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Artigo de periodico
12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers (2024)
Sousa, Carlos Alberto Rodrigues de ; Nogueira, Luciana Fidalgo Ramos ; Moreno, Claudia Roberta de Castro ; Marqueze, Elaine Cristina ; Cipolla Neto, José
Source: Frontiers in Nutrition. Unidades: FSP, ICB
Subjects: FISIOLOGIA, RITMOS BIOLÓGICOS, HOMEOSTASE, GORDURAS, COMORBIDADE, DIABETES MELLITUS, OBESIDADE, MELATONINA, TRABALHO NOTURNO, MULHERES, PESOS E MEDIDAS CORPORAIS, ESTUDOS RANDOMIZADOS
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SOUSA, Carlos Alberto Rodrigues de et al. 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers. Frontiers in Nutrition, v. 11, p. 11 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fnut.2024.1285398. Acesso em: 10 jul. 2024.
APA
Sousa, C. A. R. de, Nogueira, L. F. R., Moreno, C. R. de C., Marqueze, E. C., & Cipolla Neto, J. (2024). 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers. Frontiers in Nutrition, 11, 11 . doi:10.3389/fnut.2024.1285398
NLM
Sousa CAR de, Nogueira LFR, Moreno CR de C, Marqueze EC, Cipolla Neto J. 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers [Internet]. Frontiers in Nutrition. 2024 ; 11 11 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.3389/fnut.2024.1285398
Vancouver
Sousa CAR de, Nogueira LFR, Moreno CR de C, Marqueze EC, Cipolla Neto J. 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers [Internet]. Frontiers in Nutrition. 2024 ; 11 11 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.3389/fnut.2024.1285398
Artigo de periodico
Rs4862705 in the melatonin receptor 1A gene is associated with renal function decline in type 1 diabetes individuals (2024)
Daher, Gustavo; Bezerra, Daniele Pereira Santos ; Cavaleiro, Ana Mercedes; Pelaes, Tatiana Souza ; Admoni, Sharon Nina ; Perez, Ricardo Vessoni ; Machado, Cleide Guimarães ; Amaral, Fernanda Gaspar do ; Gianella, Maria Lúcia Corrêa ; Cipolla Neto, José
Source: Frontiers in Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, MELATONINA, ANTIOXIDANTES, DIABETES MELLITUS, POLIMORFISMO, ESTRESSE OXIDATIVO, ESTUDOS TRANSVERSAIS, ESTUDOS LONGITUDINAIS, NEFROPATIAS
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DAHER, Gustavo et al. Rs4862705 in the melatonin receptor 1A gene is associated with renal function decline in type 1 diabetes individuals. Frontiers in Endocrinology, 2024Tradução . . Disponível em: https://doi.org/10.3389/fendo.2024.1331012. Acesso em: 10 jul. 2024.
APA
Daher, G., Bezerra, D. P. S., Cavaleiro, A. M., Pelaes, T. S., Admoni, S. N., Perez, R. V., et al. (2024). Rs4862705 in the melatonin receptor 1A gene is associated with renal function decline in type 1 diabetes individuals. Frontiers in Endocrinology. doi:10.3389/fendo.2024.1331012
NLM
Daher G, Bezerra DPS, Cavaleiro AM, Pelaes TS, Admoni SN, Perez RV, Machado CG, Amaral FG do, Gianella MLC, Cipolla Neto J. Rs4862705 in the melatonin receptor 1A gene is associated with renal function decline in type 1 diabetes individuals [Internet]. Frontiers in Endocrinology. 2024 ;[citado 2024 jul. 10 ] Available from: https://doi.org/10.3389/fendo.2024.1331012
Vancouver
Daher G, Bezerra DPS, Cavaleiro AM, Pelaes TS, Admoni SN, Perez RV, Machado CG, Amaral FG do, Gianella MLC, Cipolla Neto J. Rs4862705 in the melatonin receptor 1A gene is associated with renal function decline in type 1 diabetes individuals [Internet]. Frontiers in Endocrinology. 2024 ;[citado 2024 jul. 10 ] Available from: https://doi.org/10.3389/fendo.2024.1331012
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Experimental Neurology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
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FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 10 jul. 2024.
APA
Ferreira, A. F. F., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells (2024)
Garcia, Bruna Ruschel Ewald Vega ; Makiyama, Edson Naoto ; Sampaio, Geni Rodrigues ; Freitas, Rosana Aparecida Manólio Soares ; Bovini, Andrea ; Amaral, Andressa Godoy ; Fock, Ricardo Ambrósio ; Rogero, Marcelo Macedo ; Bordin, Silvana
Source: Metabolites. Unidades: FCF, FSP, ICB
Subjects: FISIOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS EPITELIAIS E GLANDULARES, ADENOCARCINOMA, AMINOÁCIDOS, LIPOPOLISSACARÍDEOS, INFLAMAÇÃO, ANTI-INFLAMATÓRIOS, MUCOSA INTESTINAL, GLUTATIONA TRANSFERASE
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GARCIA, Bruna Ruschel Ewald Vega et al. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells. Metabolites, v. 14, n. 1, p. 13 , 2024Tradução . . Disponível em: https://doi.org/10.3390/metabo14010076. Acesso em: 10 jul. 2024.
APA
Garcia, B. R. E. V., Makiyama, E. N., Sampaio, G. R., Freitas, R. A. M. S., Bovini, A., Amaral, A. G., et al. (2024). Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells. Metabolites, 14( 1), 13 . doi:10.3390/metabo14010076
NLM
Garcia BREV, Makiyama EN, Sampaio GR, Freitas RAMS, Bovini A, Amaral AG, Fock RA, Rogero MM, Bordin S. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells [Internet]. Metabolites. 2024 ; 14( 1): 13 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.3390/metabo14010076
Vancouver
Garcia BREV, Makiyama EN, Sampaio GR, Freitas RAMS, Bovini A, Amaral AG, Fock RA, Rogero MM, Bordin S. Effects of branched-chain amino acids on the inflammatory response induced by LPS in caco-2 cells [Internet]. Metabolites. 2024 ; 14( 1): 13 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.3390/metabo14010076
Artigo de periodico
Ironing out obesity (2024)
Souza, Gabriel Orefice de ; Santos, Willian Oliveira dos ; Donato Junior, José
Source: Trends in Endocrinology and Metabolism. Unidade: ICB
Subjects: FISIOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, OBESIDADE, RESISTÊNCIA À INSULINA, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO, FERRO
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SOUZA, Gabriel Orefice de e SANTOS, Willian Oliveira dos e DONATO JUNIOR, José. Ironing out obesity. Trends in Endocrinology and Metabolism, v. 35, n. 6, p. 456-458, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.tem.2024.04.001. Acesso em: 10 jul. 2024.
APA
Souza, G. O. de, Santos, W. O. dos, & Donato Junior, J. (2024). Ironing out obesity. Trends in Endocrinology and Metabolism, 35( 6), 456-458. doi:10.1016/j.tem.2024.04.001
NLM
Souza GO de, Santos WO dos, Donato Junior J. Ironing out obesity [Internet]. Trends in Endocrinology and Metabolism. 2024 ; 35( 6): 456-458.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.tem.2024.04.001
Vancouver
Souza GO de, Santos WO dos, Donato Junior J. Ironing out obesity [Internet]. Trends in Endocrinology and Metabolism. 2024 ; 35( 6): 456-458.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.tem.2024.04.001
Artigo de periodico
Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats (2024)
Dellacqua, Lais Oliveira; Gomes, Paula Magalhães ; Batista, Julia Santos; Michelini, Lisete Compagno ; Antunes, Vagner Roberto
Source: Journal of Applied Physiology. Unidade: ICB
Subjects: FISIOLOGIA, BARORREFLEXO, FISIOLOGIA CARDIOVASCULAR, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, HIPERTENSÃO, ENVELHECIMENTO, TREINAMENTO AERÓBIO, PLASTICIDADE NEURONAL, RATOS WISTAR
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DELLACQUA, Lais Oliveira et al. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats. Journal of Applied Physiology, v. 136, n. 1, p. 189-198, 2024Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.00433.2023. Acesso em: 10 jul. 2024.
APA
Dellacqua, L. O., Gomes, P. M., Batista, J. S., Michelini, L. C., & Antunes, V. R. (2024). Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats. Journal of Applied Physiology, 136( 1), 189-198. doi:10.1152/japplphysiol.00433.2023
NLM
Dellacqua LO, Gomes PM, Batista JS, Michelini LC, Antunes VR. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats [Internet]. Journal of Applied Physiology. 2024 ; 136( 1): 189-198.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1152/japplphysiol.00433.2023
Vancouver
Dellacqua LO, Gomes PM, Batista JS, Michelini LC, Antunes VR. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats [Internet]. Journal of Applied Physiology. 2024 ; 136( 1): 189-198.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1152/japplphysiol.00433.2023
Artigo de periodico
Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure (2023)
Fontes, Milene T.; Arruda Junior, Daniel F.; Santos, Danúbia Silva dos ; Silva, João Carlos Ribeiro ; Antônio, Edinei L.; Tucci, Paulo F. J. ; Rossoni, Luciana Venturini ; Girardi, Adriana C. C.
Source: Life Sciences. Unidades: ICB, FM
Assunto: FISIOLOGIA
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FONTES, Milene T. et al. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, v. 323, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121648. Acesso em: 10 jul. 2024.
APA
Fontes, M. T., Arruda Junior, D. F., Santos, D. S. dos, Silva, J. C. R., Antônio, E. L., Tucci, P. F. J., et al. (2023). Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, 323, 1-10. doi:10.1016/j.lfs.2023.121648
NLM
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Vancouver
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Artigo de periodico
miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction (2023)
Silva, Camila Stephanie Balbino ; Couto, Gisele Kruger ; Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Huang, Zhan-Peng ; Wang, Da Zhi ; Festuccia, William Tadeu Lara ; Barreto-Chaves, Maria Luiza Morais ; Rossoni, Luciana Venturini ; Diniz, Gabriela Placoná
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, FISIOLOGIA, AORTA DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, VASOS SANGUÍNEOS, TECIDO ADIPOSO, MICRORNAS, OBESIDADE, REAÇÃO EM CADEIA POR POLIMERASE, WESTERN BLOTTING
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SILVA, Camila Stephanie Balbino et al. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, v. 316, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121416. Acesso em: 10 jul. 2024.
APA
Silva, C. S. B., Couto, G. K., Lino, C. A., Silva, T. de O., Lunardon, G., Huang, Z. -P., et al. (2023). miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, 316, 1-10. doi:10.1016/j.lfs.2023.121416
NLM
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Vancouver
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Artigo de periodico
Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats (2023)
Gomes, Paula Magalhães ; Batista, Julia Santos; Sá, Renato Willian Martins ; Antunes, Vagner Roberto
Source: Experimental Physiology. Unidade: ICB
Subjects: FISIOLOGIA, LÍQUIDO CEFALORRAQUIDIANO, PRESSÃO SANGUÍNEA, HIPERTENSÃO, DOENÇAS CARDIOVASCULARES EM ANIMAL, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, NEUROGENÉTICA, FLUXO SANGUÍNEO, RESISTÊNCIA VASCULAR
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GOMES, Paula Magalhães et al. Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats. Experimental Physiology, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1113/EP090912op. Acesso em: 10 jul. 2024.
APA
Gomes, P. M., Batista, J. S., Sá, R. W. M., & Antunes, V. R. (2023). Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats. Experimental Physiology, 1-10. doi:10.1113/EP090912op
NLM
Gomes PM, Batista JS, Sá RWM, Antunes VR. Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats [Internet]. Experimental Physiology. 2023 ; 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/EP090912op
Vancouver
Gomes PM, Batista JS, Sá RWM, Antunes VR. Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats [Internet]. Experimental Physiology. 2023 ; 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1113/EP090912op
Artigo de periodico
Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy (2023)
Chaves Filho, Adriano de Britto ; Peixoto, Albert Souza ; Castro, Érique de ; Silva, Tiago Eugênio Oliveira da; Perandini, Luiz Augusto Buoro ; Moreira, Rafael Junges ; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da; Moretti, Eduardo Hermógenes ; Steiner, Alexandre Alarcon ; Miyamoto, Sayuri ; Yoshinaga, Marcos Yukio ; Festuccia, William Tadeu Lara
Source: Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. Unidades: ICB, IQ
Subjects: FISIOLOGIA, IMUNOLOGIA, PEROXISSOMOS, OXIDAÇÃO, TECIDO ADIPOSO, ADIPOSIDADE, FÍGADO GORDUROSO, CAMUNDONGOS, ÁCIDOS GRAXOS
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CHAVES FILHO, Adriano de Britto et al. Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids, v. 1868, n. 3, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.bbalip.2022.159264. Acesso em: 10 jul. 2024.
APA
Chaves Filho, A. de B., Peixoto, A. S., Castro, É. de, Silva, T. E. O. da, Perandini, L. A. B., Moreira, R. J., et al. (2023). Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids, 1868( 3), 1-14. doi:10.1016/j.bbalip.2022.159264
NLM
Chaves Filho A de B, Peixoto AS, Castro É de, Silva TEO da, Perandini LAB, Moreira RJ, Silva RP da, Silva BP da, Moretti EH, Steiner AA, Miyamoto S, Yoshinaga MY, Festuccia WTL. Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy [Internet]. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. 2023 ; 1868( 3): 1-14.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.bbalip.2022.159264
Vancouver
Chaves Filho A de B, Peixoto AS, Castro É de, Silva TEO da, Perandini LAB, Moreira RJ, Silva RP da, Silva BP da, Moretti EH, Steiner AA, Miyamoto S, Yoshinaga MY, Festuccia WTL. Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy [Internet]. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. 2023 ; 1868( 3): 1-14.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.bbalip.2022.159264
Artigo de periodico
Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback (2022)
Mansano, Naira da Silva ; Paradela, Regina Silva; Bohlen, Tabata Mariz ; Zanardi, Izabela Mara da Silva; Chaves, Fernanda Machado; Silveira, Marina Augusto; Tavares, Mariana Rosolen ; Donato Junior, José ; Frazão, Renata
Source: Molecular and Cellular Endocrinology. Unidades: ICB, FM
Subjects: FISIOLOGIA, ANATOMIA, ESTRÓGENOS, ESTEROIDES, HIPOTÁLAMO, NEURÔNIOS, GONADOTROFINAS, PEPTÍDEO INTESTINAL VASOATIVO, NEUROTRANSMISSORES, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS
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ABNT
MANSANO, Naira da Silva et al. Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback. Molecular and Cellular Endocrinology, v. 542, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2021.111532. Acesso em: 10 jul. 2024.
APA
Mansano, N. da S., Paradela, R. S., Bohlen, T. M., Zanardi, I. M. da S., Chaves, F. M., Silveira, M. A., et al. (2022). Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback. Molecular and Cellular Endocrinology, 542, 1-10. doi:10.1016/j.mce.2021.111532
NLM
Mansano N da S, Paradela RS, Bohlen TM, Zanardi IM da S, Chaves FM, Silveira MA, Tavares MR, Donato Junior J, Frazão R. Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback [Internet]. Molecular and Cellular Endocrinology. 2022 ; 542 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2021.111532
Vancouver
Mansano N da S, Paradela RS, Bohlen TM, Zanardi IM da S, Chaves FM, Silveira MA, Tavares MR, Donato Junior J, Frazão R. Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback [Internet]. Molecular and Cellular Endocrinology. 2022 ; 542 1-10.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mce.2021.111532
Artigo de periodico
Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats (2022)
Sá, Renato Willian Martins ; Theparambil, Shefeeq M. ; Santos, Karoline Martins dos ; Christie, Isabel N. ; Marina, Nephtali ; Cardoso, Barbara V.; Hosford, Patrick S. ; Antunes, Vagner Roberto
Source: Molecular and Cellular Neuroscience. Unidade: ICB
Subjects: FISIOLOGIA, HIPOTÁLAMO, RATOS, PRESSÃO SANGUÍNEA, ADENOSINA, ASTRÓCITOS, IMUNOHISTOQUÍMICA, NUCLEOTÍDEOS
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ABNT
SÁ, Renato Willian Martins et al. Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats. Molecular and Cellular Neuroscience, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mcn.2022.103806. Acesso em: 10 jul. 2024.
APA
Sá, R. W. M., Theparambil, S. M., Santos, K. M. dos, Christie, I. N., Marina, N., Cardoso, B. V., et al. (2022). Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats. Molecular and Cellular Neuroscience. doi:10.1016/j.mcn.2022.103806
NLM
Sá RWM, Theparambil SM, Santos KM dos, Christie IN, Marina N, Cardoso BV, Hosford PS, Antunes VR. Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats [Internet]. Molecular and Cellular Neuroscience. 2022 ;[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mcn.2022.103806
Vancouver
Sá RWM, Theparambil SM, Santos KM dos, Christie IN, Marina N, Cardoso BV, Hosford PS, Antunes VR. Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats [Internet]. Molecular and Cellular Neuroscience. 2022 ;[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.mcn.2022.103806
Artigo de periodico
Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model (2022)
Ferreira, Ana Flávia Fernandes ; Singulani, Monique Patricio ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB, FM
Subjects: FISIOLOGIA, NEUROBIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, MESENCÉFALO, GÂNGLIOS, RECEPTORES DE DOPAMINA, CAMUNDONGOS, IMUNOHISTOQUÍMICA, IMMUNOBLOTTING
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, v. 59, p. 1543–1559, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02723-8. Acesso em: 10 jul. 2024.
APA
Ferreira, A. F. F., Singulani, M. P., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2022). Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, 59, 1543–1559. doi:10.1007/s12035-022-02723-8
NLM
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Vancouver
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Artigo de periodico
Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats (2022)
Moraes, Ruan Carlos Macêdo de ; Lima, Gabriely Cristina Alves; Cardinali, Camila Aparecida Errerias Fernandes ; Gonçalves, Alisson Carvalho de ; Portari, Guilherme Vannucchi ; Guerra-Shinohara, Elvira Maria ; Leboucher, Antoine ; Donato Junior, José ; Kleinridders, André ; Torrão, Andréa da Silva
Source: Life Sciences. Unidades: FCF, ICB, FM
Subjects: DOENÇA DE ALZHEIMER, HIPOTÁLAMO, FISIOLOGIA, VITAMINAS, DEMÊNCIA, INSULINA, DOENÇAS METABÓLICAS, METABOLISMO ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇAS NEURODEGENERATIVAS
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ABNT
MORAES, Ruan Carlos Macêdo de et al. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, v. 306, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120841. Acesso em: 10 jul. 2024.
APA
Moraes, R. C. M. de, Lima, G. C. A., Cardinali, C. A. E. F., Gonçalves, A. C. de, Portari, G. V., Guerra-Shinohara, E. M., et al. (2022). Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, 306, 1-13. doi:10.1016/j.lfs.2022.120841
NLM
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Vancouver
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Artigo de periodico
Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction (2021)
Fragas, Matheus Garcia ; Cândido, Vanessa Brito ; Davanzo, Gustavo Gastão ; Santos, Carla Rocha; Ceroni, Alexandre; Michelini, Lisete Compagno
Source: AJP: Regulatory, Integrative and Comparative Physiology. Unidade: ICB
Subjects: FISIOLOGIA, BARREIRA HEMATO-ENCEFÁLICA, EXERCÍCIO FÍSICO, HIPERTENSÃO, TREINAMENTO AERÓBIO, RATOS, MODELOS ANIMAIS DE DOENÇAS, HIPOTÁLAMO, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRAGAS, Matheus Garcia et al. Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction. AJP: Regulatory, Integrative and Comparative Physiology, v. 321, n. 5, p. R732–R741, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00154.2020. Acesso em: 10 jul. 2024.
APA
Fragas, M. G., Cândido, V. B., Davanzo, G. G., Santos, C. R., Ceroni, A., & Michelini, L. C. (2021). Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction. AJP: Regulatory, Integrative and Comparative Physiology, 321( 5), R732–R741. doi:10.1152/ajpregu.00154.2020
NLM
Fragas MG, Cândido VB, Davanzo GG, Santos CR, Ceroni A, Michelini LC. Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction [Internet]. AJP: Regulatory, Integrative and Comparative Physiology. 2021 ; 321( 5): R732–R741.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1152/ajpregu.00154.2020
Vancouver
Fragas MG, Cândido VB, Davanzo GG, Santos CR, Ceroni A, Michelini LC. Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction [Internet]. AJP: Regulatory, Integrative and Comparative Physiology. 2021 ; 321( 5): R732–R741.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1152/ajpregu.00154.2020
Artigo de periodico
Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice (2021)
Castro, Érique de ; Vieira, Thayna dos Santos ; Oliveira, Tiago E.; Silva, Milene Ortiz ; Andrade, Maynara Lucca; Tomazelli, Caroline Aparecida ; Peixoto, Albert Souza ; Sobrinho, Cleyton Roberto ; Moreno, Mayara Franzoi ; Gilio, Gustavo Renan ; Moreira, Rafael Junges ; Guimarães, Raphael Campos ; Perandini, Luiz Augusto Buoro ; Chimin, Patricia ; Reckziegel, Patricia ; Moretti, Eduardo Hermógenes ; Steiner, Alexandre Alarcon ; Laplante, Mathieu ; Festuccia, William Tadeu Lara
Source: AJP: Endocrinology and Metabolism. Unidade: ICB
Subjects: IMUNOLOGIA, FISIOLOGIA, FÁRMACOS IMUNOSSUPRESSORES, TECIDO ADIPOSO, METABOLISMO ENERGÉTICO, EXPRESSÃO GÊNICA, GLICOSE, CAMUNDONGOS, TERMOGÊNESE, GENES, GORDURAS
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ABNT
CASTRO, Érique de et al. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice. AJP: Endocrinology and Metabolism, v. 321, p. E592–E605, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00587.2020. Acesso em: 10 jul. 2024.
APA
Castro, É. de, Vieira, T. dos S., Oliveira, T. E., Silva, M. O., Andrade, M. L., Tomazelli, C. A., et al. (2021). Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice. AJP: Endocrinology and Metabolism, 321, E592–E605. doi:10.1152/ajpendo.00587.2020
NLM
Castro É de, Vieira T dos S, Oliveira TE, Silva MO, Andrade ML, Tomazelli CA, Peixoto AS, Sobrinho CR, Moreno MF, Gilio GR, Moreira RJ, Guimarães RC, Perandini LAB, Chimin P, Reckziegel P, Moretti EH, Steiner AA, Laplante M, Festuccia WTL. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice [Internet]. AJP: Endocrinology and Metabolism. 2021 ; 321 E592–E605.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1152/ajpendo.00587.2020
Vancouver
Castro É de, Vieira T dos S, Oliveira TE, Silva MO, Andrade ML, Tomazelli CA, Peixoto AS, Sobrinho CR, Moreno MF, Gilio GR, Moreira RJ, Guimarães RC, Perandini LAB, Chimin P, Reckziegel P, Moretti EH, Steiner AA, Laplante M, Festuccia WTL. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice [Internet]. AJP: Endocrinology and Metabolism. 2021 ; 321 E592–E605.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1152/ajpendo.00587.2020
Artigo de periodico
Pro-inflammatory interleukin-6 signaling links cognitive impairments and peripheral metabolic alterations in Alzheimer’s disease (2021)
Silva, Natalia M. Lyra e ; Gonçalves, Rafaella A.; Pascoal, Tharick A. ; Lima Filho, Ricardo A. S. ; Resende, Elisa de Paula França ; Vieira, Erica L. M.; Teixeira, Antonio L. ; Souza, Leonardo C. de ; Peny, Julyanna A.; Fortuna, Juliana T. S. ; Furigo, Isadora Clivatti ; Hashiguchi, Debora; Coreixas, Vivian S. Miya ; Clarke, Julia R.; Abisambra, José F. ; Longo, Beatriz M. ; Donato Junior, José ; Fraser, Paul E.; Rosa Neto, Pedro ; Caramelli, Paulo ; Ferreira, Sergio T. ; Felice, Fernanda G. De
Source: Translational Psychiatry. Unidade: ICB
Subjects: FISIOLOGIA, INTERLEUCINA 6, DOENÇA DE ALZHEIMER, METABOLISMO, MEMÓRIA PROCEDIMENTOS, RESSONÂNCIA MAGNÉTICA, HIPOTÁLAMO, PROCESSOS COGNITIVOS, ASTRÓCITOS, HIPOCAMPU DE ANIMAL, CAMUNDONGOS, MEMÓRIA ANIMAL
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ABNT
SILVA, Natalia M. Lyra e et al. Pro-inflammatory interleukin-6 signaling links cognitive impairments and peripheral metabolic alterations in Alzheimer’s disease. Translational Psychiatry, v. 11, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41398-021-01349-z. Acesso em: 10 jul. 2024.
APA
Silva, N. M. L. e, Gonçalves, R. A., Pascoal, T. A., Lima Filho, R. A. S., Resende, E. de P. F., Vieira, E. L. M., et al. (2021). Pro-inflammatory interleukin-6 signaling links cognitive impairments and peripheral metabolic alterations in Alzheimer’s disease. Translational Psychiatry, 11, 1-15. doi:10.1038/s41398-021-01349-z
NLM
Silva NML e, Gonçalves RA, Pascoal TA, Lima Filho RAS, Resende E de PF, Vieira ELM, Teixeira AL, Souza LC de, Peny JA, Fortuna JTS, Furigo IC, Hashiguchi D, Coreixas VSM, Clarke JR, Abisambra JF, Longo BM, Donato Junior J, Fraser PE, Rosa Neto P, Caramelli P, Ferreira ST, Felice FGD. Pro-inflammatory interleukin-6 signaling links cognitive impairments and peripheral metabolic alterations in Alzheimer’s disease [Internet]. Translational Psychiatry. 2021 ; 11 1-15.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1038/s41398-021-01349-z
Vancouver
Silva NML e, Gonçalves RA, Pascoal TA, Lima Filho RAS, Resende E de PF, Vieira ELM, Teixeira AL, Souza LC de, Peny JA, Fortuna JTS, Furigo IC, Hashiguchi D, Coreixas VSM, Clarke JR, Abisambra JF, Longo BM, Donato Junior J, Fraser PE, Rosa Neto P, Caramelli P, Ferreira ST, Felice FGD. Pro-inflammatory interleukin-6 signaling links cognitive impairments and peripheral metabolic alterations in Alzheimer’s disease [Internet]. Translational Psychiatry. 2021 ; 11 1-15.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1038/s41398-021-01349-z
Artigo de periodico
Fructose consumption by adult rats exposed to dexamethasone in utero changes the phenotype of intestinal epithelial cells and exacerbates intestinal gluconeogenesis (2020)
Zonta, Gizela Maria Agostini; Sodré, Frhancielly Shirley Souza ; Murata, Gilson Masahiro ; Amaral, Andressa Godoy; Payolla, Tanyara Baliani ; Campos, Carolina V.; Sato, Fabio Takeo ; Anhê, Gabriel Forato ; Bordin, Silvana
Source: Nutrients. Unidades: FM, ICB
Subjects: FISIOLOGIA, RATOS WISTAR, ROEDORES, LIPÍDEOS, GLICOGENOSE, HORMÔNIOS GLICOCORTICOIDES, ÚTERO, JEJUNO DE ANIMAL, FENÓTIPOS, INTESTINO DE ANIMAL, CÉLULAS EPITELIAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZONTA, Gizela Maria Agostini et al. Fructose consumption by adult rats exposed to dexamethasone in utero changes the phenotype of intestinal epithelial cells and exacerbates intestinal gluconeogenesis. Nutrients, v. 12, n. 10, p. 17 , 2020Tradução . . Disponível em: https://doi.org/10.3390/nu12103062. Acesso em: 10 jul. 2024.
APA
Zonta, G. M. A., Sodré, F. S. S., Murata, G. M., Amaral, A. G., Payolla, T. B., Campos, C. V., et al. (2020). Fructose consumption by adult rats exposed to dexamethasone in utero changes the phenotype of intestinal epithelial cells and exacerbates intestinal gluconeogenesis. Nutrients, 12( 10), 17 . doi:10.3390/nu12103062
NLM
Zonta GMA, Sodré FSS, Murata GM, Amaral AG, Payolla TB, Campos CV, Sato FT, Anhê GF, Bordin S. Fructose consumption by adult rats exposed to dexamethasone in utero changes the phenotype of intestinal epithelial cells and exacerbates intestinal gluconeogenesis [Internet]. Nutrients. 2020 ; 12( 10): 17 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.3390/nu12103062
Vancouver
Zonta GMA, Sodré FSS, Murata GM, Amaral AG, Payolla TB, Campos CV, Sato FT, Anhê GF, Bordin S. Fructose consumption by adult rats exposed to dexamethasone in utero changes the phenotype of intestinal epithelial cells and exacerbates intestinal gluconeogenesis [Internet]. Nutrients. 2020 ; 12( 10): 17 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.3390/nu12103062
Artigo de periodico
Role of microRNAs on the regulation of mitochondrial biogenesis and insulin signalingin skeletal muscle (2017)
Lima, Tanes Iamamura ; Araujo, Hygor Nunes ; Menezes, Eveline Soares ; Sponton, Carlos Henrique Grossi; Araújo, Michel Barbosa de; Bonfim, Lucas Henrique Montes; Queiroz, André Lima ; Passos, Magla Adami dos; Sousa, Thais Amaral e; Hirabara, Sandro Massao ; Martins, Amanda Roque ; Sampaio, Helena Cristina de Lima Barbosa ; Rodrigues, Alice Cristina ; Curi, Rui ; Carneiro, Everardo Magalhães ; Boschero, Antonio Carlos ; Silveira, Leonardo dos Reis
Source: Journal of Cellular Physiology. Unidades: ICB, FMRP
Subjects: FISIOLOGIA, HOMEOSTASE, DOENÇAS METABÓLICAS, RNA MENSAGEIRO, DIABETES MELLITUS, MÚSCULO ESQUELÉTICO, MITOCÔNDRIAS, INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Tanes Iamamura et al. Role of microRNAs on the regulation of mitochondrial biogenesis and insulin signalingin skeletal muscle. Journal of Cellular Physiology, v. 232, n. 5, p. 958-966, 2017Tradução . . Disponível em: https://doi.org/10.1002/jcp.25645. Acesso em: 10 jul. 2024.
APA
Lima, T. I., Araujo, H. N., Menezes, E. S., Sponton, C. H. G., Araújo, M. B. de, Bonfim, L. H. M., et al. (2017). Role of microRNAs on the regulation of mitochondrial biogenesis and insulin signalingin skeletal muscle. Journal of Cellular Physiology, 232( 5), 958-966. doi:10.1002/jcp.25645
NLM
Lima TI, Araujo HN, Menezes ES, Sponton CHG, Araújo MB de, Bonfim LHM, Queiroz AL, Passos MA dos, Sousa TA e, Hirabara SM, Martins AR, Sampaio HC de LB, Rodrigues AC, Curi R, Carneiro EM, Boschero AC, Silveira L dos R. Role of microRNAs on the regulation of mitochondrial biogenesis and insulin signalingin skeletal muscle [Internet]. Journal of Cellular Physiology. 2017 ; 232( 5): 958-966.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1002/jcp.25645
Vancouver
Lima TI, Araujo HN, Menezes ES, Sponton CHG, Araújo MB de, Bonfim LHM, Queiroz AL, Passos MA dos, Sousa TA e, Hirabara SM, Martins AR, Sampaio HC de LB, Rodrigues AC, Curi R, Carneiro EM, Boschero AC, Silveira L dos R. Role of microRNAs on the regulation of mitochondrial biogenesis and insulin signalingin skeletal muscle [Internet]. Journal of Cellular Physiology. 2017 ; 232( 5): 958-966.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1002/jcp.25645

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