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Artigo de periodico
Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis (2010)
Celes, Mara Rúbia Nunes; Malvestio, Lygia M.; Torres-Dueñas, Diego; Blefari, Valdecir; Campos, Érica Carolina; Ramos, Simone Gusmão; Prado, Cibele Maria; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Source: Laboratory Investigation. Unidade: FMRP
Subjects: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
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ABNT
CELES, Mara Rúbia Nunes et al. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, v. 90, n. 4, p. 531-542, 2010Tradução . . Disponível em: https://doi.org/10.1186/cc9157. Acesso em: 02 nov. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Torres-Dueñas, D., Blefari, V., Campos, É. C., Ramos, S. G., et al. (2010). Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, 90( 4), 531-542. doi:10.1186/cc9157
NLM
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/cc9157
Vancouver
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/cc9157
Artigo de periodico
Early identification of infectious complications in bariatric surgery by the determination of peritoneal and systemic cytokines (2009)
Salgado Júnior, Wilson; Cunha, Fernando de Queiroz; Santos, José Sebastião dos; Borges, Carla Barbosa Nonino; Sankarankutty, Ajith Kumar; Castro e Silva Júnior, Orlando de; Ceneviva, Reginaldo
Source: Obesity Surgery. Unidade: FMRP
Subjects: CIRURGIA BARIÁTRICA (COMPLICAÇÕES), CITOCINAS
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SALGADO JÚNIOR, Wilson et al. Early identification of infectious complications in bariatric surgery by the determination of peritoneal and systemic cytokines. Obesity Surgery, v. 19, n. 7, p. 867-872, 2009Tradução . . Acesso em: 02 nov. 2024.
APA
Salgado Júnior, W., Cunha, F. de Q., Santos, J. S. dos, Borges, C. B. N., Sankarankutty, A. K., Castro e Silva Júnior, O. de, & Ceneviva, R. (2009). Early identification of infectious complications in bariatric surgery by the determination of peritoneal and systemic cytokines. Obesity Surgery, 19( 7), 867-872.
NLM
Salgado Júnior W, Cunha F de Q, Santos JS dos, Borges CBN, Sankarankutty AK, Castro e Silva Júnior O de, Ceneviva R. Early identification of infectious complications in bariatric surgery by the determination of peritoneal and systemic cytokines. Obesity Surgery. 2009 ; 19( 7): 867-872.[citado 2024 nov. 02 ]
Vancouver
Salgado Júnior W, Cunha F de Q, Santos JS dos, Borges CBN, Sankarankutty AK, Castro e Silva Júnior O de, Ceneviva R. Early identification of infectious complications in bariatric surgery by the determination of peritoneal and systemic cytokines. Obesity Surgery. 2009 ; 19( 7): 867-872.[citado 2024 nov. 02 ]
Artigo de periodico
Interleukin-17 and kidney allograft outcome (2009)
Crispim, Janaína Cristiana de Oliveira; Palomino, Gustavo Martelli; Rassi, Diane Meyre; Costa, Roberto Silva; Saber, L. T.; Cunha, Fernando de Queiroz; Donadi, Eduardo Antônio; Grespan, Renata
Source: Transplantation Proceedings. Unidade: FMRP
Subjects: TRANSPLANTE DE RIM, FISIOPATOLOGIA, CITOCINAS
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ABNT
CRISPIM, Janaína Cristiana de Oliveira et al. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings, v. 41, n. 5, p. 1562-1564, 2009Tradução . . Acesso em: 02 nov. 2024.
APA
Crispim, J. C. de O., Palomino, G. M., Rassi, D. M., Costa, R. S., Saber, L. T., Cunha, F. de Q., et al. (2009). Interleukin-17 and kidney allograft outcome. Transplantation Proceedings, 41( 5), 1562-1564.
NLM
Crispim JC de O, Palomino GM, Rassi DM, Costa RS, Saber LT, Cunha F de Q, Donadi EA, Grespan R. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings. 2009 ; 41( 5): 1562-1564.[citado 2024 nov. 02 ]
Vancouver
Crispim JC de O, Palomino GM, Rassi DM, Costa RS, Saber LT, Cunha F de Q, Donadi EA, Grespan R. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings. 2009 ; 41( 5): 1562-1564.[citado 2024 nov. 02 ]
Artigo de periodico
IL-4 regulates susceptibility to intestinal inflammation in murine food allergy (2009)
Cardoso, Cristina Ribeiro; Provinciatto, Pauline Rossetti; Godoi, Dannielle Fernandes; Ferreira, Beatriz Rossetti ; Teixeira, Gerlinde; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: American Journal of Physiology. Unidades: EERP, FMRP
Subjects: HIPERSENSIBILIDADE ALIMENTAR, INTESTINOS, INFLAMAÇÃO
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ABNT
CARDOSO, Cristina Ribeiro et al. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy. American Journal of Physiology, v. 296, n. 3, p. G593-G600, 2009Tradução . . Disponível em: https://doi.org/10.1152/ajpgi.90431.2008. Acesso em: 02 nov. 2024.
APA
Cardoso, C. R., Provinciatto, P. R., Godoi, D. F., Ferreira, B. R., Teixeira, G., Rossi, M. A., et al. (2009). IL-4 regulates susceptibility to intestinal inflammation in murine food allergy. American Journal of Physiology, 296( 3), G593-G600. doi:10.1152/ajpgi.90431.2008
NLM
Cardoso CR, Provinciatto PR, Godoi DF, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy [Internet]. American Journal of Physiology. 2009 ; 296( 3): G593-G600.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1152/ajpgi.90431.2008
Vancouver
Cardoso CR, Provinciatto PR, Godoi DF, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy [Internet]. American Journal of Physiology. 2009 ; 296( 3): G593-G600.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1152/ajpgi.90431.2008
Artigo de periodico
The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron (2009)
Villarreal, Cristiane Flora; Sachs, Daniela; Funez, Mani Indiana; Parada, Carlos Amílcar; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: FARMACOLOGIA, DOR
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ABNT
VILLARREAL, Cristiane Flora et al. The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron. Biochemical Pharmacology, v. 77, n. 5, p. 867-877, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2008.11.015. Acesso em: 02 nov. 2024.
APA
Villarreal, C. F., Sachs, D., Funez, M. I., Parada, C. A., Cunha, F. de Q., & Ferreira, S. H. (2009). The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron. Biochemical Pharmacology, 77( 5), 867-877. doi:10.1016/j.bcp.2008.11.015
NLM
Villarreal CF, Sachs D, Funez MI, Parada CA, Cunha F de Q, Ferreira SH. The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron [Internet]. Biochemical Pharmacology. 2009 ; 77( 5): 867-877.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.bcp.2008.11.015
Vancouver
Villarreal CF, Sachs D, Funez MI, Parada CA, Cunha F de Q, Ferreira SH. The peripheral pro-nociceptive state induced by repetitive inflammatory stimuli involves continuous activation of protein kinase A and protein kinase C epsilon and its 'NA IND. v'1.8 sodium channel functional regulation in the primary sensory neuron [Internet]. Biochemical Pharmacology. 2009 ; 77( 5): 867-877.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.bcp.2008.11.015
Artigo de periodico
Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production (2009)
Lemos, Henrique de Paula; Grespan, Renata; Vieira, Sílvio Manfredo; Cunha, Thiago Mattar; Verri Júnior, Waldiceu Aparecido; Fernandes, Karla Simone S.; Souto, Fabrício Oliveira; Mclnnes, Iain B.; Ferreira, Sérgio Henrique; Liew, Foo Y.; Cunha, Fernando de Queiroz
Source: Proceedings of the Nacional Academy of Sciences. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, CITOCINAS, NEUTRÓFILOS
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ABNT
LEMOS, Henrique de Paula et al. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production. Proceedings of the Nacional Academy of Sciences, v. 106, n. 14, p. 5954-5959, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0812782106. Acesso em: 02 nov. 2024.
APA
Lemos, H. de P., Grespan, R., Vieira, S. M., Cunha, T. M., Verri Júnior, W. A., Fernandes, K. S. S., et al. (2009). Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production. Proceedings of the Nacional Academy of Sciences, 106( 14), 5954-5959. doi:10.1073/pnas.0812782106
NLM
Lemos H de P, Grespan R, Vieira SM, Cunha TM, Verri Júnior WA, Fernandes KSS, Souto FO, Mclnnes IB, Ferreira SH, Liew FY, Cunha F de Q. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production [Internet]. Proceedings of the Nacional Academy of Sciences. 2009 ; 106( 14): 5954-5959.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0812782106
Vancouver
Lemos H de P, Grespan R, Vieira SM, Cunha TM, Verri Júnior WA, Fernandes KSS, Souto FO, Mclnnes IB, Ferreira SH, Liew FY, Cunha F de Q. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production [Internet]. Proceedings of the Nacional Academy of Sciences. 2009 ; 106( 14): 5954-5959.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0812782106
Artigo de periodico
The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception (2009)
Sachs, Daniela; Villarreal, Cristiane Floral; Cunha, Fernando de Queiroz; Parada, Carlos Amílcar; Ferreira, Sérgio Henrique
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: PROTEÍNAS QUINASES, DOR, FARMACOLOGIA
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ABNT
SACHS, Daniela et al. The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology, v. 156, n. 5, p. 826-834, 2009Tradução . . Acesso em: 02 nov. 2024.
APA
Sachs, D., Villarreal, C. F., Cunha, F. de Q., Parada, C. A., & Ferreira, S. H. (2009). The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology, 156( 5), 826-834.
NLM
Sachs D, Villarreal CF, Cunha F de Q, Parada CA, Ferreira SH. The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology. 2009 ; 156( 5): 826-834.[citado 2024 nov. 02 ]
Vancouver
Sachs D, Villarreal CF, Cunha F de Q, Parada CA, Ferreira SH. The role of PKA and PKC'épsilon' pathways in prostaglandin 'E IND.2'-mediated hypernociception. British Journal of Pharmacology. 2009 ; 156( 5): 826-834.[citado 2024 nov. 02 ]
Artigo de periodico
Streptozotocin-induced mechanical hypernociception is not dependent on hyperglycemia (2009)
Cunha, J. M.; Funez, M. I.; Cunha, Fernando de Queiroz; Parada, C. A.; Ferreira, S. H.
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Subjects: NEUROPATIAS DIABÉTICAS, MEDICAMENTO, INSULINA
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ABNT
CUNHA, J. M. et al. Streptozotocin-induced mechanical hypernociception is not dependent on hyperglycemia. Brazilian Journal of Medical and Biological Research, v. 42, n. 2, p. 197-206, 2009Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2009000200008. Acesso em: 02 nov. 2024.
APA
Cunha, J. M., Funez, M. I., Cunha, F. de Q., Parada, C. A., & Ferreira, S. H. (2009). Streptozotocin-induced mechanical hypernociception is not dependent on hyperglycemia. Brazilian Journal of Medical and Biological Research, 42( 2), 197-206. doi:10.1590/s0100-879x2009000200008
NLM
Cunha JM, Funez MI, Cunha F de Q, Parada CA, Ferreira SH. Streptozotocin-induced mechanical hypernociception is not dependent on hyperglycemia [Internet]. Brazilian Journal of Medical and Biological Research. 2009 ; 42( 2): 197-206.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/s0100-879x2009000200008
Vancouver
Cunha JM, Funez MI, Cunha F de Q, Parada CA, Ferreira SH. Streptozotocin-induced mechanical hypernociception is not dependent on hyperglycemia [Internet]. Brazilian Journal of Medical and Biological Research. 2009 ; 42( 2): 197-206.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/s0100-879x2009000200008
Artigo de periodico
Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis (2009)
Alves-Filho, José Carlos; Freitas, Andressa; Souto, Fabrício Oliveira; Spiller, Fernando; Paula Neto, Heitor; Silva, João Santana da; Gazzinelli, Ricardo T.; Teixeira, Mauro Martins; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
ALVES-FILHO, José Carlos et al. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, v. 106, n. 10, p. 4018-4023, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0900196106. Acesso em: 02 nov. 2024.
APA
Alves-Filho, J. C., Freitas, A., Souto, F. O., Spiller, F., Paula Neto, H., Silva, J. S. da, et al. (2009). Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, 106( 10), 4018-4023. doi:10.1073/pnas.0900196106
NLM
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0900196106
Vancouver
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0900196106
Artigo de periodico
Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii (2008)
Fernandes, Karla Simone S.; Helal Neto, Edward; Brito, Marcelly M. S.; Silva, João Santana da; Cunha, Fernando de Queiroz; Barja-Fidalga, Christina
Source: Immunology. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, FUNGOS, MACRÓFAGOS, FAGOCITOSE
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ABNT
FERNANDES, Karla Simone S. et al. Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii. Immunology, v. 123, n. 4, p. 469-479, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2567.2007.02712.x. Acesso em: 02 nov. 2024.
APA
Fernandes, K. S. S., Helal Neto, E., Brito, M. M. S., Silva, J. S. da, Cunha, F. de Q., & Barja-Fidalga, C. (2008). Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii. Immunology, 123( 4), 469-479. doi:10.1111/j.1365-2567.2007.02712.x
NLM
Fernandes KSS, Helal Neto E, Brito MMS, Silva JS da, Cunha F de Q, Barja-Fidalga C. Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii [Internet]. Immunology. 2008 ; 123( 4): 469-479.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/j.1365-2567.2007.02712.x
Vancouver
Fernandes KSS, Helal Neto E, Brito MMS, Silva JS da, Cunha F de Q, Barja-Fidalga C. Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii [Internet]. Immunology. 2008 ; 123( 4): 469-479.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/j.1365-2567.2007.02712.x
Artigo de periodico
Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice (2008)
Cunha, T. M.; Barsante, M. M.; Guerrero, A. T.; Verri Júnior, Waldiceu Aparecido; Ferreira, Sérgio Henrique; Coelho, F. M.; Bertini, R.; Giacinto, C. Di; Allegretti, M.; Cunha, Fernando de Queiroz; Teixeira, M. M.
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: ARTRITE, DOR, NEUTRÓFILOS
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ABNT
CUNHA, T. M. et al. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice. British Journal of Pharmacology, v. 154, n. 2, p. 460-470, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.94. Acesso em: 02 nov. 2024.
APA
Cunha, T. M., Barsante, M. M., Guerrero, A. T., Verri Júnior, W. A., Ferreira, S. H., Coelho, F. M., et al. (2008). Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice. British Journal of Pharmacology, 154( 2), 460-470. doi:10.1038/bjp.2008.94
NLM
Cunha TM, Barsante MM, Guerrero AT, Verri Júnior WA, Ferreira SH, Coelho FM, Bertini R, Giacinto CD, Allegretti M, Cunha F de Q, Teixeira MM. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice [Internet]. British Journal of Pharmacology. 2008 ; 154( 2): 460-470.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/bjp.2008.94
Vancouver
Cunha TM, Barsante MM, Guerrero AT, Verri Júnior WA, Ferreira SH, Coelho FM, Bertini R, Giacinto CD, Allegretti M, Cunha F de Q, Teixeira MM. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice [Internet]. British Journal of Pharmacology. 2008 ; 154( 2): 460-470.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/bjp.2008.94
Artigo de periodico
Commensal microbiota is fundamental for the development of inflammatory pain (2008)
Amatal, F. A.; Sachs, D.; Costa, V. V.; Fagundes, C. T.; Cisalpino, D.; Cunha, T. M.; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Silva, T. A.; Nicoli, J. R.; Vieira, L. Q.; Souza, D. G.; Teixeira, M. M.
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: CITOCINAS, DOR, NOCICEPTORES
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ABNT
AMATAL, F. A. et al. Commensal microbiota is fundamental for the development of inflammatory pain. Proceedings of the National Academy of Sciences, v. 105, n. 6, p. 2193-2197, 2008Tradução . . Disponível em: https://doi.org/10.1073/pnas.0711891105. Acesso em: 02 nov. 2024.
APA
Amatal, F. A., Sachs, D., Costa, V. V., Fagundes, C. T., Cisalpino, D., Cunha, T. M., et al. (2008). Commensal microbiota is fundamental for the development of inflammatory pain. Proceedings of the National Academy of Sciences, 105( 6), 2193-2197. doi:10.1073/pnas.0711891105
NLM
Amatal FA, Sachs D, Costa VV, Fagundes CT, Cisalpino D, Cunha TM, Ferreira SH, Cunha F de Q, Silva TA, Nicoli JR, Vieira LQ, Souza DG, Teixeira MM. Commensal microbiota is fundamental for the development of inflammatory pain [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 6): 2193-2197.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0711891105
Vancouver
Amatal FA, Sachs D, Costa VV, Fagundes CT, Cisalpino D, Cunha TM, Ferreira SH, Cunha F de Q, Silva TA, Nicoli JR, Vieira LQ, Souza DG, Teixeira MM. Commensal microbiota is fundamental for the development of inflammatory pain [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 6): 2193-2197.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0711891105
Artigo de periodico
Crucial role of neutrophils in the development of mechanical inflammatory hypernociception (2008)
Cunha, Thiago M.; Verri Júnior, Waldiceu A.; Schivo, Ieda R.; Napimoga, Marcelo H.; Parada, Carlos Amilcar; Poole, Stephen; Teixeira, Mauro M.; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Journal of Leukocyte Biology. Unidade: FMRP
Subjects: DOR, NOCICEPTORES, NEUTRÓFILOS
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ABNT
CUNHA, Thiago M. et al. Crucial role of neutrophils in the development of mechanical inflammatory hypernociception. Journal of Leukocyte Biology, v. 83, n. 4, p. 824-832, 2008Tradução . . Disponível em: https://doi.org/10.1189/jlb.0907654. Acesso em: 02 nov. 2024.
APA
Cunha, T. M., Verri Júnior, W. A., Schivo, I. R., Napimoga, M. H., Parada, C. A., Poole, S., et al. (2008). Crucial role of neutrophils in the development of mechanical inflammatory hypernociception. Journal of Leukocyte Biology, 83( 4), 824-832. doi:10.1189/jlb.0907654
NLM
Cunha TM, Verri Júnior WA, Schivo IR, Napimoga MH, Parada CA, Poole S, Teixeira MM, Ferreira SH, Cunha F de Q. Crucial role of neutrophils in the development of mechanical inflammatory hypernociception [Internet]. Journal of Leukocyte Biology. 2008 ; 83( 4): 824-832.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1189/jlb.0907654
Vancouver
Cunha TM, Verri Júnior WA, Schivo IR, Napimoga MH, Parada CA, Poole S, Teixeira MM, Ferreira SH, Cunha F de Q. Crucial role of neutrophils in the development of mechanical inflammatory hypernociception [Internet]. Journal of Leukocyte Biology. 2008 ; 83( 4): 824-832.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1189/jlb.0907654
Artigo de periodico
Leukotriene'B IND.4'is essential for selective eosinophil recruitment following allergen challenge of 'CD4 POT.+' cells in a model of chronic eosinophilic inflammation (2008)
Cheraim, Alessandra Bonacini; Xavier-Elsas, Pedro; Oliveira, Sandra Helena Penha de; Batistella, Tiago; Russo, Momtchilo; Gaspar-Elsas, Maria Ignez; Cunha, Fernando de Queiroz
Source: Life Sciences. Unidades: FMRP, ICB
Subjects: INFLAMAÇÃO, LEUCOTRIENOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHERAIM, Alessandra Bonacini et al. Leukotriene'B IND.4'is essential for selective eosinophil recruitment following allergen challenge of 'CD4 POT.+' cells in a model of chronic eosinophilic inflammation. Life Sciences, v. 83, n. 5-6, p. 214-222, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2008.06.004. Acesso em: 02 nov. 2024.
APA
Cheraim, A. B., Xavier-Elsas, P., Oliveira, S. H. P. de, Batistella, T., Russo, M., Gaspar-Elsas, M. I., & Cunha, F. de Q. (2008). Leukotriene'B IND.4'is essential for selective eosinophil recruitment following allergen challenge of 'CD4 POT.+' cells in a model of chronic eosinophilic inflammation. Life Sciences, 83( 5-6), 214-222. doi:10.1016/j.lfs.2008.06.004
NLM
Cheraim AB, Xavier-Elsas P, Oliveira SHP de, Batistella T, Russo M, Gaspar-Elsas MI, Cunha F de Q. Leukotriene'B IND.4'is essential for selective eosinophil recruitment following allergen challenge of 'CD4 POT.+' cells in a model of chronic eosinophilic inflammation [Internet]. Life Sciences. 2008 ; 83( 5-6): 214-222.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2008.06.004
Vancouver
Cheraim AB, Xavier-Elsas P, Oliveira SHP de, Batistella T, Russo M, Gaspar-Elsas MI, Cunha F de Q. Leukotriene'B IND.4'is essential for selective eosinophil recruitment following allergen challenge of 'CD4 POT.+' cells in a model of chronic eosinophilic inflammation [Internet]. Life Sciences. 2008 ; 83( 5-6): 214-222.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2008.06.004
Artigo de periodico
CXCR2-specific chemokines mediate leukotriene 'B IND.4'-dependent recruitment of neutrophils to inflamed joints in mice with antigen-induced arthritis (2008)
Grespan, Renata; Fukada, Sandra Y.; Lemos, Henrique P.; Vieira, Silvio M.; Napimoga, Marcelo H.; Teixeira, Mauro M.; Fraser, Alasdair R.; Liew, Foo Y.; McInnes, Iain B.; Cunha, Fernando de Queiroz
Source: Arthritis & Rheumatism. Unidade: FMRP
Subjects: NEUTRÓFILOS, ARTRITE REUMATOIDE, TERAPÊUTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GRESPAN, Renata et al. CXCR2-specific chemokines mediate leukotriene 'B IND.4'-dependent recruitment of neutrophils to inflamed joints in mice with antigen-induced arthritis. Arthritis & Rheumatism, v. 58, n. 7, p. 230-240, 2008Tradução . . Acesso em: 02 nov. 2024.
APA
Grespan, R., Fukada, S. Y., Lemos, H. P., Vieira, S. M., Napimoga, M. H., Teixeira, M. M., et al. (2008). CXCR2-specific chemokines mediate leukotriene 'B IND.4'-dependent recruitment of neutrophils to inflamed joints in mice with antigen-induced arthritis. Arthritis & Rheumatism, 58( 7), 230-240.
NLM
Grespan R, Fukada SY, Lemos HP, Vieira SM, Napimoga MH, Teixeira MM, Fraser AR, Liew FY, McInnes IB, Cunha F de Q. CXCR2-specific chemokines mediate leukotriene 'B IND.4'-dependent recruitment of neutrophils to inflamed joints in mice with antigen-induced arthritis. Arthritis & Rheumatism. 2008 ; 58( 7): 230-240.[citado 2024 nov. 02 ]
Vancouver
Grespan R, Fukada SY, Lemos HP, Vieira SM, Napimoga MH, Teixeira MM, Fraser AR, Liew FY, McInnes IB, Cunha F de Q. CXCR2-specific chemokines mediate leukotriene 'B IND.4'-dependent recruitment of neutrophils to inflamed joints in mice with antigen-induced arthritis. Arthritis & Rheumatism. 2008 ; 58( 7): 230-240.[citado 2024 nov. 02 ]
Artigo de periodico
IL-33 mediates antigen-induced cutaneous and articular hypernociception in mice (2008)
Verri, Waldiceu A.; GUerrero, Ana T. G.; Fukada, Sandra Y.; Valerio, Daniel A.; Cunha, Thiago M.; Xu, Damo; Ferreira, Sérgio Henrique; Liew, Foo Y.; Cunha, Fernando de Queiroz
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: CITOCINAS, INFLAMAÇÃO, ARTRITE REUMATOIDE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VERRI, Waldiceu A. et al. IL-33 mediates antigen-induced cutaneous and articular hypernociception in mice. Proceedings of the National Academy of Sciences, v. 105, n. 7, p. 2723-2728, 2008Tradução . . Disponível em: https://doi.org/10.1073/pnas.0712116105. Acesso em: 02 nov. 2024.
APA
Verri, W. A., GUerrero, A. T. G., Fukada, S. Y., Valerio, D. A., Cunha, T. M., Xu, D., et al. (2008). IL-33 mediates antigen-induced cutaneous and articular hypernociception in mice. Proceedings of the National Academy of Sciences, 105( 7), 2723-2728. doi:10.1073/pnas.0712116105
NLM
Verri WA, GUerrero ATG, Fukada SY, Valerio DA, Cunha TM, Xu D, Ferreira SH, Liew FY, Cunha F de Q. IL-33 mediates antigen-induced cutaneous and articular hypernociception in mice [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 7): 2723-2728.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0712116105
Vancouver
Verri WA, GUerrero ATG, Fukada SY, Valerio DA, Cunha TM, Xu D, Ferreira SH, Liew FY, Cunha F de Q. IL-33 mediates antigen-induced cutaneous and articular hypernociception in mice [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 7): 2723-2728.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0712116105
Artigo de periodico
Teleantagonism: a pharmacodynamic property of the primary nociceptive neuron (2008)
Funez, Mani Indiana; Ferrari, Luiz Fernando; Duarte, Djane Braz; Sachs, Daniela; Cunha, Fernando de Queiroz; Lorenzetti, Berenice Borges; Parada, Carlos Amílcar; Ferreira, Sérgio Henrique
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: FARMACODINÂMICA, ANTAGONISMO DE DROGAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FUNEZ, Mani Indiana et al. Teleantagonism: a pharmacodynamic property of the primary nociceptive neuron. Proceedings of the National Academy of Sciences, v. 105, n. 49, p. 19038-19043, 2008Tradução . . Disponível em: https://doi.org/10.1073/pnas.0807922105. Acesso em: 02 nov. 2024.
APA
Funez, M. I., Ferrari, L. F., Duarte, D. B., Sachs, D., Cunha, F. de Q., Lorenzetti, B. B., et al. (2008). Teleantagonism: a pharmacodynamic property of the primary nociceptive neuron. Proceedings of the National Academy of Sciences, 105( 49), 19038-19043. doi:10.1073/pnas.0807922105
NLM
Funez MI, Ferrari LF, Duarte DB, Sachs D, Cunha F de Q, Lorenzetti BB, Parada CA, Ferreira SH. Teleantagonism: a pharmacodynamic property of the primary nociceptive neuron [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 49): 19038-19043.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0807922105
Vancouver
Funez MI, Ferrari LF, Duarte DB, Sachs D, Cunha F de Q, Lorenzetti BB, Parada CA, Ferreira SH. Teleantagonism: a pharmacodynamic property of the primary nociceptive neuron [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 49): 19038-19043.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1073/pnas.0807922105
Artigo de periodico
The role of neutrophils in severe sepsis (2008)
Alves-Filho, José C.; Freitas, Andressa de; Spiller, Fernando; Souto, Fabrício O.; Cunha, Fernando de Queiroz
Source: Shock : Injury, Inflammation, and Sepsis : laboratory and clinical approaches. Unidade: FMRP
Subjects: SEPSE (TRATAMENTO), NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALVES-FILHO, José C. et al. The role of neutrophils in severe sepsis. Shock : Injury, Inflammation, and Sepsis : laboratory and clinical approaches, v. 30, p. 3-9, 2008Tradução . . Acesso em: 02 nov. 2024.
APA
Alves-Filho, J. C., Freitas, A. de, Spiller, F., Souto, F. O., & Cunha, F. de Q. (2008). The role of neutrophils in severe sepsis. Shock : Injury, Inflammation, and Sepsis : laboratory and clinical approaches, 30, 3-9.
NLM
Alves-Filho JC, Freitas A de, Spiller F, Souto FO, Cunha F de Q. The role of neutrophils in severe sepsis. Shock : Injury, Inflammation, and Sepsis : laboratory and clinical approaches. 2008 ; 30 3-9.[citado 2024 nov. 02 ]
Vancouver
Alves-Filho JC, Freitas A de, Spiller F, Souto FO, Cunha F de Q. The role of neutrophils in severe sepsis. Shock : Injury, Inflammation, and Sepsis : laboratory and clinical approaches. 2008 ; 30 3-9.[citado 2024 nov. 02 ]
Artigo de periodico
Role of IL-18 in overt pain-like behaviour in mice (2008)
Verri Júnior, Waldiceu A.; Cunha, Thiago M.; Magro, Danilo A.; Domingues, Andressa C.; Vieira, Silvio M.; Souza, Guilherme R.; Liew, Foo Y.; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: DOR, NOCICEPTORES, ENDOTÉLIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VERRI JÚNIOR, Waldiceu A. et al. Role of IL-18 in overt pain-like behaviour in mice. European Journal of Pharmacology, v. 588, n. 2-3, p. 207-212, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.04.010. Acesso em: 02 nov. 2024.
APA
Verri Júnior, W. A., Cunha, T. M., Magro, D. A., Domingues, A. C., Vieira, S. M., Souza, G. R., et al. (2008). Role of IL-18 in overt pain-like behaviour in mice. European Journal of Pharmacology, 588( 2-3), 207-212. doi:10.1016/j.ejphar.2008.04.010
NLM
Verri Júnior WA, Cunha TM, Magro DA, Domingues AC, Vieira SM, Souza GR, Liew FY, Ferreira SH, Cunha F de Q. Role of IL-18 in overt pain-like behaviour in mice [Internet]. European Journal of Pharmacology. 2008 ; 588( 2-3): 207-212.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2008.04.010
Vancouver
Verri Júnior WA, Cunha TM, Magro DA, Domingues AC, Vieira SM, Souza GR, Liew FY, Ferreira SH, Cunha F de Q. Role of IL-18 in overt pain-like behaviour in mice [Internet]. European Journal of Pharmacology. 2008 ; 588( 2-3): 207-212.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2008.04.010
Artigo de periodico
Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway (2008)
Napimoga, Marcelo H.; Vieira, Silvio M.; Dal-Secco, Daniela; Freitas, Andressa; Souto, Fabrício O.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves-Filho, José C.; Grespan, Renata; Kawai, Toshihisa; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NAPIMOGA, Marcelo H. et al. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, v. 180, n. 1, p. 609-617, 2008Tradução . . Acesso em: 02 nov. 2024.
APA
Napimoga, M. H., Vieira, S. M., Dal-Secco, D., Freitas, A., Souto, F. O., Mestriner, F. L. A. C., et al. (2008). Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, 180( 1), 609-617.
NLM
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 nov. 02 ]
Vancouver
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 nov. 02 ]

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