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  • Source: Biology Open. Unidade: ICB

    Subjects: ANURA, OÓCITOS, XENOPUS, PROTEÍNAS DA MEMBRANA

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      ROSA, Jessica Kabutomori et al. Lithobates catesbeianus (American Bullfrog) oocytes: a novelheterologous expression system for aquaporins. Biology Open, v. 7, n. 4, p. 9 , 2018Tradução . . Disponível em: https://doi.org/10.1242/bio.031880. Acesso em: 05 jul. 2024.
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      Rosa, J. K., Silva, O. B., Geyer, R. R., & Musa-Aziz, R. (2018). Lithobates catesbeianus (American Bullfrog) oocytes: a novelheterologous expression system for aquaporins. Biology Open, 7( 4), 9 . doi:10.1242/bio.031880
    • NLM

      Rosa JK, Silva OB, Geyer RR, Musa-Aziz R. Lithobates catesbeianus (American Bullfrog) oocytes: a novelheterologous expression system for aquaporins [Internet]. Biology Open. 2018 ; 7( 4): 9 .[citado 2024 jul. 05 ] Available from: https://doi.org/10.1242/bio.031880
    • Vancouver

      Rosa JK, Silva OB, Geyer RR, Musa-Aziz R. Lithobates catesbeianus (American Bullfrog) oocytes: a novelheterologous expression system for aquaporins [Internet]. Biology Open. 2018 ; 7( 4): 9 .[citado 2024 jul. 05 ] Available from: https://doi.org/10.1242/bio.031880
  • Source: Archives endocrinology and Metabolism. Unidade: ICB

    Subjects: MELATONINA, GLÂNDULA PINEAL, HORMÔNIO DA GLÂNDULA PINEAL

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      AMARAL, Fernanda Gaspar do e CIPOLLA NETO, José. A brief about melatonin, a pineal hormone. Archives endocrinology and Metabolism, v. 62, n. 4, p. 472-479, 2018Tradução . . Disponível em: https://doi.org/10.20945/2359-3997000000066. Acesso em: 05 jul. 2024.
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      Amaral, F. G. do, & Cipolla Neto, J. (2018). A brief about melatonin, a pineal hormone. Archives endocrinology and Metabolism, 62( 4), 472-479. doi:10.20945/2359-3997000000066
    • NLM

      Amaral FG do, Cipolla Neto J. A brief about melatonin, a pineal hormone [Internet]. Archives endocrinology and Metabolism. 2018 ; 62( 4): 472-479.[citado 2024 jul. 05 ] Available from: https://doi.org/10.20945/2359-3997000000066
    • Vancouver

      Amaral FG do, Cipolla Neto J. A brief about melatonin, a pineal hormone [Internet]. Archives endocrinology and Metabolism. 2018 ; 62( 4): 472-479.[citado 2024 jul. 05 ] Available from: https://doi.org/10.20945/2359-3997000000066
  • Source: Journal of Comparative Neurology. Unidade: ICB

    Subjects: SISTEMA NERVOSO AUTÔNOMO, FISIOLOGIA

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      GOKOZAN, Hamza Numan et al. Area postrema undergoes dynamic postnatal changes in mice and humans. Journal of Comparative Neurology, v. 524, n. 6, p. 1259-1269, 2016Tradução . . Disponível em: https://doi.org/10.1002/cne.23903. Acesso em: 05 jul. 2024.
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      Gokozan, H. N., Baig, F., Corcoran, S., Catacutan, F. P., Gygli, P. E., Takakura, A. C., et al. (2016). Area postrema undergoes dynamic postnatal changes in mice and humans. Journal of Comparative Neurology, 524( 6), 1259-1269. doi:10.1002/cne.23903
    • NLM

      Gokozan HN, Baig F, Corcoran S, Catacutan FP, Gygli PE, Takakura AC, Moreira T dos S, Czeisler C, Otero JJ. Area postrema undergoes dynamic postnatal changes in mice and humans [Internet]. Journal of Comparative Neurology. 2016 ; 524( 6): 1259-1269.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/cne.23903
    • Vancouver

      Gokozan HN, Baig F, Corcoran S, Catacutan FP, Gygli PE, Takakura AC, Moreira T dos S, Czeisler C, Otero JJ. Area postrema undergoes dynamic postnatal changes in mice and humans [Internet]. Journal of Comparative Neurology. 2016 ; 524( 6): 1259-1269.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/cne.23903
  • Source: Journal of Neurophysiology. Unidade: ICB

    Subjects: RECEPTORES ADRENÉRGICOS, NOREPINEFRINA, RESPIRAÇÃO

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      SANTOS, Luiz Marcelo Oliveira et al. α1- and α2-adrenergic receptors in the retrotrapezoid nucleus differentially regulate breathing in anesthetized adult rats. Journal of Neurophysiology, v. 116, n. 3, p. 1036-1048, 2016Tradução . . Disponível em: https://doi.org/10.1152/jn.00023.2016. Acesso em: 05 jul. 2024.
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      Santos, L. M. O., Moreira, T. dos S., Kuo, F. -S., Mulkey, D. K., & Takakura, A. C. (2016). α1- and α2-adrenergic receptors in the retrotrapezoid nucleus differentially regulate breathing in anesthetized adult rats. Journal of Neurophysiology, 116( 3), 1036-1048. doi:10.1152/jn.00023.2016
    • NLM

      Santos LMO, Moreira T dos S, Kuo F-S, Mulkey DK, Takakura AC. α1- and α2-adrenergic receptors in the retrotrapezoid nucleus differentially regulate breathing in anesthetized adult rats [Internet]. Journal of Neurophysiology. 2016 ; 116( 3): 1036-1048.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1152/jn.00023.2016
    • Vancouver

      Santos LMO, Moreira T dos S, Kuo F-S, Mulkey DK, Takakura AC. α1- and α2-adrenergic receptors in the retrotrapezoid nucleus differentially regulate breathing in anesthetized adult rats [Internet]. Journal of Neurophysiology. 2016 ; 116( 3): 1036-1048.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1152/jn.00023.2016
  • Source: Hypertension. Unidade: ICB

    Subjects: FISIOLOGIA, FISIOLOGIA CARDIOVASCULAR, HIPERTENSÃO, HIPERTENSÃO, INIBIDORES QUÍMICOS

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      MARTÍNEZ-MARTÍNEZ, Ernesto et al. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, v. 66, n. 4, p. 767-775, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876. Acesso em: 05 jul. 2024.
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      Martínez-Martínez, E., Calvier, L., Fernández-Celis, A., Rousseau, E., Jurado-López, R., Rossoni, L. V., et al. (2015). Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, 66( 4), 767-775. doi:10.1161/HYPERTENSIONAHA.115.05876
    • NLM

      Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
    • Vancouver

      Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
  • Source: Journal of Nutritional Biochemistry. Unidades: ICB, FCF

    Subjects: SEPSE, ESTRESSE OXIDATIVO, GLUTAMINA

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      CRUZAT, Vinicius Fernandes et al. Oral supplementations with free and dipeptide forms of L-glutamine in endotoxemic mice: effects on muscle glutamine-glutathione axis and heat shock proteins. Journal of Nutritional Biochemistry, v. 25, n. 3, p. 345-352, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2013.11.009. Acesso em: 05 jul. 2024.
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      Cruzat, V. F., Pantaleão, L. C., Donato Junior, J., Bittencourt Junior, P. I. H. de, & Tirapegui, J. (2014). Oral supplementations with free and dipeptide forms of L-glutamine in endotoxemic mice: effects on muscle glutamine-glutathione axis and heat shock proteins. Journal of Nutritional Biochemistry, 25( 3), 345-352. doi:10.1016/j.jnutbio.2013.11.009
    • NLM

      Cruzat VF, Pantaleão LC, Donato Junior J, Bittencourt Junior PIH de, Tirapegui J. Oral supplementations with free and dipeptide forms of L-glutamine in endotoxemic mice: effects on muscle glutamine-glutathione axis and heat shock proteins [Internet]. Journal of Nutritional Biochemistry. 2014 ; 25( 3): 345-352.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.jnutbio.2013.11.009
    • Vancouver

      Cruzat VF, Pantaleão LC, Donato Junior J, Bittencourt Junior PIH de, Tirapegui J. Oral supplementations with free and dipeptide forms of L-glutamine in endotoxemic mice: effects on muscle glutamine-glutathione axis and heat shock proteins [Internet]. Journal of Nutritional Biochemistry. 2014 ; 25( 3): 345-352.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.jnutbio.2013.11.009
  • Source: Regulatory Peptides. Unidade: ICB

    Assunto: FISIOLOGIA

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      ALVES, E. S. et al. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets. Regulatory Peptides, v. 175, n. 1-3, p. 1-6, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.regpep.2012.01.003. Acesso em: 05 jul. 2024.
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      Alves, E. S., Haidar, A. A., Quadros, C. D., Carvalho, D. S., Morgan, D., Rocha, M. S., et al. (2012). Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets. Regulatory Peptides, 175( 1-3), 1-6. doi:10.1016/j.regpep.2012.01.003
    • NLM

      Alves ES, Haidar AA, Quadros CD, Carvalho DS, Morgan D, Rocha MS, Curi R, Carpinelli AR, Hirata AE. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets [Internet]. Regulatory Peptides. 2012 ; 175( 1-3): 1-6.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.regpep.2012.01.003
    • Vancouver

      Alves ES, Haidar AA, Quadros CD, Carvalho DS, Morgan D, Rocha MS, Curi R, Carpinelli AR, Hirata AE. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets [Internet]. Regulatory Peptides. 2012 ; 175( 1-3): 1-6.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.regpep.2012.01.003
  • Source: Intensive Care Medicine. Unidades: ICB, FM

    Subjects: FISIOLOGIA, LIPÍDEOS, GORDURAS, SEPSE, GLICEMIA, ESTUDOS PROSPECTIVOS, ESTUDOS RANDOMIZADOS, LIPOPROTEÍNAS

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      CAPPI, Sylas Bezerra et al. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis. Intensive Care Medicine, v. 38, p. 634-641, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00134-011-2458-z. Acesso em: 05 jul. 2024.
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      Cappi, S. B., Noritomi, D. T., Velasco, I. T., Curi, R., Loureiro, T. C. A., & Soriano, F. G. (2012). Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis. Intensive Care Medicine, 38, 634-641. doi:10.1007/s00134-011-2458-z
    • NLM

      Cappi SB, Noritomi DT, Velasco IT, Curi R, Loureiro TCA, Soriano FG. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis [Internet]. Intensive Care Medicine. 2012 ; 38 634-641.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00134-011-2458-z
    • Vancouver

      Cappi SB, Noritomi DT, Velasco IT, Curi R, Loureiro TCA, Soriano FG. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis [Internet]. Intensive Care Medicine. 2012 ; 38 634-641.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00134-011-2458-z
  • Source: Journal of Biomedicine and Biotechnology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      HIRABARA, Sandro M. et al. Molecular targets related to inflammation and insulin resistance and potential interventions. Journal of Biomedicine and Biotechnology, v. 2012, n. 379024, p. 1-16, 2012Tradução . . Disponível em: https://doi.org/10.1155/2012/379024. Acesso em: 05 jul. 2024.
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      Hirabara, S. M., Gorjão, R., Vinolo, M. A., Rodrigues, A. C., Nachbar, R. T., & Curi, R. (2012). Molecular targets related to inflammation and insulin resistance and potential interventions. Journal of Biomedicine and Biotechnology, 2012( 379024), 1-16. doi:10.1155/2012/379024
    • NLM

      Hirabara SM, Gorjão R, Vinolo MA, Rodrigues AC, Nachbar RT, Curi R. Molecular targets related to inflammation and insulin resistance and potential interventions [Internet]. Journal of Biomedicine and Biotechnology. 2012 ; 2012( 379024): 1-16.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1155/2012/379024
    • Vancouver

      Hirabara SM, Gorjão R, Vinolo MA, Rodrigues AC, Nachbar RT, Curi R. Molecular targets related to inflammation and insulin resistance and potential interventions [Internet]. Journal of Biomedicine and Biotechnology. 2012 ; 2012( 379024): 1-16.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1155/2012/379024
  • Source: Thyroid. Unidade: ICB

    Assunto: FISIOLOGIA

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      RACT, Erika Lia Brunetto et al. T3 rapidly increases slc2a4 gene expression. and GLUT4 trafficking to the plasma membrane in skeletal muscle of rat and improves glucose homeostasis. Thyroid, v. 22, n. 1, p. 70-79, 2012Tradução . . Disponível em: https://doi.org/10.1089/thy.2010.0409. Acesso em: 05 jul. 2024.
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      Ract, E. L. B., Teixeira, S. da S., Giannocco, G., Machado, U. F., & Nunes, M. T. (2012). T3 rapidly increases slc2a4 gene expression. and GLUT4 trafficking to the plasma membrane in skeletal muscle of rat and improves glucose homeostasis. Thyroid, 22( 1), 70-79. doi:10.1089/thy.2010.0409
    • NLM

      Ract ELB, Teixeira S da S, Giannocco G, Machado UF, Nunes MT. T3 rapidly increases slc2a4 gene expression. and GLUT4 trafficking to the plasma membrane in skeletal muscle of rat and improves glucose homeostasis [Internet]. Thyroid. 2012 ; 22( 1): 70-79.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1089/thy.2010.0409
    • Vancouver

      Ract ELB, Teixeira S da S, Giannocco G, Machado UF, Nunes MT. T3 rapidly increases slc2a4 gene expression. and GLUT4 trafficking to the plasma membrane in skeletal muscle of rat and improves glucose homeostasis [Internet]. Thyroid. 2012 ; 22( 1): 70-79.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1089/thy.2010.0409
  • Source: Behavioural Brain Research. Unidades: ICB, FMVZ

    Subjects: HIPOTÁLAMO, ALCALOIDES

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      MOTA-ORTIZ, Sandra Regina et al. The periaqueductal gray as a critical site to mediate reward seeking during predatory hunting. Behavioural Brain Research, v. 226, n. 1, p. 32-40, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2011.08.034. Acesso em: 05 jul. 2024.
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      Mota-Ortiz, S. R., Sukikara, M. H., Bittencourt, J. C., Baldo, M. V. C., Elias, C. F., Felício, L. F., & Canteras, N. S. (2012). The periaqueductal gray as a critical site to mediate reward seeking during predatory hunting. Behavioural Brain Research, 226( 1), 32-40. doi:10.1016/j.bbr.2011.08.034
    • NLM

      Mota-Ortiz SR, Sukikara MH, Bittencourt JC, Baldo MVC, Elias CF, Felício LF, Canteras NS. The periaqueductal gray as a critical site to mediate reward seeking during predatory hunting [Internet]. Behavioural Brain Research. 2012 ; 226( 1): 32-40.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.bbr.2011.08.034
    • Vancouver

      Mota-Ortiz SR, Sukikara MH, Bittencourt JC, Baldo MVC, Elias CF, Felício LF, Canteras NS. The periaqueductal gray as a critical site to mediate reward seeking during predatory hunting [Internet]. Behavioural Brain Research. 2012 ; 226( 1): 32-40.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.bbr.2011.08.034
  • Source: European Journal of Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

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      ANHÊ, Gabriel F. et al. Quercetin decreases inflammatory response and increases insulin action in skeletal muscle of ob/ob mice and in L6 myotubes. European Journal of Pharmacology, v. 689, n. 1-3, p. 285-293, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2012.06.007. Acesso em: 05 jul. 2024.
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      Anhê, G. F., Okamoto, M. M., Kinote, A., Sollon, C., Lellis-Santos, C., Anhê, F. F., et al. (2012). Quercetin decreases inflammatory response and increases insulin action in skeletal muscle of ob/ob mice and in L6 myotubes. European Journal of Pharmacology, 689( 1-3), 285-293. doi:10.1016/j.ejphar.2012.06.007
    • NLM

      Anhê GF, Okamoto MM, Kinote A, Sollon C, Lellis-Santos C, Anhê FF, Lima GA, Hirabara SM, Velloso LA, Bordin S, Machado UF. Quercetin decreases inflammatory response and increases insulin action in skeletal muscle of ob/ob mice and in L6 myotubes [Internet]. European Journal of Pharmacology. 2012 ; 689( 1-3): 285-293.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.ejphar.2012.06.007
    • Vancouver

      Anhê GF, Okamoto MM, Kinote A, Sollon C, Lellis-Santos C, Anhê FF, Lima GA, Hirabara SM, Velloso LA, Bordin S, Machado UF. Quercetin decreases inflammatory response and increases insulin action in skeletal muscle of ob/ob mice and in L6 myotubes [Internet]. European Journal of Pharmacology. 2012 ; 689( 1-3): 285-293.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.ejphar.2012.06.007
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      CALIL-SILVEIRA, Jamile e SERRANO-NASCIMENTO, Caroline e NUNES, Maria Tereza. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms. Molecular and Cellular Endocrinology, v. 350, n. 1, p. 118-124, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2011.12.002. Acesso em: 05 jul. 2024.
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      Calil-Silveira, J., Serrano-Nascimento, C., & Nunes, M. T. (2012). Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms. Molecular and Cellular Endocrinology, 350( 1), 118-124. doi:10.1016/j.mce.2011.12.002
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Nunes MT. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms [Internet]. Molecular and Cellular Endocrinology. 2012 ; 350( 1): 118-124.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.mce.2011.12.002
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Nunes MT. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms [Internet]. Molecular and Cellular Endocrinology. 2012 ; 350( 1): 118-124.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.mce.2011.12.002
  • Source: Toxicology in Vitro. Unidade: ICB

    Assunto: FISIOLOGIA

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      MASI, Laureane Nunes et al. Toxicity of fatty acids on ECV-304 endothelial cells. Toxicology in Vitro, v. 25, n. 8, p. 2140-2146, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2011.06.011. Acesso em: 05 jul. 2024.
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      Masi, L. N., Portioli-Sanches, É. P., Lima-Salgado, T. M., & Curi, R. (2011). Toxicity of fatty acids on ECV-304 endothelial cells. Toxicology in Vitro, 25( 8), 2140-2146. doi:10.1016/j.tiv.2011.06.011
    • NLM

      Masi LN, Portioli-Sanches ÉP, Lima-Salgado TM, Curi R. Toxicity of fatty acids on ECV-304 endothelial cells [Internet]. Toxicology in Vitro. 2011 ; 25( 8): 2140-2146.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.tiv.2011.06.011
    • Vancouver

      Masi LN, Portioli-Sanches ÉP, Lima-Salgado TM, Curi R. Toxicity of fatty acids on ECV-304 endothelial cells [Internet]. Toxicology in Vitro. 2011 ; 25( 8): 2140-2146.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.tiv.2011.06.011
  • Source: PloS Biology. Unidade: ICB

    Subjects: FISIOLOGIA, IMUNOLOGIA

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      CARICILLI, Andréa M. et al. Gut microbiota is a key modulator of insulin resistance in TLR 2 knockout mice. PloS Biology, v. 9, n. 12, p. 1-21, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pbio.1001212. Acesso em: 05 jul. 2024.
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      Caricilli, A. M., Picardi, P. K., Abreu, L. L. de, Ueno, M., Prada, P. O., Ropelle, E. R., et al. (2011). Gut microbiota is a key modulator of insulin resistance in TLR 2 knockout mice. PloS Biology, 9( 12), 1-21. doi:10.1371/journal.pbio.1001212
    • NLM

      Caricilli AM, Picardi PK, Abreu LL de, Ueno M, Prada PO, Ropelle ER, Hirabara SM, Castoldi Â, Vieira P, Câmara NOS, Curi R, Carvalheira JB, Saad MJA. Gut microbiota is a key modulator of insulin resistance in TLR 2 knockout mice [Internet]. PloS Biology. 2011 ; 9( 12): 1-21.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1371/journal.pbio.1001212
    • Vancouver

      Caricilli AM, Picardi PK, Abreu LL de, Ueno M, Prada PO, Ropelle ER, Hirabara SM, Castoldi Â, Vieira P, Câmara NOS, Curi R, Carvalheira JB, Saad MJA. Gut microbiota is a key modulator of insulin resistance in TLR 2 knockout mice [Internet]. PloS Biology. 2011 ; 9( 12): 1-21.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1371/journal.pbio.1001212
  • Source: Physiology & Behavior. Unidades: FMVZ, ICB

    Subjects: IMMUNOBLOTTING, COMPORTAMENTO MATERNO ANIMAL, TÉCNICAS E PROCEDIMENTOS DE LABORATÓRIO, OLFATO (PERCEPÇÃO)

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      KIRSTEN, Thiago Berti et al. Prenatal LPS exposure reduces olfactory perception in neonatal and adult rats. Physiology & Behavior, v. 104, n. 3, p. 417-422, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.physbeh.2011.04.049. Acesso em: 05 jul. 2024.
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      Kirsten, T. B., Chaves, G. P., Taricano, M., Martins, D. O., Florio, J. C., Britto, L. R. G. de, et al. (2011). Prenatal LPS exposure reduces olfactory perception in neonatal and adult rats. Physiology & Behavior, 104( 3), 417-422. doi:10.1016/j.physbeh.2011.04.049
    • NLM

      Kirsten TB, Chaves GP, Taricano M, Martins DO, Florio JC, Britto LRG de, Torrão A da S, Palermo-Neto J, Bernardi MM. Prenatal LPS exposure reduces olfactory perception in neonatal and adult rats [Internet]. Physiology & Behavior. 2011 ; 104( 3): 417-422.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.physbeh.2011.04.049
    • Vancouver

      Kirsten TB, Chaves GP, Taricano M, Martins DO, Florio JC, Britto LRG de, Torrão A da S, Palermo-Neto J, Bernardi MM. Prenatal LPS exposure reduces olfactory perception in neonatal and adult rats [Internet]. Physiology & Behavior. 2011 ; 104( 3): 417-422.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.physbeh.2011.04.049
  • Source: Diabetes. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

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      COLLINS, Stephan C. et al. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, v. 59, n. 5, p. 1192-1201, 2010Tradução . . Acesso em: 05 jul. 2024.
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      Collins, S. C., Hoppa, M. B., Walker, J. N., Amisten, S., Abdulkader, F. R. de M., Bengtsson, M., et al. (2010). Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, 59( 5), 1192-1201.
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      Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 jul. 05 ]
    • Vancouver

      Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 jul. 05 ]
  • Source: Neuroscience. Unidades: ICB, FMRP

    Subjects: ANATOMIA, FISIOLOGIA

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      FURIGO, I. C. et al. The role of the superior colliculus in predatory hunting. Neuroscience, v. 165, n. 1, p. 1-15, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2009.10.004. Acesso em: 05 jul. 2024.
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      Furigo, I. C., Oliveira, W. F. de, Oliveira, A. R., Comoli, E., Baldo, M. V. C., Mota-Ortiz, S. R., & Canteras, N. S. (2010). The role of the superior colliculus in predatory hunting. Neuroscience, 165( 1), 1-15. doi:10.1016/j.neuroscience.2009.10.004
    • NLM

      Furigo IC, Oliveira WF de, Oliveira AR, Comoli E, Baldo MVC, Mota-Ortiz SR, Canteras NS. The role of the superior colliculus in predatory hunting [Internet]. Neuroscience. 2010 ; 165( 1): 1-15.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.neuroscience.2009.10.004
    • Vancouver

      Furigo IC, Oliveira WF de, Oliveira AR, Comoli E, Baldo MVC, Mota-Ortiz SR, Canteras NS. The role of the superior colliculus in predatory hunting [Internet]. Neuroscience. 2010 ; 165( 1): 1-15.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.neuroscience.2009.10.004
  • Source: Cell Biochemistry and Function. Unidade: ICB

    Assunto: FISIOLOGIA

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      MARIN, Douglas Popp et al. Testosterone suppresses oxidative stress in human neutrophils. Cell Biochemistry and Function, v. 28, n. 5, p. 394-402, 2010Tradução . . Disponível em: https://doi.org/10.1002/cbf.1669. Acesso em: 05 jul. 2024.
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      Marin, D. P., Bolin, A. P., Santos, R. de C. M. dos, Curi, R., & Otton, R. (2010). Testosterone suppresses oxidative stress in human neutrophils. Cell Biochemistry and Function, 28( 5), 394-402. doi:10.1002/cbf.1669
    • NLM

      Marin DP, Bolin AP, Santos R de CM dos, Curi R, Otton R. Testosterone suppresses oxidative stress in human neutrophils [Internet]. Cell Biochemistry and Function. 2010 ; 28( 5): 394-402.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/cbf.1669
    • Vancouver

      Marin DP, Bolin AP, Santos R de CM dos, Curi R, Otton R. Testosterone suppresses oxidative stress in human neutrophils [Internet]. Cell Biochemistry and Function. 2010 ; 28( 5): 394-402.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/cbf.1669
  • Source: European Journal of Applied Physiology. Unidades: ICB, EEFERP

    Assunto: FISIOLOGIA

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      BASSIT, Reinaldo Abunasser et al. Effect of short-term creatine supplementation on markers of skeletal muscle damage after strenuous contractile activity. European Journal of Applied Physiology, v. 108, n. 5, p. 945-55, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00421-009-1305-1. Acesso em: 05 jul. 2024.
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      Bassit, R. A., Pinheiro, C. H. da J., Vitzel, K. F., Sproesser, A. J., Silveira, L. R., & Curi, R. (2010). Effect of short-term creatine supplementation on markers of skeletal muscle damage after strenuous contractile activity. European Journal of Applied Physiology, 108( 5), 945-55. doi:10.1007/s00421-009-1305-1
    • NLM

      Bassit RA, Pinheiro CH da J, Vitzel KF, Sproesser AJ, Silveira LR, Curi R. Effect of short-term creatine supplementation on markers of skeletal muscle damage after strenuous contractile activity [Internet]. European Journal of Applied Physiology. 2010 ; 108( 5): 945-55.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00421-009-1305-1
    • Vancouver

      Bassit RA, Pinheiro CH da J, Vitzel KF, Sproesser AJ, Silveira LR, Curi R. Effect of short-term creatine supplementation on markers of skeletal muscle damage after strenuous contractile activity [Internet]. European Journal of Applied Physiology. 2010 ; 108( 5): 945-55.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00421-009-1305-1

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