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Artigo de periodico
Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation (2022)
Veras, Flávio Protásio ; Públio, Gabriel Azevedo ; Melo, Bruno Marcel Silva de ; Prado, Douglas da Silva ; Silva, Thainá Norbiato ; Cecilio, Nerry Tatiana ; Hiroki, Carlos Hiroji ; Damasceno, Luis Eduardo Alves ; Jung, Rebecca; Toller-kawahisa, Juliana Escher ; Martins, Timna Varela ; Assunção, Stella Francy Vicente de ; Lima, Diogenes; Alves, Márcia Gaião; Vieira, Gabriel Viliod ; Tavares, Lucas Alves ; Rezende, Ana Laura Rosifini Alves ; Karbach, Susanne H ; Nakaya, Helder Takashi Imoto ; Cunha, Thiago Mattar ; Souza, Cacilda da Silva ; Cunha, Fernando de Queiroz ; Sales, Katiuchia Uzzun Sales ; Waisman, Ari ; Alves Filho, José Carlos Farias
Source: Cell Reports. Unidades: FCFRP, FMRP
Assunto: PSORÍASE
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ABNT
VERAS, Flávio Protásio et al. Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation. Cell Reports, v. 41, n. 13, p. 1-23, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2022.111897. Acesso em: 17 nov. 2024.
APA
Veras, F. P., Públio, G. A., Melo, B. M. S. de, Prado, D. da S., Silva, T. N., Cecilio, N. T., et al. (2022). Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation. Cell Reports, 41( 13), 1-23. doi:10.1016/j.celrep.2022.111897
NLM
Veras FP, Públio GA, Melo BMS de, Prado D da S, Silva TN, Cecilio NT, Hiroki CH, Damasceno LEA, Jung R, Toller-kawahisa JE, Martins TV, Assunção SFV de, Lima D, Alves MG, Vieira GV, Tavares LA, Rezende ALRA, Karbach SH, Nakaya HTI, Cunha TM, Souza C da S, Cunha F de Q, Sales KUS, Waisman A, Alves Filho JCF. Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation [Internet]. Cell Reports. 2022 ; 41( 13): 1-23.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.celrep.2022.111897
Vancouver
Veras FP, Públio GA, Melo BMS de, Prado D da S, Silva TN, Cecilio NT, Hiroki CH, Damasceno LEA, Jung R, Toller-kawahisa JE, Martins TV, Assunção SFV de, Lima D, Alves MG, Vieira GV, Tavares LA, Rezende ALRA, Karbach SH, Nakaya HTI, Cunha TM, Souza C da S, Cunha F de Q, Sales KUS, Waisman A, Alves Filho JCF. Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation [Internet]. Cell Reports. 2022 ; 41( 13): 1-23.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.celrep.2022.111897
Artigo de periodico
Morphological, cellular, and molecular basis of brain infection in COVID-19 patients (2022)
Crunfli, Fernanda ; Carregari, Victor Corasolla ; Veras, Flávio Protásio; Silva, Lucas S.; Nogueira, Mateus Henrique ; Antunes, André Saraiva Leão Marcelo ; Vendramini, Pedro Henrique; Valença, Aline Gazzola Fragnani; Teles, Caroline Brandão; Munhoz, Carolina Demarchi ; Nakaya, Helder Takashi Imoto ; Melo, Bruno Marcel; Almeida, Glaucia Maria de; Paiva, Isadora Marques; Silva, Bruna Manuella Souza; Guimarães, Rafaela Mano; Mendes, Niele Dias; Batah, Sabrina Setembre ; Siyuan, Li; Tavares, Lucas Alves ; Viana, Rosa Maria Mendes; Alves Filho, José Carlos Farias ; Arruda Neto, Eurico de; Podolsky-Gondim, Guilherme Gozzoli ; Santos, Marcelo Volpon ; Neder, Luciano; Louzada Júnior, Paulo; Oliveira, Renê Donizeti Ribeiro de; Cunha, Fernando de Queiroz ; Fabro, Alexandre Todorovic; Sebollela, Adriano Silva ; Cunha, Thiago Mattar
Source: Proceedings of the National Academy of Sciences. Unidades: ICB, FCF, FCFRP, FMRP
Subjects: FARMACOLOGIA, COVID 19
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ABNT
CRUNFLI, Fernanda et al. Morphological, cellular, and molecular basis of brain infection in COVID-19 patients. Proceedings of the National Academy of Sciences, v. 119, n. 35, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1073/pnas.2200960119. Acesso em: 17 nov. 2024.
APA
Crunfli, F., Carregari, V. C., Veras, F. P., Silva, L. S., Nogueira, M. H., Antunes, A. S. L. M., et al. (2022). Morphological, cellular, and molecular basis of brain infection in COVID-19 patients. Proceedings of the National Academy of Sciences, 119( 35), 1-12. doi:10.1073/pnas.2200960119
NLM
Crunfli F, Carregari VC, Veras FP, Silva LS, Nogueira MH, Antunes ASLM, Vendramini PH, Valença AGF, Teles CB, Munhoz CD, Nakaya HTI, Melo BM, Almeida GM de, Paiva IM, Silva BMS, Guimarães RM, Mendes ND, Batah SS, Siyuan L, Tavares LA, Viana RMM, Alves Filho JCF, Arruda Neto E de, Podolsky-Gondim GG, Santos MV, Neder L, Louzada Júnior P, Oliveira RDR de, Cunha F de Q, Fabro AT, Sebollela AS, Cunha TM. Morphological, cellular, and molecular basis of brain infection in COVID-19 patients [Internet]. Proceedings of the National Academy of Sciences. 2022 ; 119( 35): 1-12.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1073/pnas.2200960119
Vancouver
Crunfli F, Carregari VC, Veras FP, Silva LS, Nogueira MH, Antunes ASLM, Vendramini PH, Valença AGF, Teles CB, Munhoz CD, Nakaya HTI, Melo BM, Almeida GM de, Paiva IM, Silva BMS, Guimarães RM, Mendes ND, Batah SS, Siyuan L, Tavares LA, Viana RMM, Alves Filho JCF, Arruda Neto E de, Podolsky-Gondim GG, Santos MV, Neder L, Louzada Júnior P, Oliveira RDR de, Cunha F de Q, Fabro AT, Sebollela AS, Cunha TM. Morphological, cellular, and molecular basis of brain infection in COVID-19 patients [Internet]. Proceedings of the National Academy of Sciences. 2022 ; 119( 35): 1-12.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1073/pnas.2200960119
Artigo de periodico
Toxic mechanisms of cigarette smoke and heat-not-burn tobacco vapor inhalation on rheumatoid arthritis (2022)
Heluany, Cíntia Scucuglia; Scharf, Pablo Rhasan dos Santos ; Schneider, Ayda Henriques ; Donate, Paula Barbim ; Pedreira Filho, Walter R ; Oliveira, Tiago Franco de ; Cunha, Fernando de Queiroz ; Farsky, Sandra Helena Poliselli
Source: Science of the Total Environment. Unidades: FCF, FMRP
Subjects: ARTRITE REUMATOIDE, FUMO, ALCALOIDES
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ABNT
HELUANY, Cíntia Scucuglia et al. Toxic mechanisms of cigarette smoke and heat-not-burn tobacco vapor inhalation on rheumatoid arthritis. Science of the Total Environment, v. 809, p. 1-10 art. 151097, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.scitotenv.2021.151097. Acesso em: 17 nov. 2024.
APA
Heluany, C. S., Scharf, P. R. dos S., Schneider, A. H., Donate, P. B., Pedreira Filho, W. R., Oliveira, T. F. de, et al. (2022). Toxic mechanisms of cigarette smoke and heat-not-burn tobacco vapor inhalation on rheumatoid arthritis. Science of the Total Environment, 809, 1-10 art. 151097. doi:10.1016/j.scitotenv.2021.151097
NLM
Heluany CS, Scharf PR dos S, Schneider AH, Donate PB, Pedreira Filho WR, Oliveira TF de, Cunha F de Q, Farsky SHP. Toxic mechanisms of cigarette smoke and heat-not-burn tobacco vapor inhalation on rheumatoid arthritis [Internet]. Science of the Total Environment. 2022 ; 809 1-10 art. 151097.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.scitotenv.2021.151097
Vancouver
Heluany CS, Scharf PR dos S, Schneider AH, Donate PB, Pedreira Filho WR, Oliveira TF de, Cunha F de Q, Farsky SHP. Toxic mechanisms of cigarette smoke and heat-not-burn tobacco vapor inhalation on rheumatoid arthritis [Internet]. Science of the Total Environment. 2022 ; 809 1-10 art. 151097.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.scitotenv.2021.151097
Trabalho de evento-resumo
Cigarette smoke and heat-notburn tobacco vapor exposures alter granulopoiesis and neutrophil functions during experimental arthritis (2022)
Scharf, Pablo Rhasan dos Santos ; Heluany, Cíntia Scucuglia; Sandri, Silvana ; Schneider, Ayda Henriques ; Barbim, Paula Donate ; Fock, Ricardo Ambrósio ; Cunha, Fernando de Queiroz ; Farsky, Sandra Helena Poliselli
Source: Livro de Resumos. Conference titles: Congresso Brasileiro de Toxicologia/CBTox. Unidades: FCF, FMRP
Subjects: FUMO, ARTRITE REUMATOIDE
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ABNT
SCHARF, Pablo Rhasan dos Santos et al. Cigarette smoke and heat-notburn tobacco vapor exposures alter granulopoiesis and neutrophil functions during experimental arthritis. 2022, Anais.. São Paulo: Sociedade Brasileira de Toxicologia, 2022. Disponível em: https://cbtox2021.com.br/XXII_CBTOX.pdf. Acesso em: 17 nov. 2024.
APA
Scharf, P. R. dos S., Heluany, C. S., Sandri, S., Schneider, A. H., Barbim, P. D., Fock, R. A., et al. (2022). Cigarette smoke and heat-notburn tobacco vapor exposures alter granulopoiesis and neutrophil functions during experimental arthritis. In Livro de Resumos. São Paulo: Sociedade Brasileira de Toxicologia. Recuperado de https://cbtox2021.com.br/XXII_CBTOX.pdf
NLM
Scharf PR dos S, Heluany CS, Sandri S, Schneider AH, Barbim PD, Fock RA, Cunha F de Q, Farsky SHP. Cigarette smoke and heat-notburn tobacco vapor exposures alter granulopoiesis and neutrophil functions during experimental arthritis [Internet]. Livro de Resumos. 2022 ;[citado 2024 nov. 17 ] Available from: https://cbtox2021.com.br/XXII_CBTOX.pdf
Vancouver
Scharf PR dos S, Heluany CS, Sandri S, Schneider AH, Barbim PD, Fock RA, Cunha F de Q, Farsky SHP. Cigarette smoke and heat-notburn tobacco vapor exposures alter granulopoiesis and neutrophil functions during experimental arthritis [Internet]. Livro de Resumos. 2022 ;[citado 2024 nov. 17 ] Available from: https://cbtox2021.com.br/XXII_CBTOX.pdf
Artigo de periodico
Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota (2022)
Arruda, José Alcides Almeida de ; Corrêa, Jôice Dias ; Singh, Youvika ; Oliveira, Sicília Rezende ; Machado, Caio Cavalcante ; Schneider, Ayda Henriques ; Medeiros, Julliane Dutra ; Fernandes, Gabriel R; Macari, Soraia ; Barrioni, Breno Rocha ; Santos, Mariana de Souza; Rodrigues, Letícia Fernanda Duffles ; Nakaya, Helder Takashi Imoto ; Fukada, Sandra Yasuyo ; Graves, Dana T ; Cunha, Fernando Queiroz ; Silva, Tarcília Aparecida
Source: Anaerobe. Unidades: FMRP, FCFRP, FCF, FORP
Subjects: ARTRITE REUMATOIDE, FARMACOTERAPIA, METOTREXATO
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ABNT
ARRUDA, José Alcides Almeida de et al. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota. Anaerobe, v. 75, p. 1-11 art. 102577, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.anaerobe.2022.102577. Acesso em: 17 nov. 2024.
APA
Arruda, J. A. A. de, Corrêa, J. D., Singh, Y., Oliveira, S. R., Machado, C. C., Schneider, A. H., et al. (2022). Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota. Anaerobe, 75, 1-11 art. 102577. doi:10.1016/j.anaerobe.2022.102577
NLM
Arruda JAA de, Corrêa JD, Singh Y, Oliveira SR, Machado CC, Schneider AH, Medeiros JD, Fernandes GR, Macari S, Barrioni BR, Santos M de S, Rodrigues LFD, Nakaya HTI, Fukada SY, Graves DT, Cunha FQ, Silva TA. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota [Internet]. Anaerobe. 2022 ; 75 1-11 art. 102577.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.anaerobe.2022.102577
Vancouver
Arruda JAA de, Corrêa JD, Singh Y, Oliveira SR, Machado CC, Schneider AH, Medeiros JD, Fernandes GR, Macari S, Barrioni BR, Santos M de S, Rodrigues LFD, Nakaya HTI, Fukada SY, Graves DT, Cunha FQ, Silva TA. Methotrexate promotes recovery of arthritis-induced alveolar bone loss and modifies the composition of the oral-gut microbiota [Internet]. Anaerobe. 2022 ; 75 1-11 art. 102577.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.anaerobe.2022.102577
Artigo de periodico
Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity (2021)
Nascimento, Daniele Carvalho ; Viacava, Paula Ramos; Ferreira, Raphael Gomes; Damaceno, Marina Alves; Piñeros, Annie Rocío Alvarez ; Melo, Paulo Henrique de; Donate, Paula Barbim ; Toller-Kawahisa, Juliana Escher; Zoppi, Daniel ; Veras, Flávio Protásio ; Peres, Raphael S. ; Silva, Luisa Menezes ; Caetité, Diego ; Oliveira, Antonio Edson Rocha; Castro, Icaro Maia Santos ; Kauffenstein, Gilles ; Nakaya, Helder Takashi Imoto ; Borges, Marcos de Carvalho ; Zamboni, Dario Simões ; Fonseca, Denise Morais da ; Paschoal, Jonas Augusto Rizzato ; Cunha, Thiago Mattar ; Quesniaux, Valerie ; Linden, Joel ; Cunha, Fernando Queiróz ; Ryffe, Bernhard; Alves-Filho, José Carlos
Source: Immunity. Unidades: FMRP, FCFRP, FCF, ICB, BIOINFORMÁTICA
Subjects: ADENOSINA, FÁRMACOS IMUNOSSUPRESSORES, SEPSE, CAMUNDONGOS, INTERLEUCINA 10, MACRÓFAGOS
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ABNT
NASCIMENTO, Daniele Carvalho et al. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, v. 54, n. 9, p. 2024-2041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.immuni.2021.08.005. Acesso em: 17 nov. 2024.
APA
Nascimento, D. C., Viacava, P. R., Ferreira, R. G., Damaceno, M. A., Piñeros, A. R. A., Melo, P. H. de, et al. (2021). Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, 54( 9), 2024-2041. doi:10.1016/j.immuni.2021.08.005
NLM
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Vancouver
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Artigo de periodico
In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19 (2021)
Caten, Felipe Ten ; Dias, Patricia Gonzalez ; Castro, Icaro; Ogava, Rodrigo Luiz Tomio; Giddaluru, Jeevan ; Silva, Juan Carlo Santos e ; Martins, Felipe; Gonçalves, André Nicolau Aquime ; Martins, André Guilherme da Costa; Araújo, José Deney A ; Viegas, Ana Carolina Nakaya ; Cunha, Fernando de Queiroz ; Farsky, Sandra Helena Poliselli ; Bozza, Fernando A; Levin, Anna Sara Shafferman ; Pannaraj, Pia S ; Silva, Thushan I. de; Minoprio, Paola; Silva, Fabiano Pinheiro da; Andrade, Bruno B; Nakaya, Helder Takashi Imoto
Source: International Journal of Infectious Diseases. Unidades: FCF, FM, FMRP, BIOINFORMÁTICA
Subjects: COVID-19, NEUTRÓFILOS, TRANSTORNOS DA COAGULAÇÃO SANGUÍNEA, IDADE, SEXO
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ABNT
CATEN, Felipe Ten et al. In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19. International Journal of Infectious Diseases, v. 105, p. 579–587, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.ijid.2021.03.016. Acesso em: 17 nov. 2024.
APA
Caten, F. T., Dias, P. G., Castro, I., Ogava, R. L. T., Giddaluru, J., Silva, J. C. S. e, et al. (2021). In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19. International Journal of Infectious Diseases, 105, 579–587. doi:10.1016/j.ijid.2021.03.016
NLM
Caten FT, Dias PG, Castro I, Ogava RLT, Giddaluru J, Silva JCS e, Martins F, Gonçalves ANA, Martins AG da C, Araújo JDA, Viegas ACN, Cunha F de Q, Farsky SHP, Bozza FA, Levin ASS, Pannaraj PS, Silva TI de, Minoprio P, Silva FP da, Andrade BB, Nakaya HTI. In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19 [Internet]. International Journal of Infectious Diseases. 2021 ; 105 579–587.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ijid.2021.03.016
Vancouver
Caten FT, Dias PG, Castro I, Ogava RLT, Giddaluru J, Silva JCS e, Martins F, Gonçalves ANA, Martins AG da C, Araújo JDA, Viegas ACN, Cunha F de Q, Farsky SHP, Bozza FA, Levin ASS, Pannaraj PS, Silva TI de, Minoprio P, Silva FP da, Andrade BB, Nakaya HTI. In-depth analysis of laboratory parameters reveals the interplay between sex, age, and systemic inflammation in individuals with COVID-19 [Internet]. International Journal of Infectious Diseases. 2021 ; 105 579–587.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.ijid.2021.03.016
Artigo de periodico
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis (2018)
Peres, Raphael Sanches; Donate, Paula Barbim; Talbot, Jhimmy; Cecílio, Nerry Tatiana; Lobo, Patricia Rolim Mendonça; Machado, Caio Cavalcante; Lima, Kalil William Alves de; Oliveira, Renê Donizete Ribeiro de; Carregaro, Vanessa; Nakaya, Helder Takashi Imoto ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Liew, Foo Yew; Louzada Júnior, Paulo; Cunha, Fernando Queiróz
Source: Journal of Autoimmunity. Unidades: FMRP, FCF
Subjects: ARTRITE REUMATOIDE, METOTREXATO
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ABNT
PERES, Raphael Sanches et al. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, v. 90, p. 49-58, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jaut.2018.01.004. Acesso em: 17 nov. 2024.
APA
Peres, R. S., Donate, P. B., Talbot, J., Cecílio, N. T., Lobo, P. R. M., Machado, C. C., et al. (2018). TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, 90, 49-58. doi:10.1016/j.jaut.2018.01.004
NLM
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Vancouver
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Artigo de periodico
Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis (2018)
Silva, Maria C; Ferreira, Marcela Davoli; Medina, Tiago S; Costa, Renata Sesti; Silva, Grace K; Lopes, Carla D; Cardozo, Lucas Esteves; Gava, Fábio N; Lyroni, Konstantina; Dias, Fabricio C; Frade, Amanda F; Baron, Monique; Nakaya, Helder Takashi Imoto ; Figueiredo, Florêncio; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Tsatsanis, Christos; Chevillard, Christophe; Cunha-Neto, Edécio; Hirsch, Emilio; Silva, João Santana da; Cunha, Thiago Mattar
Source: Nature Communications. Unidades: FCF, FMRP, FM
Subjects: TRYPANOSOMA CRUZI, MIOCARDIOPATIAS
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ABNT
SILVA, Maria C et al. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, v. 9, p. 1-14 art. 1513, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41467-018-03986-3. Acesso em: 17 nov. 2024.
APA
Silva, M. C., Ferreira, M. D., Medina, T. S., Costa, R. S., Silva, G. K., Lopes, C. D., et al. (2018). Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, 9, 1-14 art. 1513. doi:10.1038/s41467-018-03986-3
NLM
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Vancouver
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41467-018-03986-3

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