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Artigo de periodico
Epithelium-derived 6-nitrodopamine modulates noradrenaline-induced contractions in human seminal vesicles (2024)
Britto Júnior, José ; Uramoto, Edson Hiroshi Salgado; Lima, Antonio Tiago Silva ; Ribeiro, Luiz Fernando Paulino ; Souza, Valéria Barbosa de ; Schenka, André Almeida ; Magalhães, José Cássio de Almeida; Antunes, Edson ; Fregonesi, Adriano ; De Nucci, Gilberto
Source: Life Sciences. Unidade: ICB
Subjects: FARMACOLOGIA, EPITÉLIO, CATECOLAMINAS, RECEPTORES DE DOPAMINA, CONTRAÇÃO MUSCULAR, ESPECTROMETRIA DE MASSAS, CROMATOGRAFIA LÍQUIDA, SINERGISMO DE DROGAS
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BRITTO JÚNIOR, José et al. Epithelium-derived 6-nitrodopamine modulates noradrenaline-induced contractions in human seminal vesicles. Life Sciences, v. 348, p. 8 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2024.122695. Acesso em: 11 nov. 2024.
APA
Britto Júnior, J., Uramoto, E. H. S., Lima, A. T. S., Ribeiro, L. F. P., Souza, V. B. de, Schenka, A. A., et al. (2024). Epithelium-derived 6-nitrodopamine modulates noradrenaline-induced contractions in human seminal vesicles. Life Sciences, 348, 8 . doi:10.1016/j.lfs.2024.122695
NLM
Britto Júnior J, Uramoto EHS, Lima ATS, Ribeiro LFP, Souza VB de, Schenka AA, Magalhães JC de A, Antunes E, Fregonesi A, De Nucci G. Epithelium-derived 6-nitrodopamine modulates noradrenaline-induced contractions in human seminal vesicles [Internet]. Life Sciences. 2024 ; 348 8 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2024.122695
Vancouver
Britto Júnior J, Uramoto EHS, Lima ATS, Ribeiro LFP, Souza VB de, Schenka AA, Magalhães JC de A, Antunes E, Fregonesi A, De Nucci G. Epithelium-derived 6-nitrodopamine modulates noradrenaline-induced contractions in human seminal vesicles [Internet]. Life Sciences. 2024 ; 348 8 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2024.122695
Artigo de periodico
Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure (2023)
Fontes, Milene T.; Arruda Junior, Daniel F.; Santos, Danúbia Silva dos ; Silva, João Carlos Ribeiro ; Antônio, Edinei L.; Tucci, Paulo F. J. ; Rossoni, Luciana Venturini ; Girardi, Adriana C. C.
Source: Life Sciences. Unidades: ICB, FM
Assunto: FISIOLOGIA
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FONTES, Milene T. et al. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, v. 323, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121648. Acesso em: 11 nov. 2024.
APA
Fontes, M. T., Arruda Junior, D. F., Santos, D. S. dos, Silva, J. C. R., Antônio, E. L., Tucci, P. F. J., et al. (2023). Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, 323, 1-10. doi:10.1016/j.lfs.2023.121648
NLM
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Vancouver
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Artigo de periodico
Effect of stress on the chronotropic and inotropic responses to β-adrenergic agonists in isolated atria of KOβ2 mice (2023)
Moura, Andre Luiz; Brum, Patrícia Chakur ; Carvalho, Ana Elisa Teofilo Saturi; Spadari, Regina Celia
Source: Life Sciences. Unidade: EEFE
Subjects: ADRENORECEPTORES, BETA-BLOQUEADORES, ESTRESSE, FREQUÊNCIA CARDÍACA, CAMUNDONGOS, RATOS WISTAR
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MOURA, Andre Luiz et al. Effect of stress on the chronotropic and inotropic responses to β-adrenergic agonists in isolated atria of KOβ2 mice. Life Sciences, v. 32, n. ju 2023, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121644. Acesso em: 11 nov. 2024.
APA
Moura, A. L., Brum, P. C., Carvalho, A. E. T. S., & Spadari, R. C. (2023). Effect of stress on the chronotropic and inotropic responses to β-adrenergic agonists in isolated atria of KOβ2 mice. Life Sciences, 32( ju 2023). doi:10.1016/j.lfs.2023.121644
NLM
Moura AL, Brum PC, Carvalho AETS, Spadari RC. Effect of stress on the chronotropic and inotropic responses to β-adrenergic agonists in isolated atria of KOβ2 mice [Internet]. Life Sciences. 2023 ; 32( ju 2023):[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121644
Vancouver
Moura AL, Brum PC, Carvalho AETS, Spadari RC. Effect of stress on the chronotropic and inotropic responses to β-adrenergic agonists in isolated atria of KOβ2 mice [Internet]. Life Sciences. 2023 ; 32( ju 2023):[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121644
Artigo de periodico
miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction (2023)
Silva, Camila Stephanie Balbino ; Couto, Gisele Kruger ; Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Huang, Zhan-Peng ; Wang, Da Zhi ; Festuccia, William Tadeu Lara ; Barreto-Chaves, Maria Luiza Morais ; Rossoni, Luciana Venturini ; Diniz, Gabriela Placoná
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, FISIOLOGIA, AORTA DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, VASOS SANGUÍNEOS, TECIDO ADIPOSO, MICRORNAS, OBESIDADE, REAÇÃO EM CADEIA POR POLIMERASE, WESTERN BLOTTING
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SILVA, Camila Stephanie Balbino et al. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, v. 316, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121416. Acesso em: 11 nov. 2024.
APA
Silva, C. S. B., Couto, G. K., Lino, C. A., Silva, T. de O., Lunardon, G., Huang, Z. -P., et al. (2023). miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, 316, 1-10. doi:10.1016/j.lfs.2023.121416
NLM
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Vancouver
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Artigo de periodico
Resistance exercise training benefits pulmonary, cardiac, and muscular structure and function in rats with stable pulmonary artery hypertension (2023)
Soares, Leôncio Lopes ; Leite, Luciano Bernardes ; Ervilha, Luiz Otávio Guimarães; Pelozin, Bruno Rocha Avila ; Pereira, Noemy Pinto ; Silva, Bruna Aparecida Fonseca da; Portes, Alexandre Martins Oliveira; Drummond, Filipe Rios ; Rezende, Leonardo Mateus Teixeira de ; Fernandes, Tiago ; Oliveira, Edilamar Menezes de ; Neves, Mariana Machado; Reis, Emily Correna Carlo ; Natali, Antônio José
Source: Life Sciences. Unidade: EEFE
Subjects: HIPERTENSÃO PULMONAR, MICRORNAS, TREINAMENTO DE FORÇA, CAPACIDADE PULMONAR, MÚSCULO ESQUELÉTICO, RATOS WISTAR
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SOARES, Leôncio Lopes et al. Resistance exercise training benefits pulmonary, cardiac, and muscular structure and function in rats with stable pulmonary artery hypertension. Life Sciences, v. 332, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.122128. Acesso em: 11 nov. 2024.
APA
Soares, L. L., Leite, L. B., Ervilha, L. O. G., Pelozin, B. R. A., Pereira, N. P., Silva, B. A. F. da, et al. (2023). Resistance exercise training benefits pulmonary, cardiac, and muscular structure and function in rats with stable pulmonary artery hypertension. Life Sciences, 332. doi:10.1016/j.lfs.2023.122128
NLM
Soares LL, Leite LB, Ervilha LOG, Pelozin BRA, Pereira NP, Silva BAF da, Portes AMO, Drummond FR, Rezende LMT de, Fernandes T, Oliveira EM de, Neves MM, Reis ECC, Natali AJ. Resistance exercise training benefits pulmonary, cardiac, and muscular structure and function in rats with stable pulmonary artery hypertension [Internet]. Life Sciences. 2023 ; 332[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.122128
Vancouver
Soares LL, Leite LB, Ervilha LOG, Pelozin BRA, Pereira NP, Silva BAF da, Portes AMO, Drummond FR, Rezende LMT de, Fernandes T, Oliveira EM de, Neves MM, Reis ECC, Natali AJ. Resistance exercise training benefits pulmonary, cardiac, and muscular structure and function in rats with stable pulmonary artery hypertension [Internet]. Life Sciences. 2023 ; 332[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.122128
Artigo de periodico
Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation (2022)
Pinto, Ana Paula ; Rocha, Alisson Luiz da ; Teixeira, Giovana Rampazzo; Rovina, Rafael Lemes ; Veras, Allice S. C.; Frantz, Fabiani Gai ; Pauli, José Rodrigo; Moura, Leandro Pereira de; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Quadrilatero, Joe; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidades: FCFRP, EEFERP, FMRP
Subjects: DEGRANULAÇÃO CELULAR, FÍGADO, HEPATOPATIAS, REAÇÃO EM CADEIA POR POLIMERASE
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PINTO, Ana Paula et al. Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation. Life Sciences, v. 306, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120800. Acesso em: 11 nov. 2024.
APA
Pinto, A. P., Rocha, A. L. da, Teixeira, G. R., Rovina, R. L., Veras, A. S. C., Frantz, F. G., et al. (2022). Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation. Life Sciences, 306, 1-12. doi:10.1016/j.lfs.2022.120800
NLM
Pinto AP, Rocha AL da, Teixeira GR, Rovina RL, Veras ASC, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Quadrilatero J, Silva ASR da. Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation [Internet]. Life Sciences. 2022 ; 306 1-12.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120800
Vancouver
Pinto AP, Rocha AL da, Teixeira GR, Rovina RL, Veras ASC, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Quadrilatero J, Silva ASR da. Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation [Internet]. Life Sciences. 2022 ; 306 1-12.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120800
Artigo de periodico
Resistance training prevents damage to the mitochondrial function of the skeletal muscle of rats exposed to secondary cigarette smoke (2022)
Moreno, Ana Caroline Rippi ; Oliveira, André Olean ; Oliveira, Tiago Olean ; Nunes, Maria Tereza ; Teixeira, Marcos F. S.; Seraphim, Patricia Monteiro
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, RATOS WISTAR, FUMO, FUMAÇA, MÚSCULO ESQUELÉTICO, OXIGÊNIO, EXERCÍCIO FÍSICO, MODELOS ANIMAIS DE DOENÇAS
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MORENO, Ana Caroline Rippi et al. Resistance training prevents damage to the mitochondrial function of the skeletal muscle of rats exposed to secondary cigarette smoke. Life Sciences, v. 309, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.121017. Acesso em: 11 nov. 2024.
APA
Moreno, A. C. R., Oliveira, A. O., Oliveira, T. O., Nunes, M. T., Teixeira, M. F. S., & Seraphim, P. M. (2022). Resistance training prevents damage to the mitochondrial function of the skeletal muscle of rats exposed to secondary cigarette smoke. Life Sciences, 309, 1-10. doi:10.1016/j.lfs.2022.121017
NLM
Moreno ACR, Oliveira AO, Oliveira TO, Nunes MT, Teixeira MFS, Seraphim PM. Resistance training prevents damage to the mitochondrial function of the skeletal muscle of rats exposed to secondary cigarette smoke [Internet]. Life Sciences. 2022 ; 309 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.121017
Vancouver
Moreno ACR, Oliveira AO, Oliveira TO, Nunes MT, Teixeira MFS, Seraphim PM. Resistance training prevents damage to the mitochondrial function of the skeletal muscle of rats exposed to secondary cigarette smoke [Internet]. Life Sciences. 2022 ; 309 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.121017
Artigo de periodico
Sex differences in the participation of endothelial mediators and signaling pathways involved in the vasodilator effect of a selective GPER agonist in resistance arteries of gonadectomized Wistar rats (2022)
Peixoto, Pollyana ; Alves, Ildernandes Vieira ; Couto, Gisele Kruger ; Lemos, Virgínia Soares ; Rossoni, Luciana Venturini ; Bissoli, Nazaré Souza ; Santos, Roger Lyrio dos
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, VASODILATAÇÃO, ENDOTÉLIO VASCULAR, ESTRÓGENOS, DIFERENÇAS SEXUAIS (PSICOGÊNESE), PROTEÍNAS QUINASES, PERÓXIDO DE HIDROGÊNIO, ÓXIDO NÍTRICO, PROTEÍNAS PROTO-ONCOGÊNICAS, PROTEÍNAS G, RATOS WISTAR, ARTÉRIAS
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PEIXOTO, Pollyana et al. Sex differences in the participation of endothelial mediators and signaling pathways involved in the vasodilator effect of a selective GPER agonist in resistance arteries of gonadectomized Wistar rats. Life Sciences, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120917. Acesso em: 11 nov. 2024.
APA
Peixoto, P., Alves, I. V., Couto, G. K., Lemos, V. S., Rossoni, L. V., Bissoli, N. S., & Santos, R. L. dos. (2022). Sex differences in the participation of endothelial mediators and signaling pathways involved in the vasodilator effect of a selective GPER agonist in resistance arteries of gonadectomized Wistar rats. Life Sciences. doi:10.1016/j.lfs.2022.120917
NLM
Peixoto P, Alves IV, Couto GK, Lemos VS, Rossoni LV, Bissoli NS, Santos RL dos. Sex differences in the participation of endothelial mediators and signaling pathways involved in the vasodilator effect of a selective GPER agonist in resistance arteries of gonadectomized Wistar rats [Internet]. Life Sciences. 2022 ;[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120917
Vancouver
Peixoto P, Alves IV, Couto GK, Lemos VS, Rossoni LV, Bissoli NS, Santos RL dos. Sex differences in the participation of endothelial mediators and signaling pathways involved in the vasodilator effect of a selective GPER agonist in resistance arteries of gonadectomized Wistar rats [Internet]. Life Sciences. 2022 ;[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120917
Artigo de periodico
Characterization of the metabolic differences between male and female C57BL/6 mice (2022)
Souza, Gabriel Orefice de ; Wasinski, Frederick ; Donato Junior, José
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIOS DO PÂNCREAS ENDÓCRINO, HORMÔNIO DO CRESCIMENTO, LEPTINA, HIPOTÁLAMO, INGESTÃO, OBESIDADE, DIFERENÇAS SEXUAIS ANIMAL, CAMUNDONGOS
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SOUZA, Gabriel Orefice de e WASINSKI, Frederick e DONATO JUNIOR, José. Characterization of the metabolic differences between male and female C57BL/6 mice. Life Sciences, v. 301, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120636. Acesso em: 11 nov. 2024.
APA
Souza, G. O. de, Wasinski, F., & Donato Junior, J. (2022). Characterization of the metabolic differences between male and female C57BL/6 mice. Life Sciences, 301, 1-13. doi:10.1016/j.lfs.2022.120636
NLM
Souza GO de, Wasinski F, Donato Junior J. Characterization of the metabolic differences between male and female C57BL/6 mice [Internet]. Life Sciences. 2022 ; 301 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120636
Vancouver
Souza GO de, Wasinski F, Donato Junior J. Characterization of the metabolic differences between male and female C57BL/6 mice [Internet]. Life Sciences. 2022 ; 301 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120636
Artigo de periodico
AD80, a multikinase inhibitor, as a potential drug candidate for colorectal cancer therapy (2022)
Almeida, Larissa Costa de ; Carlos, Jorge Antonio Elias Godoy ; Teixeira, Paula Rezende ; Machado Neto, João Agostinho ; Costa-Lotufo, Letícia Veras
Source: Life Sciences. Unidade: ICB
Subjects: FARMACOLOGIA, NEOPLASIAS COLORRETAIS, PROTEÍNAS PROTO-ONCOGÊNICAS, RNA MENSAGEIRO, APOPTOSE, ANTINEOPLÁSICOS, CÉLULAS CULTIVADAS DE TUMOR, INIBIDORES DE ENZIMAS, ATIVAÇÃO ENZIMÁTICA, PROTEÍNAS QUINASES, TRANSDUÇÃO DE SINAL CELULAR, CARCINOGÊNESE, PROLIFERAÇÃO CELULAR
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ALMEIDA, Larissa Costa de et al. AD80, a multikinase inhibitor, as a potential drug candidate for colorectal cancer therapy. Life Sciences, v. 308, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120911. Acesso em: 11 nov. 2024.
APA
Almeida, L. C. de, Carlos, J. A. E. G., Teixeira, P. R., Machado Neto, J. A., & Costa-Lotufo, L. V. (2022). AD80, a multikinase inhibitor, as a potential drug candidate for colorectal cancer therapy. Life Sciences, 308, 1-9. doi:10.1016/j.lfs.2022.120911
NLM
Almeida LC de, Carlos JAEG, Teixeira PR, Machado Neto JA, Costa-Lotufo LV. AD80, a multikinase inhibitor, as a potential drug candidate for colorectal cancer therapy [Internet]. Life Sciences. 2022 ; 308 1-9.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120911
Vancouver
Almeida LC de, Carlos JAEG, Teixeira PR, Machado Neto JA, Costa-Lotufo LV. AD80, a multikinase inhibitor, as a potential drug candidate for colorectal cancer therapy [Internet]. Life Sciences. 2022 ; 308 1-9.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120911
Artigo de periodico
Ischemia-reperfusion damage is attenuated by GQ-11, a peroxisome proliferator-activated receptor (PPAR)-α/γ agonist, after aorta clamping in rats (2022)
Silva, Jacqueline Cavalcante ; Bavestrello, Margherita; Gazzola, Valerio; Spinella, Giovanni ; Pane, Bianca; Grasselli, Elena ; Demori, Ilaria; Canesi, Laura ; Emionite, Laura; Cilli, Michele ; Buschiazzo, Ambra; Sambuceti, Gianmario ; Pitta, Ivan Rocha; Pitta, Marina Galdino ; Perego, Patrizia; Perego, Patrizia ; Palombo, Domenico; Abdalla, Dulcinéia Saes Parra
Source: Life Sciences. Unidade: FCF
Subjects: ISQUEMIA, AORTA, ESTRESSE OXIDATIVO, INFLAMAÇÃO
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ABNT
SILVA, Jacqueline Cavalcante et al. Ischemia-reperfusion damage is attenuated by GQ-11, a peroxisome proliferator-activated receptor (PPAR)-α/γ agonist, after aorta clamping in rats. Life Sciences, v. 297, p. 1-11 art. 120468, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120468. Acesso em: 11 nov. 2024.
APA
Silva, J. C., Bavestrello, M., Gazzola, V., Spinella, G., Pane, B., Grasselli, E., et al. (2022). Ischemia-reperfusion damage is attenuated by GQ-11, a peroxisome proliferator-activated receptor (PPAR)-α/γ agonist, after aorta clamping in rats. Life Sciences, 297, 1-11 art. 120468. doi:10.1016/j.lfs.2022.120468
NLM
Silva JC, Bavestrello M, Gazzola V, Spinella G, Pane B, Grasselli E, Demori I, Canesi L, Emionite L, Cilli M, Buschiazzo A, Sambuceti G, Pitta IR, Pitta MG, Perego P, Perego P, Palombo D, Abdalla DSP. Ischemia-reperfusion damage is attenuated by GQ-11, a peroxisome proliferator-activated receptor (PPAR)-α/γ agonist, after aorta clamping in rats [Internet]. Life Sciences. 2022 ; 297 1-11 art. 120468.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120468
Vancouver
Silva JC, Bavestrello M, Gazzola V, Spinella G, Pane B, Grasselli E, Demori I, Canesi L, Emionite L, Cilli M, Buschiazzo A, Sambuceti G, Pitta IR, Pitta MG, Perego P, Perego P, Palombo D, Abdalla DSP. Ischemia-reperfusion damage is attenuated by GQ-11, a peroxisome proliferator-activated receptor (PPAR)-α/γ agonist, after aorta clamping in rats [Internet]. Life Sciences. 2022 ; 297 1-11 art. 120468.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120468
Artigo de periodico
Comparison between aerobic exercise training and enalapril treatment as tools to improve diet-induced metabolic-associated fatty liver disease: Effects on endoplasmic reticulum stress markers (2022)
Sepúlveda-Fragoso, Vinicius ; Alexandre-Santos, Beatriz ; Barreto-Reis, Emanuelle; Fernandes, Tiago ; Oliveira, Edilamar Menezes de ; Cavalcanti, Soraia das Neves Vieira; Atella, Georgia Correa ; Miranda-Alves, Leandro ; Vázquez-Carrera, Manuel ; Nóbrega, Antonio Claudio Lucas da; Frantz, Eliete Dalla Corte ; Magliano, D'Angelo Carlo
Source: Life Sciences. Unidade: EEFE
Subjects: HEPATOPATIAS, SISTEMA RENINA-ANGIOTENSINA, RETÍCULO ENDOPLASMÁTICO, TREINAMENTO AERÓBIO, OBESIDADE
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ABNT
SEPÚLVEDA-FRAGOSO, Vinicius et al. Comparison between aerobic exercise training and enalapril treatment as tools to improve diet-induced metabolic-associated fatty liver disease: Effects on endoplasmic reticulum stress markers. Life Sciences, v. 311, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.121136. Acesso em: 11 nov. 2024.
APA
Sepúlveda-Fragoso, V., Alexandre-Santos, B., Barreto-Reis, E., Fernandes, T., Oliveira, E. M. de, Cavalcanti, S. das N. V., et al. (2022). Comparison between aerobic exercise training and enalapril treatment as tools to improve diet-induced metabolic-associated fatty liver disease: Effects on endoplasmic reticulum stress markers. Life Sciences, 311. doi:10.1016/j.lfs.2022.121136
NLM
Sepúlveda-Fragoso V, Alexandre-Santos B, Barreto-Reis E, Fernandes T, Oliveira EM de, Cavalcanti S das NV, Atella GC, Miranda-Alves L, Vázquez-Carrera M, Nóbrega ACL da, Frantz EDC, Magliano D'AC. Comparison between aerobic exercise training and enalapril treatment as tools to improve diet-induced metabolic-associated fatty liver disease: Effects on endoplasmic reticulum stress markers [Internet]. Life Sciences. 2022 ; 311[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.121136
Vancouver
Sepúlveda-Fragoso V, Alexandre-Santos B, Barreto-Reis E, Fernandes T, Oliveira EM de, Cavalcanti S das NV, Atella GC, Miranda-Alves L, Vázquez-Carrera M, Nóbrega ACL da, Frantz EDC, Magliano D'AC. Comparison between aerobic exercise training and enalapril treatment as tools to improve diet-induced metabolic-associated fatty liver disease: Effects on endoplasmic reticulum stress markers [Internet]. Life Sciences. 2022 ; 311[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.121136
Artigo de periodico
Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction (2022)
Peres, Emília Cristina ; Victorio, Jamaira A. ; Souza, Valéria Nunes ; Faloppa, Ana Cristina Breithaupt ; Rabelo, Luiza A.; Lima, Wothan Tavares de ; Davel, Ana Paula ; Rossoni, Luciana Venturini
Source: Life Sciences. Unidades: HU, FM, ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, ARTÉRIA PULMONAR, REPERFUSÃO, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS, SUPERÓXIDO DISMUTASE, ENDOTÉLIO VASCULAR, ISQUEMIA, INFLAMAÇÃO, ÓXIDO NÍTRICO, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERES, Emília Cristina et al. Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction. Life Sciences, v. 306, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120851. Acesso em: 11 nov. 2024.
APA
Peres, E. C., Victorio, J. A., Souza, V. N., Faloppa, A. C. B., Rabelo, L. A., Lima, W. T. de, et al. (2022). Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction. Life Sciences, 306, 1-10. doi:10.1016/j.lfs.2022.120851
NLM
Peres EC, Victorio JA, Souza VN, Faloppa ACB, Rabelo LA, Lima WT de, Davel AP, Rossoni LV. Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction [Internet]. Life Sciences. 2022 ; 306 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120851
Vancouver
Peres EC, Victorio JA, Souza VN, Faloppa ACB, Rabelo LA, Lima WT de, Davel AP, Rossoni LV. Simvastatin protects against intestinal ischemia/reperfusion-induced pulmonary artery dysfunction [Internet]. Life Sciences. 2022 ; 306 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120851
Artigo de periodico
Monocarboxylate transporters (MCTs) in skeletal muscle and hypothalamus of less or more physically active mice exposed to aerobic training (2022)
Scariot, Pedro Paulo Menezes; Manchado-Gobatto, Fúlvia Barros; Beck, W. R.; Papoti, Marcelo ; Van Ginkel, P. R.; Gobatto, Cláudio Alexandre
Source: Life Sciences. Unidade: EEFERP
Subjects: TREINAMENTO FÍSICO, CAPACIDADE AERÓBICA, HIPOTÁLAMO, MÚSCULO ESQUELÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SCARIOT, Pedro Paulo Menezes et al. Monocarboxylate transporters (MCTs) in skeletal muscle and hypothalamus of less or more physically active mice exposed to aerobic training. Life Sciences, v. 307, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120872. Acesso em: 11 nov. 2024.
APA
Scariot, P. P. M., Manchado-Gobatto, F. B., Beck, W. R., Papoti, M., Van Ginkel, P. R., & Gobatto, C. A. (2022). Monocarboxylate transporters (MCTs) in skeletal muscle and hypothalamus of less or more physically active mice exposed to aerobic training. Life Sciences, 307, 1-11. doi:10.1016/j.lfs.2022.120872
NLM
Scariot PPM, Manchado-Gobatto FB, Beck WR, Papoti M, Van Ginkel PR, Gobatto CA. Monocarboxylate transporters (MCTs) in skeletal muscle and hypothalamus of less or more physically active mice exposed to aerobic training [Internet]. Life Sciences. 2022 ; 307 1-11.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120872
Vancouver
Scariot PPM, Manchado-Gobatto FB, Beck WR, Papoti M, Van Ginkel PR, Gobatto CA. Monocarboxylate transporters (MCTs) in skeletal muscle and hypothalamus of less or more physically active mice exposed to aerobic training [Internet]. Life Sciences. 2022 ; 307 1-11.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120872
Artigo de periodico
Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats (2022)
Moraes, Ruan Carlos Macêdo de ; Lima, Gabriely Cristina Alves; Cardinali, Camila Aparecida Errerias Fernandes ; Gonçalves, Alisson Carvalho de ; Portari, Guilherme Vannucchi ; Guerra-Shinohara, Elvira Maria ; Leboucher, Antoine ; Donato Junior, José ; Kleinridders, André ; Torrão, Andréa da Silva
Source: Life Sciences. Unidades: FCF, ICB, FM
Subjects: DOENÇA DE ALZHEIMER, HIPOTÁLAMO, FISIOLOGIA, VITAMINAS, DEMÊNCIA, INSULINA, DOENÇAS METABÓLICAS, METABOLISMO ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAES, Ruan Carlos Macêdo de et al. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, v. 306, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120841. Acesso em: 11 nov. 2024.
APA
Moraes, R. C. M. de, Lima, G. C. A., Cardinali, C. A. E. F., Gonçalves, A. C. de, Portari, G. V., Guerra-Shinohara, E. M., et al. (2022). Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, 306, 1-13. doi:10.1016/j.lfs.2022.120841
NLM
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Vancouver
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Artigo de periodico
Phosphodiesterase 4 inhibition restrains muscle proteolysis in diabetic rats by activating PKA and EPAC/Akt effectors and inhibiting FoxO factors (2021)
Arcaro, Carlos Alberto; Assis, Renata Pires; Oliveira, Juliana Oriel; Zanon, Neusa Maria; Paula-Gomes, Silvia; Navegantes, Luiz Carlos Carvalho ; Kettelhut, Isis do Carmo ; Brunetti, Iguatemy Lourenço; Baviera, Amanda Martins
Source: Life Sciences. Unidade: FMRP
Subjects: ANTIDEPRESSIVOS, MÚSCULO ESQUELÉTICO, METABOLISMO DE PROTEÍNA, PROTEÍNAS DO TECIDO NERVOSO, TRANSDUÇÃO DE SINAL CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARCARO, Carlos Alberto et al. Phosphodiesterase 4 inhibition restrains muscle proteolysis in diabetic rats by activating PKA and EPAC/Akt effectors and inhibiting FoxO factors. Life Sciences, v. 278, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119563. Acesso em: 11 nov. 2024.
APA
Arcaro, C. A., Assis, R. P., Oliveira, J. O., Zanon, N. M., Paula-Gomes, S., Navegantes, L. C. C., et al. (2021). Phosphodiesterase 4 inhibition restrains muscle proteolysis in diabetic rats by activating PKA and EPAC/Akt effectors and inhibiting FoxO factors. Life Sciences, 278, 1-12. doi:10.1016/j.lfs.2021.119563
NLM
Arcaro CA, Assis RP, Oliveira JO, Zanon NM, Paula-Gomes S, Navegantes LCC, Kettelhut I do C, Brunetti IL, Baviera AM. Phosphodiesterase 4 inhibition restrains muscle proteolysis in diabetic rats by activating PKA and EPAC/Akt effectors and inhibiting FoxO factors [Internet]. Life Sciences. 2021 ; 278 1-12.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119563
Vancouver
Arcaro CA, Assis RP, Oliveira JO, Zanon NM, Paula-Gomes S, Navegantes LCC, Kettelhut I do C, Brunetti IL, Baviera AM. Phosphodiesterase 4 inhibition restrains muscle proteolysis in diabetic rats by activating PKA and EPAC/Akt effectors and inhibiting FoxO factors [Internet]. Life Sciences. 2021 ; 278 1-12.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119563
Artigo de periodico
Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility (2021)
Gil, Noemi Lourenço ; Azevedo, Gabriela A. ; Balbino, Aleksandro Martins ; Silva, Marina M. ; Carvalho, Maria Helena Catelli ; Akamine, Eliana Hiromi ; Keller, Alexandre C. ; Landgraf, Richardt G. ; Landgraf, Maristella de Almeida Vitta
Source: Life Sciences. Unidade: ICB
Subjects: FARMACOLOGIA, RATOS WISTAR, LIPOPOLISSACARÍDEOS, CRESCIMENTO ANIMAL, INFLAMAÇÃO, HOMEOSTASE, PERDA DE PESO, INTERLEUCINA 6, NEUTRÓFILOS, MACRÓFAGOS, EXPRESSÃO GÊNICA, PROSTAGLANDINAS, LEUCOTRIENOS B
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GIL, Noemi Lourenço et al. Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility. Life Sciences, v. 281, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119764. Acesso em: 11 nov. 2024.
APA
Gil, N. L., Azevedo, G. A., Balbino, A. M., Silva, M. M., Carvalho, M. H. C., Akamine, E. H., et al. (2021). Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility. Life Sciences, 281, 1-10. doi:10.1016/j.lfs.2021.119764
NLM
Gil NL, Azevedo GA, Balbino AM, Silva MM, Carvalho MHC, Akamine EH, Keller AC, Landgraf RG, Landgraf M de AV. Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility [Internet]. Life Sciences. 2021 ; 281 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119764
Vancouver
Gil NL, Azevedo GA, Balbino AM, Silva MM, Carvalho MHC, Akamine EH, Keller AC, Landgraf RG, Landgraf M de AV. Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility [Internet]. Life Sciences. 2021 ; 281 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119764
Artigo de periodico
C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta (2021)
Pernomian, Laena; Prado, Alejandro Ferraz do; Silva, Bruno Rodrigues; Paula, Tiago Dal-Cin de; Grando, Marcella Daruge; Bendhack, Lusiane Maria
Source: Life Sciences. Unidades: FMRP, FCFRP
Subjects: PEPTÍDEO NATRIURÉTICO ATRIAL, PROTEÍNAS, HIPERTENSÃO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERNOMIAN, Laena et al. C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta. Life Sciences, v. 272, p. 1-14, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119223. Acesso em: 11 nov. 2024.
APA
Pernomian, L., Prado, A. F. do, Silva, B. R., Paula, T. D. -C. de, Grando, M. D., & Bendhack, L. M. (2021). C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta. Life Sciences, 272, 1-14. doi:10.1016/j.lfs.2021.119223
NLM
Pernomian L, Prado AF do, Silva BR, Paula TD-C de, Grando MD, Bendhack LM. C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta [Internet]. Life Sciences. 2021 ; 272 1-14.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119223
Vancouver
Pernomian L, Prado AF do, Silva BR, Paula TD-C de, Grando MD, Bendhack LM. C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta [Internet]. Life Sciences. 2021 ; 272 1-14.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119223
Artigo de periodico
Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients (2021)
Potje, Simone Regina ; Costa, Tiago Januário da ; Fraga-Silva, Thais Fernanda de Campos ; Martins, Ronaldo Bragança ; Benatti, Maíra Nilson; Almado, Carlos Eduardo Lopes; Sá, Keyla Santos Guedes de ; Bonato, Vânia Luiza Deperon ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões ; Becari, Christiane; Martins, Maria Auxiliadora ; Tostes, Rita de Cássia Aleixo
Source: Life Sciences. Unidades: FMRP, FCFRP
Subjects: ENDOTÉLIO VASCULAR, COVID-19, COAGULAÇÃO SANGUÍNEA, ESTUDOS DE CASOS E CONTROLES, TROMBOSE, INTERLEUCINA 6
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
POTJE, Simone Regina et al. Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients. Life Sciences, v. 276, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119376. Acesso em: 11 nov. 2024.
APA
Potje, S. R., Costa, T. J. da, Fraga-Silva, T. F. de C., Martins, R. B., Benatti, M. N., Almado, C. E. L., et al. (2021). Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients. Life Sciences, 276. doi:10.1016/j.lfs.2021.119376
NLM
Potje SR, Costa TJ da, Fraga-Silva TF de C, Martins RB, Benatti MN, Almado CEL, Sá KSG de, Bonato VLD, Arruda E, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Becari C, Martins MA, Tostes R de CA. Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients [Internet]. Life Sciences. 2021 ; 276[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119376
Vancouver
Potje SR, Costa TJ da, Fraga-Silva TF de C, Martins RB, Benatti MN, Almado CEL, Sá KSG de, Bonato VLD, Arruda E, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Becari C, Martins MA, Tostes R de CA. Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients [Internet]. Life Sciences. 2021 ; 276[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119376
Artigo de periodico
Oxytocin induces anti-catabolic and anabolic effects on protein metabolism in the female rat oxidative skeletal muscle (2021)
Costa, Daniely Messias; Cruz-Filho, João da; Vasconcelos, Alan Bruno Silva; Gomes-Santos, João Victor; Reis, Luis Carlos; Junior, Waldecy de Lucca; Camargo, Enilton Aparecido; Lauton-Santos, Sandra; Zanon, Neusa Maria; Kettelhut, Isis do Carmo ; Navegantes, Luiz Carlos Carvalho
Source: Life Sciences. Unidade: FMRP
Subjects: OCITOCINA, MÚSCULO ESQUELÉTICO, ANABOLIZANTES, LISOSSOMOS, METABOLISMO DE PROTEÍNA, TRANSDUÇÃO DE SINAL CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Daniely Messias et al. Oxytocin induces anti-catabolic and anabolic effects on protein metabolism in the female rat oxidative skeletal muscle. Life Sciences, v. 279, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119665. Acesso em: 11 nov. 2024.
APA
Costa, D. M., Cruz-Filho, J. da, Vasconcelos, A. B. S., Gomes-Santos, J. V., Reis, L. C., Junior, W. de L., et al. (2021). Oxytocin induces anti-catabolic and anabolic effects on protein metabolism in the female rat oxidative skeletal muscle. Life Sciences, 279, 1-11. doi:10.1016/j.lfs.2021.119665
NLM
Costa DM, Cruz-Filho J da, Vasconcelos ABS, Gomes-Santos JV, Reis LC, Junior W de L, Camargo EA, Lauton-Santos S, Zanon NM, Kettelhut I do C, Navegantes LCC. Oxytocin induces anti-catabolic and anabolic effects on protein metabolism in the female rat oxidative skeletal muscle [Internet]. Life Sciences. 2021 ; 279 1-11.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119665
Vancouver
Costa DM, Cruz-Filho J da, Vasconcelos ABS, Gomes-Santos JV, Reis LC, Junior W de L, Camargo EA, Lauton-Santos S, Zanon NM, Kettelhut I do C, Navegantes LCC. Oxytocin induces anti-catabolic and anabolic effects on protein metabolism in the female rat oxidative skeletal muscle [Internet]. Life Sciences. 2021 ; 279 1-11.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2021.119665

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