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Trabalho de evento
Mitochondria, diets and calcium: a metabolic triad (2021)
Kowaltowski, Alicia Juliana
Source: Canal YouTube da Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq. Conference titles: IUPAB Congress. Unidade: IQ
Subjects: MITOCÔNDRIAS, DIETA, CÁLCIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KOWALTOWSKI, Alicia Juliana. Mitochondria, diets and calcium: a metabolic triad. 2021, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq, 2021. Disponível em: https://www.youtube.com/watch?v=3fdNdxX7BRY. Acesso em: 10 nov. 2024.
APA
Kowaltowski, A. J. (2021). Mitochondria, diets and calcium: a metabolic triad. In Canal YouTube da Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq. Recuperado de https://www.youtube.com/watch?v=3fdNdxX7BRY
NLM
Kowaltowski AJ. Mitochondria, diets and calcium: a metabolic triad [Internet]. Canal YouTube da Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq. 2021 ;[citado 2024 nov. 10 ] Available from: https://www.youtube.com/watch?v=3fdNdxX7BRY
Vancouver
Kowaltowski AJ. Mitochondria, diets and calcium: a metabolic triad [Internet]. Canal YouTube da Sociedade Brasileira de Bioquímica e Biologia Molecular - SBBq. 2021 ;[citado 2024 nov. 10 ] Available from: https://www.youtube.com/watch?v=3fdNdxX7BRY
Artigo de periodico
Liver lipidome signature and metabolic pathways in non-alcoholic fatty liver disease induced by a high-sugar diet (2021)
Oliveira, Daiane T; Chaves Filho, Adriano de Britto ; Yoshinaga, Marcos Yukio ; Paiva, Nívia Carolina Nogueira ; Carneiro, Cláudia M.; Miyamoto, Sayuri ; Festuccia, William Tadeu Lara ; Sá, Renata Guerra de
Source: Journal of Nutritional Biochemistry. Unidades: IQ, ICB
Subjects: HEPATOPATIAS, AÇÚCAR, FISIOLOGIA, LIPÍDEOS, FÍGADO, MITOCÔNDRIAS, DIETA ANIMAL, RATOS WISTAR, ESTRESSE OXIDATIVO, EXPRESSÃO GÊNICA, ÁCIDOS GRAXOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Daiane T et al. Liver lipidome signature and metabolic pathways in non-alcoholic fatty liver disease induced by a high-sugar diet. Journal of Nutritional Biochemistry, v. 87, p. 1-12 art. 108519, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2020.108519. Acesso em: 10 nov. 2024.
APA
Oliveira, D. T., Chaves Filho, A. de B., Yoshinaga, M. Y., Paiva, N. C. N., Carneiro, C. M., Miyamoto, S., et al. (2021). Liver lipidome signature and metabolic pathways in non-alcoholic fatty liver disease induced by a high-sugar diet. Journal of Nutritional Biochemistry, 87, 1-12 art. 108519. doi:10.1016/j.jnutbio.2020.108519
NLM
Oliveira DT, Chaves Filho A de B, Yoshinaga MY, Paiva NCN, Carneiro CM, Miyamoto S, Festuccia WTL, Sá RG de. Liver lipidome signature and metabolic pathways in non-alcoholic fatty liver disease induced by a high-sugar diet [Internet]. Journal of Nutritional Biochemistry. 2021 ; 87 1-12 art. 108519.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.jnutbio.2020.108519
Vancouver
Oliveira DT, Chaves Filho A de B, Yoshinaga MY, Paiva NCN, Carneiro CM, Miyamoto S, Festuccia WTL, Sá RG de. Liver lipidome signature and metabolic pathways in non-alcoholic fatty liver disease induced by a high-sugar diet [Internet]. Journal of Nutritional Biochemistry. 2021 ; 87 1-12 art. 108519.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.jnutbio.2020.108519
Artigo de periodico
Mitochondrial K+ transport: modulation and functional consequences (2021)
Pereira Junior, Osvaldo Rodrigues ; Kowaltowski, Alicia Juliana
Source: Molecules. Unidade: IQ
Subjects: MITOCÔNDRIAS, POTÁSSIO, FÁRMACOS
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ABNT
PEREIRA JUNIOR, Osvaldo Rodrigues e KOWALTOWSKI, Alicia Juliana. Mitochondrial K+ transport: modulation and functional consequences. Molecules, v. 26, p. 1-11 art. 2935, 2021Tradução . . Disponível em: https://doi.org/10.3390/molecules26102935. Acesso em: 10 nov. 2024.
APA
Pereira Junior, O. R., & Kowaltowski, A. J. (2021). Mitochondrial K+ transport: modulation and functional consequences. Molecules, 26, 1-11 art. 2935. doi:10.3390/molecules26102935
NLM
Pereira Junior OR, Kowaltowski AJ. Mitochondrial K+ transport: modulation and functional consequences [Internet]. Molecules. 2021 ; 26 1-11 art. 2935.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/molecules26102935
Vancouver
Pereira Junior OR, Kowaltowski AJ. Mitochondrial K+ transport: modulation and functional consequences [Internet]. Molecules. 2021 ; 26 1-11 art. 2935.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/molecules26102935
Artigo de periodico
Increased glycolysis is an early consequence of palmitate lipotoxicity mediated by redox signaling (2021)
Kakimoto, Pâmela Aiako Hypólito Brito ; Serna, Julian David Cualcialpud ; Ramos, Vitor de Miranda ; Zorzano, Antonio; Kowaltowski, Alicia Juliana
Source: Redox Biology. Unidade: IQ
Subjects: MITOCÔNDRIAS, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KAKIMOTO, Pâmela Aiako Hypólito Brito et al. Increased glycolysis is an early consequence of palmitate lipotoxicity mediated by redox signaling. Redox Biology, v. 45, p. 1-12 art. 102026, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2021.102026. Acesso em: 10 nov. 2024.
APA
Kakimoto, P. A. H. B., Serna, J. D. C., Ramos, V. de M., Zorzano, A., & Kowaltowski, A. J. (2021). Increased glycolysis is an early consequence of palmitate lipotoxicity mediated by redox signaling. Redox Biology, 45, 1-12 art. 102026. doi:10.1016/j.redox.2021.102026
NLM
Kakimoto PAHB, Serna JDC, Ramos V de M, Zorzano A, Kowaltowski AJ. Increased glycolysis is an early consequence of palmitate lipotoxicity mediated by redox signaling [Internet]. Redox Biology. 2021 ; 45 1-12 art. 102026.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.redox.2021.102026
Vancouver
Kakimoto PAHB, Serna JDC, Ramos V de M, Zorzano A, Kowaltowski AJ. Increased glycolysis is an early consequence of palmitate lipotoxicity mediated by redox signaling [Internet]. Redox Biology. 2021 ; 45 1-12 art. 102026.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.redox.2021.102026
Artigo de periodico
Changes in mitochondrial morphology modulate LPS-induced loss of calcium homeostasis in BV-2 microglial cells (2021)
Pereira Junior, Osvaldo Rodrigues ; Ramos, Vitor de Miranda ; Costa, João Victor Cabral ; Kowaltowski, Alicia Juliana
Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ
Subjects: HOMEOSTASE, CÁLCIO, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA JUNIOR, Osvaldo Rodrigues et al. Changes in mitochondrial morphology modulate LPS-induced loss of calcium homeostasis in BV-2 microglial cells. Journal of Bioenergetics and Biomembranes, v. 53, p. 109–118, 2021Tradução . . Disponível em: https://doi.org/10.1007/s10863-021-09878-4. Acesso em: 10 nov. 2024.
APA
Pereira Junior, O. R., Ramos, V. de M., Costa, J. V. C., & Kowaltowski, A. J. (2021). Changes in mitochondrial morphology modulate LPS-induced loss of calcium homeostasis in BV-2 microglial cells. Journal of Bioenergetics and Biomembranes, 53, 109–118. doi:10.1007/s10863-021-09878-4
NLM
Pereira Junior OR, Ramos V de M, Costa JVC, Kowaltowski AJ. Changes in mitochondrial morphology modulate LPS-induced loss of calcium homeostasis in BV-2 microglial cells [Internet]. Journal of Bioenergetics and Biomembranes. 2021 ; 53 109–118.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s10863-021-09878-4
Vancouver
Pereira Junior OR, Ramos V de M, Costa JVC, Kowaltowski AJ. Changes in mitochondrial morphology modulate LPS-induced loss of calcium homeostasis in BV-2 microglial cells [Internet]. Journal of Bioenergetics and Biomembranes. 2021 ; 53 109–118.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s10863-021-09878-4
Artigo de periodico
NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance (2021)
Ferezin, Camila de Castro ; Basei, Fernanda Luisa; Hanchuk, Talita D. Melo ; Oliveira, Ana Luisa de; Oliveira, Andressa Peres de ; Mori, Mateus P; Souza-Pinto, Nadja Cristhina de
Source: FEBS Open Bio. Unidade: IQ
Subjects: MITOCÔNDRIAS, CICLO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FEREZIN, Camila de Castro et al. NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance. FEBS Open Bio, v. 11, p. 546–563, 2021Tradução . . Disponível em: https://doi.org/10.1002/2211-5463.13108. Acesso em: 10 nov. 2024.
APA
Ferezin, C. de C., Basei, F. L., Hanchuk, T. D. M., Oliveira, A. L. de, Oliveira, A. P. de, Mori, M. P., & Souza-Pinto, N. C. de. (2021). NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance. FEBS Open Bio, 11, 546–563. doi:10.1002/2211-5463.13108
NLM
Ferezin C de C, Basei FL, Hanchuk TDM, Oliveira AL de, Oliveira AP de, Mori MP, Souza-Pinto NC de. NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance [Internet]. FEBS Open Bio. 2021 ; 11 546–563.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/2211-5463.13108
Vancouver
Ferezin C de C, Basei FL, Hanchuk TDM, Oliveira AL de, Oliveira AP de, Mori MP, Souza-Pinto NC de. NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance [Internet]. FEBS Open Bio. 2021 ; 11 546–563.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/2211-5463.13108
Artigo de periodico
Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells (2021)
Freire, Thiago de Souza; Mori, M.P; Miranda, J. N. F. A; Muta, Lais Yoshie Morikawa; Machado, F. T; Moreno, N. C; Souza-Pinto, Nadja Cristhina de
Source: Carcinogenesis. Unidade: IQ
Subjects: MITOCÔNDRIAS, CÉLULAS, PERÓXIDO DE HIDROGÊNIO
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ABNT
FREIRE, Thiago de Souza et al. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells. Carcinogenesis, v. 42, n. 11, p. 1380-1389, 2021Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgab079. Acesso em: 10 nov. 2024.
APA
Freire, T. de S., Mori, M. P., Miranda, J. N. F. A., Muta, L. Y. M., Machado, F. T., Moreno, N. C., & Souza-Pinto, N. C. de. (2021). Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells. Carcinogenesis, 42( 11), 1380-1389. doi:10.1093/carcin/bgab079
NLM
Freire T de S, Mori MP, Miranda JNFA, Muta LYM, Machado FT, Moreno NC, Souza-Pinto NC de. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells [Internet]. Carcinogenesis. 2021 ; 42( 11): 1380-1389.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/carcin/bgab079
Vancouver
Freire T de S, Mori MP, Miranda JNFA, Muta LYM, Machado FT, Moreno NC, Souza-Pinto NC de. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells [Internet]. Carcinogenesis. 2021 ; 42( 11): 1380-1389.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/carcin/bgab079

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