ReP USP - Resultado da busca

Artigo de periodico
The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition (2021)
Queiroz, André L.; Lessard, Sarah J.; Ouchida, Amanda T.; Araujo, Hygor N.; Gonçalves, Dawit A.; Guimarães, Dimitrius Santiago Passos Simões Fróes; Teodoro, Bruno G.; So, Kawai; Espreáfico, Enilza Maria ; Hirshman, Michael F.; Alberici, Luciane Carla ; Kettelhut, Isis do Carmo ; Goodyear, Laurie J.; Silveira, Leonardo R.
Source: Molecular Metabolism. Unidades: FMRP, FCFRP
Subjects: MICRORNAS, MÚSCULO ESQUELÉTICO, MITOCÔNDRIAS, REGULAÇÃO GÊNICA, PROLIFERAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUEIROZ, André L. et al. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, v. 51, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2021.101226. Acesso em: 05 nov. 2024.
APA
Queiroz, A. L., Lessard, S. J., Ouchida, A. T., Araujo, H. N., Gonçalves, D. A., Guimarães, D. S. P. S. F., et al. (2021). The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, 51. doi:10.1016/j.molmet.2021.101226
NLM
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Vancouver
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Artigo de periodico
The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes (2021)
Santos, Renato Elias Rodrigues de Souza ; Silva Júnior, Waldir P. da; Harrison, Simone; Skaar, Eric P.; Chazin, Walter J.; Silva Neto, José Freire da
Source: Infection and Immunity. Unidade: FMRP
Subjects: BIOFILMES, COMPLEXO ANTÍGENO-ANTICORPO, LEUCÓCITOS, NEUTRÓFILOS, VIRULÊNCIA, ZINCO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Renato Elias Rodrigues de Souza et al. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes. Infection and Immunity, v. 89, n. 11, 2021Tradução . . Disponível em: https://doi.org/10.1128/IAI.00311-21. Acesso em: 05 nov. 2024.
APA
Santos, R. E. R. de S., Silva Júnior, W. P. da, Harrison, S., Skaar, E. P., Chazin, W. J., & Silva Neto, J. F. da. (2021). The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes. Infection and Immunity, 89( 11). doi:10.1128/IAI.00311-21
NLM
Santos RER de S, Silva Júnior WP da, Harrison S, Skaar EP, Chazin WJ, Silva Neto JF da. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes [Internet]. Infection and Immunity. 2021 ; 89( 11):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1128/IAI.00311-21
Vancouver
Santos RER de S, Silva Júnior WP da, Harrison S, Skaar EP, Chazin WJ, Silva Neto JF da. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes [Internet]. Infection and Immunity. 2021 ; 89( 11):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1128/IAI.00311-21
Artigo de periodico
Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens (2020)
Black, Jennifer Ann; Crouch, Kathryn; Lemgruber, Leandro; Lapsley, Craig; Dickens, Nicholas; Tosi, Luíz Ricardo Orsini ; Mottram, Jeremy C.; McCulloch, Richard
Source: Cell Reports. Unidade: FMRP
Subjects: ALELOS, VARIAÇÃO GENÉTICA, NÚCLEO CELULAR, PROLIFERAÇÃO CELULAR, SOBREVIVÊNCIA CELULAR, DANO AO DNA, GENOMAS, RNA POLIMERASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BLACK, Jennifer Ann et al. Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens. Cell Reports, v. 30, n. 3, p. 836-851, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2019.12.049. Acesso em: 05 nov. 2024.
APA
Black, J. A., Crouch, K., Lemgruber, L., Lapsley, C., Dickens, N., Tosi, L. R. O., et al. (2020). Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens. Cell Reports, 30( 3), 836-851. doi:10.1016/j.celrep.2019.12.049
NLM
Black JA, Crouch K, Lemgruber L, Lapsley C, Dickens N, Tosi LRO, Mottram JC, McCulloch R. Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens [Internet]. Cell Reports. 2020 ; 30( 3): 836-851.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.celrep.2019.12.049
Vancouver
Black JA, Crouch K, Lemgruber L, Lapsley C, Dickens N, Tosi LRO, Mottram JC, McCulloch R. Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens [Internet]. Cell Reports. 2020 ; 30( 3): 836-851.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.celrep.2019.12.049
Artigo de periodico
Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma (2018)
Araujo, Luiza Ferreira de ; Siena, Ádamo Davi Diogenes; Plaça, Jessica Rodrigues ; Brotto, Danielle Barbosa ; Barros, Isabela Ichihara de ; Muys, Bruna Rodrigues; Biagi Jr, Carlos Alberto Oliveira de ; Peronni, Kamila Chagas; Sousa, Josane de Freitas; Molfetta, Greice Andreotti de ; West, L. C.; West, A. P.; Leopoldino, Andréia Machado ; Espreáfico, Enilza Maria ; Silva Júnior, Wilson Araújo da
Source: Scientific Reports. Unidades: FCFRP, FMRP
Subjects: MELANOMA, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAUJO, Luiza Ferreira de et al. Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma. Scientific Reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-31170-6. Acesso em: 05 nov. 2024.
APA
Araujo, L. F. de, Siena, Á. D. D., Plaça, J. R., Brotto, D. B., Barros, I. I. de, Muys, B. R., et al. (2018). Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma. Scientific Reports, 8. doi:10.1038/s41598-018-31170-6
NLM
Araujo LF de, Siena ÁDD, Plaça JR, Brotto DB, Barros II de, Muys BR, Biagi Jr CAO de, Peronni KC, Sousa J de F, Molfetta GA de, West LC, West AP, Leopoldino AM, Espreáfico EM, Silva Júnior WA da. Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41598-018-31170-6
Vancouver
Araujo LF de, Siena ÁDD, Plaça JR, Brotto DB, Barros II de, Muys BR, Biagi Jr CAO de, Peronni KC, Sousa J de F, Molfetta GA de, West LC, West AP, Leopoldino AM, Espreáfico EM, Silva Júnior WA da. Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41598-018-31170-6
Artigo de periodico
Intrauterine growth restriction programs the hypothalamus of adult male rats: integrated analysis of proteomic and metabolomic data (2017)
Pedroso, Amanda P.; Souza, Adriana P.; Dornellas, Ana P. S.; Oyama, Lila M.; Nascimento, Cláudia M. O.; Santos, Gianni M. S.; Rosa, José César; Bertolla, Ricardo P.; Klawitter, Jelena; Christians, Uwe; Tashima, Alexandre K.; Ribeiro, Eliane B.
Source: Journal of Proteome Research. Unidade: FMRP
Subjects: PROTEÔMICA, METABOLÔMICA, METABOLISMO ENERGÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEDROSO, Amanda P. et al. Intrauterine growth restriction programs the hypothalamus of adult male rats: integrated analysis of proteomic and metabolomic data. Journal of Proteome Research, v. 16, n. 4, p. 1515-1525, 2017Tradução . . Disponível em: https://doi.org/10.1021/acs.jproteome.6b00923. Acesso em: 05 nov. 2024.
APA
Pedroso, A. P., Souza, A. P., Dornellas, A. P. S., Oyama, L. M., Nascimento, C. M. O., Santos, G. M. S., et al. (2017). Intrauterine growth restriction programs the hypothalamus of adult male rats: integrated analysis of proteomic and metabolomic data. Journal of Proteome Research, 16( 4), 1515-1525. doi:10.1021/acs.jproteome.6b00923
NLM
Pedroso AP, Souza AP, Dornellas APS, Oyama LM, Nascimento CMO, Santos GMS, Rosa JC, Bertolla RP, Klawitter J, Christians U, Tashima AK, Ribeiro EB. Intrauterine growth restriction programs the hypothalamus of adult male rats: integrated analysis of proteomic and metabolomic data [Internet]. Journal of Proteome Research. 2017 ; 16( 4): 1515-1525.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1021/acs.jproteome.6b00923
Vancouver
Pedroso AP, Souza AP, Dornellas APS, Oyama LM, Nascimento CMO, Santos GMS, Rosa JC, Bertolla RP, Klawitter J, Christians U, Tashima AK, Ribeiro EB. Intrauterine growth restriction programs the hypothalamus of adult male rats: integrated analysis of proteomic and metabolomic data [Internet]. Journal of Proteome Research. 2017 ; 16( 4): 1515-1525.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1021/acs.jproteome.6b00923
Artigo de periodico
AIM2 engages active but unprocessed caspase-1 to induce noncanonical activation of the NLRP3 inflammasome (2017)
Cunha, Larissa D.; Silva, Alexandre L. N.; Ribeiro, Juliana M.; Mascarenhas, Danielle P. A.; Quirino, Gustavo F.S.; Santos, Leonardo L.; Flavell, Richard A.; Zamboni, Dario Simões
Source: Cell Reports. Unidade: FMRP
Subjects: SISTEMA IMUNE, INFLAMAÇÃO, ANTI-INFLAMATÓRIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, Larissa D. et al. AIM2 engages active but unprocessed caspase-1 to induce noncanonical activation of the NLRP3 inflammasome. Cell Reports, v. 20, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2017.06.086. Acesso em: 05 nov. 2024.
APA
Cunha, L. D., Silva, A. L. N., Ribeiro, J. M., Mascarenhas, D. P. A., Quirino, G. F. S., Santos, L. L., et al. (2017). AIM2 engages active but unprocessed caspase-1 to induce noncanonical activation of the NLRP3 inflammasome. Cell Reports, 20. doi:10.1016/j.celrep.2017.06.086
NLM
Cunha LD, Silva ALN, Ribeiro JM, Mascarenhas DPA, Quirino GFS, Santos LL, Flavell RA, Zamboni DS. AIM2 engages active but unprocessed caspase-1 to induce noncanonical activation of the NLRP3 inflammasome [Internet]. Cell Reports. 2017 ; 20[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.celrep.2017.06.086
Vancouver
Cunha LD, Silva ALN, Ribeiro JM, Mascarenhas DPA, Quirino GFS, Santos LL, Flavell RA, Zamboni DS. AIM2 engages active but unprocessed caspase-1 to induce noncanonical activation of the NLRP3 inflammasome [Internet]. Cell Reports. 2017 ; 20[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.celrep.2017.06.086
Artigo de periodico
Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages (2016)
Bradley, William P.; Boyer, Mark A.; Nguyen, Hieu T.; Birdwell, L. Dillon; Yu, Janet; Ribeiro, Juliana M.; Weiss, Susan R.; Zamboni, Dario Simões; Roy, Craig R.; Shin, Sunny
Source: Infection and Immunity. Unidade: FMRP
Subjects: FEBRE Q, MACRÓFAGOS, SISTEMA IMUNE, MICROBIOLOGIA, CÉLULAS CULTIVADAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRADLEY, William P. et al. Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages. Infection and Immunity, v. 84, n. 4, p. 998-1015, 2016Tradução . . Disponível em: https://doi.org/10.1128/iai.01536-15. Acesso em: 05 nov. 2024.
APA
Bradley, W. P., Boyer, M. A., Nguyen, H. T., Birdwell, L. D., Yu, J., Ribeiro, J. M., et al. (2016). Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages. Infection and Immunity, 84( 4), 998-1015. doi:10.1128/iai.01536-15
NLM
Bradley WP, Boyer MA, Nguyen HT, Birdwell LD, Yu J, Ribeiro JM, Weiss SR, Zamboni DS, Roy CR, Shin S. Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages [Internet]. Infection and Immunity. 2016 ; 84( 4): 998-1015.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1128/iai.01536-15
Vancouver
Bradley WP, Boyer MA, Nguyen HT, Birdwell LD, Yu J, Ribeiro JM, Weiss SR, Zamboni DS, Roy CR, Shin S. Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages [Internet]. Infection and Immunity. 2016 ; 84( 4): 998-1015.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1128/iai.01536-15
Artigo de periodico
Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality (2016)
Zoccal, Karina F.; Sorgi, Carlos Arterio; Hori, Juliana I.; Paula-Silva, Francisco Wanderley Garcia de; Arantes, Eliane Candiani; Serezani, Carlos H.; Zamboni, Dario Simões; Faccioli, Lucia Helena
Source: Nature Communications. Unidades: FCFRP, FORP, FMRP
Subjects: ENVENENAMENTO POR ANIMAIS PEÇONHENTOS, ESCORPIÕES, TERAPÊUTICA, FÁRMACOS, EDEMA PULMONAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZOCCAL, Karina F. et al. Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality. Nature Communications, v. 7, 2016Tradução . . Disponível em: https://doi.org/10.1038/ncomms10760. Acesso em: 05 nov. 2024.
APA
Zoccal, K. F., Sorgi, C. A., Hori, J. I., Paula-Silva, F. W. G. de, Arantes, E. C., Serezani, C. H., et al. (2016). Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality. Nature Communications, 7. doi:10.1038/ncomms10760
NLM
Zoccal KF, Sorgi CA, Hori JI, Paula-Silva FWG de, Arantes EC, Serezani CH, Zamboni DS, Faccioli LH. Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality [Internet]. Nature Communications. 2016 ; 7[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/ncomms10760
Vancouver
Zoccal KF, Sorgi CA, Hori JI, Paula-Silva FWG de, Arantes EC, Serezani CH, Zamboni DS, Faccioli LH. Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality [Internet]. Nature Communications. 2016 ; 7[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/ncomms10760

USP Schools

ReP

Filtros

Departament

Authors

USP affiliated authors

Subjects

Source title

Funding Agencies

Indexado em

Non normalized affiliation