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Parte de monografia/livro
What is the transcriptome and how it is evaluated (2022)
Assis, Amanda F.; Oliveira, Ernna Hérida Domingues de; Donate, Paula Barbim; Giuliatti, Silvana ; Nguyen, Catherine; Passos Junior, Geraldo Aleixo da Silva
Source: Transcriptomics in health and disease. Unidades: FMRP, FORP
Subjects: RNA MENSAGEIRO, SEQUENCIAMENTO GENÉTICO, ANÁLISE EM MICROSSÉRIES, GENOMAS, EXPRESSÃO GÊNICA
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ABNT
ASSIS, Amanda F. et al. What is the transcriptome and how it is evaluated. Transcriptomics in health and disease. Tradução . Cham: Springer, 2022. . Disponível em: https://doi.org/10.1007/978-3-030-87821-4_1. Acesso em: 07 nov. 2024.
APA
Assis, A. F., Oliveira, E. H. D. de, Donate, P. B., Giuliatti, S., Nguyen, C., & Passos Junior, G. A. da S. (2022). What is the transcriptome and how it is evaluated. In Transcriptomics in health and disease. Cham: Springer. doi:10.1007/978-3-030-87821-4_1
NLM
Assis AF, Oliveira EHD de, Donate PB, Giuliatti S, Nguyen C, Passos Junior GA da S. What is the transcriptome and how it is evaluated [Internet]. In: Transcriptomics in health and disease. Cham: Springer; 2022. [citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/978-3-030-87821-4_1
Vancouver
Assis AF, Oliveira EHD de, Donate PB, Giuliatti S, Nguyen C, Passos Junior GA da S. What is the transcriptome and how it is evaluated [Internet]. In: Transcriptomics in health and disease. Cham: Springer; 2022. [citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/978-3-030-87821-4_1
Artigo de periodico
Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization (2022)
Nogueira, Lucas Fabricio Bahia ; Cruz, Marcos Antônio Eufrasio ; Tovani, Camila Bussola; Lopes, Helena Bacha ; Beloti, Márcio Mateus ; Ciancaglini, Pietro ; Bottini, Massimo; Ramos, Ana Paula
Source: Colloids and Surfaces B: Biointerfaces. Unidades: FORP, FFCLRP
Subjects: NANOPARTÍCULAS, MEDICAMENTO, OSTEOCLASTO
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NOGUEIRA, Lucas Fabricio Bahia et al. Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization. Colloids and Surfaces B: Biointerfaces, v. 217, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.colsurfb.2022.112622. Acesso em: 07 nov. 2024.
APA
Nogueira, L. F. B., Cruz, M. A. E., Tovani, C. B., Lopes, H. B., Beloti, M. M., Ciancaglini, P., et al. (2022). Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization. Colloids and Surfaces B: Biointerfaces, 217, 1-9. doi:10.1016/j.colsurfb.2022.112622
NLM
Nogueira LFB, Cruz MAE, Tovani CB, Lopes HB, Beloti MM, Ciancaglini P, Bottini M, Ramos AP. Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization [Internet]. Colloids and Surfaces B: Biointerfaces. 2022 ; 217 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.colsurfb.2022.112622
Vancouver
Nogueira LFB, Cruz MAE, Tovani CB, Lopes HB, Beloti MM, Ciancaglini P, Bottini M, Ramos AP. Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization [Internet]. Colloids and Surfaces B: Biointerfaces. 2022 ; 217 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.colsurfb.2022.112622
Artigo de periodico
Doxycycline inhibits α-synuclein-associated pathologies in vitro and in vivo (2021)
Dominguez-Meijide, Antonio; Parrales, Valeria; Vasili, Eftychia; González-Lizárraga, Florencia; König, Annekatrin; Lázaro, Diana F.; Lannuzel, Annie; Haik, Stéphane; Guimarães, Elaine Aparecida Del Bel Belluz ; Chehín, Rosana; Raisman-Vozari, Rita; Michel, Patrick P.; Bizat, Nicolas; Outeiro, Tiago Fleming
Source: Neurobiology of Disease. Unidade: FORP
Subjects: CAENORHABDITIS, LINHAGEM CELULAR, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON
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DOMINGUEZ-MEIJIDE, Antonio et al. Doxycycline inhibits α-synuclein-associated pathologies in vitro and in vivo. Neurobiology of Disease, v. 151, p. 1-19, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.nbd.2021.105256. Acesso em: 07 nov. 2024.
APA
Dominguez-Meijide, A., Parrales, V., Vasili, E., González-Lizárraga, F., König, A., Lázaro, D. F., et al. (2021). Doxycycline inhibits α-synuclein-associated pathologies in vitro and in vivo. Neurobiology of Disease, 151, 1-19. doi:10.1016/j.nbd.2021.105256
NLM
Dominguez-Meijide A, Parrales V, Vasili E, González-Lizárraga F, König A, Lázaro DF, Lannuzel A, Haik S, Guimarães EADBB, Chehín R, Raisman-Vozari R, Michel PP, Bizat N, Outeiro TF. Doxycycline inhibits α-synuclein-associated pathologies in vitro and in vivo [Internet]. Neurobiology of Disease. 2021 ; 151 1-19.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.nbd.2021.105256
Vancouver
Dominguez-Meijide A, Parrales V, Vasili E, González-Lizárraga F, König A, Lázaro DF, Lannuzel A, Haik S, Guimarães EADBB, Chehín R, Raisman-Vozari R, Michel PP, Bizat N, Outeiro TF. Doxycycline inhibits α-synuclein-associated pathologies in vitro and in vivo [Internet]. Neurobiology of Disease. 2021 ; 151 1-19.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.nbd.2021.105256
Artigo de periodico
The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress (2021)
Ferreira Junior, Nilson Carlos; Pereira, Maurício dos Santos ; Francis, Nour; Ramirez, Paola; Martorell, Paula; González-Lizarraga, Florencia; Figadère, Bruno; Chehin, Rosana; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick Pierre
Source: Cells. Unidade: FORP
Subjects: ESTRESSE OXIDATIVO, GLICOSE, METABOLISMO, ANTIBIÓTICOS, AGENTES ANTIMICROBIANOS, FARMACOLOGIA EXPERIMENTAL
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FERREIRA JUNIOR, Nilson Carlos et al. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, v. 10, n. 8, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10082163. Acesso em: 07 nov. 2024.
APA
Ferreira Junior, N. C., Pereira, M. dos S., Francis, N., Ramirez, P., Martorell, P., González-Lizarraga, F., et al. (2021). The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, 10( 8), 1-16. doi:10.3390/cells10082163
NLM
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells10082163
Vancouver
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells10082163
Artigo de periodico
Doxycycline interferes with tau aggregation and reduces its neuronal toxicity (2021)
Medina, Luciana; González-Lizárraga, Florencia; Dominguez-Meijide, Antonio; Ploper, Diego; Parrales, Valeria; Sequeira, Sabrina; Cima-Omori, Maria-Sol; Zweckstetter, Markus; Guimarães, Elaine Aparecida Del Bel Belluz ; Michel, Patrick P.; Outeiro, Tiago Fleming; Raisman-Vozari, Rita; Chehín, Rosana; Socias, Sergio B.
Source: Frontiers in Aging Neuroscience. Unidade: FORP
Subjects: DOENÇA DE ALZHEIMER, ANTIBIÓTICOS, DOENÇAS NEURODEGENERATIVAS
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MEDINA, Luciana et al. Doxycycline interferes with tau aggregation and reduces its neuronal toxicity. Frontiers in Aging Neuroscience, v. 13, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.3389/fnagi.2021.635760. Acesso em: 07 nov. 2024.
APA
Medina, L., González-Lizárraga, F., Dominguez-Meijide, A., Ploper, D., Parrales, V., Sequeira, S., et al. (2021). Doxycycline interferes with tau aggregation and reduces its neuronal toxicity. Frontiers in Aging Neuroscience, 13, 1-13. doi:10.3389/fnagi.2021.635760
NLM
Medina L, González-Lizárraga F, Dominguez-Meijide A, Ploper D, Parrales V, Sequeira S, Cima-Omori M-S, Zweckstetter M, Guimarães EADBB, Michel PP, Outeiro TF, Raisman-Vozari R, Chehín R, Socias SB. Doxycycline interferes with tau aggregation and reduces its neuronal toxicity [Internet]. Frontiers in Aging Neuroscience. 2021 ; 13 1-13.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fnagi.2021.635760
Vancouver
Medina L, González-Lizárraga F, Dominguez-Meijide A, Ploper D, Parrales V, Sequeira S, Cima-Omori M-S, Zweckstetter M, Guimarães EADBB, Michel PP, Outeiro TF, Raisman-Vozari R, Chehín R, Socias SB. Doxycycline interferes with tau aggregation and reduces its neuronal toxicity [Internet]. Frontiers in Aging Neuroscience. 2021 ; 13 1-13.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fnagi.2021.635760
Artigo de periodico
Doxycycline and its derivative, COL‐3, decrease dyskinesia induced by L-DOPA in hemiparkinsonian rats (2021)
Bortolanza, Mariza ; Nascimento, Glauce Crivelaro do ; Raisman‐Vozari, Rita; Del Bel, Elaine Aparecida
Source: British Journal of Pharmacology. Unidades: FMRP, FORP
Subjects: FÁRMACOS (SISTEMA NERVOSO CENTRAL), DOENÇA DE PARKINSON, ASTRÓCITOS, INFLAMAÇÃO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
BORTOLANZA, Mariza et al. Doxycycline and its derivative, COL‐3, decrease dyskinesia induced by L-DOPA in hemiparkinsonian rats. British Journal of Pharmacology, v. 178, n. 13, p. 2595-2616, 2021Tradução . . Disponível em: https://doi.org/10.1111/bph.15439. Acesso em: 07 nov. 2024.
APA
Bortolanza, M., Nascimento, G. C. do, Raisman‐Vozari, R., & Del Bel, E. A. (2021). Doxycycline and its derivative, COL‐3, decrease dyskinesia induced by L-DOPA in hemiparkinsonian rats. British Journal of Pharmacology, 178( 13), 2595-2616. doi:10.1111/bph.15439
NLM
Bortolanza M, Nascimento GC do, Raisman‐Vozari R, Del Bel EA. Doxycycline and its derivative, COL‐3, decrease dyskinesia induced by L-DOPA in hemiparkinsonian rats [Internet]. British Journal of Pharmacology. 2021 ; 178( 13): 2595-2616.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/bph.15439
Vancouver
Bortolanza M, Nascimento GC do, Raisman‐Vozari R, Del Bel EA. Doxycycline and its derivative, COL‐3, decrease dyskinesia induced by L-DOPA in hemiparkinsonian rats [Internet]. British Journal of Pharmacology. 2021 ; 178( 13): 2595-2616.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/bph.15439
Artigo de periodico
Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease (2021)
Pereira, Maurício dos Santos ; Abreu, Gabriel Henrique Dias; Rocca, Jeremy; Hamadat, Sabah; Raisman-Vozari, Rita; Michel, Patrick Pierre; Del Bel, Elaine Aparecida
Source: Frontiers in Pharmacology. Unidade: FORP
Subjects: ASTROCITOMA, CANABINOIDES, GLUTAMATOS
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ABNT
PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 07 nov. 2024.
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Del Bel, E. A. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Artigo de periodico
Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease (2021)
Pereira, Maurício dos Santos; Abreu, Gabriel Henrique Dias; Rocca, Jeremy; Hamadat, Sabah; Raisman-Vozari, Rita; Michel, Patrick Pierre; Guimarães, Elaine Aparecida Del Bel Belluz
Source: Frontiers in Pharmacology. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, DISCINESIAS, LESÃO CEREBRAL CRÔNICA, CAMUNDONGOS
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ABNT
PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 07 nov. 2024.
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Guimarães, E. A. D. B. B. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Guimarães EADBB. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Guimarães EADBB. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 nov. 07 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Artigo de periodico
Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption (2020)
Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick P.
Source: Glia. Unidades: FMRP, FORP
Subjects: CANABINOIDES, GLICOSE, ESTRESSE OXIDATIVO
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ABNT
PEREIRA, Maurício dos Santos et al. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, v. 68, n. 3, p. 561-573, 2020Tradução . . Disponível em: https://doi.org/10.1002/glia.23738. Acesso em: 07 nov. 2024.
APA
Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., Raisman-Vozari, R., & Michel, P. P. (2020). Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, 68( 3), 561-573. doi:10.1002/glia.23738
NLM
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1002/glia.23738
Vancouver
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1002/glia.23738
Artigo de periodico
CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects (2020)
González-Lizárraga, Florencia; Ploper, Diego; Ávila, César L.; Socías, Sergio B.; Pereira, Mauricio dos Santos; Machín, Belén; Del Bel, Elaine Aparecida ; Michel, Patrick Pierre; Pietrasanta, Lía I.; Raisman-Vozari, Rita; Chehín, Rosana
Source: Scientific Reports. Unidade: FORP
Subjects: REPOSICIONAMENTO DE FÁRMACOS, INFLAMAÇÃO, DOENÇA DE PARKINSON, FARMACOTERAPIA
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ABNT
GONZÁLEZ-LIZÁRRAGA, Florencia et al. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects. Scientific Reports, v. 10, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-76927-0. Acesso em: 07 nov. 2024.
APA
González-Lizárraga, F., Ploper, D., Ávila, C. L., Socías, S. B., Pereira, M. dos S., Machín, B., et al. (2020). CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects. Scientific Reports, 10( 1). doi:10.1038/s41598-020-76927-0
NLM
González-Lizárraga F, Ploper D, Ávila CL, Socías SB, Pereira M dos S, Machín B, Del Bel EA, Michel PP, Pietrasanta LI, Raisman-Vozari R, Chehín R. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/s41598-020-76927-0
Vancouver
González-Lizárraga F, Ploper D, Ávila CL, Socías SB, Pereira M dos S, Machín B, Del Bel EA, Michel PP, Pietrasanta LI, Raisman-Vozari R, Chehín R. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/s41598-020-76927-0
Artigo de periodico
Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates (2019)
Acuña, Leonardo; Hamadat, Sabah; Corbalán, Natalia S.; González-Lizárraga, Florencia; Pereira, Maurício dos Santos ; Rocca, Jérémy; Díaz, Julia Sepúlveda; Del-Bel, Elaine Aparecida; Papy-García, Dulce; Chehín, Rosana N.; Michel, Patrick P.; Raisman-Vozari, Rita
Source: Cells. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, INFLAMAÇÃO, CITOCINAS, NEURÔNIOS, ANTIBIÓTICOS
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ABNT
ACUÑA, Leonardo et al. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates. Cells, v. 8, n. 8, p. [17] , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8080776. Acesso em: 07 nov. 2024.
APA
Acuña, L., Hamadat, S., Corbalán, N. S., González-Lizárraga, F., Pereira, M. dos S., Rocca, J., et al. (2019). Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates. Cells, 8( 8), [17] . doi:10.3390/cells8080776
NLM
Acuña L, Hamadat S, Corbalán NS, González-Lizárraga F, Pereira M dos S, Rocca J, Díaz JS, Del-Bel EA, Papy-García D, Chehín RN, Michel PP, Raisman-Vozari R. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates [Internet]. Cells. 2019 ; 8( 8): [17] .[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells8080776
Vancouver
Acuña L, Hamadat S, Corbalán NS, González-Lizárraga F, Pereira M dos S, Rocca J, Díaz JS, Del-Bel EA, Papy-García D, Chehín RN, Michel PP, Raisman-Vozari R. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates [Internet]. Cells. 2019 ; 8( 8): [17] .[citado 2024 nov. 07 ] Available from: https://doi.org/10.3390/cells8080776
Artigo de periodico
The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells (2019)
Melo-Lima, Breno Luiz ; Poras, Isabelle; Passos, Geraldo Aleixo Silva ; Carosella, Edgardo D.; Donadi, Eduardo Antônio ; Moreau, Philippe
Source: Immunology. Unidades: FORP, FMRP
Subjects: TIMO (SISTEMAS SANGUÍNEO E IMUNE), AUTOIMUNIDADE, ANTÍGENOS
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ABNT
MELO-LIMA, Breno Luiz et al. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells. Immunology, v. 158, p. 121-135, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13099. Acesso em: 07 nov. 2024.
APA
Melo-Lima, B. L., Poras, I., Passos, G. A. S., Carosella, E. D., Donadi, E. A., & Moreau, P. (2019). The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells. Immunology, 158, 121-135. doi:10.1111/imm.13099
NLM
Melo-Lima BL, Poras I, Passos GAS, Carosella ED, Donadi EA, Moreau P. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells [Internet]. Immunology. 2019 ; 158 121-135.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/imm.13099
Vancouver
Melo-Lima BL, Poras I, Passos GAS, Carosella ED, Donadi EA, Moreau P. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells [Internet]. Immunology. 2019 ; 158 121-135.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/imm.13099
Artigo de periodico
Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia (2019)
Santos, Keila Dayane Bariotto dos; Padovan‐Neto, Fernando Eduardo; Bortolanza, Mariza; Pereira, Maurício dos Santos; Raisman-Vozari, Rita; Tumas, Vitor; Del Bel, Elaine Aparecida
Source: European Journal of Neuroscience. Unidades: FMRP, FORP
Subjects: DOENÇA DE PARKINSON, AZUL DE METILENO, ÓXIDO NÍTRICO
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ABNT
SANTOS, Keila Dayane Bariotto dos et al. Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia. European Journal of Neuroscience, v. 49, n. 6, p. 869–882, 2019Tradução . . Disponível em: https://doi.org/10.1111/ejn.14079. Acesso em: 07 nov. 2024.
APA
Santos, K. D. B. dos, Padovan‐Neto, F. E., Bortolanza, M., Pereira, M. dos S., Raisman-Vozari, R., Tumas, V., & Del Bel, E. A. (2019). Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia. European Journal of Neuroscience, 49( 6), 869–882. doi:10.1111/ejn.14079
NLM
Santos KDB dos, Padovan‐Neto FE, Bortolanza M, Pereira M dos S, Raisman-Vozari R, Tumas V, Del Bel EA. Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia [Internet]. European Journal of Neuroscience. 2019 ; 49( 6): 869–882.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/ejn.14079
Vancouver
Santos KDB dos, Padovan‐Neto FE, Bortolanza M, Pereira M dos S, Raisman-Vozari R, Tumas V, Del Bel EA. Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia [Internet]. European Journal of Neuroscience. 2019 ; 49( 6): 869–882.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/ejn.14079
Artigo de periodico
Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis (2018)
Pólvora, Tábata Larissa Santos; Nobre, Átila Vinícius Vitor; Tirapelli, Camila ; Taba Júnior, Mário ; Macedo, Leandro Dorigan de; Santana, Rodrigo de Carvalho ; Pozzetto, Bruno ; Lourenço, Alan Grupioni ; Motta, Ana Carolina Fragoso
Source: Expert Review of Clinical Immunology. Unidades: FORP, FMRP, FO
Subjects: INFECÇÕES POR HIV, DOENÇAS PERIODONTAIS, SALIVA, ANTIRRETROVIRAIS, BIOMARCADORES, PERIODONTITE CRÔNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PÓLVORA, Tábata Larissa Santos et al. Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis. Expert Review of Clinical Immunology, v. 14, n. 4, p. 315-327, 2018Tradução . . Disponível em: https://doi.org/10.1080/1744666x.2018.1459571. Acesso em: 07 nov. 2024.
APA
Pólvora, T. L. S., Nobre, Á. V. V., Tirapelli, C., Taba Júnior, M., Macedo, L. D. de, Santana, R. de C., et al. (2018). Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis. Expert Review of Clinical Immunology, 14( 4), 315-327. doi:10.1080/1744666x.2018.1459571
NLM
Pólvora TLS, Nobre ÁVV, Tirapelli C, Taba Júnior M, Macedo LD de, Santana R de C, Pozzetto B, Lourenço AG, Motta ACF. Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis [Internet]. Expert Review of Clinical Immunology. 2018 ; 14( 4): 315-327.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1080/1744666x.2018.1459571
Vancouver
Pólvora TLS, Nobre ÁVV, Tirapelli C, Taba Júnior M, Macedo LD de, Santana R de C, Pozzetto B, Lourenço AG, Motta ACF. Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis [Internet]. Expert Review of Clinical Immunology. 2018 ; 14( 4): 315-327.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1080/1744666x.2018.1459571
Artigo de periodico
Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors (2018)
Sonego, Andreza B.; Prado, Douglas S.; Vale, Gabriel T.; Sepulveda-Diaz, Julia E.; Cunha, Thiago Mattar; Tirapelli, Carlos Renato ; Del Bel, Elaine A.; Raisman-Vozari, Rita; Guimarães, Francisco Silveira
Source: Brain, Behavior, and Immunity. Unidades: FMRP, EERP, FORP
Subjects: CANABINOIDES, INFLAMAÇÃO, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SONEGO, Andreza B. et al. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors. Brain, Behavior, and Immunity, v. 74, p. 241-251, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2018.09.014. Acesso em: 07 nov. 2024.
APA
Sonego, A. B., Prado, D. S., Vale, G. T., Sepulveda-Diaz, J. E., Cunha, T. M., Tirapelli, C. R., et al. (2018). Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors. Brain, Behavior, and Immunity, 74, 241-251. doi:10.1016/j.bbi.2018.09.014
NLM
Sonego AB, Prado DS, Vale GT, Sepulveda-Diaz JE, Cunha TM, Tirapelli CR, Del Bel EA, Raisman-Vozari R, Guimarães FS. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors [Internet]. Brain, Behavior, and Immunity. 2018 ; 74 241-251.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.014
Vancouver
Sonego AB, Prado DS, Vale GT, Sepulveda-Diaz JE, Cunha TM, Tirapelli CR, Del Bel EA, Raisman-Vozari R, Guimarães FS. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors [Internet]. Brain, Behavior, and Immunity. 2018 ; 74 241-251.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.014
Artigo de periodico
Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats (2017)
Santos-Junior, Nilton Nascimento; Costa, Luís Henrique Angenendt; Catalão, Carlos Henrique Rocha; Kanashiro, Alexandre; Sharshar, Tarek; Rocha, Maria José Alves da
Source: Pituitary. Unidade: FORP
Subjects: CITOCINAS, SEPSE, SISTEMA NERVOSO CENTRAL, VASOPRESSINAS, OCITOCINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS-JUNIOR, Nilton Nascimento et al. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, v. 20, n. 5, p. 515-521, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11102-017-0812-z. Acesso em: 07 nov. 2024.
APA
Santos-Junior, N. N., Costa, L. H. A., Catalão, C. H. R., Kanashiro, A., Sharshar, T., & Rocha, M. J. A. da. (2017). Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, 20( 5), 515-521. doi:10.1007/s11102-017-0812-z
NLM
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Vancouver
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Artigo de periodico
Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species (2017)
González-Lizárraga, Florencia; Socías, Sergio B; Ávila, César L; Torres-Bugeau, Clarisa M; Barbosa, Leandro Ramos Souza; Binolfi, Andres; Sepúlveda-Díaz, Julia E; Del-Bel, Elaine; Fernandez, Claudio O; Papy-Garcia, Dulce; Itri, Rosangela; Raisman-Vozari Rita; Chehín, Rosana N
Source: Scientific Reports. Unidades: IF, FORP
Subjects: DOENÇAS NEURODEGENERATIVAS, PROTEÍNAS, BIOFÍSICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZÁLEZ-LIZÁRRAGA, Florencia et al. Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species. Scientific Reports, v. 7, n. 41755, p. 1-13, 2017Tradução . . Disponível em: https://doi.org/10.1038/srep41755. Acesso em: 07 nov. 2024.
APA
González-Lizárraga, F., Socías, S. B., Ávila, C. L., Torres-Bugeau, C. M., Barbosa, L. R. S., Binolfi, A., et al. (2017). Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species. Scientific Reports, 7( 41755), 1-13. doi:10.1038/srep41755
NLM
González-Lizárraga F, Socías SB, Ávila CL, Torres-Bugeau CM, Barbosa LRS, Binolfi A, Sepúlveda-Díaz JE, Del-Bel E, Fernandez CO, Papy-Garcia D, Itri R, Raisman-Vozari Rita, Chehín RN. Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species [Internet]. Scientific Reports. 2017 ; 7( 41755): 1-13.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/srep41755
Vancouver
González-Lizárraga F, Socías SB, Ávila CL, Torres-Bugeau CM, Barbosa LRS, Binolfi A, Sepúlveda-Díaz JE, Del-Bel E, Fernandez CO, Papy-Garcia D, Itri R, Raisman-Vozari Rita, Chehín RN. Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species [Internet]. Scientific Reports. 2017 ; 7( 41755): 1-13.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/srep41755
Artigo de periodico
Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides (2016)
Reglodi, Dora; Renaud, Justine; Tamas, Andrea; Tizabi, Yousef; Socías, Sergio B; Del Bel, Elaine Aparecida; Raisman-Vozari , Rita
Source: Progress in Neurobiology. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, ANTIBIÓTICOS, ESTRESSE OXIDATIVO, ANTIOXIDANTES, TERAPÊUTICA, COMPOSTOS FENÓLICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REGLODI, Dora et al. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides. Progress in Neurobiology, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.pneurobio.2015.10.004. Acesso em: 07 nov. 2024.
APA
Reglodi, D., Renaud, J., Tamas, A., Tizabi, Y., Socías, S. B., Del Bel, E. A., & Raisman-Vozari , R. (2016). Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides. Progress in Neurobiology. doi:10.1016/j.pneurobio.2015.10.004
NLM
Reglodi D, Renaud J, Tamas A, Tizabi Y, Socías SB, Del Bel EA, Raisman-Vozari R. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides [Internet]. Progress in Neurobiology. 2016 ;[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.pneurobio.2015.10.004
Vancouver
Reglodi D, Renaud J, Tamas A, Tizabi Y, Socías SB, Del Bel EA, Raisman-Vozari R. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides [Internet]. Progress in Neurobiology. 2016 ;[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.pneurobio.2015.10.004
Artigo de periodico
CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration (2016)
Tristão, Fabrine Sales Massafera; Lazzarini, Márcio; Martin, Sabine; Amar, Majid; Stühmer, Walter; Kirchhoff, Frank; Gomes, Lucas Araújo Caldi; Lanfumey, Laurance; Prediger, Rui D.; Sepulveda, Julia E.; Del Bel, Elaine Aparecida; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, INFLAMAÇÃO, DEGENERAÇÃO NEURAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TRISTÃO, Fabrine Sales Massafera et al. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, v. 29, n. 3, p. 364-380, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9557-5. Acesso em: 07 nov. 2024.
APA
Tristão, F. S. M., Lazzarini, M., Martin, S., Amar, M., Stühmer, W., Kirchhoff, F., et al. (2016). CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, 29( 3), 364-380. doi:10.1007/s12640-015-9557-5
NLM
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Vancouver
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Artigo de periodico
Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways (2016)
Santa-Cecília, Flávia V.; Socias, Benjamin; Ouidja, Mohand O.; Sepulveda-Diaz, Julia E.; Acuña, Leonardo; Silva, Rangel L.; Michel, Patrick P.; Del Bel, Elaine Aparecida; Cunha, Thiago Mattar; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidades: FORP, FMRP
Subjects: DOENÇAS NEURODEGENERATIVAS, CITOCINAS, DOENÇA DE PARKINSON, FÁRMACOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTA-CECÍLIA, Flávia V. et al. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, v. 29, n. 4, p. 447-459, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9592-2. Acesso em: 07 nov. 2024.
APA
Santa-Cecília, F. V., Socias, B., Ouidja, M. O., Sepulveda-Diaz, J. E., Acuña, L., Silva, R. L., et al. (2016). Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, 29( 4), 447-459. doi:10.1007/s12640-015-9592-2
NLM
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Vancouver
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1007/s12640-015-9592-2

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