Filtros : "ANATOMIA" "Molecular and Cellular Endocrinology" Removidos: "PTR" "1929" Limpar

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  • Source: Molecular and Cellular Endocrinology. Unidades: ICB, FM

    Subjects: FISIOLOGIA, ANATOMIA, ESTRÓGENOS, ESTEROIDES, HIPOTÁLAMO, NEURÔNIOS, GONADOTROFINAS, PEPTÍDEO INTESTINAL VASOATIVO, NEUROTRANSMISSORES, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS

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      MANSANO, Naira da Silva et al. Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback. Molecular and Cellular Endocrinology, v. 542, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2021.111532. Acesso em: 27 jun. 2024.
    • APA

      Mansano, N. da S., Paradela, R. S., Bohlen, T. M., Zanardi, I. M. da S., Chaves, F. M., Silveira, M. A., et al. (2022). Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback. Molecular and Cellular Endocrinology, 542, 1-10. doi:10.1016/j.mce.2021.111532
    • NLM

      Mansano N da S, Paradela RS, Bohlen TM, Zanardi IM da S, Chaves FM, Silveira MA, Tavares MR, Donato Junior J, Frazão R. Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback [Internet]. Molecular and Cellular Endocrinology. 2022 ; 542 1-10.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2021.111532
    • Vancouver

      Mansano N da S, Paradela RS, Bohlen TM, Zanardi IM da S, Chaves FM, Silveira MA, Tavares MR, Donato Junior J, Frazão R. Vasoactive intestinal peptide exerts an excitatory effect on hypothalamic kisspeptin neurons during estrogen negative feedback [Internet]. Molecular and Cellular Endocrinology. 2022 ; 542 1-10.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2021.111532
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, DOENÇAS CARDIOVASCULARES, HORMÔNIOS TIREOIDIANOS, INSULINA, SISTEMA RENINA-ANGIOTENSINA, CATECOLAMINAS, INSUFICIÊNCIA CARDÍACA

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      TAKANO, Ana Paula Cremasco e SENGER, Nathalia e BARRETO-CHAVES, Maria Luiza Morais. The endocrinological component and signaling pathways associated to cardiac hypertrophy. Molecular and Cellular Endocrinology, v. 30, p. 13 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2020.110972. Acesso em: 27 jun. 2024.
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      Takano, A. P. C., Senger, N., & Barreto-Chaves, M. L. M. (2020). The endocrinological component and signaling pathways associated to cardiac hypertrophy. Molecular and Cellular Endocrinology, 30, 13 . doi:10.1016/j.mce.2020.110972
    • NLM

      Takano APC, Senger N, Barreto-Chaves MLM. The endocrinological component and signaling pathways associated to cardiac hypertrophy [Internet]. Molecular and Cellular Endocrinology. 2020 ; 30 13 .[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2020.110972
    • Vancouver

      Takano APC, Senger N, Barreto-Chaves MLM. The endocrinological component and signaling pathways associated to cardiac hypertrophy [Internet]. Molecular and Cellular Endocrinology. 2020 ; 30 13 .[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2020.110972
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, METABOLISMO DE PROTEÍNA, HORMÔNIOS TIREOIDIANOS, CORAÇÃO

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      LINO, Caroline Antunes e DEMASI, Marilene e BARRETO-CHAVES, Maria Luiza Morais. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, v. 493, p. 1-9, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110451. Acesso em: 27 jun. 2024.
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      Lino, C. A., Demasi, M., & Barreto-Chaves, M. L. M. (2019). Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, 493, 1-9. doi:10.1016/j.mce.2019.110451
    • NLM

      Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
    • Vancouver

      Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA, METABOLISMO ENERGÉTICO, HIPOGLICEMIA

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      QUARESMA, Paula Gabriele Fernandes et al. Growth hormone/STAT5 signaling in proopiomelanocortin neurons regulates glucoprivic hyperphagia. Molecular and Cellular Endocrinology, v. 498, p. 1-14, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110574. Acesso em: 27 jun. 2024.
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      Quaresma, P. G. F., Teixeira, P. D. S., Furigo, I. C., Wasinski, F., Couto, G. C. L., Frazão, R., et al. (2019). Growth hormone/STAT5 signaling in proopiomelanocortin neurons regulates glucoprivic hyperphagia. Molecular and Cellular Endocrinology, 498, 1-14. doi:10.1016/j.mce.2019.110574
    • NLM

      Quaresma PGF, Teixeira PDS, Furigo IC, Wasinski F, Couto GCL, Frazão R, List EO, Kopchick JJ, Donato Junior J. Growth hormone/STAT5 signaling in proopiomelanocortin neurons regulates glucoprivic hyperphagia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-14.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2019.110574
    • Vancouver

      Quaresma PGF, Teixeira PDS, Furigo IC, Wasinski F, Couto GCL, Frazão R, List EO, Kopchick JJ, Donato Junior J. Growth hormone/STAT5 signaling in proopiomelanocortin neurons regulates glucoprivic hyperphagia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-14.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2019.110574
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA, ANGIOTENSINA II, METABOLISMO, OBESIDADE, GLICOSE

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      LIMA, Vanessa Morais et al. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, v. 498, p. 1-10, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110576. Acesso em: 27 jun. 2024.
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      Lima, V. M., Lino, C. A., Senger, N., Silva, T. de O., Fonseca, R. I. B., Bader, M., et al. (2019). Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, 498, 1-10. doi:10.1016/j.mce.2019.110576
    • NLM

      Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
    • Vancouver

      Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
  • Source: Molecular and Cellular Endocrinology. Unidades: FMRP, ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      SILVEIRA, Marina Augusto et al. STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, v. 448, p. 55-65, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.03.024. Acesso em: 27 jun. 2024.
    • APA

      Silveira, M. A., Furigo, I. C., Zampieri, T. T., Bohlen, T. M., Paula, D. G. de, Franci, C. R., et al. (2017). STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, 448, 55-65. doi:10.1016/j.mce.2017.03.024
    • NLM

      Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
    • Vancouver

      Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      FURIGO, Isadora C. et al. Brain STAT5 signaling and behavioral control. Molecular and Cellular Endocrinology, v. 438, p. 70-76, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.04.019. Acesso em: 27 jun. 2024.
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      Furigo, I. C., Ramos-Lobo, A. M., Frazão, R., & Donato Junior, J. (2016). Brain STAT5 signaling and behavioral control. Molecular and Cellular Endocrinology, 438, 70-76. doi:10.1016/j.mce.2016.04.019
    • NLM

      Furigo IC, Ramos-Lobo AM, Frazão R, Donato Junior J. Brain STAT5 signaling and behavioral control [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 70-76.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2016.04.019
    • Vancouver

      Furigo IC, Ramos-Lobo AM, Frazão R, Donato Junior J. Brain STAT5 signaling and behavioral control [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 70-76.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2016.04.019
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      BOHLEN, Tabata M. et al. Fatness rather than leptin sensitivity determines the timing of puberty in female mice. Molecular and Cellular Endocrinology, v. 423, p. 11-21, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.12.022. Acesso em: 27 jun. 2024.
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      Bohlen, T. M., Silveira, M. A., Zampieri, T. T., Frazão, R., & Donato Junior, J. (2016). Fatness rather than leptin sensitivity determines the timing of puberty in female mice. Molecular and Cellular Endocrinology, 423, 11-21. doi:10.1016/j.mce.2015.12.022
    • NLM

      Bohlen TM, Silveira MA, Zampieri TT, Frazão R, Donato Junior J. Fatness rather than leptin sensitivity determines the timing of puberty in female mice [Internet]. Molecular and Cellular Endocrinology. 2016 ; 423 11-21.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2015.12.022
    • Vancouver

      Bohlen TM, Silveira MA, Zampieri TT, Frazão R, Donato Junior J. Fatness rather than leptin sensitivity determines the timing of puberty in female mice [Internet]. Molecular and Cellular Endocrinology. 2016 ; 423 11-21.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2015.12.022
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: ANATOMIA, HIPERTIREOIDISMO, RATOS WISTAR, DOENÇAS CARDIOVASCULARES EM ANIMAL, SISTEMA RENINA-ANGIOTENSINA

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      LINO, Caroline Antunes et al. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, v. 416, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.08.015. Acesso em: 27 jun. 2024.
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      Lino, C. A., Silva, I. B. da, Shibata, C. E. R., & Barreto-Chaves, M. L. M. (2015). Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, 416, 1-8. doi:10.1016/j.mce.2015.08.015
    • NLM

      Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
    • Vancouver

      Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      TAKANO, Ana Paula Cremasco e DINIZ, Gabriela Placoná e BARRETO-CHAVES, Maria Luiza Morais. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, v. 376, n. 1-2, p. 43-50, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.05.024. Acesso em: 27 jun. 2024.
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      Takano, A. P. C., Diniz, G. P., & Barreto-Chaves, M. L. M. (2013). AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, 376( 1-2), 43-50. doi:10.1016/j.mce.2013.05.024
    • NLM

      Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
    • Vancouver

      Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      DINIZ, Gabriela Placoná e TAKANO, Ana Paula Cremasco e BARRETO-CHAVES, Maria Luiza Morais. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, v. 374, n. 1-2, p. 117-124, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.04.010. Acesso em: 27 jun. 2024.
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      Diniz, G. P., Takano, A. P. C., & Barreto-Chaves, M. L. M. (2013). miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, 374( 1-2), 117-124. doi:10.1016/j.mce.2013.04.010
    • NLM

      Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
    • Vancouver

      Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      MENDONÇA, Pedro Omori Ribeiro de e LOTFI, Claudimara Ferini Pacicco. the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E. Molecular and Cellular Endocrinology, v. 336, n. 1-2, p. 156-161, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.12.012. Acesso em: 27 jun. 2024.
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      Mendonça, P. O. R. de, & Lotfi, C. F. P. (2011). the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E. Molecular and Cellular Endocrinology, 336( 1-2), 156-161. doi:10.1016/j.mce.2010.12.012
    • NLM

      Mendonça POR de, Lotfi CFP. the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E [Internet]. Molecular and Cellular Endocrinology. 2011 ; 336( 1-2): 156-161.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2010.12.012
    • Vancouver

      Mendonça POR de, Lotfi CFP. the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E [Internet]. Molecular and Cellular Endocrinology. 2011 ; 336( 1-2): 156-161.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2010.12.012
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      ARAUJO, A. S. R. et al. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, v. 333, n. 1, p. 78-84, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.12.018. Acesso em: 27 jun. 2024.
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      Araujo, A. S. R., Diniz, G. P., Seibel, F. E. R., Branchini, G., Ribeiro, M. F. M., Brum, I. S., et al. (2011). Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, 333( 1), 78-84. doi:10.1016/j.mce.2010.12.018
    • NLM

      Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
    • Vancouver

      Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      BARRETO-CHAVES, Maria Luiza Morais et al. E-NTPDase 3 (ATP diphosphohydrolase) from cardiomyocytes, activity and expression are modulated by thyroid hormone. Molecular and Cellular Endocrinology, v. 251, p. 49-55, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2006.02.010. Acesso em: 27 jun. 2024.
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      Barreto-Chaves, M. L. M., Carneiro-Ramos, M. S., Cotomacci, G., Coutinho Júnior, M. B., & Sarkis, J. J. F. (2006). E-NTPDase 3 (ATP diphosphohydrolase) from cardiomyocytes, activity and expression are modulated by thyroid hormone. Molecular and Cellular Endocrinology, 251, 49-55. doi:10.1016/j.mce.2006.02.010
    • NLM

      Barreto-Chaves MLM, Carneiro-Ramos MS, Cotomacci G, Coutinho Júnior MB, Sarkis JJF. E-NTPDase 3 (ATP diphosphohydrolase) from cardiomyocytes, activity and expression are modulated by thyroid hormone [Internet]. Molecular and Cellular Endocrinology. 2006 ; 251 49-55.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2006.02.010
    • Vancouver

      Barreto-Chaves MLM, Carneiro-Ramos MS, Cotomacci G, Coutinho Júnior MB, Sarkis JJF. E-NTPDase 3 (ATP diphosphohydrolase) from cardiomyocytes, activity and expression are modulated by thyroid hormone [Internet]. Molecular and Cellular Endocrinology. 2006 ; 251 49-55.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2006.02.010
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      MATTOS, Gabriele E. e LOTFI, Claudimara Ferini Pacicco. Differences between the growth regulatory pathways in primary rat adrenal cells and mouse tumor cell line. Molecular and Cellular Endocrinology, v. 245, p. 31-42, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2005.10.001. Acesso em: 27 jun. 2024.
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      Mattos, G. E., & Lotfi, C. F. P. (2005). Differences between the growth regulatory pathways in primary rat adrenal cells and mouse tumor cell line. Molecular and Cellular Endocrinology, 245, 31-42. doi:10.1016/j.mce.2005.10.001
    • NLM

      Mattos GE, Lotfi CFP. Differences between the growth regulatory pathways in primary rat adrenal cells and mouse tumor cell line [Internet]. Molecular and Cellular Endocrinology. 2005 ; 245 31-42.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2005.10.001
    • Vancouver

      Mattos GE, Lotfi CFP. Differences between the growth regulatory pathways in primary rat adrenal cells and mouse tumor cell line [Internet]. Molecular and Cellular Endocrinology. 2005 ; 245 31-42.[citado 2024 jun. 27 ] Available from: https://doi.org/10.1016/j.mce.2005.10.001
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      WINK, Márcia R. et al. Thyroid hormone upregulates ecto-5'-nucleotidase/CD73 in C6 rat. Molecular and Cellular Endocrinology, v. 205, p. 107-114, 2003Tradução . . Acesso em: 27 jun. 2024.
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      Wink, M. R., Braganhol, E., Tamajusuku, A. S. K., Casali, E. A., Barreto-Chaves, M. L. M., Sarkis, J. J. F., & Battastini, A. M. O. (2003). Thyroid hormone upregulates ecto-5'-nucleotidase/CD73 in C6 rat. Molecular and Cellular Endocrinology, 205, 107-114.
    • NLM

      Wink MR, Braganhol E, Tamajusuku ASK, Casali EA, Barreto-Chaves MLM, Sarkis JJF, Battastini AMO. Thyroid hormone upregulates ecto-5'-nucleotidase/CD73 in C6 rat. Molecular and Cellular Endocrinology. 2003 ; 205 107-114.[citado 2024 jun. 27 ]
    • Vancouver

      Wink MR, Braganhol E, Tamajusuku ASK, Casali EA, Barreto-Chaves MLM, Sarkis JJF, Battastini AMO. Thyroid hormone upregulates ecto-5'-nucleotidase/CD73 in C6 rat. Molecular and Cellular Endocrinology. 2003 ; 205 107-114.[citado 2024 jun. 27 ]

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