Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice (2017)
- Authors:
- Autor USP: DONATO JÚNIOR, JOSÉ - ICB
- Unidade: ICB
- DOI: 10.1016/j.bbi.2017.04.007
- Assunto: FISIOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Maryland Heights
- Date published: 2017
- Source:
- Título: Brain, Behavior, and Immunity
- ISSN: 1090-2139
- Volume/Número/Paginação/Ano: v. 64, p. 140-151, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
KINCHESKI, Grasielle C. et al. Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice. Brain, Behavior, and Immunity, v. 64, p. 140-151, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2017.04.007. Acesso em: 28 dez. 2025. -
APA
Kincheski, G. C., Valentim, I. S., Clarke, J. R., Cozachenco, D., Castelo-Branco, M. T. L., Ramos-Lobo, A. M., et al. (2017). Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice. Brain, Behavior, and Immunity, 64, 140-151. doi:10.1016/j.bbi.2017.04.007 -
NLM
Kincheski GC, Valentim IS, Clarke JR, Cozachenco D, Castelo-Branco MTL, Ramos-Lobo AM, Rumjanek VMBD, Donato Junior J, Felice FG de, Ferreira ST. Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice [Internet]. Brain, Behavior, and Immunity. 2017 ; 64 140-151.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/j.bbi.2017.04.007 -
Vancouver
Kincheski GC, Valentim IS, Clarke JR, Cozachenco D, Castelo-Branco MTL, Ramos-Lobo AM, Rumjanek VMBD, Donato Junior J, Felice FG de, Ferreira ST. Chronic sleep restriction promotes brain inflammation and synapse loss, and potentiates memory impairment induced by amyloid-β oligomers in mice [Internet]. Brain, Behavior, and Immunity. 2017 ; 64 140-151.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/j.bbi.2017.04.007 - Lack of SOCS3 gene in leptin receptor-expressing cells impairs fetal growth and offspring weight
- Alteration in the number of neuronal and non-neuronal cells in mouse models of obesity
- Growth hormone, hpothalamic inflammation, and aging
- Pro-inflammatory interleukin-6 signaling links cognitive impairments and peripheral metabolic alterations in Alzheimer’s disease
- Identification of prolactin-responsive cells that express leptin receptors in the mouse brain
- The central nervous system as a promising target to treat diabetes mellitus
- Cardiorespiratory fitness in adolescents with obesity: a 6-month follow-up study
- O hipotálamo em foco: em busca dos fatores reguladores do metabolismo
- Programming of metabolism by adipokines during development
- Papel da sinalização hormonal no encéfalo para o controle do balanço energético e metabolismo
Informações sobre o DOI: 10.1016/j.bbi.2017.04.007 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
