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Artigo de periodico
NLRP3 activation contributes to endothelin-1-induced erectile dysfunction (2023)
Fais, Rafael Sobrano; Costa, Rafael Menezes da ; Mendes, Allan Carvalho; Mestriner, Fabiola Leslie Antunes Cardoso; Comerma-Steffensen, Simón Gabriel; Tostes, Rita de Cássia Aleixo ; Simonsen, Ulf; Carneiro, Fernando Silva
Fonte: Journal of Cellular and Molecular Medicine. Unidade: FMRP
Assuntos: ANTAGONISTAS, ENDOTELINAS, DISFUNÇÃO ERÉTIL, ESPÉCIES REATIVAS DE OXIGÊNIO, INFLAMAÇÃO
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ABNT
FAIS, Rafael Sobrano et al. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, v. 27, n. 1, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1111/jcmm.17463. Acesso em: 04 nov. 2024.
APA
Fais, R. S., Costa, R. M. da, Mendes, A. C., Mestriner, F. L. A. C., Comerma-Steffensen, S. G., Tostes, R. de C. A., et al. (2023). NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, 27( 1), 1-14. doi:10.1111/jcmm.17463
NLM
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1111/jcmm.17463
Vancouver
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1111/jcmm.17463
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Fonte: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Assuntos: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
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SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 04 nov. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Fonte: Frontiers in Physiology. Unidade: FMRP
Assuntos: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
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COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 04 nov. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood (2022)
Vidigal, Camila B.; Moura, Kawane F.; Costa, Tiago Januário da ; Borges, Lorena I.; Figaro, Pedro M. M.; Pinto, Ingrid C.; Andrade, Fábio G. de; Gerardin, Daniela C. C.; Casagrande, Rúbia ; Tostes, Rita de Cássia Aleixo ; Franco, Maria do Carmo ; Ceravolo, Graziela S.
Fonte: Life Sciences. Unidade: FMRP
Assuntos: ANTICONVULSIVANTES, AORTA, REGULAÇÃO GÊNICA, PROSTAGLANDINA SINTASE, FATORES SEXUAIS, DOENÇAS VASCULARES
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VIDIGAL, Camila B. et al. Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood. Life Sciences, v. 288, p. 1-7, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.120189. Acesso em: 04 nov. 2024.
APA
Vidigal, C. B., Moura, K. F., Costa, T. J. da, Borges, L. I., Figaro, P. M. M., Pinto, I. C., et al. (2022). Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood. Life Sciences, 288, 1-7. doi:10.1016/j.lfs.2021.120189
NLM
Vidigal CB, Moura KF, Costa TJ da, Borges LI, Figaro PMM, Pinto IC, Andrade FG de, Gerardin DCC, Casagrande R, Tostes R de CA, Franco M do C, Ceravolo GS. Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood [Internet]. Life Sciences. 2022 ; 288 1-7.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2021.120189
Vancouver
Vidigal CB, Moura KF, Costa TJ da, Borges LI, Figaro PMM, Pinto IC, Andrade FG de, Gerardin DCC, Casagrande R, Tostes R de CA, Franco M do C, Ceravolo GS. Topiramate treatment in Wistar rats during childhood induces sex-specific vascular dysfunction in adulthood [Internet]. Life Sciences. 2022 ; 288 1-7.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2021.120189
Artigo de periodico
O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system (2022)
Passos Junior, Rinaldo Rodrigues dos; Bomfim, Gisele Facholi ; Giachini, Fernanda R.; Tostes, Rita de Cássia Aleixo ; Lima, Victor Vitorino
Fonte: Frontiers in Immunology. Unidade: FMRP
Assuntos: HIPERTENSÃO, SISTEMA IMUNE, PROTEÍNAS
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PASSOS JUNIOR, Rinaldo Rodrigues dos et al. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system. Frontiers in Immunology, v. 13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.852115. Acesso em: 04 nov. 2024.
APA
Passos Junior, R. R. dos, Bomfim, G. F., Giachini, F. R., Tostes, R. de C. A., & Lima, V. V. (2022). O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system. Frontiers in Immunology, 13. doi:10.3389/fimmu.2022.852115
NLM
Passos Junior RR dos, Bomfim GF, Giachini FR, Tostes R de CA, Lima VV. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system [Internet]. Frontiers in Immunology. 2022 ; 13[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fimmu.2022.852115
Vancouver
Passos Junior RR dos, Bomfim GF, Giachini FR, Tostes R de CA, Lima VV. O-Linked β-N-acetylglucosamine modification: linking hypertension and the immune system [Internet]. Frontiers in Immunology. 2022 ; 13[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fimmu.2022.852115
Artigo de periodico
Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats (2022)
Brazão, Vânia; Colato, Rafaela Pravato ; Santello, Fabricia Helena; Duarte, Andressa ; Goulart, Amanda; Oliveira, Pedro Alexandre Sampaio ; Silva, Carla Brigagão Pacheco da ; Tirapelli, Carlos Renato ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo ; Prado Júnior, José Clóvis do
Fonte: Experimental Gerontology. Unidades: EERP, FMRP, FCFRP
Assuntos: ANTIOXIDANTES, DOENÇA DE CHAGAS, PERÓXIDO DE HIDROGÊNIO, MELATONINA, ESTRESSE OXIDATIVO
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BRAZÃO, Vânia et al. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, v. 167, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2022.111895. Acesso em: 04 nov. 2024.
APA
Brazão, V., Colato, R. P., Santello, F. H., Duarte, A., Goulart, A., Oliveira, P. A. S., et al. (2022). Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, 167, 1-9. doi:10.1016/j.exger.2022.111895
NLM
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Vancouver
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Fonte: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Assuntos: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
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COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 04 nov. 2024.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Artigo de periodico
Programming of vascular dysfunction by maternal stress: immune system implications (2022)
Costa, Tiago Januário da ; Oliveira, Julio Cezar de; Giachini, Fernanda Regina; Lima, Victor Vitorino; Tostes, Rita de Cássia Aleixo ; Bomfim, Gisele Facholi
Fonte: Frontiers in Physiology. Unidade: FMRP
Assuntos: SISTEMA IMUNE, ESPÉCIES REATIVAS DE OXIGÊNIO, ESTRESSE, SISTEMA RENINA-ANGIOTENSINA
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ABNT
COSTA, Tiago Januário da et al. Programming of vascular dysfunction by maternal stress: immune system implications. Frontiers in Physiology, v. 13, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.787617. Acesso em: 04 nov. 2024.
APA
Costa, T. J. da, Oliveira, J. C. de, Giachini, F. R., Lima, V. V., Tostes, R. de C. A., & Bomfim, G. F. (2022). Programming of vascular dysfunction by maternal stress: immune system implications. Frontiers in Physiology, 13, 1-17. doi:10.3389/fphys.2022.787617
NLM
Costa TJ da, Oliveira JC de, Giachini FR, Lima VV, Tostes R de CA, Bomfim GF. Programming of vascular dysfunction by maternal stress: immune system implications [Internet]. Frontiers in Physiology. 2022 ; 13 1-17.[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2022.787617
Vancouver
Costa TJ da, Oliveira JC de, Giachini FR, Lima VV, Tostes R de CA, Bomfim GF. Programming of vascular dysfunction by maternal stress: immune system implications [Internet]. Frontiers in Physiology. 2022 ; 13 1-17.[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2022.787617
Artigo de periodico
Vascular stress signaling in hypertension (2021)
Cicalese, Stephanie M.; Silva, Josiane Fernandes da; Priviero, Fernanda; Webb, R. Clinton; Eguchi, Satoru; Tostes, Rita de Cássia Aleixo
Fonte: Circulation Research. Unidade: FMRP
Assuntos: RETÍCULO ENDOPLASMÁTICO, SOBREVIVÊNCIA CELULAR, HIPERTENSÃO, INFLAMAÇÃO, MITOCÔNDRIAS
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ABNT
CICALESE, Stephanie M. et al. Vascular stress signaling in hypertension. Circulation Research, v. 128, n. 7, p. 969-992, 2021Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.121.318053. Acesso em: 04 nov. 2024.
APA
Cicalese, S. M., Silva, J. F. da, Priviero, F., Webb, R. C., Eguchi, S., & Tostes, R. de C. A. (2021). Vascular stress signaling in hypertension. Circulation Research, 128( 7), 969-992. doi:10.1161/CIRCRESAHA.121.318053
NLM
Cicalese SM, Silva JF da, Priviero F, Webb RC, Eguchi S, Tostes R de CA. Vascular stress signaling in hypertension [Internet]. Circulation Research. 2021 ; 128( 7): 969-992.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1161/CIRCRESAHA.121.318053
Vancouver
Cicalese SM, Silva JF da, Priviero F, Webb RC, Eguchi S, Tostes R de CA. Vascular stress signaling in hypertension [Internet]. Circulation Research. 2021 ; 128( 7): 969-992.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1161/CIRCRESAHA.121.318053
Artigo de periodico
Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway (2019)
Amaral, Jefferson Henrich; Rizzi, Elen S.; Alves-Lopes, Rhéure; Pinheiro, Lucas Cézar ; Tostes, Rita de Cássia Aleixo ; Tanus-Santos, José Eduardo
Fonte: Free Radical Biology and Medicine. Unidades: EERP, FMRP
Assuntos: ANTIOXIDANTES, NITRITOS, HIPERTENSÃO
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ABNT
AMARAL, Jefferson Henrich et al. Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway. Free Radical Biology and Medicine, v. 141, p. 261-268, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2019.06.028. Acesso em: 04 nov. 2024.
APA
Amaral, J. H., Rizzi, E. S., Alves-Lopes, R., Pinheiro, L. C., Tostes, R. de C. A., & Tanus-Santos, J. E. (2019). Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway. Free Radical Biology and Medicine, 141, 261-268. doi:10.1016/j.freeradbiomed.2019.06.028
NLM
Amaral JH, Rizzi ES, Alves-Lopes R, Pinheiro LC, Tostes R de CA, Tanus-Santos JE. Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway [Internet]. Free Radical Biology and Medicine. 2019 ; 141 261-268.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.06.028
Vancouver
Amaral JH, Rizzi ES, Alves-Lopes R, Pinheiro LC, Tostes R de CA, Tanus-Santos JE. Antioxidant and antihypertensive responses to oral nitrite involves activation of the Nrf2 pathway [Internet]. Free Radical Biology and Medicine. 2019 ; 141 261-268.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.06.028
Artigo de periodico
Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats (2019)
Lataro, Renata M.; Silva, Marcondes Alves Barbosa da; Mestriner, Fabiola Leslie Antunes Cardoso; Cáu, Stêfany Bruno de Assis; Tostes, Rita de Cássia Aleixo ; Salgado, Hélio César
Fonte: American Journal of Hypertension. Unidade: FMRP
Assuntos: HIPERTENSÃO, PRESSÃO SANGUÍNEA, RATOS
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ABNT
LATARO, Renata M. et al. Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats. American Journal of Hypertension, v. 32, n. 6, p. 579-587, 2019Tradução . . Disponível em: https://doi.org/10.1093/ajh/hpz036. Acesso em: 04 nov. 2024.
APA
Lataro, R. M., Silva, M. A. B. da, Mestriner, F. L. A. C., Cáu, S. B. de A., Tostes, R. de C. A., & Salgado, H. C. (2019). Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats. American Journal of Hypertension, 32( 6), 579-587. doi:10.1093/ajh/hpz036
NLM
Lataro RM, Silva MAB da, Mestriner FLAC, Cáu SB de A, Tostes R de CA, Salgado HC. Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats [Internet]. American Journal of Hypertension. 2019 ; 32( 6): 579-587.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/ajh/hpz036
Vancouver
Lataro RM, Silva MAB da, Mestriner FLAC, Cáu SB de A, Tostes R de CA, Salgado HC. Chronic treatment with acetylcholinesterase inhibitors attenuates vascular dysfunction in spontaneously hypertensive rats [Internet]. American Journal of Hypertension. 2019 ; 32( 6): 579-587.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/ajh/hpz036
Artigo de periodico
Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice (2018)
Neves, Karla Bianca; Montezano, Augusto Cesar; Alves-Lopes, Rheure; Bruder-Nascimento, Thiago; Costa, Rafael Menezes; Costa, Roberto Silva; Touyz, Rhian M.; Passaglia, Rita de Cássia Aleixo Tostes
Fonte: International Journal of Molecular Sciences. Unidade: FMRP
Assuntos: RIM, DIABETES MELLITUS, ESTRESSE OXIDATIVO, RATOS
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ABNT
NEVES, Karla Bianca et al. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice. International Journal of Molecular Sciences, v. 19, n. 8, 2018Tradução . . Disponível em: https://doi.org/10.3390/ijms19082454. Acesso em: 04 nov. 2024.
APA
Neves, K. B., Montezano, A. C., Alves-Lopes, R., Bruder-Nascimento, T., Costa, R. M., Costa, R. S., et al. (2018). Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice. International Journal of Molecular Sciences, 19( 8). doi:10.3390/ijms19082454
NLM
Neves KB, Montezano AC, Alves-Lopes R, Bruder-Nascimento T, Costa RM, Costa RS, Touyz RM, Passaglia R de CAT. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 8):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/ijms19082454
Vancouver
Neves KB, Montezano AC, Alves-Lopes R, Bruder-Nascimento T, Costa RM, Costa RS, Touyz RM, Passaglia R de CAT. Upregulation of Nrf2 and decreased redox signaling contribute to renoprotective effects of chemerin receptor blockade in diabetic mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 8):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/ijms19082454
Artigo de periodico
Increased O-glcNAcylation of endothelial nitric oxide synthase compromises the anti-contractile properties of perivascular adipose tissue in metabolic syndrome (2018)
Costa, Rafael M. da; Silva, Josiane F. da; Alves, Juliano V.; Dias, Thiago B.; Rassi, Diane M.; Garcia, Luis Vicente; Lobato, Núbia de Souza; Passaglia, Rita de Cássia Aleixo Tostes
Fonte: Frontiers in Physiology. Unidade: FMRP
Assuntos: TECIDO ADIPOSO, SÍNDROME X METABÓLICA, OBESIDADE, AÇÚCAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael M. da et al. Increased O-glcNAcylation of endothelial nitric oxide synthase compromises the anti-contractile properties of perivascular adipose tissue in metabolic syndrome. Frontiers in Physiology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fphys.2018.00341. Acesso em: 04 nov. 2024.
APA
Costa, R. M. da, Silva, J. F. da, Alves, J. V., Dias, T. B., Rassi, D. M., Garcia, L. V., et al. (2018). Increased O-glcNAcylation of endothelial nitric oxide synthase compromises the anti-contractile properties of perivascular adipose tissue in metabolic syndrome. Frontiers in Physiology, 9. doi:10.3389/fphys.2018.00341
NLM
Costa RM da, Silva JF da, Alves JV, Dias TB, Rassi DM, Garcia LV, Lobato N de S, Passaglia R de CAT. Increased O-glcNAcylation of endothelial nitric oxide synthase compromises the anti-contractile properties of perivascular adipose tissue in metabolic syndrome [Internet]. Frontiers in Physiology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2018.00341
Vancouver
Costa RM da, Silva JF da, Alves JV, Dias TB, Rassi DM, Garcia LV, Lobato N de S, Passaglia R de CAT. Increased O-glcNAcylation of endothelial nitric oxide synthase compromises the anti-contractile properties of perivascular adipose tissue in metabolic syndrome [Internet]. Frontiers in Physiology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2018.00341
Artigo de periodico
Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity (2018)
Costa, Rafael M.; Neves, Karla B.; Passaglia, Rita de Cássia Aleixo Tostes; Lobato, Núbia S.
Fonte: Frontiers in Physiology. Unidade: FMRP
Assuntos: TECIDO ADIPOSO, OBESIDADE, SISTEMA CARDIOVASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael M. et al. Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity. Frontiers in Physiology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fphys.2018.00253. Acesso em: 04 nov. 2024.
APA
Costa, R. M., Neves, K. B., Passaglia, R. de C. A. T., & Lobato, N. S. (2018). Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity. Frontiers in Physiology, 9. doi:10.3389/fphys.2018.00253
NLM
Costa RM, Neves KB, Passaglia R de CAT, Lobato NS. Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity [Internet]. Frontiers in Physiology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2018.00253
Vancouver
Costa RM, Neves KB, Passaglia R de CAT, Lobato NS. Perivascular adipose tissue as a relevant fat depot for cardiovascular risk in obesity [Internet]. Frontiers in Physiology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphys.2018.00253
Artigo de periodico
O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway (2018)
Miguez, Jéssica S. G.; Dela Justina, Vanessa; Bressan, Alecsander F. M.; Marchi, Patrícia G. F.; França, Adenilda C. Honorio; Carneiro, Fernando Silva; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes; Giachini, Fernanda R.; Lima, Victor V.
Fonte: Life Sciences. Unidade: FMRP
Assuntos: CITOCINAS, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MIGUEZ, Jéssica S. G. et al. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, v. 209, p. 78-84, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.07.058. Acesso em: 04 nov. 2024.
APA
Miguez, J. S. G., Dela Justina, V., Bressan, A. F. M., Marchi, P. G. F., França, A. C. H., Carneiro, F. S., et al. (2018). O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, 209, 78-84. doi:10.1016/j.lfs.2018.07.058
NLM
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Vancouver
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Artigo de periodico
O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels (2018)
Dela Justina, Vanessa; Passos Junior, Rinaldo R. dos; Bressan, Alecsander F.; Passaglia, Rita de Cássia Aleixo Tostes; Carneiro, Fernando Silva; Soares, Thaigra S.; Volpato, Gustavo T.; Lima, Victor Vitorino; Martin, Sebastian San; Giachini, Fernanda R.
Fonte: Life Sciences. Unidade: FMRP
Assuntos: GRAVIDEZ, HIPERGLICEMIA, TROFOBLASTO, CÉLULAS ENDOTELIAIS, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DELA JUSTINA, Vanessa et al. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, v. 205, p. 18-25, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.05.013. Acesso em: 04 nov. 2024.
APA
Dela Justina, V., Passos Junior, R. R. dos, Bressan, A. F., Passaglia, R. de C. A. T., Carneiro, F. S., Soares, T. S., et al. (2018). O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, 205, 18-25. doi:10.1016/j.lfs.2018.05.013
NLM
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Vancouver
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Artigo de periodico
Caryocar brasiliense induces vasorelaxation through endothelial Ca2+/calmodulin and PI3K/Akt/eNOS-dependent signaling pathways in rats (2018)
Oliveira, Lais Moraes de; Oliveira, Thiago Sardinha de; Costa, Rafael Menezes da; Martins, José Luís Rodrigues; Freitas, Carla Santos de; Gil, Eric de Souza; Costa, Elson Alves; Passaglia, Rita de Cassia Aleixo Tostes ; Vaz, Boniek Gontijo; Filgueira, Fernando Paranaiba; Ghedini, Paulo César
Fonte: Revista Brasileira de Farmacognosia. Unidade: FMRP
Assuntos: PEQUI, CALMODULINA, COMPOSTOS FENÓLICOS, ENDOTÉLIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Lais Moraes de et al. Caryocar brasiliense induces vasorelaxation through endothelial Ca2+/calmodulin and PI3K/Akt/eNOS-dependent signaling pathways in rats. Revista Brasileira de Farmacognosia, v. 28, n. 6, p. 678-685, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bjp.2018.07.007. Acesso em: 04 nov. 2024.
APA
Oliveira, L. M. de, Oliveira, T. S. de, Costa, R. M. da, Martins, J. L. R., Freitas, C. S. de, Gil, E. de S., et al. (2018). Caryocar brasiliense induces vasorelaxation through endothelial Ca2+/calmodulin and PI3K/Akt/eNOS-dependent signaling pathways in rats. Revista Brasileira de Farmacognosia, 28( 6), 678-685. doi:10.1016/j.bjp.2018.07.007
NLM
Oliveira LM de, Oliveira TS de, Costa RM da, Martins JLR, Freitas CS de, Gil E de S, Costa EA, Passaglia R de CAT, Vaz BG, Filgueira FP, Ghedini PC. Caryocar brasiliense induces vasorelaxation through endothelial Ca2+/calmodulin and PI3K/Akt/eNOS-dependent signaling pathways in rats [Internet]. Revista Brasileira de Farmacognosia. 2018 ; 28( 6): 678-685.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.bjp.2018.07.007
Vancouver
Oliveira LM de, Oliveira TS de, Costa RM da, Martins JLR, Freitas CS de, Gil E de S, Costa EA, Passaglia R de CAT, Vaz BG, Filgueira FP, Ghedini PC. Caryocar brasiliense induces vasorelaxation through endothelial Ca2+/calmodulin and PI3K/Akt/eNOS-dependent signaling pathways in rats [Internet]. Revista Brasileira de Farmacognosia. 2018 ; 28( 6): 678-685.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.bjp.2018.07.007
Artigo de periodico
Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion (2018)
Omoto, Ana Carolina Mieko; Gava, Fábio Nelson; Silva, Carlos Alberto Aguiar da; Silva, Hadder Batista; Parente, Juliana Montenegro; Costa, Rafael Menezes; Castro, Michele Mazzaron de; Passaglia, Rita de Cássia Aleixo Tostes; Salgado, Hélio César; Fazan Júnior, Rubens
Fonte: PLOS ONE. Unidade: FMRP
Assuntos: REPERFUSÃO, PROCEDIMENTOS CIRÚRGICOS CARDÍACOS, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OMOTO, Ana Carolina Mieko et al. Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion. PLOS ONE, v. 13, n. 12, p. e0209190, 2018Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0209190. Acesso em: 04 nov. 2024.
APA
Omoto, A. C. M., Gava, F. N., Silva, C. A. A. da, Silva, H. B., Parente, J. M., Costa, R. M., et al. (2018). Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion. PLOS ONE, 13( 12), e0209190. doi:10.1371/journal.pone.0209190
NLM
Omoto ACM, Gava FN, Silva CAA da, Silva HB, Parente JM, Costa RM, Castro MM de, Passaglia R de CAT, Salgado HC, Fazan Júnior R. Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion [Internet]. PLOS ONE. 2018 ; 13( 12): e0209190.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1371/journal.pone.0209190
Vancouver
Omoto ACM, Gava FN, Silva CAA da, Silva HB, Parente JM, Costa RM, Castro MM de, Passaglia R de CAT, Salgado HC, Fazan Júnior R. Lack of scarring is not always a sign of cardiac health: functional and molecular characterization of the rat heart’s following chronic reperfusion [Internet]. PLOS ONE. 2018 ; 13( 12): e0209190.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1371/journal.pone.0209190
Artigo de periodico
Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats (2017)
Justina, Vanessa Dela; Gonçalves, Jéssica S.; Freitas, Raiany Alves de; Fonseca, Adriany Dias; Volpato, Gustavo T.; Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Giachini, Fernanda R.
Fonte: Inflammation. Unidade: FMRP
Assuntos: CITOCINAS, HIPERGLICEMIA, PLACENTA, COMPLICAÇÕES NA GRAVIDEZ
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JUSTINA, Vanessa Dela et al. Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats. Inflammation, v. 40, n. 5, p. 1773-1781, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10753-017-0620-7. Acesso em: 04 nov. 2024.
APA
Justina, V. D., Gonçalves, J. S., Freitas, R. A. de, Fonseca, A. D., Volpato, G. T., Tostes, R. de C. A., et al. (2017). Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats. Inflammation, 40( 5), 1773-1781. doi:10.1007/s10753-017-0620-7
NLM
Justina VD, Gonçalves JS, Freitas RA de, Fonseca AD, Volpato GT, Tostes R de CA, Carneiro FS, Giachini FR. Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats [Internet]. Inflammation. 2017 ; 40( 5): 1773-1781.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1007/s10753-017-0620-7
Vancouver
Justina VD, Gonçalves JS, Freitas RA de, Fonseca AD, Volpato GT, Tostes R de CA, Carneiro FS, Giachini FR. Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats [Internet]. Inflammation. 2017 ; 40( 5): 1773-1781.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1007/s10753-017-0620-7
Artigo de periodico
Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases (2016)
Gohar, Eman Y; Giachini, Fernanda R; Pollock, David M; Tostes, Rita de Cássia Aleixo
Fonte: Life Sciences. Unidade: FMRP
Assuntos: DOENÇAS CARDIOVASCULARES, NEFROPATIAS, ENDOTELINAS, DIFERENÇAS SEXUAIS (PSICOGÊNESE), CROMOSSOMOS SEXUAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOHAR, Eman Y et al. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life Sciences, v. 159, p. 20-29, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.02.093. Acesso em: 04 nov. 2024.
APA
Gohar, E. Y., Giachini, F. R., Pollock, D. M., & Tostes, R. de C. A. (2016). Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life Sciences, 159, 20-29. doi:10.1016/j.lfs.2016.02.093
NLM
Gohar EY, Giachini FR, Pollock DM, Tostes R de CA. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases [Internet]. Life Sciences. 2016 ; 159 20-29.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.093
Vancouver
Gohar EY, Giachini FR, Pollock DM, Tostes R de CA. Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases [Internet]. Life Sciences. 2016 ; 159 20-29.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.093

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