Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation (2022)
- Authors:
- Veras, Flávio Protásio
- Públio, Gabriel Azevedo
- Melo, Bruno Marcel Silva de
- Prado, Douglas da Silva
- Silva, Thainá Norbiato
- Cecilio, Nerry Tatiana
- Hiroki, Carlos Hiroji
- Damasceno, Luis Eduardo Alves
- Jung, Rebecca
- Toller-kawahisa, Juliana Escher
- Martins, Timna Varela
- Assunção, Stella Francy Vicente de
- Lima, Diogenes
- Alves, Márcia Gaião
- Vieira, Gabriel Viliod
- Tavares, Lucas Alves
- Rezende, Ana Laura Rosifini Alves
- Karbach, Susanne H
- Nakaya, Helder Takashi Imoto
- Cunha, Thiago Mattar
- Souza, Cacilda da Silva
- Cunha, Fernando de Queiroz
- Sales, Katiuchia Uzzun Sales
- Waisman, Ari
- Alves Filho, José Carlos Farias
- Veras, Flávio Protásio
- USP affiliated authors: VERAS, FLÁVIO PROTÁSIO - FCFRP ; CUNHA, THIAGO MATTAR - FMRP ; SOUZA, CACILDA DA SILVA - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; SALES, KATIUCHIA UZZUN - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; PÚBLIO, GABRIEL AZEVEDO - FMRP ; MELO, BRUNO MARCEL SILVA DE - FMRP ; DAMASCENO, LUIS EDUARDO ALVES - FMRP ; MARTINS, TIMNA VARELA - FMRP ; ASSUNÇÃO, STELLA FRANCY VICENTE DE - FMRP ; VIEIRA, GABRIEL VILIOD - FMRP ; TAVARES, LUCAS ALVES - FMRP
- Unidades: FCFRP; FMRP
- DOI: 10.1016/j.celrep.2022.111897
- Assunto: PSORÍASE
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Cell Reports
- ISSN: 2211-1247
- Volume/Número/Paginação/Ano: v. 41, n. 13, p. 1-23, art. 111897, 2022
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc-nd
-
ABNT
VERAS, Flávio Protásio et al. Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation. Cell Reports, v. 41, n. 13, p. 1-23, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2022.111897. Acesso em: 27 dez. 2025. -
APA
Veras, F. P., Públio, G. A., Melo, B. M. S. de, Prado, D. da S., Silva, T. N., Cecilio, N. T., et al. (2022). Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation. Cell Reports, 41( 13), 1-23. doi:10.1016/j.celrep.2022.111897 -
NLM
Veras FP, Públio GA, Melo BMS de, Prado D da S, Silva TN, Cecilio NT, Hiroki CH, Damasceno LEA, Jung R, Toller-kawahisa JE, Martins TV, Assunção SFV de, Lima D, Alves MG, Vieira GV, Tavares LA, Rezende ALRA, Karbach SH, Nakaya HTI, Cunha TM, Souza C da S, Cunha F de Q, Sales KUS, Waisman A, Alves Filho JCF. Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation [Internet]. Cell Reports. 2022 ; 41( 13): 1-23.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1016/j.celrep.2022.111897 -
Vancouver
Veras FP, Públio GA, Melo BMS de, Prado D da S, Silva TN, Cecilio NT, Hiroki CH, Damasceno LEA, Jung R, Toller-kawahisa JE, Martins TV, Assunção SFV de, Lima D, Alves MG, Vieira GV, Tavares LA, Rezende ALRA, Karbach SH, Nakaya HTI, Cunha TM, Souza C da S, Cunha F de Q, Sales KUS, Waisman A, Alves Filho JCF. Pyruvate kinase M2 mediates IL-17 signaling in keratinocytes driving psoriatic skin inflammation [Internet]. Cell Reports. 2022 ; 41( 13): 1-23.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1016/j.celrep.2022.111897 - S100A9 drives the chronification of psoriasiform inflammation by inducing IL-23/type 3 immunity
- A clinically-relevant STING agonist restrains human TH17 cell inflammatory profile
- The metabolic function of pyruvate kinase M2 regulates reactive oxygen species production and microbial killing by neutrophils
- Pitavastatin ameliorates autoimmune neuroinflammation by regulating the Treg/Th17 cell balance through inhibition of mevalonate metabolism
- PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation
- Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition
- MMP-9 mediates cross-talk between neutrophils and endothelial cells in psoriasis
- STING is an intrinsic checkpoint inhibitor that restrains the TH17 cell pathogenic program
- Toll-like receptor 9 activation in neutrophils impairs chemotaxis and reduces sepsis outcome
- Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice
Informações sobre o DOI: 10.1016/j.celrep.2022.111897 (Fonte: oaDOI API)
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