Journal of Physiology. Heart and Circulatory Physiology, 325( 2), H252–H263 Aug. 2023. doi:10.1152/ajpheart.00096.2023
NLM
Rodrigues D, Machado MR, Alves JV, Fraga-Silva TF de C, Martins RB, Campos LCB, Francisco DF, Couto AES do, Bonato VLD, Arruda E, Becari C, Auxiliadora-Martins M, Louzada Júnior P, Costa RM da, Tostes R de CA. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 325( 2): H252–H263 Aug. 2023.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1152/ajpheart.00096.2023
Vancouver
Rodrigues D, Machado MR, Alves JV, Fraga-Silva TF de C, Martins RB, Campos LCB, Francisco DF, Couto AES do, Bonato VLD, Arruda E, Becari C, Auxiliadora-Martins M, Louzada Júnior P, Costa RM da, Tostes R de CA. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 325( 2): H252–H263 Aug. 2023.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1152/ajpheart.00096.2023
Artigo de periodico
NLRP3 activation contributes to endothelin-1-induced erectile dysfunction (2023)
Fais, Rafael Sobrano; Costa, Rafael Menezes da ; Mendes, Allan Carvalho; Mestriner, Fabiola Leslie Antunes Cardoso; Comerma-Steffensen, Simón Gabriel; Tostes, Rita de Cássia Aleixo ; Simonsen, Ulf; Carneiro, Fernando Silva
Source: Journal of Cellular and Molecular Medicine. Unidade: FMRP
Subjects: ANTAGONISTAS, ENDOTELINAS, DISFUNÇÃO ERÉTIL, ESPÉCIES REATIVAS DE OXIGÊNIO, INFLAMAÇÃO
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ABNT
FAIS, Rafael Sobrano et al. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, v. 27, n. 1, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1111/jcmm.17463. Acesso em: 04 jul. 2024.
APA
Fais, R. S., Costa, R. M. da, Mendes, A. C., Mestriner, F. L. A. C., Comerma-Steffensen, S. G., Tostes, R. de C. A., et al. (2023). NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, 27( 1), 1-14. doi:10.1111/jcmm.17463
NLM
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1111/jcmm.17463
Vancouver
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1111/jcmm.17463
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
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ABNT
COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 04 jul. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Subjects: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
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ABNT
SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 04 jul. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system (2022)
Passos Junior, Rinaldo Rodrigues dos; Bomfim, Gisele Facholi ; Giachini, Fernanda R.; Tostes, Rita de Cássia Aleixo ; Lima, Victor Vitorino
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: HIPERTENSÃO, SISTEMA IMUNE, PROTEÍNAS
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ABNT
PASSOS JUNIOR, Rinaldo Rodrigues dos et al. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system. Frontiers in Immunology, v. 13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.852115. Acesso em: 04 jul. 2024.
APA
Passos Junior, R. R. dos, Bomfim, G. F., Giachini, F. R., Tostes, R. de C. A., & Lima, V. V. (2022). O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system. Frontiers in Immunology, 13. doi:10.3389/fimmu.2022.852115
NLM
Passos Junior RR dos, Bomfim GF, Giachini FR, Tostes R de CA, Lima VV. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system [Internet]. Frontiers in Immunology. 2022 ; 13[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fimmu.2022.852115
Vancouver
Passos Junior RR dos, Bomfim GF, Giachini FR, Tostes R de CA, Lima VV. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system [Internet]. Frontiers in Immunology. 2022 ; 13[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fimmu.2022.852115
Artigo de periodico
Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction (2022)
Silva, Josiane Fernandes da; Bolsoni, Juliana A.; Costa, Rafael Menezes da ; Alves, Juliano Vilela ; Bressan, Alecsander Fabrício Moreira ; Silva, Luiz Eduardo Virgilio da ; Costa, Tiago Januário da ; Oliveira, Antonio E. R.; Manzato, Carla Pavan; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Nakaya, Helder Takashi Imoto ; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo
Source: British Journal of Pharmacology. Unidades: FMRP, FCF
Subjects: GORDURAS NA DIETA, CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, OBESIDADE, HIDROCARBONETOS, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Josiane Fernandes da et al. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, v. 179, n. 12, p. 2938-2952, 2022Tradução . . Disponível em: https://doi.org/10.1111/bph.15789. Acesso em: 04 jul. 2024.
APA
Silva, J. F. da, Bolsoni, J. A., Costa, R. M. da, Alves, J. V., Bressan, A. F. M., Silva, L. E. V. da, et al. (2022). Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, 179( 12), 2938-2952. doi:10.1111/bph.15789
NLM
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1111/bph.15789
Vancouver
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1111/bph.15789
Artigo de periodico
Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats (2022)
Brazão, Vânia; Colato, Rafaela Pravato ; Santello, Fabricia Helena; Duarte, Andressa ; Goulart, Amanda; Oliveira, Pedro Alexandre Sampaio ; Silva, Carla Brigagão Pacheco da ; Tirapelli, Carlos Renato ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo ; Prado Júnior, José Clóvis do
Source: Experimental Gerontology. Unidades: EERP, FMRP, FCFRP
Subjects: ANTIOXIDANTES, DOENÇA DE CHAGAS, PERÓXIDO DE HIDROGÊNIO, MELATONINA, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRAZÃO, Vânia et al. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, v. 167, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2022.111895. Acesso em: 04 jul. 2024.
APA
Brazão, V., Colato, R. P., Santello, F. H., Duarte, A., Goulart, A., Oliveira, P. A. S., et al. (2022). Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, 167, 1-9. doi:10.1016/j.exger.2022.111895
NLM
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Vancouver
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Source: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Subjects: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 04 jul. 2024.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Artigo de periodico
Programming of vascular dysfunction by maternal stress: immune system implications (2022)
Costa, Tiago Januário da ; Oliveira, Julio Cezar de; Giachini, Fernanda Regina; Lima, Victor Vitorino; Tostes, Rita de Cássia Aleixo ; Bomfim, Gisele Facholi
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: SISTEMA IMUNE, ESPÉCIES REATIVAS DE OXIGÊNIO, ESTRESSE, SISTEMA RENINA-ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Programming of vascular dysfunction by maternal stress: immune system implications. Frontiers in Physiology, v. 13, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.787617. Acesso em: 04 jul. 2024.
APA
Costa, T. J. da, Oliveira, J. C. de, Giachini, F. R., Lima, V. V., Tostes, R. de C. A., & Bomfim, G. F. (2022). Programming of vascular dysfunction by maternal stress: immune system implications. Frontiers in Physiology, 13, 1-17. doi:10.3389/fphys.2022.787617
NLM
Costa TJ da, Oliveira JC de, Giachini FR, Lima VV, Tostes R de CA, Bomfim GF. Programming of vascular dysfunction by maternal stress: immune system implications [Internet]. Frontiers in Physiology. 2022 ; 13 1-17.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fphys.2022.787617
Vancouver
Costa TJ da, Oliveira JC de, Giachini FR, Lima VV, Tostes R de CA, Bomfim GF. Programming of vascular dysfunction by maternal stress: immune system implications [Internet]. Frontiers in Physiology. 2022 ; 13 1-17.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fphys.2022.787617
Artigo de periodico
The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters (2021)
Domingos-Souza, Gean; Santos-Almeida, Fernanda Machado; Meschiari, César Arruda; Ferreira, Nathanne dos Santos; Pereira, Camila André; Oliveira, Nayara Pestana de; Costa, Thaís Caroline Prates; Tostes, Rita de Cássia Aleixo ; White, Carl; Fazan Júnior, Rubens
Source: Hypertension Research. Unidade: FMRP
Subjects: PRESSORECEPTORES, HIPERTENSÃO, ESTIMULAÇÃO ELÉTRICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DOMINGOS-SOUZA, Gean et al. The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters. Hypertension Research, v. 44, n. 8, p. 932–940, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41440-021-00639-9. Acesso em: 04 jul. 2024.
APA
Domingos-Souza, G., Santos-Almeida, F. M., Meschiari, C. A., Ferreira, N. dos S., Pereira, C. A., Oliveira, N. P. de, et al. (2021). The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters. Hypertension Research, 44( 8), 932–940. doi:10.1038/s41440-021-00639-9
NLM
Domingos-Souza G, Santos-Almeida FM, Meschiari CA, Ferreira N dos S, Pereira CA, Oliveira NP de, Costa TCP, Tostes R de CA, White C, Fazan Júnior R. The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters [Internet]. Hypertension Research. 2021 ; 44( 8): 932–940.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1038/s41440-021-00639-9
Vancouver
Domingos-Souza G, Santos-Almeida FM, Meschiari CA, Ferreira N dos S, Pereira CA, Oliveira NP de, Costa TCP, Tostes R de CA, White C, Fazan Júnior R. The ability of baroreflex activation to improve blood pressure and resistance vessel function in spontaneously hypertensive rats is dependent on stimulation parameters [Internet]. Hypertension Research. 2021 ; 44( 8): 932–940.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1038/s41440-021-00639-9
Artigo de periodico
NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes (2021)
Costa, Frederico Ribeiro Campos ; Leite, Jefferson Antonio; Rassi, Diane Meyre ; Silva, Josiane Fernandes da ; Oliveira, Jefferson Elias ; Guimarães, Jhefferson Barbosa ; Freitas, Maria Cristina Foss de ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Carlos, Daniela
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, DIABETES MELLITUS, DOENÇAS AUTOIMUNES, PÂNCREAS, CAMUNDONGOS, POLIMORFISMO, SISTEMA IMUNE, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Frederico Ribeiro Campos et al. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, v. 35, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2021.109176. Acesso em: 04 jul. 2024.
APA
Costa, F. R. C., Leite, J. A., Rassi, D. M., Silva, J. F. da, Oliveira, J. E., Guimarães, J. B., et al. (2021). NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, 35, 1-16. doi:10.1016/j.celrep.2021.109176
NLM
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Vancouver
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Artigo de periodico
Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats (2021)
Troiano, Jéssica A.; Potje, Simone Regina ; Graton, Murilo E.; Gonçalves, Emily T.; Tostes, Rita de Cássia Aleixo ; Antoniali, Cristina
Source: Frontiers in Physiology. Unidades: FMRP, FCFRP
Subjects: AORTA TORÁCICA, ÓXIDO NÍTRICO, GRAVIDEZ, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TROIANO, Jéssica A. et al. Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats. Frontiers in Physiology, v. 12, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphys.2021.760237. Acesso em: 04 jul. 2024.
APA
Troiano, J. A., Potje, S. R., Graton, M. E., Gonçalves, E. T., Tostes, R. de C. A., & Antoniali, C. (2021). Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats. Frontiers in Physiology, 12, 1-13. doi:10.3389/fphys.2021.760237
NLM
Troiano JA, Potje SR, Graton ME, Gonçalves ET, Tostes R de CA, Antoniali C. Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats [Internet]. Frontiers in Physiology. 2021 ; 12 1-13.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fphys.2021.760237
Vancouver
Troiano JA, Potje SR, Graton ME, Gonçalves ET, Tostes R de CA, Antoniali C. Caveolin-1/endothelial nitric oxide synthase interaction is reduced in arteries from pregnant spontaneously hypertensive rats [Internet]. Frontiers in Physiology. 2021 ; 12 1-13.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3389/fphys.2021.760237
Artigo de periodico
Aldosterone, inflammation, immune system, and hypertension (2021)
Ferreira, Nathanne dos Santos; Tostes, Rita de Cássia Aleixo ; Paradis, Pierre; Schiffrin, Ernesto L.
Source: American Journal of Hypertension. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, SISTEMA IMUNE, LINFÓCITOS, MONÓCITOS, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Nathanne dos Santos et al. Aldosterone, inflammation, immune system, and hypertension. American Journal of Hypertension, v. 34, n. 1, p. 15-27, 2021Tradução . . Disponível em: https://doi.org/10.1093/ajh/hpaa137. Acesso em: 04 jul. 2024.
APA
Ferreira, N. dos S., Tostes, R. de C. A., Paradis, P., & Schiffrin, E. L. (2021). Aldosterone, inflammation, immune system, and hypertension. American Journal of Hypertension, 34( 1), 15-27. doi:10.1093/ajh/hpaa137
NLM
Ferreira N dos S, Tostes R de CA, Paradis P, Schiffrin EL. Aldosterone, inflammation, immune system, and hypertension [Internet]. American Journal of Hypertension. 2021 ; 34( 1): 15-27.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1093/ajh/hpaa137
Vancouver
Ferreira N dos S, Tostes R de CA, Paradis P, Schiffrin EL. Aldosterone, inflammation, immune system, and hypertension [Internet]. American Journal of Hypertension. 2021 ; 34( 1): 15-27.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1093/ajh/hpaa137
Artigo de periodico
Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone (2021)
Rodrigues, Daniel ; Costa, Tiago Januário da ; Silva, Josiane Fernandes da ; Oliveira Neto, José Teles de ; Alves, Juliano Vilela ; Fedoce, Aline Garcia ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: ALDOSTERONA, DOENÇAS VASCULARES, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Daniel et al. Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone. International Journal of Molecular Sciences, v. 22, n. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/ijms22116154. Acesso em: 04 jul. 2024.
APA
Rodrigues, D., Costa, T. J. da, Silva, J. F. da, Oliveira Neto, J. T. de, Alves, J. V., Fedoce, A. G., et al. (2021). Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone. International Journal of Molecular Sciences, 22( 11), 1-16. doi:10.3390/ijms22116154
NLM
Rodrigues D, Costa TJ da, Silva JF da, Oliveira Neto JT de, Alves JV, Fedoce AG, Costa RM da, Tostes R de CA. Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 11): 1-16.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3390/ijms22116154
Vancouver
Rodrigues D, Costa TJ da, Silva JF da, Oliveira Neto JT de, Alves JV, Fedoce AG, Costa RM da, Tostes R de CA. Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 11): 1-16.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3390/ijms22116154
Artigo de periodico
The homeostatic role of hydrogen peroxide, superoxide anion and nitric oxide in the vasculature (2021)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de; Arce, Cristina; Santos, Jeimison Duarte ; Silva Neto, Júlio Alves da; Egea, Gustavo; Dantas, Ana Paula; Tostes, Rita de Cássia Aleixo ; Jiménez-Altayó, Francesc
Source: Free Radical Biology and Medicine. Unidade: FMRP
Subjects: ANTIOXIDANTES, HOMEOSTASE, ESPÉCIES REATIVAS DE OXIGÊNIO, PERÓXIDO DE HIDROGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. The homeostatic role of hydrogen peroxide, superoxide anion and nitric oxide in the vasculature. Free Radical Biology and Medicine, v. 162, p. 615-635, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2020.11.021. Acesso em: 04 jul. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Arce, C., Santos, J. D., Silva Neto, J. A. da, Egea, G., et al. (2021). The homeostatic role of hydrogen peroxide, superoxide anion and nitric oxide in the vasculature. Free Radical Biology and Medicine, 162, 615-635. doi:10.1016/j.freeradbiomed.2020.11.021
NLM
Costa TJ da, Barros PR de, Arce C, Santos JD, Silva Neto JA da, Egea G, Dantas AP, Tostes R de CA, Jiménez-Altayó F. The homeostatic role of hydrogen peroxide, superoxide anion and nitric oxide in the vasculature [Internet]. Free Radical Biology and Medicine. 2021 ; 162 615-635.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.021
Vancouver
Costa TJ da, Barros PR de, Arce C, Santos JD, Silva Neto JA da, Egea G, Dantas AP, Tostes R de CA, Jiménez-Altayó F. The homeostatic role of hydrogen peroxide, superoxide anion and nitric oxide in the vasculature [Internet]. Free Radical Biology and Medicine. 2021 ; 162 615-635.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.021

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