Filtros : "Indexado na Base de Dados PubMed" "2016" Removidos: "FCF-FBC" "FFCLRP-593" "IAG-AGA" "BMC Genomics" Limpar

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  • Source: Metabolism Clinical and Experimental. Unidade: FM

    Subjects: ENZIMAS (METABOLISMO), ESTRESSE OXIDATIVO, DIABETES MELLITUS (COMPLICAÇÕES), NEFROPATIAS (ETIOLOGIA), PEROXIDASE

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      MOHAMMEDI, Kamel et al. Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes. Metabolism Clinical and Experimental, v. 65, n. 2, p. 12-19, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2015.10.004. Acesso em: 09 ago. 2024.
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      Mohammedi, K., Patente, T. A., Bellili-Muñoz, N., Driss, F., Le Nagard, H., Fumeron, F., et al. (2016). Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes. Metabolism Clinical and Experimental, 65( 2), 12-19. doi:10.1016/j.metabol.2015.10.004
    • NLM

      Mohammedi K, Patente TA, Bellili-Muñoz N, Driss F, Le Nagard H, Fumeron F, Roussel R, Hadjadj S, Corrêa-Giannella ML, Marre M, Velho G. Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes [Internet]. Metabolism Clinical and Experimental. 2016 ; 65( 2): 12-19.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.metabol.2015.10.004
    • Vancouver

      Mohammedi K, Patente TA, Bellili-Muñoz N, Driss F, Le Nagard H, Fumeron F, Roussel R, Hadjadj S, Corrêa-Giannella ML, Marre M, Velho G. Glutathione peroxidase-1 gene (GPX1) variants, oxidative stress and risk of kidney complications in people with type 1 diabetes [Internet]. Metabolism Clinical and Experimental. 2016 ; 65( 2): 12-19.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.metabol.2015.10.004
  • Source: Lausanne. Unidade: FM

    Subjects: CARCINOGÊNESE, CÉLULAS CULTIVADAS DE TUMOR, GLICOPROTEÍNAS

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      CARDOSO, Ana Carolina Ferreira et al. Galectin-3 Determines Tumor Cell Adaptive Strategies in Stressed Tumor Microenvironments. Lausanne, v. 6, n. art. 127, p. 1-12, 2016Tradução . . Disponível em: https://doi.org/10.3389/fonc.2016.00127. Acesso em: 09 ago. 2024.
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      Cardoso, A. C. F., Andrade, L. N. de S., Bustos, S. O., & Chammas, R. (2016). Galectin-3 Determines Tumor Cell Adaptive Strategies in Stressed Tumor Microenvironments. Lausanne, 6( art. 127), 1-12. doi:10.3389/fonc.2016.00127
    • NLM

      Cardoso ACF, Andrade LN de S, Bustos SO, Chammas R. Galectin-3 Determines Tumor Cell Adaptive Strategies in Stressed Tumor Microenvironments [Internet]. Lausanne. 2016 ; 6( art. 127): 1-12.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fonc.2016.00127
    • Vancouver

      Cardoso ACF, Andrade LN de S, Bustos SO, Chammas R. Galectin-3 Determines Tumor Cell Adaptive Strategies in Stressed Tumor Microenvironments [Internet]. Lausanne. 2016 ; 6( art. 127): 1-12.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fonc.2016.00127
  • Source: Behavioural Brain Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      SALAME, Samira et al. Distinct neuroplasticity processes are induced by different periods of acrobatic exercise training. Behavioural Brain Research, v. 308, p. 64-74, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2016.04.0296. Acesso em: 09 ago. 2024.
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      Salame, S., Garcia, P. C., Real, C. C., Borborema, J., Mota-Ortiz, S. R., Britto, L. R. G. de, & Pires, R. S. (2016). Distinct neuroplasticity processes are induced by different periods of acrobatic exercise training. Behavioural Brain Research, 308, 64-74. doi:10.1016/j.bbr.2016.04.0296
    • NLM

      Salame S, Garcia PC, Real CC, Borborema J, Mota-Ortiz SR, Britto LRG de, Pires RS. Distinct neuroplasticity processes are induced by different periods of acrobatic exercise training [Internet]. Behavioural Brain Research. 2016 ; 308 64-74.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.bbr.2016.04.0296
    • Vancouver

      Salame S, Garcia PC, Real CC, Borborema J, Mota-Ortiz SR, Britto LRG de, Pires RS. Distinct neuroplasticity processes are induced by different periods of acrobatic exercise training [Internet]. Behavioural Brain Research. 2016 ; 308 64-74.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.bbr.2016.04.0296
  • Source: Epilepsy & Behavior. Unidade: ICB

    Assunto: FISIOLOGIA

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      ROCHA, Anna Karynna Alves de Alencar et al. Pilocarpine-induced epilepsy alters the expression and daily variation of the nuclear receptor RORα in the hippocampus of rats. Epilepsy & Behavior, v. 55, p. 38-46, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.yebeh.2015.11.026. Acesso em: 09 ago. 2024.
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      Rocha, A. K. A. de A., Lima, E. de, Amaral, F. G. do, Peres, R., Cipolla-Neto, J., & Amado, D. (2016). Pilocarpine-induced epilepsy alters the expression and daily variation of the nuclear receptor RORα in the hippocampus of rats. Epilepsy & Behavior, 55, 38-46. doi:10.1016/j.yebeh.2015.11.026
    • NLM

      Rocha AKA de A, Lima E de, Amaral FG do, Peres R, Cipolla-Neto J, Amado D. Pilocarpine-induced epilepsy alters the expression and daily variation of the nuclear receptor RORα in the hippocampus of rats [Internet]. Epilepsy & Behavior. 2016 ; 55 38-46.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.yebeh.2015.11.026
    • Vancouver

      Rocha AKA de A, Lima E de, Amaral FG do, Peres R, Cipolla-Neto J, Amado D. Pilocarpine-induced epilepsy alters the expression and daily variation of the nuclear receptor RORα in the hippocampus of rats [Internet]. Epilepsy & Behavior. 2016 ; 55 38-46.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.yebeh.2015.11.026
  • Source: Frontiers in Physiology. Unidade: ICB

    Assunto: ANATOMIA

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      CAMPOS. JULIANE C., et al. Mitochondrial quality control in cardiac diseases. Frontiers in Physiology, v. 7, p. 1-9, 2016Tradução . . Disponível em: https://doi.org/10.3389/fphys.2016.00479. Acesso em: 09 ago. 2024.
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      Campos. Juliane C.,, Bozi, L. H. M., Bechara, L. R. G., Lima, V. M., & Ferreira, J. C. B. (2016). Mitochondrial quality control in cardiac diseases. Frontiers in Physiology, 7, 1-9. doi:10.3389/fphys.2016.00479
    • NLM

      Campos. Juliane C., Bozi LHM, Bechara LRG, Lima VM, Ferreira JCB. Mitochondrial quality control in cardiac diseases [Internet]. Frontiers in Physiology. 2016 ; 7 1-9.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fphys.2016.00479
    • Vancouver

      Campos. Juliane C., Bozi LHM, Bechara LRG, Lima VM, Ferreira JCB. Mitochondrial quality control in cardiac diseases [Internet]. Frontiers in Physiology. 2016 ; 7 1-9.[citado 2024 ago. 09 ] Available from: https://doi.org/10.3389/fphys.2016.00479
  • Source: Brazilian Dental Journal. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      NÓBREGA, Letícia M. M. et al. Molecular identification of cultivable bacteria from infected root canals associated with acute apical abscess. Brazilian Dental Journal, v. 27, n. 3, p. 318-324, 2016Tradução . . Disponível em: https://doi.org/10.1590/0103-6440201600715. Acesso em: 09 ago. 2024.
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      Nóbrega, L. M. M., Montagner, F., Ribeiro, A. C., Mayer, M. P. A., & Gomes, B. P. F. A. (2016). Molecular identification of cultivable bacteria from infected root canals associated with acute apical abscess. Brazilian Dental Journal, 27( 3), 318-324. doi:10.1590/0103-6440201600715
    • NLM

      Nóbrega LMM, Montagner F, Ribeiro AC, Mayer MPA, Gomes BPFA. Molecular identification of cultivable bacteria from infected root canals associated with acute apical abscess [Internet]. Brazilian Dental Journal. 2016 ; 27( 3): 318-324.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/0103-6440201600715
    • Vancouver

      Nóbrega LMM, Montagner F, Ribeiro AC, Mayer MPA, Gomes BPFA. Molecular identification of cultivable bacteria from infected root canals associated with acute apical abscess [Internet]. Brazilian Dental Journal. 2016 ; 27( 3): 318-324.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/0103-6440201600715
  • Source: PLOS ONE. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      MARTINS-PINHEIRO, Marinalva et al. Evolutionary and functional relationships of the dha regulon by genomic context analysis. PLOS ONE, v. 11, n. 3, p. 1-13, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0150772. Acesso em: 09 ago. 2024.
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      Martins-Pinheiro, M., Lima, W. C., Asif, H., Oller, C. A., & Menck, C. F. M. (2016). Evolutionary and functional relationships of the dha regulon by genomic context analysis. PLOS ONE, 11( 3), 1-13. doi:10.1371/journal.pone.0150772
    • NLM

      Martins-Pinheiro M, Lima WC, Asif H, Oller CA, Menck CFM. Evolutionary and functional relationships of the dha regulon by genomic context analysis [Internet]. PLOS ONE. 2016 ; 11( 3): 1-13.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0150772
    • Vancouver

      Martins-Pinheiro M, Lima WC, Asif H, Oller CA, Menck CFM. Evolutionary and functional relationships of the dha regulon by genomic context analysis [Internet]. PLOS ONE. 2016 ; 11( 3): 1-13.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pone.0150772
  • Source: Infection and Immunity. Unidade: ICB

    Subjects: MALÁRIA, DOENÇAS PARASITÁRIAS, ABORTO, PARASITOLOGIA, PLASMODIUM, GRAVIDEZ

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      MORAES, Luciana V. de et al. Murine Model for Preclinical Studies of Var2CSA-Mediated PathologyAssociated with Malaria in Pregnancy. Infection and Immunity, v. 84, n. 6, 2016Tradução . . Disponível em: https://doi.org/10.1128/IAI.01207-15. Acesso em: 09 ago. 2024.
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      Moraes, L. V. de, Dechavanne, S., Sousa, P. M., Barateiro, A., Cunha, S. F., Nunes-Silva, S., et al. (2016). Murine Model for Preclinical Studies of Var2CSA-Mediated PathologyAssociated with Malaria in Pregnancy. Infection and Immunity, 84( 6). doi:10.1128/IAI.01207-15
    • NLM

      Moraes LV de, Dechavanne S, Sousa PM, Barateiro A, Cunha SF, Nunes-Silva S, Lima FA, Murillo O, Marinho CRF, Gangnard S, Srivastava A, Braks JA, Janse CJ, Gamain B, Franke-Fayard B, Penha-Gonçalves C. Murine Model for Preclinical Studies of Var2CSA-Mediated PathologyAssociated with Malaria in Pregnancy [Internet]. Infection and Immunity. 2016 ; 84( 6):[citado 2024 ago. 09 ] Available from: https://doi.org/10.1128/IAI.01207-15
    • Vancouver

      Moraes LV de, Dechavanne S, Sousa PM, Barateiro A, Cunha SF, Nunes-Silva S, Lima FA, Murillo O, Marinho CRF, Gangnard S, Srivastava A, Braks JA, Janse CJ, Gamain B, Franke-Fayard B, Penha-Gonçalves C. Murine Model for Preclinical Studies of Var2CSA-Mediated PathologyAssociated with Malaria in Pregnancy [Internet]. Infection and Immunity. 2016 ; 84( 6):[citado 2024 ago. 09 ] Available from: https://doi.org/10.1128/IAI.01207-15
  • Source: PLoS Neglected Tropical Diseases. Unidade: ICB

    Subjects: PARASITOLOGIA, LEISHMANIA BRASILIENSIS, LEISHMANIOSE CUTÂNEA, ANTINEOPLÁSICOS

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      COELHO, Adriano C. et al. A luciferase-expressing Leishmania braziliensis line that leads to sustained skin lesions in BALB/c mice and allows monitoring of miltefosine treatment outcome. PLoS Neglected Tropical Diseases, v. 10, n. 5, p. 1-16, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0004660. Acesso em: 09 ago. 2024.
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      Coelho, A. C., Oliveira, J. C., Espada, C. R., Reimão, J. Q., Trinconi, C. T., & Uliana, S. R. B. (2016). A luciferase-expressing Leishmania braziliensis line that leads to sustained skin lesions in BALB/c mice and allows monitoring of miltefosine treatment outcome. PLoS Neglected Tropical Diseases, 10( 5), 1-16. doi:10.1371/journal.pntd.0004660
    • NLM

      Coelho AC, Oliveira JC, Espada CR, Reimão JQ, Trinconi CT, Uliana SRB. A luciferase-expressing Leishmania braziliensis line that leads to sustained skin lesions in BALB/c mice and allows monitoring of miltefosine treatment outcome [Internet]. PLoS Neglected Tropical Diseases. 2016 ; 10( 5): 1-16.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pntd.0004660
    • Vancouver

      Coelho AC, Oliveira JC, Espada CR, Reimão JQ, Trinconi CT, Uliana SRB. A luciferase-expressing Leishmania braziliensis line that leads to sustained skin lesions in BALB/c mice and allows monitoring of miltefosine treatment outcome [Internet]. PLoS Neglected Tropical Diseases. 2016 ; 10( 5): 1-16.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1371/journal.pntd.0004660
  • Source: Brazilian Dental Journal. Unidade: ICB

    Subjects: PATOLOGIA BUCAL, BIOFILMES, CÉLULAS EPITELIAIS, HIDROXIAPATITA, PROTEÍNAS SALIVARES

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      NUNES, Ana Carla Robatto e LONGO, Priscila Larcher e MAYER, Marcia Pinto Alves. Influence of Aae Autotransporter Protein on Adhesion and Biofilm Formation by Aggregatibacter actinomycetemcomitans. Brazilian Dental Journal, v. 27, n. 3, p. 255-260, 2016Tradução . . Disponível em: https://doi.org/10.1590/0103-6440201600260. Acesso em: 09 ago. 2024.
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      Nunes, A. C. R., Longo, P. L., & Mayer, M. P. A. (2016). Influence of Aae Autotransporter Protein on Adhesion and Biofilm Formation by Aggregatibacter actinomycetemcomitans. Brazilian Dental Journal, 27( 3), 255-260. doi:10.1590/0103-6440201600260
    • NLM

      Nunes ACR, Longo PL, Mayer MPA. Influence of Aae Autotransporter Protein on Adhesion and Biofilm Formation by Aggregatibacter actinomycetemcomitans [Internet]. Brazilian Dental Journal. 2016 ; 27( 3): 255-260.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/0103-6440201600260
    • Vancouver

      Nunes ACR, Longo PL, Mayer MPA. Influence of Aae Autotransporter Protein on Adhesion and Biofilm Formation by Aggregatibacter actinomycetemcomitans [Internet]. Brazilian Dental Journal. 2016 ; 27( 3): 255-260.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/0103-6440201600260
  • Source: Addiction Biology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      QUADIR, Sema G. et al. Homer2 regulates alcohol and stress cross-sensitization. Addiction Biology, v. 21, n. 3, p. 613-633, 2016Tradução . . Disponível em: https://doi.org/10.1111/adb.12252. Acesso em: 09 ago. 2024.
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      Quadir, S. G., Santos, J. R. B. dos, Campbell, R., Wroten, M. G., Singh, N., Holloway, J. J., et al. (2016). Homer2 regulates alcohol and stress cross-sensitization. Addiction Biology, 21( 3), 613-633. doi:10.1111/adb.12252
    • NLM

      Quadir SG, Santos JRB dos, Campbell R, Wroten MG, Singh N, Holloway JJ, Bal SK, Camarini R, Szumlinski K. Homer2 regulates alcohol and stress cross-sensitization [Internet]. Addiction Biology. 2016 ; 21( 3): 613-633.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1111/adb.12252
    • Vancouver

      Quadir SG, Santos JRB dos, Campbell R, Wroten MG, Singh N, Holloway JJ, Bal SK, Camarini R, Szumlinski K. Homer2 regulates alcohol and stress cross-sensitization [Internet]. Addiction Biology. 2016 ; 21( 3): 613-633.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1111/adb.12252
  • Source: Brain Research Bulletin. Unidade: ICB

    Assunto: FARMACOLOGIA

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      CAMARINI, Rosana e PAUTASSI, Ricardo Marcos. Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression. Brain Research Bulletin, v. 125, p. 53-78, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2016.04.006. Acesso em: 09 ago. 2024.
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      Camarini, R., & Pautassi, R. M. (2016). Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression. Brain Research Bulletin, 125, 53-78. doi:10.1016/j.brainresbull.2016.04.006
    • NLM

      Camarini R, Pautassi RM. Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression [Internet]. Brain Research Bulletin. 2016 ; 125 53-78.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.brainresbull.2016.04.006
    • Vancouver

      Camarini R, Pautassi RM. Behavioral sensitization to ethanol: neural basis and factors that influence its acquisition and expression [Internet]. Brain Research Bulletin. 2016 ; 125 53-78.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.brainresbull.2016.04.006
  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SIMÕES, Daniel et al. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase. Journal of Endocrinology, v. 231, n. 3, p. 235-244, 2016Tradução . . Disponível em: https://doi.org/10.1530/JOE-16-0259. Acesso em: 09 ago. 2024.
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      Simões, D., Riva, P., Peliciari-Garcia, R. A., Cruzat, V. F., Graciano, M. fernanda, Munhoz, A. C., et al. (2016). Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase. Journal of Endocrinology, 231( 3), 235-244. doi:10.1530/JOE-16-0259
    • NLM

      Simões D, Riva P, Peliciari-Garcia RA, Cruzat VF, Graciano M fernanda, Munhoz AC, Taneda M, Cipolla-Neto J, Carpinelli AR. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase [Internet]. Journal of Endocrinology. 2016 ; 231( 3): 235-244.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1530/JOE-16-0259
    • Vancouver

      Simões D, Riva P, Peliciari-Garcia RA, Cruzat VF, Graciano M fernanda, Munhoz AC, Taneda M, Cipolla-Neto J, Carpinelli AR. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase [Internet]. Journal of Endocrinology. 2016 ; 231( 3): 235-244.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1530/JOE-16-0259
  • Source: Scienctific Reports. Unidade: ICB

    Assunto: FISIOLOGIA

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      LABBÉ, Sébastien M. et al. mTORC1 is required for brown adipose tissue recruitment and metabolic adaptation to cold. Scienctific Reports, v. 6, n. 37223, p. 1-17, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep37223. Acesso em: 09 ago. 2024.
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      Labbé, S. M., Mouchiroud, M., Caron, A., Secco, B., Freinkman, E., Lamoureux, G., et al. (2016). mTORC1 is required for brown adipose tissue recruitment and metabolic adaptation to cold. Scienctific Reports, 6( 37223), 1-17. doi:10.1038/srep37223
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      Labbé SM, Mouchiroud M, Caron A, Secco B, Freinkman E, Lamoureux G, Gélinas Y, Lecomte R, Bossé Y, Chimin P, Festuccia WTL, Richard D, Laplante M. mTORC1 is required for brown adipose tissue recruitment and metabolic adaptation to cold [Internet]. Scienctific Reports. 2016 ; 6( 37223): 1-17.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1038/srep37223
    • Vancouver

      Labbé SM, Mouchiroud M, Caron A, Secco B, Freinkman E, Lamoureux G, Gélinas Y, Lecomte R, Bossé Y, Chimin P, Festuccia WTL, Richard D, Laplante M. mTORC1 is required for brown adipose tissue recruitment and metabolic adaptation to cold [Internet]. Scienctific Reports. 2016 ; 6( 37223): 1-17.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1038/srep37223
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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      MATTERA, Maria Sara de Lima Coutinho et al. Maternal periodontitis decreases plasma membrane GLUT4 content in skeletal muscle of adult offspring. Life Sciences, v. 148, p. 194-200, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.02.010. Acesso em: 09 ago. 2024.
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      Mattera, M. S. de L. C., Chiba, F. Y., Mota, M. S. de O. da, Pereira, R. F., Ervolino, E., Chaves Neto, A. H., et al. (2016). Maternal periodontitis decreases plasma membrane GLUT4 content in skeletal muscle of adult offspring. Life Sciences, 148, 194-200. doi:10.1016/j.lfs.2016.02.010
    • NLM

      Mattera MS de LC, Chiba FY, Mota MS de O da, Pereira RF, Ervolino E, Chaves Neto AH, Salzedas LMP, Scaramele NF, Silva CA, Okamoto MM, Machado UF, Sumida DH. Maternal periodontitis decreases plasma membrane GLUT4 content in skeletal muscle of adult offspring. [Internet]. Life Sciences. 2016 ; 148 194-200.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.010
    • Vancouver

      Mattera MS de LC, Chiba FY, Mota MS de O da, Pereira RF, Ervolino E, Chaves Neto AH, Salzedas LMP, Scaramele NF, Silva CA, Okamoto MM, Machado UF, Sumida DH. Maternal periodontitis decreases plasma membrane GLUT4 content in skeletal muscle of adult offspring. [Internet]. Life Sciences. 2016 ; 148 194-200.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1016/j.lfs.2016.02.010
  • Source: Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine. Unidades: FMRP, ICB

    Subjects: HOMEOSTASE, FERRO, ESTRESSE OXIDATIVO, PROTEÍNAS

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      FERREIRA, Ivan Gonçalves de Castro et al. Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine, v. 29, n. 5, p. 851-862, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10534-016-9956-y. Acesso em: 09 ago. 2024.
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      Ferreira, I. G. de C., Rodrigues, M. M., Silva Neto, J. F. da, Mazzon, R. R., & Marques, M. do V. (2016). Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine, 29( 5), 851-862. doi:10.1007/s10534-016-9956-y
    • NLM

      Ferreira IG de C, Rodrigues MM, Silva Neto JF da, Mazzon RR, Marques M do V. Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus [Internet]. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine. 2016 ; 29( 5): 851-862.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1007/s10534-016-9956-y
    • Vancouver

      Ferreira IG de C, Rodrigues MM, Silva Neto JF da, Mazzon RR, Marques M do V. Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus [Internet]. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine. 2016 ; 29( 5): 851-862.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1007/s10534-016-9956-y
  • Source: American Journal of Physiology. Cell Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      CALIL-SILVEIRA, Jamile et al. Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, v. 310, n. 7, p. C576-C582, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00210.2015. Acesso em: 09 ago. 2024.
    • APA

      Calil-Silveira, J., Serrano-Nascimento, C., Kopp, P. A., & Nunes, M. T. (2016). Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, 310( 7), C576-C582. doi:10.1152/ajpcell.00210.2015
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
  • Source: Anais da Academia Brasileira de Ciências. Unidade: ICB

    Subjects: FERRO, CORAÇÃO, MÚSCULO ESQUELÉTICO

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    • ABNT

      SOUZA, Janaina Sena de e RACT, Erika Lia Brunetto e NUNES, Maria Tereza. Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats. Anais da Academia Brasileira de Ciências, v. 88, n. 4, p. 2277-2290, 2016Tradução . . Disponível em: https://doi.org/10.1590/0001-3765201620160173. Acesso em: 09 ago. 2024.
    • APA

      Souza, J. S. de, Ract, E. L. B., & Nunes, M. T. (2016). Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats. Anais da Academia Brasileira de Ciências, 88( 4), 2277-2290. doi:10.1590/0001-3765201620160173
    • NLM

      Souza JS de, Ract ELB, Nunes MT. Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats [Internet]. Anais da Academia Brasileira de Ciências. 2016 ;88( 4): 2277-2290.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/0001-3765201620160173
    • Vancouver

      Souza JS de, Ract ELB, Nunes MT. Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats [Internet]. Anais da Academia Brasileira de Ciências. 2016 ;88( 4): 2277-2290.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1590/0001-3765201620160173
  • Source: American Journal of Physiology. Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      CASARE, Fernando Augusto Malavazzi et al. Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, v. 310, n. 11, p. F1295-F1307, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00471.2015. Acesso em: 09 ago. 2024.
    • APA

      Casare, F. A. M., Thieme, K., Costa-Pessoa, J. M., Rossoni, L. V., Couto, G. K., Fernandes, F. B., et al. (2016). Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, 310( 11), F1295-F1307. doi:10.1152/ajprenal.00471.2015
    • NLM

      Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
    • Vancouver

      Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
  • Source: Journal of Molecular Endocrinology. Unidade: ICB

    Assunto: HISTOLOGIA

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      MEYEROVICH, Kira et al. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of Molecular Endocrinology, v. 57, n. 1, p. R1-R17, 2016Tradução . . Disponível em: https://doi.org/10.1530/JME-15-0306. Acesso em: 09 ago. 2024.
    • APA

      Meyerovich, K., Ortis, F., Allagnat, F., & Cardozo, A. K. (2016). Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of Molecular Endocrinology, 57( 1), R1-R17. doi:10.1530/JME-15-0306
    • NLM

      Meyerovich K, Ortis F, Allagnat F, Cardozo AK. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation [Internet]. Journal of Molecular Endocrinology. 2016 ; 57( 1): R1-R17.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1530/JME-15-0306
    • Vancouver

      Meyerovich K, Ortis F, Allagnat F, Cardozo AK. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation [Internet]. Journal of Molecular Endocrinology. 2016 ; 57( 1): R1-R17.[citado 2024 ago. 09 ] Available from: https://doi.org/10.1530/JME-15-0306

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