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Artigo de periodico
The alternatively spliced RECK transcript variant 3 is a predictor of poor survival for melanoma patients being upregulated in aggressive cell lines and modulating MMP gene expression in vitro (2020)
Jacomasso, Thiago; Ribas, Hennrique Taborda; Lima, Marina Trombetta ; Konig, Michelle Silberspitz; Trindade, Edvaldo da Silva; Martinez, Glaucia Regina ; Sogayar, Mari Cleide ; Winnischofer, Sheila Maria Brochado
Source: Melanoma Research. Unidade: IQ
Subjects: MELANOMA, EXPRESSÃO GÊNICA, METALOPROTEINASES
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JACOMASSO, Thiago et al. The alternatively spliced RECK transcript variant 3 is a predictor of poor survival for melanoma patients being upregulated in aggressive cell lines and modulating MMP gene expression in vitro. Melanoma Research, v. 30, n. 3, p. 223-234, 2020Tradução . . Disponível em: https://doi.org/10.1097/CMR.0000000000000650. Acesso em: 12 nov. 2024.
APA
Jacomasso, T., Ribas, H. T., Lima, M. T., Konig, M. S., Trindade, E. da S., Martinez, G. R., et al. (2020). The alternatively spliced RECK transcript variant 3 is a predictor of poor survival for melanoma patients being upregulated in aggressive cell lines and modulating MMP gene expression in vitro. Melanoma Research, 30( 3), 223-234. doi:10.1097/CMR.0000000000000650
NLM
Jacomasso T, Ribas HT, Lima MT, Konig MS, Trindade E da S, Martinez GR, Sogayar MC, Winnischofer SMB. The alternatively spliced RECK transcript variant 3 is a predictor of poor survival for melanoma patients being upregulated in aggressive cell lines and modulating MMP gene expression in vitro [Internet]. Melanoma Research. 2020 ; 30( 3): 223-234.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1097/CMR.0000000000000650
Vancouver
Jacomasso T, Ribas HT, Lima MT, Konig MS, Trindade E da S, Martinez GR, Sogayar MC, Winnischofer SMB. The alternatively spliced RECK transcript variant 3 is a predictor of poor survival for melanoma patients being upregulated in aggressive cell lines and modulating MMP gene expression in vitro [Internet]. Melanoma Research. 2020 ; 30( 3): 223-234.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1097/CMR.0000000000000650
Artigo de periodico
Downregulation of reversion-inducing cysteine-rich protein with Kazal motifs in malignant melanoma: inverse correlation with membrane-type 1-matrix metalloproteinase and tissue inhibitor of metalloproteinase 2 (2014)
Jacomasso, Thiago; Trombetta-Lima, Marina; Sogayar, Mari Cleide ; Winnischofer, Sheila Maria Brochado
Source: Melanoma Research. Unidade: IQ
Subjects: MELANOMA, METALOPROTEINASES
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ABNT
JACOMASSO, Thiago et al. Downregulation of reversion-inducing cysteine-rich protein with Kazal motifs in malignant melanoma: inverse correlation with membrane-type 1-matrix metalloproteinase and tissue inhibitor of metalloproteinase 2. Melanoma Research, v. 24, n. 1, p. 32-39, 2014Tradução . . Disponível em: https://doi.org/10.1097/CMR.0000000000000039. Acesso em: 12 nov. 2024.
APA
Jacomasso, T., Trombetta-Lima, M., Sogayar, M. C., & Winnischofer, S. M. B. (2014). Downregulation of reversion-inducing cysteine-rich protein with Kazal motifs in malignant melanoma: inverse correlation with membrane-type 1-matrix metalloproteinase and tissue inhibitor of metalloproteinase 2. Melanoma Research, 24( 1), 32-39. doi:10.1097/CMR.0000000000000039
NLM
Jacomasso T, Trombetta-Lima M, Sogayar MC, Winnischofer SMB. Downregulation of reversion-inducing cysteine-rich protein with Kazal motifs in malignant melanoma: inverse correlation with membrane-type 1-matrix metalloproteinase and tissue inhibitor of metalloproteinase 2 [Internet]. Melanoma Research. 2014 ; 24( 1): 32-39.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1097/CMR.0000000000000039
Vancouver
Jacomasso T, Trombetta-Lima M, Sogayar MC, Winnischofer SMB. Downregulation of reversion-inducing cysteine-rich protein with Kazal motifs in malignant melanoma: inverse correlation with membrane-type 1-matrix metalloproteinase and tissue inhibitor of metalloproteinase 2 [Internet]. Melanoma Research. 2014 ; 24( 1): 32-39.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1097/CMR.0000000000000039
Trabalho de evento-resumo
The role of RECK in the anti-tumor activity of simvastatin in U87MG human glioblastoma cells (2012)
kenski, J. N. C; Jacomasso, Thiago; Bark, Jamil Mamedio; Lima, Marina Trombetta; Sogayar, Mari Cleide ; Winnischofer, Sheila Maria Brochado
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq). Unidade: IQ
Assunto: BIOQUÍMICA
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KENSKI, J. N. C et al. The role of RECK in the anti-tumor activity of simvastatin in U87MG human glioblastoma cells. 2012, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2012. . Acesso em: 12 nov. 2024.
APA
kenski, J. N. C., Jacomasso, T., Bark, J. M., Lima, M. T., Sogayar, M. C., & Winnischofer, S. M. B. (2012). The role of RECK in the anti-tumor activity of simvastatin in U87MG human glioblastoma cells. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
kenski JNC, Jacomasso T, Bark JM, Lima MT, Sogayar MC, Winnischofer SMB. The role of RECK in the anti-tumor activity of simvastatin in U87MG human glioblastoma cells. Program and Index. 2012 ;[citado 2024 nov. 12 ]
Vancouver
kenski JNC, Jacomasso T, Bark JM, Lima MT, Sogayar MC, Winnischofer SMB. The role of RECK in the anti-tumor activity of simvastatin in U87MG human glioblastoma cells. Program and Index. 2012 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Cloning and expression of three alternative isoforms of the RECK tumor suppressor gene in human malignant cell lines and E. coli BL21 DE3 (2011)
Lima, Marina Trombetta; Konig, Michelle Silberspitz; Nogueira, Heloísa Neumann; Carreira, Ana Claudia Oliveira; Demasi, Marcos Angelo Almeida; Colin, Christian; Winnischofer, Sheila Maria Brochado; Sogayar, Mari Cleide
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq). Unidade: IQ
Subjects: GENES SUPRESSORES DE TUMOR, ESCHERICHIA COLI
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ABNT
LIMA, Marina Trombetta et al. Cloning and expression of three alternative isoforms of the RECK tumor suppressor gene in human malignant cell lines and E. coli BL21 DE3. 2011, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2011. . Acesso em: 12 nov. 2024.
APA
Lima, M. T., Konig, M. S., Nogueira, H. N., Carreira, A. C. O., Demasi, M. A. A., Colin, C., et al. (2011). Cloning and expression of three alternative isoforms of the RECK tumor suppressor gene in human malignant cell lines and E. coli BL21 DE3. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Lima MT, Konig MS, Nogueira HN, Carreira ACO, Demasi MAA, Colin C, Winnischofer SMB, Sogayar MC. Cloning and expression of three alternative isoforms of the RECK tumor suppressor gene in human malignant cell lines and E. coli BL21 DE3. Program and Index. 2011 ;[citado 2024 nov. 12 ]
Vancouver
Lima MT, Konig MS, Nogueira HN, Carreira ACO, Demasi MAA, Colin C, Winnischofer SMB, Sogayar MC. Cloning and expression of three alternative isoforms of the RECK tumor suppressor gene in human malignant cell lines and E. coli BL21 DE3. Program and Index. 2011 ;[citado 2024 nov. 12 ]
Artigo de periodico
Involvement of catalase in the apoptotic mechanism induced by apigenin in HepG2 human hepatoma cells (2011)
Valdameri, Glaucio; Trombetta-Lima, Marina; Worfel, Paulo R; Pires, Amanda do Rocio Andrade; Martinez, Glaucia Regina; Noleto, Guilhermina R; Cadena, Silvia Maria Suter Correia; Sogayar, Mari Cleide ; Winnischofer, Sheila Maria Brochado
Source: Chemico-Biological Interactions. Unidade: IQ
Subjects: APOPTOSE, CATÁLISE
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ABNT
VALDAMERI, Glaucio et al. Involvement of catalase in the apoptotic mechanism induced by apigenin in HepG2 human hepatoma cells. Chemico-Biological Interactions, v. 193, n. 2, p. 180-189, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2011.06.009. Acesso em: 12 nov. 2024.
APA
Valdameri, G., Trombetta-Lima, M., Worfel, P. R., Pires, A. do R. A., Martinez, G. R., Noleto, G. R., et al. (2011). Involvement of catalase in the apoptotic mechanism induced by apigenin in HepG2 human hepatoma cells. Chemico-Biological Interactions, 193( 2), 180-189. doi:10.1016/j.cbi.2011.06.009
NLM
Valdameri G, Trombetta-Lima M, Worfel PR, Pires A do RA, Martinez GR, Noleto GR, Cadena SMSC, Sogayar MC, Winnischofer SMB. Involvement of catalase in the apoptotic mechanism induced by apigenin in HepG2 human hepatoma cells [Internet]. Chemico-Biological Interactions. 2011 ; 193( 2): 180-189.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.cbi.2011.06.009
Vancouver
Valdameri G, Trombetta-Lima M, Worfel PR, Pires A do RA, Martinez GR, Noleto GR, Cadena SMSC, Sogayar MC, Winnischofer SMB. Involvement of catalase in the apoptotic mechanism induced by apigenin in HepG2 human hepatoma cells [Internet]. Chemico-Biological Interactions. 2011 ; 193( 2): 180-189.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.cbi.2011.06.009
Trabalho de evento-resumo
Quercetin inhibits glioma cell proliferation and migration: role of MMP2 and TIMP1 (2011)
Amstalden, Hudson Gouveia; Jacomasso, Thiago; Lima, Marina Trombetta; Sogayar, Mari Cleide ; Martinez, Glaucia Regina; Winnischofer, Sheila Maria Brochado
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq). Unidade: IQ
Subjects: PROLIFERAÇÃO CELULAR, BIOQUÍMICA
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AMSTALDEN, Hudson Gouveia et al. Quercetin inhibits glioma cell proliferation and migration: role of MMP2 and TIMP1. 2011, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2011. . Acesso em: 12 nov. 2024.
APA
Amstalden, H. G., Jacomasso, T., Lima, M. T., Sogayar, M. C., Martinez, G. R., & Winnischofer, S. M. B. (2011). Quercetin inhibits glioma cell proliferation and migration: role of MMP2 and TIMP1. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Amstalden HG, Jacomasso T, Lima MT, Sogayar MC, Martinez GR, Winnischofer SMB. Quercetin inhibits glioma cell proliferation and migration: role of MMP2 and TIMP1. Program and Index. 2011 ;[citado 2024 nov. 12 ]
Vancouver
Amstalden HG, Jacomasso T, Lima MT, Sogayar MC, Martinez GR, Winnischofer SMB. Quercetin inhibits glioma cell proliferation and migration: role of MMP2 and TIMP1. Program and Index. 2011 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo periodico
Characterization of three novel splice variants of the RECK tumor and metastasis suppressor gene: correlation with glioma progression (2011)
Lima, Marina Trombetta; Winnischofer, Sheila Maria Brochado; Carreira, Ana Claudia Oliveira; Demasi, Marcos Angelo Almeida; Coli, Christian; Oba-Shinjo, Sueli Mieko; Marie, Suely Kazue Nagahashi; Sogayar, Mari Cleide
Source: Clinical & Experimental Metastasis. Conference titles: Joint Metastasis Research Society (MRS) and American Association for Cancer Research (AACR) Conference: Metastais and the Tumor Microenvironment. Unidades: FM, IQ
Subjects: GENES SUPRESSORES DE TUMOR, METÁSTASE NEOPLÁSICA
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LIMA, Marina Trombetta et al. Characterization of three novel splice variants of the RECK tumor and metastasis suppressor gene: correlation with glioma progression. Clinical & Experimental Metastasis. Dordrecht: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1007/s10585-010-9361-9. Acesso em: 12 nov. 2024. , 2011
APA
Lima, M. T., Winnischofer, S. M. B., Carreira, A. C. O., Demasi, M. A. A., Coli, C., Oba-Shinjo, S. M., et al. (2011). Characterization of three novel splice variants of the RECK tumor and metastasis suppressor gene: correlation with glioma progression. Clinical & Experimental Metastasis. Dordrecht: Faculdade de Medicina, Universidade de São Paulo. doi:10.1007/s10585-010-9361-9
NLM
Lima MT, Winnischofer SMB, Carreira ACO, Demasi MAA, Coli C, Oba-Shinjo SM, Marie SKN, Sogayar MC. Characterization of three novel splice variants of the RECK tumor and metastasis suppressor gene: correlation with glioma progression [Internet]. Clinical & Experimental Metastasis. 2011 ; 28( 2): 231 res. B65.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s10585-010-9361-9
Vancouver
Lima MT, Winnischofer SMB, Carreira ACO, Demasi MAA, Coli C, Oba-Shinjo SM, Marie SKN, Sogayar MC. Characterization of three novel splice variants of the RECK tumor and metastasis suppressor gene: correlation with glioma progression [Internet]. Clinical & Experimental Metastasis. 2011 ; 28( 2): 231 res. B65.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s10585-010-9361-9
Trabalho de evento-resumo
Three novel splice variants of the RECK tumor and metastasis supressor gene (2011)
Lima, Marina Trombetta; Demasi, Marcos Angelo Almeida; Carreira, Ana Claudia Oliveira; Colin, Christian; Marie, Suely Kazue Nagahashi; Shinjo, Sueli Mieko Oba; Winnischofer, Sheila Maria Brochado; Sogayar, Mari Cleide
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq). Unidades: FM, IQ
Subjects: GENES SUPRESSORES DE TUMOR, BIOQUÍMICA
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ABNT
LIMA, Marina Trombetta et al. Three novel splice variants of the RECK tumor and metastasis supressor gene. 2011, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2011. . Acesso em: 12 nov. 2024.
APA
Lima, M. T., Demasi, M. A. A., Carreira, A. C. O., Colin, C., Marie, S. K. N., Shinjo, S. M. O., et al. (2011). Three novel splice variants of the RECK tumor and metastasis supressor gene. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Lima MT, Demasi MAA, Carreira ACO, Colin C, Marie SKN, Shinjo SMO, Winnischofer SMB, Sogayar MC. Three novel splice variants of the RECK tumor and metastasis supressor gene. Program and Index. 2011 ;[citado 2024 nov. 12 ]
Vancouver
Lima MT, Demasi MAA, Carreira ACO, Colin C, Marie SKN, Shinjo SMO, Winnischofer SMB, Sogayar MC. Three novel splice variants of the RECK tumor and metastasis supressor gene. Program and Index. 2011 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Involvement of ROS on the apoptotic mechanism induced by apigenin in HepG2 cells (2010)
Valdameri, G; Lima, Maria Trombeta; Sogayar, Mari Cleide ; Pires, A. R. A; Winnischofer, Sheila Maria Brochado; Rocha, M. E. M
Source: Abstracts. Conference titles: Annual Meeting of Brazilian Biochemistry and Molecular Biology (SBBq). Unidade: IQ
Subjects: APOPTOSE, BIOQUÍMICA
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ABNT
VALDAMERI, G et al. Involvement of ROS on the apoptotic mechanism induced by apigenin in HepG2 cells. 2010, Anais.. São Paulo: SBBq, 2010. . Acesso em: 12 nov. 2024.
APA
Valdameri, G., Lima, M. T., Sogayar, M. C., Pires, A. R. A., Winnischofer, S. M. B., & Rocha, M. E. M. (2010). Involvement of ROS on the apoptotic mechanism induced by apigenin in HepG2 cells. In Abstracts. São Paulo: SBBq.
NLM
Valdameri G, Lima MT, Sogayar MC, Pires ARA, Winnischofer SMB, Rocha MEM. Involvement of ROS on the apoptotic mechanism induced by apigenin in HepG2 cells. Abstracts. 2010 ;[citado 2024 nov. 12 ]
Vancouver
Valdameri G, Lima MT, Sogayar MC, Pires ARA, Winnischofer SMB, Rocha MEM. Involvement of ROS on the apoptotic mechanism induced by apigenin in HepG2 cells. Abstracts. 2010 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Transcrito alternativo de RECK é regulado negativamente por quercetina em células de glioma humano U373MG (2010)
Amstalden, H. G; Sene, Reginaldo Vieira de; Jacomasso, T; kenski, J. N. C; Valdameri, G; Lima, M. T; Sogayar, Mari Cleide ; Martinez, G. R; Winnischofer, Sheila Maria Brochado
Source: Resumos. Conference titles: Congresso Brasileiro de Genética. Unidade: IQ
Subjects: GLICOPROTEÍNAS, NEOPLASIAS
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ABNT
AMSTALDEN, H. G et al. Transcrito alternativo de RECK é regulado negativamente por quercetina em células de glioma humano U373MG. 2010, Anais.. Ribeirão Preto: Sociedade Brasileira de Genética (SBG), 2010. . Acesso em: 12 nov. 2024.
APA
Amstalden, H. G., Sene, R. V. de, Jacomasso, T., kenski, J. N. C., Valdameri, G., Lima, M. T., et al. (2010). Transcrito alternativo de RECK é regulado negativamente por quercetina em células de glioma humano U373MG. In Resumos. Ribeirão Preto: Sociedade Brasileira de Genética (SBG).
NLM
Amstalden HG, Sene RV de, Jacomasso T, kenski JNC, Valdameri G, Lima MT, Sogayar MC, Martinez GR, Winnischofer SMB. Transcrito alternativo de RECK é regulado negativamente por quercetina em células de glioma humano U373MG. Resumos. 2010 ;[citado 2024 nov. 12 ]
Vancouver
Amstalden HG, Sene RV de, Jacomasso T, kenski JNC, Valdameri G, Lima MT, Sogayar MC, Martinez GR, Winnischofer SMB. Transcrito alternativo de RECK é regulado negativamente por quercetina em células de glioma humano U373MG. Resumos. 2010 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Alternatively spliced transcripts of the RECK gene are differentially expressed in a melanoma progression model (2010)
Jacomasso, T; Lima, M. T; kenski, J. N. C; Sogayar, Mari Cleide ; Winnischofer, Sheila Maria Brochado
Source: Abstracts. Conference titles: Annual Meeting of Brazilian Biochemistry and Molecular Biology (SBBq). Unidade: IQ
Subjects: MELANOMA, BIOQUÍMICA
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ABNT
JACOMASSO, T et al. Alternatively spliced transcripts of the RECK gene are differentially expressed in a melanoma progression model. 2010, Anais.. São Paulo: SBBq, 2010. . Acesso em: 12 nov. 2024.
APA
Jacomasso, T., Lima, M. T., kenski, J. N. C., Sogayar, M. C., & Winnischofer, S. M. B. (2010). Alternatively spliced transcripts of the RECK gene are differentially expressed in a melanoma progression model. In Abstracts. São Paulo: SBBq.
NLM
Jacomasso T, Lima MT, kenski JNC, Sogayar MC, Winnischofer SMB. Alternatively spliced transcripts of the RECK gene are differentially expressed in a melanoma progression model. Abstracts. 2010 ;[citado 2024 nov. 12 ]
Vancouver
Jacomasso T, Lima MT, kenski JNC, Sogayar MC, Winnischofer SMB. Alternatively spliced transcripts of the RECK gene are differentially expressed in a melanoma progression model. Abstracts. 2010 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Characterization on three novel splice variants of the reck tumor and metastasis supressor gene: correlation with glioma progression and MMP expression (2010)
Lima, Marina Trombetta; Winnischofer, Sheila Maria Brochado; Carreira, Ana Claudia Oliveira; Demasi, Marcos Angelo Almeida; Nogueira, H. N; Colin, Christian; Sogayar, Mari Cleide
Source: Abstracts. Conference titles: Annual Meeting of Brazilian Biochemistry and Molecular Biology (SBBq). Unidade: IQ
Subjects: METÁSTASE NEOPLÁSICA, GENES SUPRESSORES DE TUMOR
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ABNT
LIMA, Marina Trombetta et al. Characterization on three novel splice variants of the reck tumor and metastasis supressor gene: correlation with glioma progression and MMP expression. 2010, Anais.. São Paulo: SBBq, 2010. . Acesso em: 12 nov. 2024.
APA
Lima, M. T., Winnischofer, S. M. B., Carreira, A. C. O., Demasi, M. A. A., Nogueira, H. N., Colin, C., & Sogayar, M. C. (2010). Characterization on three novel splice variants of the reck tumor and metastasis supressor gene: correlation with glioma progression and MMP expression. In Abstracts. São Paulo: SBBq.
NLM
Lima MT, Winnischofer SMB, Carreira ACO, Demasi MAA, Nogueira HN, Colin C, Sogayar MC. Characterization on three novel splice variants of the reck tumor and metastasis supressor gene: correlation with glioma progression and MMP expression. Abstracts. 2010 ;[citado 2024 nov. 12 ]
Vancouver
Lima MT, Winnischofer SMB, Carreira ACO, Demasi MAA, Nogueira HN, Colin C, Sogayar MC. Characterization on three novel splice variants of the reck tumor and metastasis supressor gene: correlation with glioma progression and MMP expression. Abstracts. 2010 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Isolation and characterization of three novel isoforms of the RECK tumor suppressor gene (2009)
Lima, Marina Trombetta; Winnischofer, Sheila Maria Brochado; Colin, C.; Sogayar, Mari Cleide
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society. Unidade: IQ
Assunto: GENES SUPRESSORES DE TUMOR
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ABNT
LIMA, Marina Trombetta et al. Isolation and characterization of three novel isoforms of the RECK tumor suppressor gene. 2009, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq, 2009. . Acesso em: 12 nov. 2024.
APA
Lima, M. T., Winnischofer, S. M. B., Colin, C., & Sogayar, M. C. (2009). Isolation and characterization of three novel isoforms of the RECK tumor suppressor gene. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq.
NLM
Lima MT, Winnischofer SMB, Colin C, Sogayar MC. Isolation and characterization of three novel isoforms of the RECK tumor suppressor gene. Program and Index. 2009 ;[citado 2024 nov. 12 ]
Vancouver
Lima MT, Winnischofer SMB, Colin C, Sogayar MC. Isolation and characterization of three novel isoforms of the RECK tumor suppressor gene. Program and Index. 2009 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Characterization of RECK gene expression and its alternative isoforms in melanoma cells (2009)
Jacomasso, T.; Barbosa, F. A. L.; Lima, Marina Trombetta; Sogayar, Mari Cleide ; Winnischofer, Sheila Maria Brochado
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society. Unidade: IQ
Subjects: METALOPROTEINASES, MELANOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JACOMASSO, T. et al. Characterization of RECK gene expression and its alternative isoforms in melanoma cells. 2009, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq, 2009. . Acesso em: 12 nov. 2024.
APA
Jacomasso, T., Barbosa, F. A. L., Lima, M. T., Sogayar, M. C., & Winnischofer, S. M. B. (2009). Characterization of RECK gene expression and its alternative isoforms in melanoma cells. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq.
NLM
Jacomasso T, Barbosa FAL, Lima MT, Sogayar MC, Winnischofer SMB. Characterization of RECK gene expression and its alternative isoforms in melanoma cells. Program and Index. 2009 ;[citado 2024 nov. 12 ]
Vancouver
Jacomasso T, Barbosa FAL, Lima MT, Sogayar MC, Winnischofer SMB. Characterization of RECK gene expression and its alternative isoforms in melanoma cells. Program and Index. 2009 ;[citado 2024 nov. 12 ]
Trabalho de evento-resumo
Characterization of RECK transcriptional repression mediated by the c -myc oncogene product (2008)
Lima, Marina Trombetta; Winnischofer, Sheila Maria Brochado; Figueira, Rita de Cássia Savio; Sogayar, Mari Cleide
Source: Abstracts. Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). Unidade: IQ
Subjects: BIOQUÍMICA, ONCOLOGIA, EXPRESSÃO GÊNICA, GENES SUPRESSORES DE TUMOR
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ABNT
LIMA, Marina Trombetta et al. Characterization of RECK transcriptional repression mediated by the c -myc oncogene product. 2008, Anais.. São Paulo: SBBq, 2008. . Acesso em: 12 nov. 2024.
APA
Lima, M. T., Winnischofer, S. M. B., Figueira, R. de C. S., & Sogayar, M. C. (2008). Characterization of RECK transcriptional repression mediated by the c -myc oncogene product. In Abstracts. São Paulo: SBBq.
NLM
Lima MT, Winnischofer SMB, Figueira R de CS, Sogayar MC. Characterization of RECK transcriptional repression mediated by the c -myc oncogene product. Abstracts. 2008 ;[citado 2024 nov. 12 ]
Vancouver
Lima MT, Winnischofer SMB, Figueira R de CS, Sogayar MC. Characterization of RECK transcriptional repression mediated by the c -myc oncogene product. Abstracts. 2008 ;[citado 2024 nov. 12 ]
Artigo de periodico
ADAM23 methylation and expression analysis in brain tumors (2005)
Costa, Fabrício F.; Colin, Christian; Shinjo, Sueli Mieko Oba; Zanata, Silvio Marques; Marie, Suely Kazue Nagahashi; Sogayar, Mari Cleide ; Camargo, Anamaria Aranha
Source: Neuroscience Letters. Unidades: FM, IQ
Subjects: GENES SUPRESSORES DE TUMOR, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Fabrício F. et al. ADAM23 methylation and expression analysis in brain tumors. Neuroscience Letters, v. 380, n. 3, p. 260-264, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2005.01.050. Acesso em: 12 nov. 2024.
APA
Costa, F. F., Colin, C., Shinjo, S. M. O., Zanata, S. M., Marie, S. K. N., Sogayar, M. C., & Camargo, A. A. (2005). ADAM23 methylation and expression analysis in brain tumors. Neuroscience Letters, 380( 3), 260-264. doi:10.1016/j.neulet.2005.01.050
NLM
Costa FF, Colin C, Shinjo SMO, Zanata SM, Marie SKN, Sogayar MC, Camargo AA. ADAM23 methylation and expression analysis in brain tumors [Internet]. Neuroscience Letters. 2005 ; 380( 3): 260-264.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.neulet.2005.01.050
Vancouver
Costa FF, Colin C, Shinjo SMO, Zanata SM, Marie SKN, Sogayar MC, Camargo AA. ADAM23 methylation and expression analysis in brain tumors [Internet]. Neuroscience Letters. 2005 ; 380( 3): 260-264.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.neulet.2005.01.050
Trabalho de evento-resumo periodico
Loss of ADAM23, a putative new metastasis suppressor gene, leads to enhanced migration of the MDA-MB-435 breast cancer cell line possibly by modulating the alfavbeta3 integrin activation (2005)
Verbisck, Newton Valério; Muras, Angelita Gonzalez; Moura, Ricardo Pereira de; Costa, F. F.; Melo, F. H. M.; Zanata, Silvio Marques; Sogayar, Mari Cleide ; Chammas, Roger; Camargo, Anamaria Aranha
Source: Applied Cancer Research. Conference titles: São Paulo research Conference Cancer Today: From Molecular Biology to Treatment. Unidades: IQ, FM
Assunto: BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VERBISCK, Newton Valério et al. Loss of ADAM23, a putative new metastasis suppressor gene, leads to enhanced migration of the MDA-MB-435 breast cancer cell line possibly by modulating the alfavbeta3 integrin activation. Applied Cancer Research. Ribeirão Preto: Instituto de Química, Universidade de São Paulo. . Acesso em: 12 nov. 2024. , 2005
APA
Verbisck, N. V., Muras, A. G., Moura, R. P. de, Costa, F. F., Melo, F. H. M., Zanata, S. M., et al. (2005). Loss of ADAM23, a putative new metastasis suppressor gene, leads to enhanced migration of the MDA-MB-435 breast cancer cell line possibly by modulating the alfavbeta3 integrin activation. Applied Cancer Research. Ribeirão Preto: Instituto de Química, Universidade de São Paulo.
NLM
Verbisck NV, Muras AG, Moura RP de, Costa FF, Melo FHM, Zanata SM, Sogayar MC, Chammas R, Camargo AA. Loss of ADAM23, a putative new metastasis suppressor gene, leads to enhanced migration of the MDA-MB-435 breast cancer cell line possibly by modulating the alfavbeta3 integrin activation. Applied Cancer Research. 2005 ;( 2): 69 res.P-080.[citado 2024 nov. 12 ]
Vancouver
Verbisck NV, Muras AG, Moura RP de, Costa FF, Melo FHM, Zanata SM, Sogayar MC, Chammas R, Camargo AA. Loss of ADAM23, a putative new metastasis suppressor gene, leads to enhanced migration of the MDA-MB-435 breast cancer cell line possibly by modulating the alfavbeta3 integrin activation. Applied Cancer Research. 2005 ;( 2): 69 res.P-080.[citado 2024 nov. 12 ]
Artigo de periodico
Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors (2004)
Costa, Fabrício F.; Verbisck, Newton Valério; Salim, Anna Christina de Matos; Ierardi, Daniela Filippini; Pires, Lilian Campos Pires; Sasahara, Regina Maki; Sogayar, Mari Cleide ; Zanata, Silvio Marques; Mackay, Alan; O'Hare, Michael; Soares, Fernando Augusto; Simpson, Andrew John George; Camargo, Anamaria Aranha
Source: Oncogene. Unidade: IQ
Subjects: GENES SUPRESSORES DE TUMOR, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Fabrício F. et al. Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors. Oncogene, v. 23, n. 7, p. 1481-1488, 2004Tradução . . Disponível em: https://doi.org/10.1038/sj.onc.1207263. Acesso em: 12 nov. 2024.
APA
Costa, F. F., Verbisck, N. V., Salim, A. C. de M., Ierardi, D. F., Pires, L. C. P., Sasahara, R. M., et al. (2004). Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors. Oncogene, 23( 7), 1481-1488. doi:10.1038/sj.onc.1207263
NLM
Costa FF, Verbisck NV, Salim AC de M, Ierardi DF, Pires LCP, Sasahara RM, Sogayar MC, Zanata SM, Mackay A, O'Hare M, Soares FA, Simpson AJG, Camargo AA. Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors [Internet]. Oncogene. 2004 ; 23( 7): 1481-1488.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1038/sj.onc.1207263
Vancouver
Costa FF, Verbisck NV, Salim AC de M, Ierardi DF, Pires LCP, Sasahara RM, Sogayar MC, Zanata SM, Mackay A, O'Hare M, Soares FA, Simpson AJG, Camargo AA. Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors [Internet]. Oncogene. 2004 ; 23( 7): 1481-1488.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1038/sj.onc.1207263

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