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Artigo de periodico
Diosmin reduces chronic constriction injury-induced neuropathic pain in mice (2017)
Bertozzi, Mariana M.; Rossaneis, Ana C.; Fattori, Victor; Longhi-Balbinot, Daniela T.; Freitas, Andressa; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Chemico-Biological Interactions. Unidade: FMRP
Subjects: DOR CRÔNICA, CLAMPEAMENTO, FLAVONOIDES, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, ESTRESSE OXIDATIVO
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ABNT
BERTOZZI, Mariana M. et al. Diosmin reduces chronic constriction injury-induced neuropathic pain in mice. Chemico-Biological Interactions, v. 273, p. 180-189, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2017.06.014. Acesso em: 11 out. 2024.
APA
Bertozzi, M. M., Rossaneis, A. C., Fattori, V., Longhi-Balbinot, D. T., Freitas, A., Cunha, F. de Q., et al. (2017). Diosmin reduces chronic constriction injury-induced neuropathic pain in mice. Chemico-Biological Interactions, 273, 180-189. doi:10.1016/j.cbi.2017.06.014
NLM
Bertozzi MM, Rossaneis AC, Fattori V, Longhi-Balbinot DT, Freitas A, Cunha F de Q, Alves Filho JCF, Cunha TM, Casagrande R, Verri Júnior WA. Diosmin reduces chronic constriction injury-induced neuropathic pain in mice [Internet]. Chemico-Biological Interactions. 2017 ; 273 180-189.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.cbi.2017.06.014
Vancouver
Bertozzi MM, Rossaneis AC, Fattori V, Longhi-Balbinot DT, Freitas A, Cunha F de Q, Alves Filho JCF, Cunha TM, Casagrande R, Verri Júnior WA. Diosmin reduces chronic constriction injury-induced neuropathic pain in mice [Internet]. Chemico-Biological Interactions. 2017 ; 273 180-189.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.cbi.2017.06.014
Artigo de periodico
MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response (2014)
Sônego, Fabiane; Castanheira, Fernanda V. S.; Czaikoski, Paula G.; Kanashiro, Alexandre; Souto, Fabricio O.; França, Rafael O.; Nascimento, Daniele C.; Freitas, Andressa; Spiller, Fernando; Cunha, Larissa D.; Zamboni, Dario Simões; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Source: PLOS ONE. Unidade: FMRP
Subjects: SEPSE, INFLAMAÇÃO
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ABNT
SÔNEGO, Fabiane et al. MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response. PLOS ONE, v. 9, n. 8, p. e103734-1-e103734-13, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0103734. Acesso em: 11 out. 2024.
APA
Sônego, F., Castanheira, F. V. S., Czaikoski, P. G., Kanashiro, A., Souto, F. O., França, R. O., et al. (2014). MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response. PLOS ONE, 9( 8), e103734-1-e103734-13. doi:10.1371/journal.pone.0103734
NLM
Sônego F, Castanheira FVS, Czaikoski PG, Kanashiro A, Souto FO, França RO, Nascimento DC, Freitas A, Spiller F, Cunha LD, Zamboni DS, Alves Filho JCF, Cunha F de Q. MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response [Internet]. PLOS ONE. 2014 ; 9( 8): e103734-1-e103734-13.[citado 2024 out. 11 ] Available from: https://doi.org/10.1371/journal.pone.0103734
Vancouver
Sônego F, Castanheira FVS, Czaikoski PG, Kanashiro A, Souto FO, França RO, Nascimento DC, Freitas A, Spiller F, Cunha LD, Zamboni DS, Alves Filho JCF, Cunha F de Q. MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response [Internet]. PLOS ONE. 2014 ; 9( 8): e103734-1-e103734-13.[citado 2024 out. 11 ] Available from: https://doi.org/10.1371/journal.pone.0103734
Artigo de periodico
Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis (2011)
Paula Neto, Heitor Affonso de; Alves Filho, José Carlos Farias; Souto, Fabrício Oliveira; Spiller, Fernando; Amêndola, Rafael S.; Freitas, Andressa; Cunha, Fernando de Queiroz; Barja-Fidalgo, Christina
Source: Shock. Unidade: FMRP
Subjects: UNIDADES DE TERAPIA INTENSIVA, SEPSE, INFECÇÃO HOSPITALAR
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ABNT
PAULA NETO, Heitor Affonso de et al. Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock, v. 35, n. 1, p. 17-27, 2011Tradução . . Acesso em: 11 out. 2024.
APA
Paula Neto, H. A. de, Alves Filho, J. C. F., Souto, F. O., Spiller, F., Amêndola, R. S., Freitas, A., et al. (2011). Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock, 35( 1), 17-27.
NLM
Paula Neto HA de, Alves Filho JCF, Souto FO, Spiller F, Amêndola RS, Freitas A, Cunha F de Q, Barja-Fidalgo C. Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock. 2011 ; 35( 1): 17-27.[citado 2024 out. 11 ]
Vancouver
Paula Neto HA de, Alves Filho JCF, Souto FO, Spiller F, Amêndola RS, Freitas A, Cunha F de Q, Barja-Fidalgo C. Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock. 2011 ; 35( 1): 17-27.[citado 2024 out. 11 ]
Artigo de periodico
Divergent role of heme oxygenase inhibition in the pathogenesis of sepsis (2011)
Freitas, Andressa; Alves Filho, José Carlos Farias; Trevelin, Silvia Cellone; Spiller, Fernando; Suavinha, Marina Moreira; Nascimento, Daniele Carvalho; Pestana, Cezar Rangel; Dal-Secco, Daniela; Sônego, Fabiane; Czaikoski, Paula Giselle; Curti, Carlos; Barja-Fidalgo, Christina; Cunha, Fernando de Queiroz
Source: Shock. Unidades: FMRP, FCFRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
FREITAS, Andressa et al. Divergent role of heme oxygenase inhibition in the pathogenesis of sepsis. Shock, v. 35, n. 6, p. 550-559, 2011Tradução . . Acesso em: 11 out. 2024.
APA
Freitas, A., Alves Filho, J. C. F., Trevelin, S. C., Spiller, F., Suavinha, M. M., Nascimento, D. C., et al. (2011). Divergent role of heme oxygenase inhibition in the pathogenesis of sepsis. Shock, 35( 6), 550-559.
NLM
Freitas A, Alves Filho JCF, Trevelin SC, Spiller F, Suavinha MM, Nascimento DC, Pestana CR, Dal-Secco D, Sônego F, Czaikoski PG, Curti C, Barja-Fidalgo C, Cunha F de Q. Divergent role of heme oxygenase inhibition in the pathogenesis of sepsis. Shock. 2011 ; 35( 6): 550-559.[citado 2024 out. 11 ]
Vancouver
Freitas A, Alves Filho JCF, Trevelin SC, Spiller F, Suavinha MM, Nascimento DC, Pestana CR, Dal-Secco D, Sônego F, Czaikoski PG, Curti C, Barja-Fidalgo C, Cunha F de Q. Divergent role of heme oxygenase inhibition in the pathogenesis of sepsis. Shock. 2011 ; 35( 6): 550-559.[citado 2024 out. 11 ]
Artigo de periodico
Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice (2011)
Spiller, Fernando; Costa, Carlotta; Souto, Fabrício Oliveira; Vinchi, Francesca; Mestriner, Fabiola Leslie Antunes Cardoso; Laure, Hélen Julie; Alves Filho, José C.; Freitas, Andressa; Rosa, José César; Ferreira, Sérgio Henrique; Altruda, Fiorella; Hirsch, Emilio; Greene, Lewis Joel; Tolosano, Emanuela; Cunha, Fernando de Queiroz
Source: American Journal of Respiratory and Critical Care Medicine. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
SPILLER, Fernando et al. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, v. 183, n. 7, p. 922-931, 2011Tradução . . Acesso em: 11 out. 2024.
APA
Spiller, F., Costa, C., Souto, F. O., Vinchi, F., Mestriner, F. L. A. C., Laure, H. J., et al. (2011). Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, 183( 7), 922-931.
NLM
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 out. 11 ]
Vancouver
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 out. 11 ]
Artigo de periodico
Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration (2010)
Dal Secco, Daniela; Freitas, Andressa; Abreu, Monica A.; Garlet, Thiago P.; Rossi, Marcos Antonio; Ferreira, Sérgio Henrique; Silva, João Santana da; Alves-Filho, José C.; Cunha, Fernando Queiroz
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO, FARMACOTERAPIA
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ABNT
DAL SECCO, Daniela et al. Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 381, n. 6, p. 483-493, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00210-010-0500-2. Acesso em: 11 out. 2024.
APA
Dal Secco, D., Freitas, A., Abreu, M. A., Garlet, T. P., Rossi, M. A., Ferreira, S. H., et al. (2010). Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration. Naunyn-Schmiedeberg's Archives of Pharmacology, 381( 6), 483-493. doi:10.1007/s00210-010-0500-2
NLM
Dal Secco D, Freitas A, Abreu MA, Garlet TP, Rossi MA, Ferreira SH, Silva JS da, Alves-Filho JC, Cunha FQ. Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2010 ; 381( 6): 483-493.[citado 2024 out. 11 ] Available from: https://doi.org/10.1007/s00210-010-0500-2
Vancouver
Dal Secco D, Freitas A, Abreu MA, Garlet TP, Rossi MA, Ferreira SH, Silva JS da, Alves-Filho JC, Cunha FQ. Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2010 ; 381( 6): 483-493.[citado 2024 out. 11 ] Available from: https://doi.org/10.1007/s00210-010-0500-2
Artigo de periodico
Hydrogen sulfide improves neutrophil migration and survival in sepsis via 'K POT.+' ATP channel activation (2010)
Spiller, Fernando; Orrico, Maria I. L.; Nascimento, Daniele C.; Czaikoski, Paula G.; Souto, Fabrício O.; Alves-Filho, José C.; Freitas, Andressa; Carlos, Daniela; Montenegro, Marcelo F.; Neto, Alberto F.; Ferreira, Sérgio Henrique; Rossi, Marcos Antonio; Hothersall, John S.; Assreuy, Jamil; Cunha, Fernando de Queiroz
Source: American Journal of Respiratory and Critical Care Medicine. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
SPILLER, Fernando et al. Hydrogen sulfide improves neutrophil migration and survival in sepsis via 'K POT.+' ATP channel activation. American Journal of Respiratory and Critical Care Medicine, v. 182, n. 3, p. 360-368, 2010Tradução . . Acesso em: 11 out. 2024.
APA
Spiller, F., Orrico, M. I. L., Nascimento, D. C., Czaikoski, P. G., Souto, F. O., Alves-Filho, J. C., et al. (2010). Hydrogen sulfide improves neutrophil migration and survival in sepsis via 'K POT.+' ATP channel activation. American Journal of Respiratory and Critical Care Medicine, 182( 3), 360-368.
NLM
Spiller F, Orrico MIL, Nascimento DC, Czaikoski PG, Souto FO, Alves-Filho JC, Freitas A, Carlos D, Montenegro MF, Neto AF, Ferreira SH, Rossi MA, Hothersall JS, Assreuy J, Cunha F de Q. Hydrogen sulfide improves neutrophil migration and survival in sepsis via 'K POT.+' ATP channel activation. American Journal of Respiratory and Critical Care Medicine. 2010 ; 182( 3): 360-368.[citado 2024 out. 11 ]
Vancouver
Spiller F, Orrico MIL, Nascimento DC, Czaikoski PG, Souto FO, Alves-Filho JC, Freitas A, Carlos D, Montenegro MF, Neto AF, Ferreira SH, Rossi MA, Hothersall JS, Assreuy J, Cunha F de Q. Hydrogen sulfide improves neutrophil migration and survival in sepsis via 'K POT.+' ATP channel activation. American Journal of Respiratory and Critical Care Medicine. 2010 ; 182( 3): 360-368.[citado 2024 out. 11 ]
Artigo de periodico
IL-17 receptor signaling is required to control polymicrobial sepsis (2009)
Freitas, Andressa; Alves-Filho, José C.; Victoni, Tatiana; Secher, Thomas; Lemos, Henrique P.; Sônego, Fabiane; Cunha, Fernando de Queiroz; Ryffel, Bernhard
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
FREITAS, Andressa et al. IL-17 receptor signaling is required to control polymicrobial sepsis. Journal of Immunology, v. 182, n. 12, p. 7846-7854, 2009Tradução . . Disponível em: https://doi.org/10.1186/cc7810. Acesso em: 11 out. 2024.
APA
Freitas, A., Alves-Filho, J. C., Victoni, T., Secher, T., Lemos, H. P., Sônego, F., et al. (2009). IL-17 receptor signaling is required to control polymicrobial sepsis. Journal of Immunology, 182( 12), 7846-7854. doi:10.1186/cc7810
NLM
Freitas A, Alves-Filho JC, Victoni T, Secher T, Lemos HP, Sônego F, Cunha F de Q, Ryffel B. IL-17 receptor signaling is required to control polymicrobial sepsis [Internet]. Journal of Immunology. 2009 ; 182( 12): 7846-7854.[citado 2024 out. 11 ] Available from: https://doi.org/10.1186/cc7810
Vancouver
Freitas A, Alves-Filho JC, Victoni T, Secher T, Lemos HP, Sônego F, Cunha F de Q, Ryffel B. IL-17 receptor signaling is required to control polymicrobial sepsis [Internet]. Journal of Immunology. 2009 ; 182( 12): 7846-7854.[citado 2024 out. 11 ] Available from: https://doi.org/10.1186/cc7810
Artigo de periodico
Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis (2009)
Alves-Filho, José Carlos; Freitas, Andressa; Souto, Fabrício Oliveira; Spiller, Fernando; Paula Neto, Heitor; Silva, João Santana da; Gazzinelli, Ricardo T.; Teixeira, Mauro Martins; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
ALVES-FILHO, José Carlos et al. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, v. 106, n. 10, p. 4018-4023, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0900196106. Acesso em: 11 out. 2024.
APA
Alves-Filho, J. C., Freitas, A., Souto, F. O., Spiller, F., Paula Neto, H., Silva, J. S. da, et al. (2009). Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, 106( 10), 4018-4023. doi:10.1073/pnas.0900196106
NLM
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 out. 11 ] Available from: https://doi.org/10.1073/pnas.0900196106
Vancouver
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 out. 11 ] Available from: https://doi.org/10.1073/pnas.0900196106
Artigo de periodico
Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway (2008)
Napimoga, Marcelo H.; Vieira, Silvio M.; Dal-Secco, Daniela; Freitas, Andressa; Souto, Fabrício O.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves-Filho, José C.; Grespan, Renata; Kawai, Toshihisa; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO
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ABNT
NAPIMOGA, Marcelo H. et al. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, v. 180, n. 1, p. 609-617, 2008Tradução . . Acesso em: 11 out. 2024.
APA
Napimoga, M. H., Vieira, S. M., Dal-Secco, D., Freitas, A., Souto, F. O., Mestriner, F. L. A. C., et al. (2008). Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, 180( 1), 609-617.
NLM
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 out. 11 ]
Vancouver
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 out. 11 ]
Artigo de periodico
Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels (2008)
Dal-Secco, Daniela; Cunha, Thiago M.; Freitas, Andressa; Alves-Filho, José Carlos; Souto, Fabrício O.; Fukada, Sandra Y.; Grespan, Renata; Alencar, Nylane M. N.; Neto, Alberto F.; Rossi, Marcos Antonio; Ferreira, S. H.; Hothersall, John S.; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAL-SECCO, Daniela et al. Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology, v. 181, n. 6, p. 4287-4298, 2008Tradução . . Acesso em: 11 out. 2024.
APA
Dal-Secco, D., Cunha, T. M., Freitas, A., Alves-Filho, J. C., Souto, F. O., Fukada, S. Y., et al. (2008). Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology, 181( 6), 4287-4298.
NLM
Dal-Secco D, Cunha TM, Freitas A, Alves-Filho JC, Souto FO, Fukada SY, Grespan R, Alencar NMN, Neto AF, Rossi MA, Ferreira SH, Hothersall JS, Cunha F de Q. Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology. 2008 ; 181( 6): 4287-4298.[citado 2024 out. 11 ]
Vancouver
Dal-Secco D, Cunha TM, Freitas A, Alves-Filho JC, Souto FO, Fukada SY, Grespan R, Alencar NMN, Neto AF, Rossi MA, Ferreira SH, Hothersall JS, Cunha F de Q. Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology. 2008 ; 181( 6): 4287-4298.[citado 2024 out. 11 ]
Artigo de periodico
Down-regulation of CXCR2 on neutrophils in severe sepsis is mediated by inducible nitric oxide synthase-derived nitric oxide (2007)
Rios-Santos, Fabrício; Alves-Filho, José C.; Souto, Fabrício Oliveira; Spiller, Fernando; Freitas, Andressa; Lotufo, Celina Monteiro C.; Soares, Milena Botelho Pereira; Santos, Ricardo Ribeiro dos; Teixeira, Mauro M.; Cunha, Fernando de Queiroz
Source: American Journal of Respiratory and Critical Care Medicine. Unidade: FMRP
Subjects: SEPSE, NEUTRÓFILOS, ÓXIDO NÍTRICO
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ABNT
RIOS-SANTOS, Fabrício et al. Down-regulation of CXCR2 on neutrophils in severe sepsis is mediated by inducible nitric oxide synthase-derived nitric oxide. American Journal of Respiratory and Critical Care Medicine, v. 175, p. 490-497, 2007Tradução . . Acesso em: 11 out. 2024.
APA
Rios-Santos, F., Alves-Filho, J. C., Souto, F. O., Spiller, F., Freitas, A., Lotufo, C. M. C., et al. (2007). Down-regulation of CXCR2 on neutrophils in severe sepsis is mediated by inducible nitric oxide synthase-derived nitric oxide. American Journal of Respiratory and Critical Care Medicine, 175, 490-497.
NLM
Rios-Santos F, Alves-Filho JC, Souto FO, Spiller F, Freitas A, Lotufo CMC, Soares MBP, Santos RR dos, Teixeira MM, Cunha F de Q. Down-regulation of CXCR2 on neutrophils in severe sepsis is mediated by inducible nitric oxide synthase-derived nitric oxide. American Journal of Respiratory and Critical Care Medicine. 2007 ; 175 490-497.[citado 2024 out. 11 ]
Vancouver
Rios-Santos F, Alves-Filho JC, Souto FO, Spiller F, Freitas A, Lotufo CMC, Soares MBP, Santos RR dos, Teixeira MM, Cunha F de Q. Down-regulation of CXCR2 on neutrophils in severe sepsis is mediated by inducible nitric oxide synthase-derived nitric oxide. American Journal of Respiratory and Critical Care Medicine. 2007 ; 175 490-497.[citado 2024 out. 11 ]
Artigo de periodico
Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase (2006)
Dal Secco, Daniela; Moreira, Ana P.; Freitas, Andressa; Silva, João Santana da; Rossi, Marcos Antonio; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Nitric Oxide. Unidade: FMRP
Subjects: ADERÊNCIA CELULAR, ÓXIDO NÍTRICO, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAL SECCO, Daniela et al. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase. Nitric Oxide, v. 15, p. 77-86, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2006.02.004. Acesso em: 11 out. 2024.
APA
Dal Secco, D., Moreira, A. P., Freitas, A., Silva, J. S. da, Rossi, M. A., Ferreira, S. H., & Cunha, F. de Q. (2006). Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase. Nitric Oxide, 15, 77-86. doi:10.1016/j.niox.2006.02.004
NLM
Dal Secco D, Moreira AP, Freitas A, Silva JS da, Rossi MA, Ferreira SH, Cunha F de Q. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase [Internet]. Nitric Oxide. 2006 ; 15 77-86.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.niox.2006.02.004
Vancouver
Dal Secco D, Moreira AP, Freitas A, Silva JS da, Rossi MA, Ferreira SH, Cunha F de Q. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase [Internet]. Nitric Oxide. 2006 ; 15 77-86.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.niox.2006.02.004

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