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Artigo de periodico
Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala (2018)
Lima, Leandro Bueno; Haubenthal, Fernanda Tais; Silveira, Marina Augusto; Bohlen, Tabata Mariz ; Metzger, Martin Andreas ; Donato Junior, José ; Frazão, Renata
Source: Neuroscience Letters. Unidade: ICB
Subjects: AMÍGDALA DO CEREBELO, NEURÔNIOS, OLFATO, ESTEROIDES, RECEPTORES DE ESTEROIDES
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ABNT
LIMA, Leandro Bueno et al. Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala. Neuroscience Letters, v. 681, p. 12-16, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2018.05.023. Acesso em: 10 nov. 2024.
APA
Lima, L. B., Haubenthal, F. T., Silveira, M. A., Bohlen, T. M., Metzger, M. A., Donato Junior, J., & Frazão, R. (2018). Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala. Neuroscience Letters, 681, 12-16. doi:10.1016/j.neulet.2018.05.023
NLM
Lima LB, Haubenthal FT, Silveira MA, Bohlen TM, Metzger MA, Donato Junior J, Frazão R. Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala [Internet]. Neuroscience Letters. 2018 ; 681 12-16.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.neulet.2018.05.023
Vancouver
Lima LB, Haubenthal FT, Silveira MA, Bohlen TM, Metzger MA, Donato Junior J, Frazão R. Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala [Internet]. Neuroscience Letters. 2018 ; 681 12-16.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.neulet.2018.05.023
Artigo de periodico
Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats (2018)
Flores, Rafael Appel; Silva, Eduardo Simão da; Ribas, Anderson Savaris; Taschetto, Ana Paula Dambros; Zampieri, Thais Tessari; Donato Junior, José ; Paschoalini, Marta Aparecida
Source: Brain Research. Unidade: ICB
Subjects: ADRENALINA, SEROTONINA, INGESTÃO, ALIMENTOS, NEURÔNIOS, RATOS
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FLORES, Rafael Appel et al. Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats. Brain Research, v. 1678, p. 153-163, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2017.10.021. Acesso em: 10 nov. 2024.
APA
Flores, R. A., Silva, E. S. da, Ribas, A. S., Taschetto, A. P. D., Zampieri, T. T., Donato Junior, J., & Paschoalini, M. A. (2018). Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats. Brain Research, 1678, 153-163. doi:10.1016/j.brainres.2017.10.021
NLM
Flores RA, Silva ES da, Ribas AS, Taschetto APD, Zampieri TT, Donato Junior J, Paschoalini MA. Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats [Internet]. Brain Research. 2018 ; 1678 153-163.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.brainres.2017.10.021
Vancouver
Flores RA, Silva ES da, Ribas AS, Taschetto APD, Zampieri TT, Donato Junior J, Paschoalini MA. Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats [Internet]. Brain Research. 2018 ; 1678 153-163.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.brainres.2017.10.021
Artigo de periodico
Maternal metabolic adaptations are necessary for normal offspring growth and brain development (2018)
Lobo, Angela Maria Ramos; Furigo, Isadora Clivatti; Teixeria, Pryscila Dryelle Sousa; Zampieri, Thais Tessari; Wasinski, Frederick; Buonfiglio, Daniella do Carmo; Donato Junior, José
Source: Physiological Reports. Unidade: ICB
Subjects: BALANÇO DE ENERGIA, HOMEOSTASE, GLICOSE, HIPOTÁLAMO, METABOLISMO, INSULINA, LEPTINA
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LOBO, Angela Maria Ramos et al. Maternal metabolic adaptations are necessary for normal offspring growth and brain development. Physiological Reports, v. 6, n. 5, p. 15, 2018Tradução . . Disponível em: https://doi.org/10.14814/phy2.13643. Acesso em: 10 nov. 2024.
APA
Lobo, A. M. R., Furigo, I. C., Teixeria, P. D. S., Zampieri, T. T., Wasinski, F., Buonfiglio, D. do C., & Donato Junior, J. (2018). Maternal metabolic adaptations are necessary for normal offspring growth and brain development. Physiological Reports, 6( 5), 15. doi:10.14814/phy2.13643
NLM
Lobo AMR, Furigo IC, Teixeria PDS, Zampieri TT, Wasinski F, Buonfiglio D do C, Donato Junior J. Maternal metabolic adaptations are necessary for normal offspring growth and brain development [Internet]. Physiological Reports. 2018 ; 6( 5): 15.[citado 2024 nov. 10 ] Available from: https://doi.org/10.14814/phy2.13643
Vancouver
Lobo AMR, Furigo IC, Teixeria PDS, Zampieri TT, Wasinski F, Buonfiglio D do C, Donato Junior J. Maternal metabolic adaptations are necessary for normal offspring growth and brain development [Internet]. Physiological Reports. 2018 ; 6( 5): 15.[citado 2024 nov. 10 ] Available from: https://doi.org/10.14814/phy2.13643
Artigo de periodico
Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD (2018)
Araujo, Layanne Cabral da Cunha ; Feitosa, Karla Barroso; Murata, Gilson Masahiro ; Furigo, Isadora Clivatti ; Teixeira, Simone Aparecida ; Monaco, Camila Ferraz Lucena ; Ribeiro, Luciene Maria; Muscará, Marcelo Nicolás (Orientador) ; Costa, Soraia Katia Pereira ; Donato Junior, José ; Bordin, Silvana ; Curi, Rui ; Carvalho, Carla Roberta de Oliveira
Source: Scientific reports. Unidade: ICB
Subjects: PLANTAS MEDICINAIS, HEPATOPATIAS, INSULINA, EXTRATOS (FORMAS FARMACÊUTICAS), GLICOSE, CAMUNDONGOS, HOMEOSTASE
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ARAUJO, Layanne Cabral da Cunha et al. Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD. Scientific reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-29044-y. Acesso em: 10 nov. 2024.
APA
Araujo, L. C. da C., Feitosa, K. B., Murata, G. M., Furigo, I. C., Teixeira, S. A., Monaco, C. F. L., et al. (2018). Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD. Scientific reports, 8. doi:10.1038/s41598-018-29044-y
NLM
Araujo LC da C, Feitosa KB, Murata GM, Furigo IC, Teixeira SA, Monaco CFL, Ribeiro LM, Costa SKP, Donato Junior J, Bordin S, Curi R, Carvalho CR de O. Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD [Internet]. Scientific reports. 2018 ; 8[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-018-29044-y
Vancouver
Araujo LC da C, Feitosa KB, Murata GM, Furigo IC, Teixeira SA, Monaco CFL, Ribeiro LM, Costa SKP, Donato Junior J, Bordin S, Curi R, Carvalho CR de O. Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD [Internet]. Scientific reports. 2018 ; 8[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-018-29044-y
Artigo de periodico
Brain STAT5 signaling modulates learning and memory formation (2018)
Furigo, Isadora Clivatti; Melo, Helen Maciqueira; Silva, Natalia de Menezes Lyra e; Lobo, Angela Maria Ramos; Teixeira, Pryscila Dryelle Sousa; Buonfiglio, Daniella do Carmo; Wasinski, Frederick; Lima Filha, Eliana Rosa; Higuti, Eliza; Peroni, Cibele Nunes; Bartolini, Paolo; Soares, Carlos Roberto Jorge; Metzger, Martin Andreas ; Felice, Fernanda Guarino De; Donato Junior, José
Source: Brain Structure and Function. Unidade: ICB
Subjects: CITOCINAS, MEMÓRIA, CÉREBRO, APRENDIZAGEM ANIMAL, METABOLISMO ANIMAL, LIPÍDEOS, HIPOCAMPU DE ANIMAL, HIPOTÁLAMO
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FURIGO, Isadora Clivatti et al. Brain STAT5 signaling modulates learning and memory formation. Brain Structure and Function, v. 223, n. 5, p. 2229-2241, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00429-018-1627-z. Acesso em: 10 nov. 2024.
APA
Furigo, I. C., Melo, H. M., Silva, N. de M. L. e, Lobo, A. M. R., Teixeira, P. D. S., Buonfiglio, D. do C., et al. (2018). Brain STAT5 signaling modulates learning and memory formation. Brain Structure and Function, 223( 5), 2229-2241. doi:10.1007/s00429-018-1627-z
NLM
Furigo IC, Melo HM, Silva N de ML e, Lobo AMR, Teixeira PDS, Buonfiglio D do C, Wasinski F, Lima Filha ER, Higuti E, Peroni CN, Bartolini P, Soares CRJ, Metzger MA, Felice FGD, Donato Junior J. Brain STAT5 signaling modulates learning and memory formation [Internet]. Brain Structure and Function. 2018 ; 223( 5): 2229-2241.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00429-018-1627-z
Vancouver
Furigo IC, Melo HM, Silva N de ML e, Lobo AMR, Teixeira PDS, Buonfiglio D do C, Wasinski F, Lima Filha ER, Higuti E, Peroni CN, Bartolini P, Soares CRJ, Metzger MA, Felice FGD, Donato Junior J. Brain STAT5 signaling modulates learning and memory formation [Internet]. Brain Structure and Function. 2018 ; 223( 5): 2229-2241.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00429-018-1627-z
Artigo de periodico
A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system (2018)
Bohlen, Tabata Mariz ; Buonfiglio, Daniella do Carmo ; Ferreira Neto, Hildebrando Candido; Cipolla Neto, José ; Donato Junior, José ; Frazão, Renata
Source: Frontiers in Endocrinology. Unidade: ICB
Subjects: PUBERDADE, FOTOPERIODISMO, MELATONINA, BALANÇO DE ENERGIA, CAMUNDONGOS, EXPRESSÃO GÊNICA, HIPOTÁLAMO, NEUROPEPTÍDEOS
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ABNT
BOHLEN, Tabata Mariz et al. A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system. Frontiers in Endocrinology, v. 9, p. 9 , 2018Tradução . . Disponível em: https://doi.org/10.3389/fendo.2018.00044. Acesso em: 10 nov. 2024.
APA
Bohlen, T. M., Buonfiglio, D. do C., Ferreira Neto, H. C., Cipolla Neto, J., Donato Junior, J., & Frazão, R. (2018). A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system. Frontiers in Endocrinology, 9, 9 . doi:10.3389/fendo.2018.00044
NLM
Bohlen TM, Buonfiglio D do C, Ferreira Neto HC, Cipolla Neto J, Donato Junior J, Frazão R. A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system [Internet]. Frontiers in Endocrinology. 2018 ; 9 9 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2018.00044
Vancouver
Bohlen TM, Buonfiglio D do C, Ferreira Neto HC, Cipolla Neto J, Donato Junior J, Frazão R. A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system [Internet]. Frontiers in Endocrinology. 2018 ; 9 9 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2018.00044
Artigo de periodico
Melatonin absence leads to long-term leptin resistance and overweight in rats (2018)
Buonfiglio, Daniella do Carmo; Parthimos, Rafaela Panaiotta; Ferreira, Rosana Fátima Dantas; Silva, Raysa Cerqueira; Gomes, Guilherme; Silva, Jéssica Andrade da; Lobo, Angela Maria Ramos; Amaral, Fernada Gaspar do; Matos, Raphael Afonso de; Silva Junior, José Sinésio; Teixeira, Lívia Clemente Motta; Donato Junior, José ; Reiter, Russel J; Cipolla Neto, José
Source: Frontiers in Endocrinology. Unidade: ICB
Subjects: MELATONINA, LEPTINA, RATOS WISTAR, BALANÇO DE ENERGIA, HIPOTÁLAMO, TECIDO ADIPOSO, NEUROPEPTÍDEOS
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ABNT
BUONFIGLIO, Daniella do Carmo et al. Melatonin absence leads to long-term leptin resistance and overweight in rats. Frontiers in Endocrinology, v. 9, p. 12 , 2018Tradução . . Disponível em: https://doi.org/10.3389/fendo.2018.00122. Acesso em: 10 nov. 2024.
APA
Buonfiglio, D. do C., Parthimos, R. P., Ferreira, R. F. D., Silva, R. C., Gomes, G., Silva, J. A. da, et al. (2018). Melatonin absence leads to long-term leptin resistance and overweight in rats. Frontiers in Endocrinology, 9, 12 . doi:10.3389/fendo.2018.00122
NLM
Buonfiglio D do C, Parthimos RP, Ferreira RFD, Silva RC, Gomes G, Silva JA da, Lobo AMR, Amaral FG do, Matos RA de, Silva Junior JS, Teixeira LCM, Donato Junior J, Reiter RJ, Cipolla Neto J. Melatonin absence leads to long-term leptin resistance and overweight in rats [Internet]. Frontiers in Endocrinology. 2018 ; 9 12 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2018.00122
Vancouver
Buonfiglio D do C, Parthimos RP, Ferreira RFD, Silva RC, Gomes G, Silva JA da, Lobo AMR, Amaral FG do, Matos RA de, Silva Junior JS, Teixeira LCM, Donato Junior J, Reiter RJ, Cipolla Neto J. Melatonin absence leads to long-term leptin resistance and overweight in rats [Internet]. Frontiers in Endocrinology. 2018 ; 9 12 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2018.00122
Artigo de periodico
TGF-β1 down-regulation in the mediobasal hypothalamus attenuates hypothalamic inflammation and protects against diet-induced obesity (2018)
Mendes, Natália Ferreira; Gaspar, Joana Margarida Navalho; Lima Júnior, José Carlos de; Donato Junior, José ; Velloso, Lício Augusto; Araújo, Eliana Pereira de
Source: Metabolism: Clinical and Experimental. Unidade: ICB
Subjects: HIPOTÁLAMO, OBESIDADE, HOMEOSTASE, CAMUNDONGOS, LIPÍDEOS, METABOLISMO ENERGÉTICO, INFLAMAÇÃO, ADIPOSIDADE
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ABNT
MENDES, Natália Ferreira et al. TGF-β1 down-regulation in the mediobasal hypothalamus attenuates hypothalamic inflammation and protects against diet-induced obesity. Metabolism: Clinical and Experimental, v. 85, p. 171-182, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2018.09.007. Acesso em: 10 nov. 2024.
APA
Mendes, N. F., Gaspar, J. M. N., Lima Júnior, J. C. de, Donato Junior, J., Velloso, L. A., & Araújo, E. P. de. (2018). TGF-β1 down-regulation in the mediobasal hypothalamus attenuates hypothalamic inflammation and protects against diet-induced obesity. Metabolism: Clinical and Experimental, 85, 171-182. doi:10.1016/j.metabol.2018.09.007
NLM
Mendes NF, Gaspar JMN, Lima Júnior JC de, Donato Junior J, Velloso LA, Araújo EP de. TGF-β1 down-regulation in the mediobasal hypothalamus attenuates hypothalamic inflammation and protects against diet-induced obesity [Internet]. Metabolism: Clinical and Experimental. 2018 ; 85 171-182.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.metabol.2018.09.007
Vancouver
Mendes NF, Gaspar JMN, Lima Júnior JC de, Donato Junior J, Velloso LA, Araújo EP de. TGF-β1 down-regulation in the mediobasal hypothalamus attenuates hypothalamic inflammation and protects against diet-induced obesity [Internet]. Metabolism: Clinical and Experimental. 2018 ; 85 171-182.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.metabol.2018.09.007
Artigo de periodico
Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats (2018)
Oliveira, Ariclécio Cunha de ; Andreotti, Sandra ; Sertie, Rogério Antônio Laurato ; Campaña, Amanda Baron; Proença, André Ricardo Gomes de ; Vasconcelos, Renata Prado; Oliveira, Keciany Alves de ; Souza, Andrelina Noronha Coelho de ; Donato Junior, José ; Lima, Fabio Bessa
Source: Life Sciences. Unidade: ICB
Subjects: MELATONINA, INSULINA, GLICEMIA, TECIDO ADIPOSO, GORDURAS, HIPOTÁLAMO, SANGUE
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ABNT
OLIVEIRA, Ariclécio Cunha de et al. Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats. Life Sciences, v. 199, p. 158-166, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.02.040. Acesso em: 10 nov. 2024.
APA
Oliveira, A. C. de, Andreotti, S., Sertie, R. A. L., Campaña, A. B., Proença, A. R. G. de, Vasconcelos, R. P., et al. (2018). Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats. Life Sciences, 199, 158-166. doi:10.1016/j.lfs.2018.02.040
NLM
Oliveira AC de, Andreotti S, Sertie RAL, Campaña AB, Proença ARG de, Vasconcelos RP, Oliveira KA de, Souza ANC de, Donato Junior J, Lima FB. Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats [Internet]. Life Sciences. 2018 ; 199 158-166.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.lfs.2018.02.040
Vancouver
Oliveira AC de, Andreotti S, Sertie RAL, Campaña AB, Proença ARG de, Vasconcelos RP, Oliveira KA de, Souza ANC de, Donato Junior J, Lima FB. Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats [Internet]. Life Sciences. 2018 ; 199 158-166.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.lfs.2018.02.040
Artigo de periodico
Afferent and efferent connections of the interpeduncular nucleus with special reference to circuits involving the habenula and raphe nuclei (2017)
Lima, Leandro B.; Bueno, Debora; Leite, Fernanda; Souza, Stefani; Gonçalves, Luciano; Furigo, Isadora C.; Donato Junior, José ; Metzger, Martin Andreas
Source: Journal of Comparative Neurology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
LIMA, Leandro B. et al. Afferent and efferent connections of the interpeduncular nucleus with special reference to circuits involving the habenula and raphe nuclei. Journal of Comparative Neurology, v. 525, n. 10, p. 2411-2442, 2017Tradução . . Disponível em: https://doi.org/10.1002/cne.24217. Acesso em: 10 nov. 2024.
APA
Lima, L. B., Bueno, D., Leite, F., Souza, S., Gonçalves, L., Furigo, I. C., et al. (2017). Afferent and efferent connections of the interpeduncular nucleus with special reference to circuits involving the habenula and raphe nuclei. Journal of Comparative Neurology, 525( 10), 2411-2442. doi:10.1002/cne.24217
NLM
Lima LB, Bueno D, Leite F, Souza S, Gonçalves L, Furigo IC, Donato Junior J, Metzger MA. Afferent and efferent connections of the interpeduncular nucleus with special reference to circuits involving the habenula and raphe nuclei [Internet]. Journal of Comparative Neurology. 2017 ; 525( 10): 2411-2442.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/cne.24217
Vancouver
Lima LB, Bueno D, Leite F, Souza S, Gonçalves L, Furigo IC, Donato Junior J, Metzger MA. Afferent and efferent connections of the interpeduncular nucleus with special reference to circuits involving the habenula and raphe nuclei [Internet]. Journal of Comparative Neurology. 2017 ; 525( 10): 2411-2442.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/cne.24217
Artigo de periodico
Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice (2017)
Kincheski, Grasielle C.; Valentim, Isabela S.; Clarke, Julia R.; Cozachenco, Danielle; Castelo-Branco, Morgana T. L.; Ramos-Lobo, Angela M.; Rumjanek, Vivian M. B. D.; Donato Junior, José ; Felice, Fernanda G. de; Ferreira, Sergio T.
Source: Brain, Behavior, and Immunity. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
KINCHESKI, Grasielle C. et al. Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice. Brain, Behavior, and Immunity, v. 64, p. 140-151, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2017.04.007. Acesso em: 10 nov. 2024.
APA
Kincheski, G. C., Valentim, I. S., Clarke, J. R., Cozachenco, D., Castelo-Branco, M. T. L., Ramos-Lobo, A. M., et al. (2017). Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice. Brain, Behavior, and Immunity, 64, 140-151. doi:10.1016/j.bbi.2017.04.007
NLM
Kincheski GC, Valentim IS, Clarke JR, Cozachenco D, Castelo-Branco MTL, Ramos-Lobo AM, Rumjanek VMBD, Donato Junior J, Felice FG de, Ferreira ST. Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice [Internet]. Brain, Behavior, and Immunity. 2017 ; 64 140-151.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbi.2017.04.007
Vancouver
Kincheski GC, Valentim IS, Clarke JR, Cozachenco D, Castelo-Branco MTL, Ramos-Lobo AM, Rumjanek VMBD, Donato Junior J, Felice FG de, Ferreira ST. Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice [Internet]. Brain, Behavior, and Immunity. 2017 ; 64 140-151.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbi.2017.04.007
Artigo de periodico
SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance (2017)
Pedroso, João A. B.; Mendonca, Pedro O. R. de; Fortes, Marco A. S.; Tomaz, Igor; Pecorali, Vitor L.; Auricino, Thais B.; Costa, Ismael C.; Lima, Leandro B.; Furigo, Isadora C.; Bueno, Debora N.; Ramos-Lobo, Angela M.; Lotfi, Claudimara Ferini Pacicco ; Donato Junior, José
Source: Journal of Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
PEDROSO, João A. B. et al. SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance. Journal of Endocrinology, v. 235, n. 3, p. 207-222, 2017Tradução . . Disponível em: https://doi.org/10.1530/JOE-17-0255. Acesso em: 10 nov. 2024.
APA
Pedroso, J. A. B., Mendonca, P. O. R. de, Fortes, M. A. S., Tomaz, I., Pecorali, V. L., Auricino, T. B., et al. (2017). SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance. Journal of Endocrinology, 235( 3), 207-222. doi:10.1530/JOE-17-0255
NLM
Pedroso JAB, Mendonca POR de, Fortes MAS, Tomaz I, Pecorali VL, Auricino TB, Costa IC, Lima LB, Furigo IC, Bueno DN, Ramos-Lobo AM, Lotfi CFP, Donato Junior J. SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance [Internet]. Journal of Endocrinology. 2017 ; 235( 3): 207-222.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1530/JOE-17-0255
Vancouver
Pedroso JAB, Mendonca POR de, Fortes MAS, Tomaz I, Pecorali VL, Auricino TB, Costa IC, Lima LB, Furigo IC, Bueno DN, Ramos-Lobo AM, Lotfi CFP, Donato Junior J. SOCS3 expression in SF1 cells regulates adrenal differentiation and exercise performance [Internet]. Journal of Endocrinology. 2017 ; 235( 3): 207-222.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1530/JOE-17-0255
Artigo de periodico
The role of leptin in health and disease (2017)
Ramos-Lobo, Angela M.; Donato Junior, José
Source: Temperature. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
RAMOS-LOBO, Angela M. e DONATO JUNIOR, José. The role of leptin in health and disease. Temperature, v. 4, n. 3, p. 258-291, 2017Tradução . . Disponível em: https://doi.org/10.1080/23328940.2017.1327003. Acesso em: 10 nov. 2024.
APA
Ramos-Lobo, A. M., & Donato Junior, J. (2017). The role of leptin in health and disease. Temperature, 4( 3), 258-291. doi:10.1080/23328940.2017.1327003
NLM
Ramos-Lobo AM, Donato Junior J. The role of leptin in health and disease [Internet]. Temperature. 2017 ; 4( 3): 258-291.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/23328940.2017.1327003
Vancouver
Ramos-Lobo AM, Donato Junior J. The role of leptin in health and disease [Internet]. Temperature. 2017 ; 4( 3): 258-291.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/23328940.2017.1327003
Artigo de periodico
Zinc supplementation improves glucose homeostasis in high fat-fed mice by enhancing pancreatic β-cell function (2017)
Cooper-Capetini, Vinícius; Vasconcelos, Diogo Antonio Alves de; Martins, Amanda Roque; Hirabara , Sandro Massao; Donato Junior, José ; Carpinelli, Angelo Rafael ; Abdulkader, Fernando Rodrigues de Moraes
Source: Nutrients. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COOPER-CAPETINI, Vinícius et al. Zinc supplementation improves glucose homeostasis in high fat-fed mice by enhancing pancreatic β-cell function. Nutrients, v. 9, n. 10, p. 1-12, 2017Tradução . . Disponível em: https://doi.org/10.3390/nu9101150. Acesso em: 10 nov. 2024.
APA
Cooper-Capetini, V., Vasconcelos, D. A. A. de, Martins, A. R., Hirabara , S. M., Donato Junior, J., Carpinelli, A. R., & Abdulkader, F. R. de M. (2017). Zinc supplementation improves glucose homeostasis in high fat-fed mice by enhancing pancreatic β-cell function. Nutrients, 9( 10), 1-12. doi:10.3390/nu9101150
NLM
Cooper-Capetini V, Vasconcelos DAA de, Martins AR, Hirabara SM, Donato Junior J, Carpinelli AR, Abdulkader FR de M. Zinc supplementation improves glucose homeostasis in high fat-fed mice by enhancing pancreatic β-cell function [Internet]. Nutrients. 2017 ; 9( 10): 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/nu9101150
Vancouver
Cooper-Capetini V, Vasconcelos DAA de, Martins AR, Hirabara SM, Donato Junior J, Carpinelli AR, Abdulkader FR de M. Zinc supplementation improves glucose homeostasis in high fat-fed mice by enhancing pancreatic β-cell function [Internet]. Nutrients. 2017 ; 9( 10): 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/nu9101150
Artigo de periodico
Distribution of growth hormone-responsive cells in the mouse brain (2017)
Furigo, Isadora C.; Metzger, Martin Andreas ; Teixeira, Pryscila D. S.; Soares, Carlos R. J.; Donato Junior, José
Source: Brain Structure & Function. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FURIGO, Isadora C. et al. Distribution of growth hormone-responsive cells in the mouse brain. Brain Structure & Function, v. 222, n. 1, p. 341-363, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00429-016-1221-1. Acesso em: 10 nov. 2024.
APA
Furigo, I. C., Metzger, M. A., Teixeira, P. D. S., Soares, C. R. J., & Donato Junior, J. (2017). Distribution of growth hormone-responsive cells in the mouse brain. Brain Structure & Function, 222( 1), 341-363. doi:10.1007/s00429-016-1221-1
NLM
Furigo IC, Metzger MA, Teixeira PDS, Soares CRJ, Donato Junior J. Distribution of growth hormone-responsive cells in the mouse brain [Internet]. Brain Structure & Function. 2017 ; 222( 1): 341-363.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00429-016-1221-1
Vancouver
Furigo IC, Metzger MA, Teixeira PDS, Soares CRJ, Donato Junior J. Distribution of growth hormone-responsive cells in the mouse brain [Internet]. Brain Structure & Function. 2017 ; 222( 1): 341-363.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00429-016-1221-1
Artigo de periodico
SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation (2017)
Ramos-Lobo, Angela M.; Teixeira, Pryscila D. S.; Furigo, Isadora C.; Donato Junior, José
Source: Neuroscience. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAMOS-LOBO, Angela M. et al. SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation. Neuroscience, v. 365, p. 114-124, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.09.048. Acesso em: 10 nov. 2024.
APA
Ramos-Lobo, A. M., Teixeira, P. D. S., Furigo, I. C., & Donato Junior, J. (2017). SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation. Neuroscience, 365, 114-124. doi:10.1016/j.neuroscience.2017.09.048
NLM
Ramos-Lobo AM, Teixeira PDS, Furigo IC, Donato Junior J. SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation [Internet]. Neuroscience. 2017 ; 365 114-124.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.09.048
Vancouver
Ramos-Lobo AM, Teixeira PDS, Furigo IC, Donato Junior J. SOCS3 ablation in SF1 cells causes modest metabolic effects during pregnancy and lactation [Internet]. Neuroscience. 2017 ; 365 114-124.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.09.048
Artigo de periodico
PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction (2017)
Garcia-Galiano, David; Borges, Beatriz C.; Donato Junior, José ; Allen, Susan J.; Bellefontaine, Nicole; Wang, Mengjie; Zhao, Jean J.; Kozloff, Kenneth M.; Hill, Jennifer W.; Elias, Carol F.
Source: Journal of Clinical Investigation Insight - JCI insight. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GARCIA-GALIANO, David et al. PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction. Journal of Clinical Investigation Insight - JCI insight, v. 2, n. 23, p. 1-19, 2017Tradução . . Disponível em: https://doi.org/10.1172/jci.insight.96728. Acesso em: 10 nov. 2024.
APA
Garcia-Galiano, D., Borges, B. C., Donato Junior, J., Allen, S. J., Bellefontaine, N., Wang, M., et al. (2017). PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction. Journal of Clinical Investigation Insight - JCI insight, 2( 23), 1-19. doi:10.1172/jci.insight.96728
NLM
Garcia-Galiano D, Borges BC, Donato Junior J, Allen SJ, Bellefontaine N, Wang M, Zhao JJ, Kozloff KM, Hill JW, Elias CF. PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction [Internet]. Journal of Clinical Investigation Insight - JCI insight. 2017 ; 2( 23): 1-19.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1172/jci.insight.96728
Vancouver
Garcia-Galiano D, Borges BC, Donato Junior J, Allen SJ, Bellefontaine N, Wang M, Zhao JJ, Kozloff KM, Hill JW, Elias CF. PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction [Internet]. Journal of Clinical Investigation Insight - JCI insight. 2017 ; 2( 23): 1-19.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1172/jci.insight.96728
Artigo de periodico
Expression, purification and characterization of the authentic form of human growth hormone receptor antagonist G120R-hGH obtained in Escherichia coli periplasmic space (2017)
Menezes, Ana C. S. C.; Suzuki, Miriam F.; Oliveira, João E.; Ribela, Maria Teresa de Carvalho Pinto; Furigo, Isadora C.; Donato Junior, José ; Bartolini, Paolo; Soares, Carlos R. J.
Source: Protein Expression and Purification. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MENEZES, Ana C. S. C. et al. Expression, purification and characterization of the authentic form of human growth hormone receptor antagonist G120R-hGH obtained in Escherichia coli periplasmic space. Protein Expression and Purification, v. 131, p. 91-100, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0085026. Acesso em: 10 nov. 2024.
APA
Menezes, A. C. S. C., Suzuki, M. F., Oliveira, J. E., Ribela, M. T. de C. P., Furigo, I. C., Donato Junior, J., et al. (2017). Expression, purification and characterization of the authentic form of human growth hormone receptor antagonist G120R-hGH obtained in Escherichia coli periplasmic space. Protein Expression and Purification, 131, 91-100. doi:10.1371/journal.pone.0085026
NLM
Menezes ACSC, Suzuki MF, Oliveira JE, Ribela MT de CP, Furigo IC, Donato Junior J, Bartolini P, Soares CRJ. Expression, purification and characterization of the authentic form of human growth hormone receptor antagonist G120R-hGH obtained in Escherichia coli periplasmic space [Internet]. Protein Expression and Purification. 2017 ; 131 91-100.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0085026
Vancouver
Menezes ACSC, Suzuki MF, Oliveira JE, Ribela MT de CP, Furigo IC, Donato Junior J, Bartolini P, Soares CRJ. Expression, purification and characterization of the authentic form of human growth hormone receptor antagonist G120R-hGH obtained in Escherichia coli periplasmic space [Internet]. Protein Expression and Purification. 2017 ; 131 91-100.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0085026
Artigo de periodico
Injections of the of the α1-adrenoceptor antagonist prazosin into the median raphe nucleus increase food intake and Fos expression in orexin neurons of free-feeding rats (2017)
Silva, Eduardo Simão da; Flores, Rafael Appel; Ribas, Anderson Savaris; Taschetto, Ana Paula; Faria, Moacir Serralvo; Lima, Leandro Bueno; Metzger, Martin Andreas ; Donato Junior, José ; Paschoalini, Marta Aparecida
Source: Behavioural Brain Research. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Eduardo Simão da et al. Injections of the of the α1-adrenoceptor antagonist prazosin into the median raphe nucleus increase food intake and Fos expression in orexin neurons of free-feeding rats. Behavioural Brain Research, v. 324, p. 87-95, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2017.02.021. Acesso em: 10 nov. 2024.
APA
Silva, E. S. da, Flores, R. A., Ribas, A. S., Taschetto, A. P., Faria, M. S., Lima, L. B., et al. (2017). Injections of the of the α1-adrenoceptor antagonist prazosin into the median raphe nucleus increase food intake and Fos expression in orexin neurons of free-feeding rats. Behavioural Brain Research, 324, 87-95. doi:10.1016/j.bbr.2017.02.021
NLM
Silva ES da, Flores RA, Ribas AS, Taschetto AP, Faria MS, Lima LB, Metzger MA, Donato Junior J, Paschoalini MA. Injections of the of the α1-adrenoceptor antagonist prazosin into the median raphe nucleus increase food intake and Fos expression in orexin neurons of free-feeding rats [Internet]. Behavioural Brain Research. 2017 ; 324 87-95.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbr.2017.02.021
Vancouver
Silva ES da, Flores RA, Ribas AS, Taschetto AP, Faria MS, Lima LB, Metzger MA, Donato Junior J, Paschoalini MA. Injections of the of the α1-adrenoceptor antagonist prazosin into the median raphe nucleus increase food intake and Fos expression in orexin neurons of free-feeding rats [Internet]. Behavioural Brain Research. 2017 ; 324 87-95.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.bbr.2017.02.021
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 10 nov. 2024.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1042/CS20171368

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