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Artigo de periodico
Repurposing of the nootropic drug vinpocetine as an analgesic and anti-inflammatory agent: evidence in a mouse model of superoxide anion-triggered inflammation (2019)
Lourenco-Gonzalez, Yuri ; Fattori, Victor; Domiciano, Talita Perdigão ; Rossaneis, Ana Carolina; Borghi, Sergio M.; Zaninelli, Tiago H.; Bernardy, Catia Campaner Ferrari; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Mediators of Inflammation. Unidade: FMRP
Subjects: NEUROTRANSMISSORES, MODELOS ANIMAIS, DOR, ANALGÉSICOS
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ABNT
LOURENCO-GONZALEZ, Yuri et al. Repurposing of the nootropic drug vinpocetine as an analgesic and anti-inflammatory agent: evidence in a mouse model of superoxide anion-triggered inflammation. Mediators of Inflammation, v. 2019, 2019Tradução . . Disponível em: https://doi.org/10.1155/2019/6481812. Acesso em: 12 nov. 2024.
APA
Lourenco-Gonzalez, Y., Fattori, V., Domiciano, T. P., Rossaneis, A. C., Borghi, S. M., Zaninelli, T. H., et al. (2019). Repurposing of the nootropic drug vinpocetine as an analgesic and anti-inflammatory agent: evidence in a mouse model of superoxide anion-triggered inflammation. Mediators of Inflammation, 2019. doi:10.1155/2019/6481812
NLM
Lourenco-Gonzalez Y, Fattori V, Domiciano TP, Rossaneis AC, Borghi SM, Zaninelli TH, Bernardy CCF, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Repurposing of the nootropic drug vinpocetine as an analgesic and anti-inflammatory agent: evidence in a mouse model of superoxide anion-triggered inflammation [Internet]. Mediators of Inflammation. 2019 ; 2019[citado 2024 nov. 12 ] Available from: https://doi.org/10.1155/2019/6481812
Vancouver
Lourenco-Gonzalez Y, Fattori V, Domiciano TP, Rossaneis AC, Borghi SM, Zaninelli TH, Bernardy CCF, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Repurposing of the nootropic drug vinpocetine as an analgesic and anti-inflammatory agent: evidence in a mouse model of superoxide anion-triggered inflammation [Internet]. Mediators of Inflammation. 2019 ; 2019[citado 2024 nov. 12 ] Available from: https://doi.org/10.1155/2019/6481812
Artigo de periodico
Differential regulation of oxidative stress and cytokine production by endothelin ETA and ETB receptors in superoxide anion-induced inflammation and pain in mice (2017)
Fattori, Victor; Serafim, Karla G. G.; Zarpelon, Ana C.; Borghi, Sergio M.; Pinho-Ribeiro, Felipe A.; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Journal of Drug Targeting. Unidade: FMRP
Subjects: CITOCINAS, ESTRESSE OXIDATIVO, ENDOTELINAS, INFLAMAÇÃO
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ABNT
FATTORI, Victor et al. Differential regulation of oxidative stress and cytokine production by endothelin ETA and ETB receptors in superoxide anion-induced inflammation and pain in mice. Journal of Drug Targeting, v. 25, n. 3, p. 264-274, 2017Tradução . . Disponível em: https://doi.org/10.1080/1061186x.2016.1245308. Acesso em: 12 nov. 2024.
APA
Fattori, V., Serafim, K. G. G., Zarpelon, A. C., Borghi, S. M., Pinho-Ribeiro, F. A., Alves Filho, J. C. F., et al. (2017). Differential regulation of oxidative stress and cytokine production by endothelin ETA and ETB receptors in superoxide anion-induced inflammation and pain in mice. Journal of Drug Targeting, 25( 3), 264-274. doi:10.1080/1061186x.2016.1245308
NLM
Fattori V, Serafim KGG, Zarpelon AC, Borghi SM, Pinho-Ribeiro FA, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Differential regulation of oxidative stress and cytokine production by endothelin ETA and ETB receptors in superoxide anion-induced inflammation and pain in mice [Internet]. Journal of Drug Targeting. 2017 ; 25( 3): 264-274.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1080/1061186x.2016.1245308
Vancouver
Fattori V, Serafim KGG, Zarpelon AC, Borghi SM, Pinho-Ribeiro FA, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Differential regulation of oxidative stress and cytokine production by endothelin ETA and ETB receptors in superoxide anion-induced inflammation and pain in mice [Internet]. Journal of Drug Targeting. 2017 ; 25( 3): 264-274.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1080/1061186x.2016.1245308
Artigo de periodico
Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects (2017)
Zucoloto, Amanda Z.; Manchope, Marília F.; Staurengo-Ferrari, Larissa; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Antunes, Maísa Mota; Menezes, Gustavo B.; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Inflammation Research. Unidade: FMRP
Subjects: ANALGÉSICOS, ANTI-INFLAMATÓRIOS, LEUCÓCITOS, DOR, CAVIDADE PERITONEAL, ESTIMULAÇÃO FÍSICA
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ABNT
ZUCOLOTO, Amanda Z. et al. Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects. Inflammation Research, v. 66, n. 7, p. 591-602, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00011-017-1040-8. Acesso em: 12 nov. 2024.
APA
Zucoloto, A. Z., Manchope, M. F., Staurengo-Ferrari, L., Alves Filho, J. C. F., Cunha, T. M., Antunes, M. M., et al. (2017). Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects. Inflammation Research, 66( 7), 591-602. doi:10.1007/s00011-017-1040-8
NLM
Zucoloto AZ, Manchope MF, Staurengo-Ferrari L, Alves Filho JCF, Cunha TM, Antunes MM, Menezes GB, Cunha F de Q, Casagrande R, Verri Júnior WA. Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects [Internet]. Inflammation Research. 2017 ; 66( 7): 591-602.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s00011-017-1040-8
Vancouver
Zucoloto AZ, Manchope MF, Staurengo-Ferrari L, Alves Filho JCF, Cunha TM, Antunes MM, Menezes GB, Cunha F de Q, Casagrande R, Verri Júnior WA. Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects [Internet]. Inflammation Research. 2017 ; 66( 7): 591-602.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s00011-017-1040-8
Artigo de periodico
The nitroxyl donor, Angeli's salt, reduces chronic constriction injury-induced neuropathic pain (2016)
Longhi-Balbinot, Daniela T.; Rossaneis, Ana C.; Pinho-Ribeiro, Felipe A.; Bertozzi, Mariana M.; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Peron, Jean Pierre Schatzmann ; Miranda, Katrina M.; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Chemico-Biological Interactions. Unidades: ICB, FMRP
Subjects: ADENOSINA TRIFOSFATO, ASTRÓCITOS, TRANSDUÇÃO DE SINAL CELULAR, INTERLEUCINAS
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ABNT
LONGHI-BALBINOT, Daniela T. et al. The nitroxyl donor, Angeli's salt, reduces chronic constriction injury-induced neuropathic pain. Chemico-Biological Interactions, v. 256, p. 1-8, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2016.06.009. Acesso em: 12 nov. 2024.
APA
Longhi-Balbinot, D. T., Rossaneis, A. C., Pinho-Ribeiro, F. A., Bertozzi, M. M., Cunha, F. de Q., Alves Filho, J. C. F., et al. (2016). The nitroxyl donor, Angeli's salt, reduces chronic constriction injury-induced neuropathic pain. Chemico-Biological Interactions, 256, 1-8. doi:10.1016/j.cbi.2016.06.009
NLM
Longhi-Balbinot DT, Rossaneis AC, Pinho-Ribeiro FA, Bertozzi MM, Cunha F de Q, Alves Filho JCF, Cunha TM, Peron JPS, Miranda KM, Casagrande R, Verri Júnior WA. The nitroxyl donor, Angeli's salt, reduces chronic constriction injury-induced neuropathic pain [Internet]. Chemico-Biological Interactions. 2016 ; 256 1-8.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.cbi.2016.06.009
Vancouver
Longhi-Balbinot DT, Rossaneis AC, Pinho-Ribeiro FA, Bertozzi MM, Cunha F de Q, Alves Filho JCF, Cunha TM, Peron JPS, Miranda KM, Casagrande R, Verri Júnior WA. The nitroxyl donor, Angeli's salt, reduces chronic constriction injury-induced neuropathic pain [Internet]. Chemico-Biological Interactions. 2016 ; 256 1-8.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.cbi.2016.06.009
Artigo de periodico
The citrus flavonone naringenin reduces lipopolysaccharide-induced inflammatory pain and leukocyte recruitment by inhibiting NF-κB activation (2016)
Pinho-Ribeiro, Felipe A.; Zarpelon, Ana C.; Mizokami, Sandra S.; Borghi, Sergio M.; Bordignon, Juliano; Silva, Rangel L.; Cunha, Thiago Mattar; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Journal of Nutritional Biochemistry. Unidade: FMRP
Subjects: CITOCINAS, INFLAMAÇÃO, DOR, LEUCÓCITOS, ESTRESSE OXIDATIVO
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ABNT
PINHO-RIBEIRO, Felipe A. et al. The citrus flavonone naringenin reduces lipopolysaccharide-induced inflammatory pain and leukocyte recruitment by inhibiting NF-κB activation. Journal of Nutritional Biochemistry, v. 33, p. 8-14, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2016.03.013. Acesso em: 12 nov. 2024.
APA
Pinho-Ribeiro, F. A., Zarpelon, A. C., Mizokami, S. S., Borghi, S. M., Bordignon, J., Silva, R. L., et al. (2016). The citrus flavonone naringenin reduces lipopolysaccharide-induced inflammatory pain and leukocyte recruitment by inhibiting NF-κB activation. Journal of Nutritional Biochemistry, 33, 8-14. doi:10.1016/j.jnutbio.2016.03.013
NLM
Pinho-Ribeiro FA, Zarpelon AC, Mizokami SS, Borghi SM, Bordignon J, Silva RL, Cunha TM, Alves-Filho JC, Cunha F de Q, Casagrande R, Verri Júnior WA. The citrus flavonone naringenin reduces lipopolysaccharide-induced inflammatory pain and leukocyte recruitment by inhibiting NF-κB activation [Internet]. Journal of Nutritional Biochemistry. 2016 ; 33 8-14.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.jnutbio.2016.03.013
Vancouver
Pinho-Ribeiro FA, Zarpelon AC, Mizokami SS, Borghi SM, Bordignon J, Silva RL, Cunha TM, Alves-Filho JC, Cunha F de Q, Casagrande R, Verri Júnior WA. The citrus flavonone naringenin reduces lipopolysaccharide-induced inflammatory pain and leukocyte recruitment by inhibiting NF-κB activation [Internet]. Journal of Nutritional Biochemistry. 2016 ; 33 8-14.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.jnutbio.2016.03.013
Artigo de periodico
Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain (2016)
Zarpelon, Ana C.; Rodrigues, Francielle C.; Lopes, Alexandre H.; Souza, Guilherme R.; Carvalho, Thacyana T.; Pinto, Larissa G.; Xu, Damo; Ferreira, Sérgio Henrique; Alves Filho, José Carlos Farias; McInnes, Iain B.; Ryffel, Bernhard; Quesniaux, Valérie F. J.; Reverchon, Flora; Mortaud, Stéphane; Menuet, Arnaud; Liew, Foo Y.; Cunha, Fernando de Queiroz; Cunha, Thiago Mattar; Verri Júnior, Waldiceu A.
Source: FASEB Journal. Unidade: FMRP
Subjects: INTERLEUCINAS, RECEPTORES, DOENÇAS DO SISTEMA NERVOSO, DOR, MODELOS ANIMAIS
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ABNT
ZARPELON, Ana C. et al. Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain. FASEB Journal, v. 30, n. 1, p. 54-65, 2016Tradução . . Disponível em: https://doi.org/10.1096/fj.14-267146. Acesso em: 12 nov. 2024.
APA
Zarpelon, A. C., Rodrigues, F. C., Lopes, A. H., Souza, G. R., Carvalho, T. T., Pinto, L. G., et al. (2016). Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain. FASEB Journal, 30( 1), 54-65. doi:10.1096/fj.14-267146
NLM
Zarpelon AC, Rodrigues FC, Lopes AH, Souza GR, Carvalho TT, Pinto LG, Xu D, Ferreira SH, Alves Filho JCF, McInnes IB, Ryffel B, Quesniaux VFJ, Reverchon F, Mortaud S, Menuet A, Liew FY, Cunha F de Q, Cunha TM, Verri Júnior WA. Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain [Internet]. FASEB Journal. 2016 ; 30( 1): 54-65.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1096/fj.14-267146
Vancouver
Zarpelon AC, Rodrigues FC, Lopes AH, Souza GR, Carvalho TT, Pinto LG, Xu D, Ferreira SH, Alves Filho JCF, McInnes IB, Ryffel B, Quesniaux VFJ, Reverchon F, Mortaud S, Menuet A, Liew FY, Cunha F de Q, Cunha TM, Verri Júnior WA. Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain [Internet]. FASEB Journal. 2016 ; 30( 1): 54-65.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1096/fj.14-267146
Artigo de periodico
DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain (2016)
Lopes, Alexandre H.; Brandolini, Laura; Aramini, Andrea; Bianchini, Gianluca; Silva, Rangel L.; Zaperlon, Ana C.; Verri Júnior, Waldiceu Aparecido; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Teixeira, Mauro M.; Allegretti, Marcello; Cunha, Thiago Mattar
Source: Pharmacological Research. Unidade: FMRP
Subjects: DOR, COMPLICAÇÕES PÓS-OPERATÓRIAS, QUIMIOCINAS, NEUTRÓFILOS, QUIMIOTAXIA
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ABNT
LOPES, Alexandre H. et al. DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain. Pharmacological Research, v. 103, p. 69-79, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2015.11.005. Acesso em: 12 nov. 2024.
APA
Lopes, A. H., Brandolini, L., Aramini, A., Bianchini, G., Silva, R. L., Zaperlon, A. C., et al. (2016). DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain. Pharmacological Research, 103, 69-79. doi:10.1016/j.phrs.2015.11.005
NLM
Lopes AH, Brandolini L, Aramini A, Bianchini G, Silva RL, Zaperlon AC, Verri Júnior WA, Alves Filho JCF, Cunha F de Q, Teixeira MM, Allegretti M, Cunha TM. DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain [Internet]. Pharmacological Research. 2016 ; 103 69-79.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.phrs.2015.11.005
Vancouver
Lopes AH, Brandolini L, Aramini A, Bianchini G, Silva RL, Zaperlon AC, Verri Júnior WA, Alves Filho JCF, Cunha F de Q, Teixeira MM, Allegretti M, Cunha TM. DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain [Internet]. Pharmacological Research. 2016 ; 103 69-79.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.phrs.2015.11.005
Artigo de periodico
Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress (2016)
Pinho-Ribeiro, Felipe A.; Fattori, Victor; Zarpelon, Ana C.; Borghi, Sergio M.; Staurengo-Ferrari, Larissa; Carvalho, Thacyana T.; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Cunha, Thiago Mattar; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Inflammopharmacology. Unidade: FMRP
Subjects: CITOCINAS, DOR, INFLAMAÇÃO, ESTRESSE OXIDATIVO
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ABNT
PINHO-RIBEIRO, Felipe A. et al. Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress. Inflammopharmacology, v. 24, p. 97-107, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10787-016-0266-3. Acesso em: 12 nov. 2024.
APA
Pinho-Ribeiro, F. A., Fattori, V., Zarpelon, A. C., Borghi, S. M., Staurengo-Ferrari, L., Carvalho, T. T., et al. (2016). Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress. Inflammopharmacology, 24, 97-107. doi:10.1007/s10787-016-0266-3
NLM
Pinho-Ribeiro FA, Fattori V, Zarpelon AC, Borghi SM, Staurengo-Ferrari L, Carvalho TT, Alves-Filho JC, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress [Internet]. Inflammopharmacology. 2016 ; 24 97-107.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s10787-016-0266-3
Vancouver
Pinho-Ribeiro FA, Fattori V, Zarpelon AC, Borghi SM, Staurengo-Ferrari L, Carvalho TT, Alves-Filho JC, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress [Internet]. Inflammopharmacology. 2016 ; 24 97-107.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s10787-016-0266-3
Artigo de periodico
Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice (2015)
Yamacita-Borin, Fabiane Y.; Zarpelon, Ana C.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Cunha, Thiago Mattar; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Neuroscience Letters. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS (FARMACOLOGIA), NEUTRÓFILOS (FISIOLOGIA), INFLAMAÇÃO, ESTRESSE OXIDATIVO, DOR (METABOLISMO), ESTIMULAÇÃO FÍSICA
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ABNT
YAMACITA-BORIN, Fabiane Y. et al. Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice. Neuroscience Letters, v. 605, p. 53-58, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2015.08.015. Acesso em: 12 nov. 2024.
APA
Yamacita-Borin, F. Y., Zarpelon, A. C., Pinho-Ribeiro, F. A., Fattori, V., Alves Filho, J. C. F., Cunha, F. de Q., et al. (2015). Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice. Neuroscience Letters, 605, 53-58. doi:10.1016/j.neulet.2015.08.015
NLM
Yamacita-Borin FY, Zarpelon AC, Pinho-Ribeiro FA, Fattori V, Alves Filho JCF, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice [Internet]. Neuroscience Letters. 2015 ; 605 53-58.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.neulet.2015.08.015
Vancouver
Yamacita-Borin FY, Zarpelon AC, Pinho-Ribeiro FA, Fattori V, Alves Filho JCF, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice [Internet]. Neuroscience Letters. 2015 ; 605 53-58.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.neulet.2015.08.015
Artigo de periodico
Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice (2015)
Serafim, Karla G. G.; Navarro, Suelen A.; Zarpelon, Ana C.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: HIPERTENSÃO PULMONAR, ESTRESSE OXIDATIVO, DOR, ENDOTELINAS, ANTI-INFLAMATÓRIOS
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ABNT
SERAFIM, Karla G. G. et al. Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 388, n. 11, p. 1211-1221, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00210-015-1160-z. Acesso em: 12 nov. 2024.
APA
Serafim, K. G. G., Navarro, S. A., Zarpelon, A. C., Pinho-Ribeiro, F. A., Fattori, V., Cunha, T. M., et al. (2015). Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice. Naunyn-Schmiedeberg's Archives of Pharmacology, 388( 11), 1211-1221. doi:10.1007/s00210-015-1160-z
NLM
Serafim KGG, Navarro SA, Zarpelon AC, Pinho-Ribeiro FA, Fattori V, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2015 ; 388( 11): 1211-1221.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s00210-015-1160-z
Vancouver
Serafim KGG, Navarro SA, Zarpelon AC, Pinho-Ribeiro FA, Fattori V, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2015 ; 388( 11): 1211-1221.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s00210-015-1160-z
Artigo de periodico
Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-kB activation (2015)
Fattori, Victor; Pinho-Ribeiro, Felipe A.; Borghi, Sergio M.; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Inflammation Research. Unidade: FMRP
Subjects: DOR, ANTI-INFLAMATÓRIOS, NOCICEPTORES, AÇAFRÃO
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ABNT
FATTORI, Victor et al. Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-kB activation. Inflammation Research, v. 64, n. 12, p. 993-1003, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00011-015-0885-y. Acesso em: 12 nov. 2024.
APA
Fattori, V., Pinho-Ribeiro, F. A., Borghi, S. M., Alves Filho, J. C. F., Cunha, T. M., Cunha, F. de Q., et al. (2015). Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-kB activation. Inflammation Research, 64( 12), 993-1003. doi:10.1007/s00011-015-0885-y
NLM
Fattori V, Pinho-Ribeiro FA, Borghi SM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-kB activation [Internet]. Inflammation Research. 2015 ; 64( 12): 993-1003.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s00011-015-0885-y
Vancouver
Fattori V, Pinho-Ribeiro FA, Borghi SM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-kB activation [Internet]. Inflammation Research. 2015 ; 64( 12): 993-1003.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1007/s00011-015-0885-y
Artigo de periodico
Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain (2015)
Lopes, Alexandre H.; Talbot, Jhimmy; Silva, Rangel L.; Lima, Jonilson B.; França, Rafael O.; Verri Júnior, Waldiceu Aparecido; Mascarenhas, Danielle Pini Alves; Ryffel, Bernhard; Cunha, Fernando de Queiroz; Zamboni, Dario Simões; Cunha, Thiago Mattar
Source: Pain. Unidade: FMRP
Subjects: APOPTOSE, PROTEÍNAS DE LIGAÇÃO DO CÁLCIO, INFLAMAÇÃO, MODELOS ANIMAIS DE DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Alexandre H. et al. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain. Pain, v. 156, n. 3, p. 451-459, 2015Tradução . . Disponível em: https://doi.org/10.1097/01.j.pain.0000460322.72396.53. Acesso em: 12 nov. 2024.
APA
Lopes, A. H., Talbot, J., Silva, R. L., Lima, J. B., França, R. O., Verri Júnior, W. A., et al. (2015). Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain. Pain, 156( 3), 451-459. doi:10.1097/01.j.pain.0000460322.72396.53
NLM
Lopes AH, Talbot J, Silva RL, Lima JB, França RO, Verri Júnior WA, Mascarenhas DPA, Ryffel B, Cunha F de Q, Zamboni DS, Cunha TM. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain [Internet]. Pain. 2015 ; 156( 3): 451-459.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1097/01.j.pain.0000460322.72396.53
Vancouver
Lopes AH, Talbot J, Silva RL, Lima JB, França RO, Verri Júnior WA, Mascarenhas DPA, Ryffel B, Cunha F de Q, Zamboni DS, Cunha TM. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain [Internet]. Pain. 2015 ; 156( 3): 451-459.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1097/01.j.pain.0000460322.72396.53
Artigo de periodico
Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB (2015)
Ruiz-Miyazawa, Kenji W.; Pinho-Ribeiro, Felipe A.; Zarpelon, Ana C.; Staurengo-Ferrari, Larissa; Silva, Rangel L.; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Chemico-Biological Interactions. Unidade: FMRP
Subjects: CITOCINAS, LIPOPOLISSACARÍDEOS, MACRÓFAGOS, NEUTRÓFILOS, CAVIDADE PERITONEAL, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RUIZ-MIYAZAWA, Kenji W. et al. Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB. Chemico-Biological Interactions, v. 237, p. 9-17, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.05.007. Acesso em: 12 nov. 2024.
APA
Ruiz-Miyazawa, K. W., Pinho-Ribeiro, F. A., Zarpelon, A. C., Staurengo-Ferrari, L., Silva, R. L., Alves Filho, J. C. F., et al. (2015). Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB. Chemico-Biological Interactions, 237, 9-17. doi:10.1016/j.cbi.2015.05.007
NLM
Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Zarpelon AC, Staurengo-Ferrari L, Silva RL, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB [Internet]. Chemico-Biological Interactions. 2015 ; 237 9-17.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.cbi.2015.05.007
Vancouver
Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Zarpelon AC, Staurengo-Ferrari L, Silva RL, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB [Internet]. Chemico-Biological Interactions. 2015 ; 237 9-17.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.cbi.2015.05.007
Artigo de periodico
Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception (2015)
Souza, Guilherme R.; Cunha, Thiago Mattar; Silva, Rangel L.; Lotufo, Celina M.; Verri Júnior, Waldiceu Aparecido; Funez, Mani Indiana; Villarreal, Christiane F.; Talbot, Jhimmy; Sousa, Lirlândia P.; Parada, Carlos Amílcar; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique
Source: Pharmacology, Biochemistry and Behavior. Unidade: FMRP
Subjects: DOR CRÔNICA, INFLAMAÇÃO, PROSTAGLANDINAS, NOCICEPTORES, NEURÔNIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Guilherme R. et al. Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception. Pharmacology, Biochemistry and Behavior, v. 134, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2015.04.005. Acesso em: 12 nov. 2024.
APA
Souza, G. R., Cunha, T. M., Silva, R. L., Lotufo, C. M., Verri Júnior, W. A., Funez, M. I., et al. (2015). Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception. Pharmacology, Biochemistry and Behavior, 134, 49-56. doi:10.1016/j.pbb.2015.04.005
NLM
Souza GR, Cunha TM, Silva RL, Lotufo CM, Verri Júnior WA, Funez MI, Villarreal CF, Talbot J, Sousa LP, Parada CA, Cunha F de Q, Ferreira SH. Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception [Internet]. Pharmacology, Biochemistry and Behavior. 2015 ; 134 49-56.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.pbb.2015.04.005
Vancouver
Souza GR, Cunha TM, Silva RL, Lotufo CM, Verri Júnior WA, Funez MI, Villarreal CF, Talbot J, Sousa LP, Parada CA, Cunha F de Q, Ferreira SH. Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception [Internet]. Pharmacology, Biochemistry and Behavior. 2015 ; 134 49-56.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.pbb.2015.04.005
Artigo de periodico
Targeting interleukin-1'beta' reduces intense acute swimming-induced muscle mechanical hyperalgesia in mice (2014)
Borghi, Sergio M.; Zarpelon, Ana C.; Pinho-Ribeiro, Felipe A.; Cardoso, Renato D. R.; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Journal of Pharmacy and Pharmacology. Unidade: FMRP
Subjects: INTERLEUCINA 1 (FARMACOLOGIA), MÚSCULO ESQUELÉTICO, PEROXIDASE (METABOLISMO), MEDULA ESPINHAL (EFEITOS DE DROGAS), NATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGHI, Sergio M. et al. Targeting interleukin-1'beta' reduces intense acute swimming-induced muscle mechanical hyperalgesia in mice. Journal of Pharmacy and Pharmacology, v. 66, n. 7, p. 1009-1020, 2014Tradução . . Disponível em: https://doi.org/10.1111/jphp.12226. Acesso em: 12 nov. 2024.
APA
Borghi, S. M., Zarpelon, A. C., Pinho-Ribeiro, F. A., Cardoso, R. D. R., Cunha, T. M., Alves Filho, J. C. F., et al. (2014). Targeting interleukin-1'beta' reduces intense acute swimming-induced muscle mechanical hyperalgesia in mice. Journal of Pharmacy and Pharmacology, 66( 7), 1009-1020. doi:10.1111/jphp.12226
NLM
Borghi SM, Zarpelon AC, Pinho-Ribeiro FA, Cardoso RDR, Cunha TM, Alves Filho JCF, Ferreira SH, Cunha F de Q, Casagrande R, Verri Júnior WA. Targeting interleukin-1'beta' reduces intense acute swimming-induced muscle mechanical hyperalgesia in mice [Internet]. Journal of Pharmacy and Pharmacology. 2014 ; 66( 7): 1009-1020.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/jphp.12226
Vancouver
Borghi SM, Zarpelon AC, Pinho-Ribeiro FA, Cardoso RDR, Cunha TM, Alves Filho JCF, Ferreira SH, Cunha F de Q, Casagrande R, Verri Júnior WA. Targeting interleukin-1'beta' reduces intense acute swimming-induced muscle mechanical hyperalgesia in mice [Internet]. Journal of Pharmacy and Pharmacology. 2014 ; 66( 7): 1009-1020.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1111/jphp.12226
Artigo de periodico
Role of TNF-α/TNFR1 in intense acute swimming-induced delayed onset muscle soreness in mice (2014)
Borghi, Sergio M; Zarpelon, Ana C; Pinho-Ribeiro, Felipe A; Cardoso, Renato D R; Martins-Pinge, Marli C; Tatakihara, Roberto I; Cunha, Thiago Mattar; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu A
Source: Physiology & Behavior. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS NÃO ESTEROIDES (FARMACOLOGIA), GLICOSE (SANGUE;ANÁLISE), IMUNOGLOBULINAS, LEUCÓCITOS, MÚSCULO ESQUELÉTICO, NATAÇÃO (FISIOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGHI, Sergio M et al. Role of TNF-α/TNFR1 in intense acute swimming-induced delayed onset muscle soreness in mice. Physiology & Behavior, v. 128, p. 277-287, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.physbeh.2014.01.023. Acesso em: 12 nov. 2024.
APA
Borghi, S. M., Zarpelon, A. C., Pinho-Ribeiro, F. A., Cardoso, R. D. R., Martins-Pinge, M. C., Tatakihara, R. I., et al. (2014). Role of TNF-α/TNFR1 in intense acute swimming-induced delayed onset muscle soreness in mice. Physiology & Behavior, 128, 277-287. doi:10.1016/j.physbeh.2014.01.023
NLM
Borghi SM, Zarpelon AC, Pinho-Ribeiro FA, Cardoso RDR, Martins-Pinge MC, Tatakihara RI, Cunha TM, Ferreira SH, Cunha F de Q, Casagrande R, Verri Júnior WA. Role of TNF-α/TNFR1 in intense acute swimming-induced delayed onset muscle soreness in mice [Internet]. Physiology & Behavior. 2014 ; 128 277-287.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.physbeh.2014.01.023
Vancouver
Borghi SM, Zarpelon AC, Pinho-Ribeiro FA, Cardoso RDR, Martins-Pinge MC, Tatakihara RI, Cunha TM, Ferreira SH, Cunha F de Q, Casagrande R, Verri Júnior WA. Role of TNF-α/TNFR1 in intense acute swimming-induced delayed onset muscle soreness in mice [Internet]. Physiology & Behavior. 2014 ; 128 277-287.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.physbeh.2014.01.023
Artigo de periodico
Nitroxyl inhibits overt pain-like behavior in mice: role of cGMP/PKG/ATP-sensitive potassium channel signaling pathway (2014)
Staurengo-Ferrari, Larissa; Zarpelon, Ana C; Longhi-Balbinot, Daniela T; Marchesi, Mario; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique; Casagrande, Rubia; Miranda, Katrina M; Verri Junior, Waldiceu A
Source: Pharmacological Reports. Unidade: FMRP
Subjects: ANALGÉSICOS (FARMACOLOGIA), DOR (MEDIÇÃO), TRANSDUÇÃO DE SINAL CELULAR (EFEITOS DE DROGAS), COMPORTAMENTO ANIMAL (EFEITOS DE DROGAS), PROTEÍNAS QUINASES (METABOLISMO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
STAURENGO-FERRARI, Larissa et al. Nitroxyl inhibits overt pain-like behavior in mice: role of cGMP/PKG/ATP-sensitive potassium channel signaling pathway. Pharmacological Reports, v. 66, n. 4, p. 691-698, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.pharep.2014.04.003. Acesso em: 12 nov. 2024.
APA
Staurengo-Ferrari, L., Zarpelon, A. C., Longhi-Balbinot, D. T., Marchesi, M., Cunha, T. M., Alves Filho, J. C. F., et al. (2014). Nitroxyl inhibits overt pain-like behavior in mice: role of cGMP/PKG/ATP-sensitive potassium channel signaling pathway. Pharmacological Reports, 66( 4), 691-698. doi:10.1016/j.pharep.2014.04.003
NLM
Staurengo-Ferrari L, Zarpelon AC, Longhi-Balbinot DT, Marchesi M, Cunha TM, Alves Filho JCF, Cunha F de Q, Ferreira SH, Casagrande R, Miranda KM, Verri Junior WA. Nitroxyl inhibits overt pain-like behavior in mice: role of cGMP/PKG/ATP-sensitive potassium channel signaling pathway [Internet]. Pharmacological Reports. 2014 ; 66( 4): 691-698.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.pharep.2014.04.003
Vancouver
Staurengo-Ferrari L, Zarpelon AC, Longhi-Balbinot DT, Marchesi M, Cunha TM, Alves Filho JCF, Cunha F de Q, Ferreira SH, Casagrande R, Miranda KM, Verri Junior WA. Nitroxyl inhibits overt pain-like behavior in mice: role of cGMP/PKG/ATP-sensitive potassium channel signaling pathway [Internet]. Pharmacological Reports. 2014 ; 66( 4): 691-698.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.pharep.2014.04.003
Artigo de periodico
An interleukin-33/ST2 signaling deficiency reduces overt pain-like behaviors in mice (2013)
Magro, D. A. C.; Hohmann, M. S. N.; Mizokami, S. S.; Cunha, T. M.; Alves-Filho, José Carlos Farias; Casagrande, R.; Ferreira, Sérgio Henrique; Liew, F. Y.; Cunha, Fernando de Queiroz; Verri Júnior, Waldiceu Aparecido
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Subjects: INTERLEUCINAS, DOR, NOCICEPTORES, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGRO, D. A. C. et al. An interleukin-33/ST2 signaling deficiency reduces overt pain-like behaviors in mice. Brazilian Journal of Medical and Biological Research, v. 46, n. 7, p. 601-606, 2013Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20132894. Acesso em: 12 nov. 2024.
APA
Magro, D. A. C., Hohmann, M. S. N., Mizokami, S. S., Cunha, T. M., Alves-Filho, J. C. F., Casagrande, R., et al. (2013). An interleukin-33/ST2 signaling deficiency reduces overt pain-like behaviors in mice. Brazilian Journal of Medical and Biological Research, 46( 7), 601-606. doi:10.1590/1414-431X20132894
NLM
Magro DAC, Hohmann MSN, Mizokami SS, Cunha TM, Alves-Filho JCF, Casagrande R, Ferreira SH, Liew FY, Cunha F de Q, Verri Júnior WA. An interleukin-33/ST2 signaling deficiency reduces overt pain-like behaviors in mice [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 7): 601-606.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1590/1414-431X20132894
Vancouver
Magro DAC, Hohmann MSN, Mizokami SS, Cunha TM, Alves-Filho JCF, Casagrande R, Ferreira SH, Liew FY, Cunha F de Q, Verri Júnior WA. An interleukin-33/ST2 signaling deficiency reduces overt pain-like behaviors in mice [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 7): 601-606.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1590/1414-431X20132894
Artigo de periodico
Toll-like receptor 2/MyD88 signaling mediates zymosan-induced joint hypernociception in mice: participation of TNF-'alfa', IL-1'beta' and CXCL1/KC (2012)
Guerrero, Ana T. G.; Cunha, Thiago Mattar; Verri Júnior, Waldiceu Aparecido; Guazzinelli, Ricardo T.; Teixeira, Mauro M.; Cunha, Fernando de Queiroz; Ferreira, Sérgio Henrique
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: ARTICULAÇÕES, DOR, ARTRITE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUERRERO, Ana T. G. et al. Toll-like receptor 2/MyD88 signaling mediates zymosan-induced joint hypernociception in mice: participation of TNF-'alfa', IL-1'beta' and CXCL1/KC. European Journal of Pharmacology, v. 674, n. 1, p. 51-57, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2011.10.023. Acesso em: 12 nov. 2024.
APA
Guerrero, A. T. G., Cunha, T. M., Verri Júnior, W. A., Guazzinelli, R. T., Teixeira, M. M., Cunha, F. de Q., & Ferreira, S. H. (2012). Toll-like receptor 2/MyD88 signaling mediates zymosan-induced joint hypernociception in mice: participation of TNF-'alfa', IL-1'beta' and CXCL1/KC. European Journal of Pharmacology, 674( 1), 51-57. doi:10.1016/j.ejphar.2011.10.023
NLM
Guerrero ATG, Cunha TM, Verri Júnior WA, Guazzinelli RT, Teixeira MM, Cunha F de Q, Ferreira SH. Toll-like receptor 2/MyD88 signaling mediates zymosan-induced joint hypernociception in mice: participation of TNF-'alfa', IL-1'beta' and CXCL1/KC [Internet]. European Journal of Pharmacology. 2012 ; 674( 1): 51-57.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.ejphar.2011.10.023
Vancouver
Guerrero ATG, Cunha TM, Verri Júnior WA, Guazzinelli RT, Teixeira MM, Cunha F de Q, Ferreira SH. Toll-like receptor 2/MyD88 signaling mediates zymosan-induced joint hypernociception in mice: participation of TNF-'alfa', IL-1'beta' and CXCL1/KC [Internet]. European Journal of Pharmacology. 2012 ; 674( 1): 51-57.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.ejphar.2011.10.023
Artigo de periodico
Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice (2012)
Pavão-de-Souza, Gabriela F.; Zarpelon, Ana C.; Tedeschi, Giovana C.; Mizokami, Sandra S.; Sanson, Joice S.; Cunha, Thiago Mattar; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Pharmacology, Biochemistry and Behavior. Unidade: FMRP
Subjects: DOR (FISIOPATOLOGIA), NOCICEPTORES, MEDULA ESPINHAL, FLAVONÓIDES (FARMACOLOGIA), MODELOS ANIMAIS DE DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAVÃO-DE-SOUZA, Gabriela F. et al. Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice. Pharmacology, Biochemistry and Behavior, v. 101, n. 3, p. 320-328, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2012.01.018. Acesso em: 12 nov. 2024.
APA
Pavão-de-Souza, G. F., Zarpelon, A. C., Tedeschi, G. C., Mizokami, S. S., Sanson, J. S., Cunha, T. M., et al. (2012). Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice. Pharmacology, Biochemistry and Behavior, 101( 3), 320-328. doi:10.1016/j.pbb.2012.01.018
NLM
Pavão-de-Souza GF, Zarpelon AC, Tedeschi GC, Mizokami SS, Sanson JS, Cunha TM, Ferreira SH, Cunha F de Q, Casagrande R, Verri Júnior WA. Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice [Internet]. Pharmacology, Biochemistry and Behavior. 2012 ; 101( 3): 320-328.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.pbb.2012.01.018
Vancouver
Pavão-de-Souza GF, Zarpelon AC, Tedeschi GC, Mizokami SS, Sanson JS, Cunha TM, Ferreira SH, Cunha F de Q, Casagrande R, Verri Júnior WA. Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice [Internet]. Pharmacology, Biochemistry and Behavior. 2012 ; 101( 3): 320-328.[citado 2024 nov. 12 ] Available from: https://doi.org/10.1016/j.pbb.2012.01.018

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