Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality (2016)
- Authors:
- USP affiliated authors: SORGI, CARLOS ARTERIO - FCFRP ; SILVA, FRANCISCO WANDERLEY GARCIA DE PAULA E - FORP ; BRAGA, ELIANE CANDIANI ARANTES - FCFRP ; ZAMBONI, DARIO SIMÕES - FMRP ; FACCIOLI, LUCIA HELENA - FCFRP
- Unidades: FCFRP; FORP; FMRP
- DOI: 10.1038/ncomms10760
- Subjects: ENVENENAMENTO POR ANIMAIS PEÇONHENTOS; ESCORPIÕES; TERAPÊUTICA; FÁRMACOS; EDEMA PULMONAR
- Language: Inglês
- Imprenta:
- Source:
- Título: Nature Communications
- ISSN: 2041-1723
- Volume/Número/Paginação/Ano: v. 7, art. 10760, 2016
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
ZOCCAL, Karina F. et al. Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality. Nature Communications, v. 7, 2016Tradução . . Disponível em: https://doi.org/10.1038/ncomms10760. Acesso em: 25 jan. 2026. -
APA
Zoccal, K. F., Sorgi, C. A., Hori, J. I., Paula-Silva, F. W. G. de, Arantes, E. C., Serezani, C. H., et al. (2016). Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality. Nature Communications, 7. doi:10.1038/ncomms10760 -
NLM
Zoccal KF, Sorgi CA, Hori JI, Paula-Silva FWG de, Arantes EC, Serezani CH, Zamboni DS, Faccioli LH. Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality [Internet]. Nature Communications. 2016 ; 7[citado 2026 jan. 25 ] Available from: https://doi.org/10.1038/ncomms10760 -
Vancouver
Zoccal KF, Sorgi CA, Hori JI, Paula-Silva FWG de, Arantes EC, Serezani CH, Zamboni DS, Faccioli LH. Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality [Internet]. Nature Communications. 2016 ; 7[citado 2026 jan. 25 ] Available from: https://doi.org/10.1038/ncomms10760 - CD14,TLR2 and TLR4 are essential to macrophages recognize Tityus serrulatus venom and its toxin 1 and to release lipid mediators, IL-6 and TNFA
- PPAR-'gama' activation by Tityus serrulatus venom regulates lipid body formation and lipid mediator production
- TLR2, TLR4 and CD14 recognize venom-associated molecular patterns from Tityus serrulatus to induce macrophage-derived inflammatory mediators
- Hyaluronidase decreases neutrophils infiltration to the inflammatory site
- Prostaglandins D2 and E2 have opposite effects on alveolar macrophages infected with Histoplasma capsulatum
- 5-LO, FLAP, and BLT1 expression in human T cell lymphotropic virus type 1
- The role of 'beta'-2 integrin in alveolar macrophage phagocytic activity in histoplasmosis
- Role of 5-lipoxigenase on lipid body formation induced by Histoplasma capsulatum cell wall 'beta'-glucan and coinfected immunity
- Papel do TLR2 na histoplasmose experimental
- Leukotrienes in the susceptibility and resistence to infection by Histoplasma capsulatum
Informações sobre o DOI: 10.1038/ncomms10760 (Fonte: oaDOI API)
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