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Artigo de periodico
Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats (2022)
Brazão, Vânia; Colato, Rafaela Pravato ; Santello, Fabricia Helena; Duarte, Andressa ; Goulart, Amanda; Oliveira, Pedro Alexandre Sampaio ; Silva, Carla Brigagão Pacheco da ; Tirapelli, Carlos Renato ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo ; Prado Júnior, José Clóvis do
Source: Experimental Gerontology. Unidades: EERP, FMRP, FCFRP
Subjects: ANTIOXIDANTES, DOENÇA DE CHAGAS, PERÓXIDO DE HIDROGÊNIO, MELATONINA, ESTRESSE OXIDATIVO
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ABNT
BRAZÃO, Vânia et al. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, v. 167, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2022.111895. Acesso em: 26 jun. 2024.
APA
Brazão, V., Colato, R. P., Santello, F. H., Duarte, A., Goulart, A., Oliveira, P. A. S., et al. (2022). Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, 167, 1-9. doi:10.1016/j.exger.2022.111895
NLM
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Vancouver
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Artigo de periodico
Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis (2021)
Valle, Gabriel Tavares do; Ricci, Sthefany Teodoro; Silva, Alessandra Oliveira; Tirapelli, Carlos Renato ; Ceron, Carla Speroni
Source: Canadian Journal of Physiology and Pharmacology. Unidade: EERP
Subjects: ANIMAIS, PERÓXIDO DE HIDROGÊNIO, RATOS, ESTRESSE OXIDATIVO, SEPSE
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ABNT
VALLE, Gabriel Tavares do et al. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis. Canadian Journal of Physiology and Pharmacology, v. 99, n. 7, p. 699-707, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2020-0564. Acesso em: 26 jun. 2024.
APA
Valle, G. T. do, Ricci, S. T., Silva, A. O., Tirapelli, C. R., & Ceron, C. S. (2021). Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis. Canadian Journal of Physiology and Pharmacology, 99( 7), 699-707. doi:10.1139/cjpp-2020-0564
NLM
Valle GT do, Ricci ST, Silva AO, Tirapelli CR, Ceron CS. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 699-707.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1139/cjpp-2020-0564
Vancouver
Valle GT do, Ricci ST, Silva AO, Tirapelli CR, Ceron CS. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 699-707.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1139/cjpp-2020-0564
Artigo de periodico
Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats (2020)
Gonzaga, Natália de Almeida ; Vale, Gabriel Tavares do ; Silva, Carla Beatriz Pereira da ; Pinheiro, Lucas Cezar ; Leite, Letícia N.; Carneiro, Fernando Silva ; Tanus-Santos, José Eduardo ; Tirapelli, Carlos Renato
Source: Nitric Oxide. Unidades: EERP, FMRP
Subjects: DISFUNÇÃO ERÉTIL, HIPERTENSÃO, NITRITOS, ESPÉCIES REATIVAS DE OXIGÊNIO, ANTIOXIDANTES
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ABNT
GONZAGA, Natália de Almeida et al. Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats. Nitric Oxide, v. 94, p. 19-26, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.10.006. Acesso em: 26 jun. 2024.
APA
Gonzaga, N. de A., Vale, G. T. do, Silva, C. B. P. da, Pinheiro, L. C., Leite, L. N., Carneiro, F. S., et al. (2020). Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats. Nitric Oxide, 94, 19-26. doi:10.1016/j.niox.2019.10.006
NLM
Gonzaga N de A, Vale GT do, Silva CBP da, Pinheiro LC, Leite LN, Carneiro FS, Tanus-Santos JE, Tirapelli CR. Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats [Internet]. Nitric Oxide. 2020 ; 94 19-26.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.niox.2019.10.006
Vancouver
Gonzaga N de A, Vale GT do, Silva CBP da, Pinheiro LC, Leite LN, Carneiro FS, Tanus-Santos JE, Tirapelli CR. Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats [Internet]. Nitric Oxide. 2020 ; 94 19-26.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.niox.2019.10.006
Artigo de periodico
Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors (2020)
Vale, Gabriel Tavares do; Leoni, Drieli; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Uliana, Daniela L; La Gata, Davi C; Resstel, Leonardo Barbosa Moraes; Padovan, Cláudia Maria ; Tirapelli, Carlos Renato
Source: Stress. Unidades: FMRP, FFCLRP, EERP
Subjects: ESTRESSE, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
VALE, Gabriel Tavares do et al. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors. Stress, v. 23, n. 3, p. 328-337, 2020Tradução . . Disponível em: https://doi.org/10.1080/10253890.2019.1675627. Acesso em: 26 jun. 2024.
APA
Vale, G. T. do, Leoni, D., Sousa, A. H. de, Gonzaga, N. de A., Uliana, D. L., La Gata, D. C., et al. (2020). Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors. Stress, 23( 3), 328-337. doi:10.1080/10253890.2019.1675627
NLM
Vale GT do, Leoni D, Sousa AH de, Gonzaga N de A, Uliana DL, La Gata DC, Resstel LBM, Padovan CM, Tirapelli CR. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors [Internet]. Stress. 2020 ; 23( 3): 328-337.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1080/10253890.2019.1675627
Vancouver
Vale GT do, Leoni D, Sousa AH de, Gonzaga N de A, Uliana DL, La Gata DC, Resstel LBM, Padovan CM, Tirapelli CR. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors [Internet]. Stress. 2020 ; 23( 3): 328-337.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1080/10253890.2019.1675627
Artigo de periodico
Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection (2020)
Malacco, Nathalia Luisa Sousa de Oliveira; Souza, Jessica Amanda Marques; Martins, Flavia Rayssa Braga; Rachid, Milene Alvarenga; Simplicio, Janaina Aparecida; Tirapelli, Carlos Renato ; Sabino, Adriano de Paula; Queiroz-Junior, Celso Martins; Goes, Grazielle Ribeiro; Vieira, Leda Quercia; Souza, Danielle Glória; Pinho, Vanessa; Teixeira, Mauro Martins; Soriani, Frederico Marianetti
Source: eLife. Unidade: EERP
Subjects: ASPERGILLUS, ETANOL, CONSUMO, IMUNOLOGIA, DOENÇAS INFECCIOSAS, INFLAMAÇÃO, MICROBIOLOGIA, NEUTRÓFILOS
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ABNT
MALACCO, Nathalia Luisa Sousa de Oliveira et al. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection. eLife, v. 9, 2020Tradução . . Disponível em: https://doi.org/10.7554/eLife.58855. Acesso em: 26 jun. 2024.
APA
Malacco, N. L. S. de O., Souza, J. A. M., Martins, F. R. B., Rachid, M. A., Simplicio, J. A., Tirapelli, C. R., et al. (2020). Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection. eLife, 9. doi:10.7554/eLife.58855
NLM
Malacco NLS de O, Souza JAM, Martins FRB, Rachid MA, Simplicio JA, Tirapelli CR, Sabino A de P, Queiroz-Junior CM, Goes GR, Vieira LQ, Souza DG, Pinho V, Teixeira MM, Soriani FM. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection [Internet]. eLife. 2020 ; 9[citado 2024 jun. 26 ] Available from: https://doi.org/10.7554/eLife.58855
Vancouver
Malacco NLS de O, Souza JAM, Martins FRB, Rachid MA, Simplicio JA, Tirapelli CR, Sabino A de P, Queiroz-Junior CM, Goes GR, Vieira LQ, Souza DG, Pinho V, Teixeira MM, Soriani FM. Chronic ethanol consumption compromises neutrophil function in acute pulmonary Aspergillus fumigatus infection [Internet]. eLife. 2020 ; 9[citado 2024 jun. 26 ] Available from: https://doi.org/10.7554/eLife.58855
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 26 jun. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder (2019)
Vale, Gabriel Tavares do; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Oliveira, Mariana G. de; Justo, Alberto F. O; Alexandre, Eduardo C.; Tanus-Santos, José Eduardo ; Antunes, Edson; Tirapelli, Carlos Renato
Source: Canadian Journal of Physiology and Pharmacology. Unidades: FMRP, EERP
Subjects: ETANOL, ESTRESSE OXIDATIVO, BEXIGA
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ABNT
VALE, Gabriel Tavares do et al. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, v. 97, n. 12, p. 1103-1114, 2019Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2019-0143. Acesso em: 26 jun. 2024.
APA
Vale, G. T. do, Sousa, A. H. de, Gonzaga, N. de A., Oliveira, M. G. de, Justo, A. F. O., Alexandre, E. C., et al. (2019). Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, 97( 12), 1103-1114. doi:10.1139/cjpp-2019-0143
NLM
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Vancouver
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Artigo de periodico
Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms (2019)
Nakashima, Marcelo A.; Silva, Carla Beatriz Pereira da ; Gonzaga, Natália de Almeida; Simplicio, Janaina A.; Omoto, Ana Carolina Mieko ; Tirapelli, Luis Fernando; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Source: Cytokine. Unidades: FMRP, EERP
Subjects: ESTRESSE OXIDATIVO, ETANOL, CITOTOXINAS, SEPSE, INTERLEUCINAS
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ABNT
NAKASHIMA, Marcelo A. et al. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, v. 121, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2019.154734. Acesso em: 26 jun. 2024.
APA
Nakashima, M. A., Silva, C. B. P. da, Gonzaga, N. de A., Simplicio, J. A., Omoto, A. C. M., Tirapelli, L. F., et al. (2019). Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, 121. doi:10.1016/j.cyto.2019.154734
NLM
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Vancouver
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Artigo de periodico
Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin (2019)
Gonzaga, Natália de Almeida; Awata, Wanessa Mayumi Carvalho; Tanus-Santos, José Eduardo ; Padovan, Júlio C.; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: AORTA, ETANOL (REMOÇÃO)
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ABNT
GONZAGA, Natália de Almeida et al. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin. European Journal of Pharmacology, v. 847, p. 103-112, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.01.055. Acesso em: 26 jun. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Tanus-Santos, J. E., Padovan, J. C., & Tirapelli, C. R. (2019). Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin. European Journal of Pharmacology, 847, 103-112. doi:10.1016/j.ejphar.2019.01.055
NLM
Gonzaga N de A, Awata WMC, Tanus-Santos JE, Padovan JC, Tirapelli CR. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin [Internet]. European Journal of Pharmacology. 2019 ; 847 103-112.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2019.01.055
Vancouver
Gonzaga N de A, Awata WMC, Tanus-Santos JE, Padovan JC, Tirapelli CR. Mechanisms underlying vascular hypocontractility induced by ethanol withdrawal: role of cyclooxygenase 2-derived prostacyclin [Internet]. European Journal of Pharmacology. 2019 ; 847 103-112.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2019.01.055
Artigo de periodico
Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS (2018)
Ceron, Carla S.; Vale, Gabriel T. do; Simplicio, Janaina A.; Ricci, Sthefany T.; Martinis, Bruno Spinosa de; Freitas, Andressa de; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FFCLRP, EERP
Subjects: ESTRESSE OXIDATIVO, ETANOL, CITOCINAS, SEPSE, INTERLEUCINAS
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ABNT
CERON, Carla S. et al. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, v. 825, p. 39-47, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2018.02.008. Acesso em: 26 jun. 2024.
APA
Ceron, C. S., Vale, G. T. do, Simplicio, J. A., Ricci, S. T., Martinis, B. S. de, Freitas, A. de, & Tirapelli, C. R. (2018). Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, 825, 39-47. doi:10.1016/j.ejphar.2018.02.008
NLM
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008
Vancouver
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008
Artigo de periodico
Effects of melatonin on thymic and oxidative stress dysfunctions during Trypanosoma cruzi infection (2018)
Muniz, Vânia Brazão; Colato, Rafaela Pravato; Santello, Fabricia Helena; Vale, Gabriel Tavares do; Gonzaga, Natália de Almeida; Tirapelli, Carlos Renato; Prado Júnior, José Clóvis do
Source: Journal of Pineal Research. Unidades: EERP, FCFRP
Subjects: ANTIOXIDANTES, DOENÇA DE CHAGAS, ESTRESSE OXIDATIVO, TRYPANOSOMA CRUZI, TIMO (GLÂNDULA ENDÓCRINA)
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ABNT
MUNIZ, Vânia Brazão et al. Effects of melatonin on thymic and oxidative stress dysfunctions during Trypanosoma cruzi infection. Journal of Pineal Research, v. 65, n. 3, p. [12 ], 2018Tradução . . Disponível em: https://doi.org/10.1111/jpi.12510. Acesso em: 26 jun. 2024.
APA
Muniz, V. B., Colato, R. P., Santello, F. H., Vale, G. T. do, Gonzaga, N. de A., Tirapelli, C. R., & Prado Júnior, J. C. do. (2018). Effects of melatonin on thymic and oxidative stress dysfunctions during Trypanosoma cruzi infection. Journal of Pineal Research, 65( 3), [12 ]. doi:10.1111/jpi.12510
NLM
Muniz VB, Colato RP, Santello FH, Vale GT do, Gonzaga N de A, Tirapelli CR, Prado Júnior JC do. Effects of melatonin on thymic and oxidative stress dysfunctions during Trypanosoma cruzi infection [Internet]. Journal of Pineal Research. 2018 ; 65( 3): [12 ].[citado 2024 jun. 26 ] Available from: https://doi.org/10.1111/jpi.12510
Vancouver
Muniz VB, Colato RP, Santello FH, Vale GT do, Gonzaga N de A, Tirapelli CR, Prado Júnior JC do. Effects of melatonin on thymic and oxidative stress dysfunctions during Trypanosoma cruzi infection [Internet]. Journal of Pineal Research. 2018 ; 65( 3): [12 ].[citado 2024 jun. 26 ] Available from: https://doi.org/10.1111/jpi.12510
Artigo de periodico
Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide (2018)
Gonzaga, Natália de Almeida; Awata, Wanessa M. C.; Vale, Gabriel Tavares do; Marchi, Kátia Colombo; Muniz, Jaqueline Jóice ; Santos, José Eduardo Tanus dos ; Tirapelli, Carlos Renato
Source: Vascular Pharmacology. Unidades: FMRP, EERP
Subjects: ETANOL, PERÓXIDO DE HIDROGÊNIO, TECIDO ADIPOSO, CATALASE, ENDOTÉLIO VASCULAR
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ABNT
GONZAGA, Natália de Almeida et al. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide. Vascular Pharmacology, v. 111, p. 44-53, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2018.08.010. Acesso em: 26 jun. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Vale, G. T. do, Marchi, K. C., Muniz, J. J., Santos, J. E. T. dos, & Tirapelli, C. R. (2018). Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide. Vascular Pharmacology, 111, 44-53. doi:10.1016/j.vph.2018.08.010
NLM
Gonzaga N de A, Awata WMC, Vale GT do, Marchi KC, Muniz JJ, Santos JET dos, Tirapelli CR. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide [Internet]. Vascular Pharmacology. 2018 ; 111 44-53.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.vph.2018.08.010
Vancouver
Gonzaga N de A, Awata WMC, Vale GT do, Marchi KC, Muniz JJ, Santos JET dos, Tirapelli CR. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide [Internet]. Vascular Pharmacology. 2018 ; 111 44-53.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.vph.2018.08.010
Artigo de periodico
Quercetin decreases the activity of matrix metalloproteinase-2 and ameliorates vascular remodeling in renovascular hypertension (2018)
Pereira, Sherliane C.; Parente, Juliana M.; Belo, Vanessa A.; Mendes, Atlante S.; Gonzaga, Natália A.; Vale, Gabriel T. do; Ceron, Carla S.; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato; Castro, Michele Mazzaron de
Source: Atherosclerosis. Unidades: FMRP, EERP
Subjects: ANTIOXIDANTES, METALOPROTEINASES, HIPERTENSÃO, ESTRESSE OXIDATIVO
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ABNT
PEREIRA, Sherliane C. et al. Quercetin decreases the activity of matrix metalloproteinase-2 and ameliorates vascular remodeling in renovascular hypertension. Atherosclerosis, v. 270, p. 146-153, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.atherosclerosis.2018.01.031. Acesso em: 26 jun. 2024.
APA
Pereira, S. C., Parente, J. M., Belo, V. A., Mendes, A. S., Gonzaga, N. A., Vale, G. T. do, et al. (2018). Quercetin decreases the activity of matrix metalloproteinase-2 and ameliorates vascular remodeling in renovascular hypertension. Atherosclerosis, 270, 146-153. doi:10.1016/j.atherosclerosis.2018.01.031
NLM
Pereira SC, Parente JM, Belo VA, Mendes AS, Gonzaga NA, Vale GT do, Ceron CS, Tanus-Santos JE, Tirapelli CR, Castro MM de. Quercetin decreases the activity of matrix metalloproteinase-2 and ameliorates vascular remodeling in renovascular hypertension [Internet]. Atherosclerosis. 2018 ; 270 146-153.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2018.01.031
Vancouver
Pereira SC, Parente JM, Belo VA, Mendes AS, Gonzaga NA, Vale GT do, Ceron CS, Tanus-Santos JE, Tirapelli CR, Castro MM de. Quercetin decreases the activity of matrix metalloproteinase-2 and ameliorates vascular remodeling in renovascular hypertension [Internet]. Atherosclerosis. 2018 ; 270 146-153.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2018.01.031
Artigo de periodico
Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers (2018)
Pinto, Carina Guidi; Marchi, Kátia Colombo; Arizza, Ailton Amarante; Leite, Ana Paula Silveira; Tirapelli, Carlos Renato ; Matheus, Selma Maria Michelin
Source: Acta Histochemica. Unidades: EERP, FMRP
Subjects: FIBRAS MUSCULARES, RATOS, ABUSO DE ÁLCOOL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINTO, Carina Guidi et al. Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers. Acta Histochemica, v. 120, n. 8, p. 789-796, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.acthis.2018.09.004. Acesso em: 26 jun. 2024.
APA
Pinto, C. G., Marchi, K. C., Arizza, A. A., Leite, A. P. S., Tirapelli, C. R., & Matheus, S. M. M. (2018). Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers. Acta Histochemica, 120( 8), 789-796. doi:10.1016/j.acthis.2018.09.004
NLM
Pinto CG, Marchi KC, Arizza AA, Leite APS, Tirapelli CR, Matheus SMM. Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers [Internet]. Acta Histochemica. 2018 ; 120( 8): 789-796.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.acthis.2018.09.004
Vancouver
Pinto CG, Marchi KC, Arizza AA, Leite APS, Tirapelli CR, Matheus SMM. Cholecalciferol in ethanol-preferring rats muscle fibers increases the number and area of type II fibers [Internet]. Acta Histochemica. 2018 ; 120( 8): 789-796.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.acthis.2018.09.004
Artigo de periodico
Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection (2018)
Colato, Rafaela Pravato; Muniz, Vânia Brazão; Gabriel Tavares do Vale; Fabricia Helena Santello; Pedro Alexandre Sampaio; Tirapelli, Carlos Renato; Bisson, Gabriela Silva ; Prado Junior, José Clóvis do
Source: Cytokine. Unidades: EERP, FCFRP
Subjects: ESTRESSE OXIDATIVO, ENVELHECIMENTO, CITOCINAS, TIMO (SISTEMAS SANGUÍNEO E IMUNE), TRYPANOSOMA CRUZI
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COLATO, Rafaela Pravato et al. Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection. Cytokine, v. 111, p. 88-96, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2018.08.004. Acesso em: 26 jun. 2024.
APA
Colato, R. P., Muniz, V. B., Gabriel Tavares do Vale,, Fabricia Helena Santello,, Pedro Alexandre Sampaio,, Tirapelli, C. R., et al. (2018). Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection. Cytokine, 111, 88-96. doi:10.1016/j.cyto.2018.08.004
NLM
Colato RP, Muniz VB, Gabriel Tavares do Vale, Fabricia Helena Santello, Pedro Alexandre Sampaio, Tirapelli CR, Bisson GS, Prado Junior JC do. Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection [Internet]. Cytokine. 2018 ; 111 88-96.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.cyto.2018.08.004
Vancouver
Colato RP, Muniz VB, Gabriel Tavares do Vale, Fabricia Helena Santello, Pedro Alexandre Sampaio, Tirapelli CR, Bisson GS, Prado Junior JC do. Cytokine modulation, oxidative stress and thymic dysfunctions: role of age-related changes in the experimental Trypanosoma cruzi infection Age-related thymic dysfunctions and Trypanosoma cruzi infection [Internet]. Cytokine. 2018 ; 111 88-96.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.cyto.2018.08.004
Artigo de periodico
Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection (2017)
Brazão, Vânia; Santello, Fabrícia H.; Colato, Rafaela P.; Mazotti, Tamires T.; Tazinafo, Lucas F.; Toldo, Miriam Paula Alonso; Vale, Gabriel T. do; Tirapelli, Carlos Renato; Prado Júnior, José Clóvis do
Source: Journal of Pineal Research. Unidades: EERP, FCFRP
Subjects: TRYPANOSOMA CRUZI, MELATONINA, DOENÇA DE CHAGAS, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRAZÃO, Vânia et al. Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection. Journal of Pineal Research, v. 63, 2017Tradução . . Disponível em: https://doi.org/10.1111/jpi.12409. Acesso em: 26 jun. 2024.
APA
Brazão, V., Santello, F. H., Colato, R. P., Mazotti, T. T., Tazinafo, L. F., Toldo, M. P. A., et al. (2017). Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection. Journal of Pineal Research, 63. doi:10.1111/jpi.12409
NLM
Brazão V, Santello FH, Colato RP, Mazotti TT, Tazinafo LF, Toldo MPA, Vale GT do, Tirapelli CR, Prado Júnior JC do. Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection [Internet]. Journal of Pineal Research. 2017 ; 63[citado 2024 jun. 26 ] Available from: https://doi.org/10.1111/jpi.12409
Vancouver
Brazão V, Santello FH, Colato RP, Mazotti TT, Tazinafo LF, Toldo MPA, Vale GT do, Tirapelli CR, Prado Júnior JC do. Melatonin: antioxidant and modulatory properties in age‐related changes during Trypanosoma cruzi infection [Internet]. Journal of Pineal Research. 2017 ; 63[citado 2024 jun. 26 ] Available from: https://doi.org/10.1111/jpi.12409
Artigo de periodico
Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids (2017)
Diniz, Mariana C.; Olivon, Vania C.; Tavares, Lívia D.; Simplicio, Janaina A.; Gonzaga, Natália A.; Souza, Daniele G. de; Bendhack, Lusiane Maria; Tirapelli, Carlos Renato; Bonaventura, Daniella
Source: Life Sciences. Unidades: FCFRP, EERP
Subjects: NITRATOS, AORTA, VASODILATAÇÃO, OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Mariana C. et al. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, v. 176, p. 26-34, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.03.016. Acesso em: 26 jun. 2024.
APA
Diniz, M. C., Olivon, V. C., Tavares, L. D., Simplicio, J. A., Gonzaga, N. A., Souza, D. G. de, et al. (2017). Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, 176, 26-34. doi:10.1016/j.lfs.2017.03.016
NLM
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Vancouver
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016
Artigo de periodico
Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species (2017)
Côco, Hariane; Pernomian, Larissa; Pereira, Priscila C.; Gomes, Mayara Santos; Marchi, Katia C.; Lopes, Alexandre H.; Cunha, Thiago Mattar; Tirapelli, Carlos Renato; Oliveira, Ana Maria de
Source: Journal of Pharmacy and Pharmacology. Unidades: FCFRP, FMRP, EERP
Subjects: ANGIOTENSINA II, ARTÉRIAS CARÓTIDAS, ENDOTÉLIO VASCULAR, ESTRESSE PSICOLÓGICO, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CÔCO, Hariane et al. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species. Journal of Pharmacy and Pharmacology, v. 69, n. 1, p. 52-65, 2017Tradução . . Disponível em: https://doi.org/10.1111/jphp.12659. Acesso em: 26 jun. 2024.
APA
Côco, H., Pernomian, L., Pereira, P. C., Gomes, M. S., Marchi, K. C., Lopes, A. H., et al. (2017). Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species. Journal of Pharmacy and Pharmacology, 69( 1), 52-65. doi:10.1111/jphp.12659
NLM
Côco H, Pernomian L, Pereira PC, Gomes MS, Marchi KC, Lopes AH, Cunha TM, Tirapelli CR, Oliveira AM de. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species [Internet]. Journal of Pharmacy and Pharmacology. 2017 ; 69( 1): 52-65.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1111/jphp.12659
Vancouver
Côco H, Pernomian L, Pereira PC, Gomes MS, Marchi KC, Lopes AH, Cunha TM, Tirapelli CR, Oliveira AM de. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species [Internet]. Journal of Pharmacy and Pharmacology. 2017 ; 69( 1): 52-65.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1111/jphp.12659
Artigo de periodico
Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats (2017)
Hott, Sara C.; Gomes, Felipe V.; Uliana, Daniela L.; Vale, Gabriel T.; Tirapelli, Carlos Renato; Resstel, Leonardo Barbosa Moraes
Source: Neuropharmacology. Unidades: EERP, FMRP
Subjects: ÓXIDO NÍTRICO, SISTEMA CARDIOVASCULAR, GLUTAMATOS, MEDO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HOTT, Sara C. et al. Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats. Neuropharmacology, v. 112, n. Pt. A, p. 135-143, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2016.05.022. Acesso em: 26 jun. 2024.
APA
Hott, S. C., Gomes, F. V., Uliana, D. L., Vale, G. T., Tirapelli, C. R., & Resstel, L. B. M. (2017). Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats. Neuropharmacology, 112( Pt. A), 135-143. doi:10.1016/j.neuropharm.2016.05.022
NLM
Hott SC, Gomes FV, Uliana DL, Vale GT, Tirapelli CR, Resstel LBM. Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats [Internet]. Neuropharmacology. 2017 ; 112( Pt. A): 135-143.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.neuropharm.2016.05.022
Vancouver
Hott SC, Gomes FV, Uliana DL, Vale GT, Tirapelli CR, Resstel LBM. Bed nucleus of the stria terminalis NMDA receptors and nitric oxide modulate contextual fear conditioning in rats [Internet]. Neuropharmacology. 2017 ; 112( Pt. A): 135-143.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.neuropharm.2016.05.022
Artigo de periodico
Pharmacological characterization of the mechanisms underlying the vascular effects of succinate (2016)
Leite, Letícia N.; Gonzaga, Natália A.; Simplicio, Janaina A.; Vale, Gabriel T. do; Carballido, José M.; Alves Filho, José Carlos Farias; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: AORTA TORÁCICA, ENDOTÉLIO VASCULAR, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Letícia N. et al. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, v. 789, p. 334-343, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2016.07.045. Acesso em: 26 jun. 2024.
APA
Leite, L. N., Gonzaga, N. A., Simplicio, J. A., Vale, G. T. do, Carballido, J. M., Alves Filho, J. C. F., & Tirapelli, C. R. (2016). Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, 789, 334-343. doi:10.1016/j.ejphar.2016.07.045
NLM
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
Vancouver
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 jun. 26 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045

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