Lack of SOCS3 gene in leptin receptor-expressing cells impairs fetal growth and offspring weight (2012)
- Authors:
- Autor USP: DONATO JÚNIOR, JOSÉ - ICB
- Unidade: ICB
- Assunto: FISIOLOGIA HUMANA
- Language: Inglês
- Imprenta:
- Publisher: Dep/Physiol/Biophys/ICB/USP
- Publisher place: São Paulo
- Date published: 2012
- Source:
- Título: Abstracts
- Conference titles: Thematic Symposium: new frontiers in applied physiology
-
ABNT
ZAMPIERI, Thais Tessari e DONATO JUNIOR, José. Lack of SOCS3 gene in leptin receptor-expressing cells impairs fetal growth and offspring weight. 2012, Anais.. São Paulo: Dep/Physiol/Biophys/ICB/USP, 2012. . Acesso em: 11 fev. 2026. -
APA
Zampieri, T. T., & Donato Junior, J. (2012). Lack of SOCS3 gene in leptin receptor-expressing cells impairs fetal growth and offspring weight. In Abstracts. São Paulo: Dep/Physiol/Biophys/ICB/USP. -
NLM
Zampieri TT, Donato Junior J. Lack of SOCS3 gene in leptin receptor-expressing cells impairs fetal growth and offspring weight. Abstracts. 2012 ;[citado 2026 fev. 11 ] -
Vancouver
Zampieri TT, Donato Junior J. Lack of SOCS3 gene in leptin receptor-expressing cells impairs fetal growth and offspring weight. Abstracts. 2012 ;[citado 2026 fev. 11 ] - Densely packed GluA2/3 immunoreactive neurons outline a complex corridor compartment in the caudal nucleus accumbens shell
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- Expression, purification and characterization of the authentic form of human growth hormone receptor antagonist G120R-hGH obtained in Escherichia coli periplasmic space
- PI3Kα inactivation in leptin receptor cells increases leptina sensitivity but disrupts growth and reproduction
- SOCS3 deficiency in leptin receptor-expressing cells mitigates the development of pregnancy-induced metabolic changes
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