Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors (2004)
- Authors:
- Costa, Fabrício F.
- Verbisck, Newton Valério
- Salim, Anna Christina de Matos
- Ierardi, Daniela Filippini
- Pires, Lilian Campos Pires
- Sasahara, Regina Maki
- Sogayar, Mari Cleide
- Zanata, Silvio Marques - Universidade Federal do Paraná (UFPR)
- Mackay, Alan
- O'Hare, Michael
- Soares, Fernando Augusto
- Simpson, Andrew John George
- Camargo, Anamaria Aranha
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- DOI: 10.1038/sj.onc.1207263
- Subjects: GENES SUPRESSORES DE TUMOR; EXPRESSÃO GÊNICA
- Language: Inglês
- Imprenta:
- Publisher place: Basingstoke
- Date published: 2004
- Source:
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
COSTA, Fabrício F. et al. Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors. Oncogene, v. 23, n. 7, p. 1481-1488, 2004Tradução . . Disponível em: https://doi.org/10.1038/sj.onc.1207263. Acesso em: 16 abr. 2024. -
APA
Costa, F. F., Verbisck, N. V., Salim, A. C. de M., Ierardi, D. F., Pires, L. C. P., Sasahara, R. M., et al. (2004). Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors. Oncogene, 23( 7), 1481-1488. doi:10.1038/sj.onc.1207263 -
NLM
Costa FF, Verbisck NV, Salim AC de M, Ierardi DF, Pires LCP, Sasahara RM, Sogayar MC, Zanata SM, Mackay A, O'Hare M, Soares FA, Simpson AJG, Camargo AA. Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors [Internet]. Oncogene. 2004 ; 23( 7): 1481-1488.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1038/sj.onc.1207263 -
Vancouver
Costa FF, Verbisck NV, Salim AC de M, Ierardi DF, Pires LCP, Sasahara RM, Sogayar MC, Zanata SM, Mackay A, O'Hare M, Soares FA, Simpson AJG, Camargo AA. Epigenetic silencing of the adhesion molecule ADAM23 is highly frequent in breast tumors [Internet]. Oncogene. 2004 ; 23( 7): 1481-1488.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1038/sj.onc.1207263 - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
Informações sobre o DOI: 10.1038/sj.onc.1207263 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
