Filtros : "ICB" "Indexado na Base de dados Science Citation Index" Removidos: "USP" "Universidade Federal de Santa Catarina (UFSC)" "Gomez, José Gregório Cabrera" "2024" "A sociedade" Limpar

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  • Source: Journal of Physiology. Unidades: ICB, FMRP

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 02 jul. 2024.
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      Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
    • NLM

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
    • Vancouver

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      DAVEL, A. P. et al. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, v. 44, n. 9, p. 920-932, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500104. Acesso em: 02 jul. 2024.
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      Davel, A. P., Wenceslau, C. F., Akamine, E. H., Xavier, F. E., Couto, G. K., Oliveira, H. T., & Rossoni, L. V. (2011). Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, 44( 9), 920-932. doi:10.1590/S0100-879X2011007500104
    • NLM

      Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
    • Vancouver

      Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
  • Source: Oncogene. Unidades: FMRP, ICB, FCFRP

    Assunto: IMUNOLOGIA

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      CARVALHO, Daniel Diniz de et al. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, v. 30, n. 2, p. 223-233, 2011Tradução . . Disponível em: https://doi.org/10.1038/onc.2010.409. Acesso em: 02 jul. 2024.
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      Carvalho, D. D. de, Binato, R., Pereira, W. de O., Leroy, J. M. G., Colassanti, M. D., Proto-Siqueira, R., et al. (2011). BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, 30( 2), 223-233. doi:10.1038/onc.2010.409
    • NLM

      Carvalho DD de, Binato R, Pereira W de O, Leroy JMG, Colassanti MD, Proto-Siqueira R, Silva AEBB da, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1038/onc.2010.409
    • Vancouver

      Carvalho DD de, Binato R, Pereira W de O, Leroy JMG, Colassanti MD, Proto-Siqueira R, Silva AEBB da, Zago MA, Zanichelli MA, Abdelhay E, Castro FA de, Jacysyn JF, Amarante-Mendes JGP. BCR–ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients [Internet]. Oncogene. 2011 ; 30( 2): 223-233.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1038/onc.2010.409
  • Source: International Journal of Developmental Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEONELLI, Mauro e MARTINS, Daniel de Oliveira e BRITTO, Luiz Roberto Giorgetti de. TRPV1 receptors modulate retinal development. International Journal of Developmental Neuroscience, v. 29, n. 4, p. 405-413, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.ijdevneu.2011.03.002. Acesso em: 02 jul. 2024.
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      Leonelli, M., Martins, D. de O., & Britto, L. R. G. de. (2011). TRPV1 receptors modulate retinal development. International Journal of Developmental Neuroscience, 29( 4), 405-413. doi:10.1016/j.ijdevneu.2011.03.002
    • NLM

      Leonelli M, Martins D de O, Britto LRG de. TRPV1 receptors modulate retinal development [Internet]. International Journal of Developmental Neuroscience. 2011 ; 29( 4): 405-413.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ijdevneu.2011.03.002
    • Vancouver

      Leonelli M, Martins D de O, Britto LRG de. TRPV1 receptors modulate retinal development [Internet]. International Journal of Developmental Neuroscience. 2011 ; 29( 4): 405-413.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ijdevneu.2011.03.002
  • Source: Antimicrobial Agents and Chemotherapy. Unidade: ICB

    Assunto: PARASITOLOGIA

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      JORDÃO, Fabiana Morandi et al. In Vitro and In Vivo antiplasmodial activities of risedronate and its interference with protein prenylation in Plasmodium falciparum. Antimicrobial Agents and Chemotherapy, v. 55, n. 5, p. 2026-2031, 2011Tradução . . Disponível em: https://doi.org/10.1128/AAC.01820-10. Acesso em: 02 jul. 2024.
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      Jordão, F. M., Saito, A. Y., Miguel, D. C., Peres, V. de J., Kimura, E. A., & Katzin, A. M. (2011). In Vitro and In Vivo antiplasmodial activities of risedronate and its interference with protein prenylation in Plasmodium falciparum. Antimicrobial Agents and Chemotherapy, 55( 5), 2026-2031. doi:10.1128/AAC.01820-10
    • NLM

      Jordão FM, Saito AY, Miguel DC, Peres V de J, Kimura EA, Katzin AM. In Vitro and In Vivo antiplasmodial activities of risedronate and its interference with protein prenylation in Plasmodium falciparum [Internet]. Antimicrobial Agents and Chemotherapy. 2011 ; 55( 5): 2026-2031.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1128/AAC.01820-10
    • Vancouver

      Jordão FM, Saito AY, Miguel DC, Peres V de J, Kimura EA, Katzin AM. In Vitro and In Vivo antiplasmodial activities of risedronate and its interference with protein prenylation in Plasmodium falciparum [Internet]. Antimicrobial Agents and Chemotherapy. 2011 ; 55( 5): 2026-2031.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1128/AAC.01820-10
  • Source: Inflammation. Unidade: ICB

    Assunto: FISIOLOGIA

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      DELBIN, Maria Andréia et al. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, v. 34, n. 5, p. 319-325, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10753-010-9237-9. Acesso em: 02 jul. 2024.
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      Delbin, M. A., Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2011). Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, 34( 5), 319-325. doi:10.1007/s10753-010-9237-9
    • NLM

      Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
    • Vancouver

      Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      MARIÚBA, M. V. et al. Effect of triiodothyronine on the maxilla and masseter muscles of the rat stomatognathic system. Brazilian Journal of Medical and Biological Research, v. 44, n. 7, p. 694-699, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500063. Acesso em: 02 jul. 2024.
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      Mariúba, M. V., Silva, F. G. da, Bordin, S., & Nunes, M. T. (2011). Effect of triiodothyronine on the maxilla and masseter muscles of the rat stomatognathic system. Brazilian Journal of Medical and Biological Research, 44( 7), 694-699. doi:10.1590/S0100-879X2011007500063
    • NLM

      Mariúba MV, Silva FG da, Bordin S, Nunes MT. Effect of triiodothyronine on the maxilla and masseter muscles of the rat stomatognathic system [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 7): 694-699.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/S0100-879X2011007500063
    • Vancouver

      Mariúba MV, Silva FG da, Bordin S, Nunes MT. Effect of triiodothyronine on the maxilla and masseter muscles of the rat stomatognathic system [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 7): 694-699.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/S0100-879X2011007500063
  • Source: Allergologia et Immunopathologia. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LEIVA, L. E. et al. Primary immunodeficiency diseases in Latin America: proceedings of the second Latin American society for immunodeficiencies (LASID) advisory board. Allergologia et Immunopathologia, v. 39, n. 2, p. 106-110, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.aller.2010.10.007. Acesso em: 02 jul. 2024.
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      Leiva, L. E., Bezrodnik, L., Oleastro, M., Condino-Neto, A., Costa-Carvalho, B. T., Grumach, A. S., et al. (2011). Primary immunodeficiency diseases in Latin America: proceedings of the second Latin American society for immunodeficiencies (LASID) advisory board. Allergologia et Immunopathologia, 39( 2), 106-110. doi:10.1016/j.aller.2010.10.007
    • NLM

      Leiva LE, Bezrodnik L, Oleastro M, Condino-Neto A, Costa-Carvalho BT, Grumach AS, Espinosa-Rosales FJ, Franco JL, King A, Inostroza J, Quezada A, Porras O, Sorensen RU. Primary immunodeficiency diseases in Latin America: proceedings of the second Latin American society for immunodeficiencies (LASID) advisory board [Internet]. Allergologia et Immunopathologia. 2011 ; 39( 2): 106-110.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.aller.2010.10.007
    • Vancouver

      Leiva LE, Bezrodnik L, Oleastro M, Condino-Neto A, Costa-Carvalho BT, Grumach AS, Espinosa-Rosales FJ, Franco JL, King A, Inostroza J, Quezada A, Porras O, Sorensen RU. Primary immunodeficiency diseases in Latin America: proceedings of the second Latin American society for immunodeficiencies (LASID) advisory board [Internet]. Allergologia et Immunopathologia. 2011 ; 39( 2): 106-110.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.aller.2010.10.007
  • Source: Respiratory Physiology and Neurobiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      FAVERO, Michele T. et al. Chemosensory control by commissural nucleus of the solitary tract in rats. Respiratory Physiology and Neurobiology, v. 179, n. 2-3, p. 227-234, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.resp.2011.08.010. Acesso em: 02 jul. 2024.
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      Favero, M. T., Takakura, A. C., Paula, P. M. de, Colombari, E., Menani, J. V., & Moreira, T. dos S. (2011). Chemosensory control by commissural nucleus of the solitary tract in rats. Respiratory Physiology and Neurobiology, 179( 2-3), 227-234. doi:10.1016/j.resp.2011.08.010
    • NLM

      Favero MT, Takakura AC, Paula PM de, Colombari E, Menani JV, Moreira T dos S. Chemosensory control by commissural nucleus of the solitary tract in rats [Internet]. Respiratory Physiology and Neurobiology. 2011 ; 179( 2-3): 227-234.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.resp.2011.08.010
    • Vancouver

      Favero MT, Takakura AC, Paula PM de, Colombari E, Menani JV, Moreira T dos S. Chemosensory control by commissural nucleus of the solitary tract in rats [Internet]. Respiratory Physiology and Neurobiology. 2011 ; 179( 2-3): 227-234.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.resp.2011.08.010
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      MOREIRA, Thiago dos Santos et al. Central chemoreceptors and neural mechanisms of cardiorespiratory control. Brazilian Journal of Medical and Biological Research, v. 44, n. 9, p. 883-889, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500094. Acesso em: 02 jul. 2024.
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      Moreira, T. dos S., Takakura, A. C., Damasceno, R. dos S., Falquetto, B., Totola, L. T., Roberto Sobrinho, C., et al. (2011). Central chemoreceptors and neural mechanisms of cardiorespiratory control. Brazilian Journal of Medical and Biological Research, 44( 9), 883-889. doi:10.1590/S0100-879X2011007500094
    • NLM

      Moreira T dos S, Takakura AC, Damasceno R dos S, Falquetto B, Totola LT, Roberto Sobrinho C, Ragioto DT, Zolezi FP. Central chemoreceptors and neural mechanisms of cardiorespiratory control [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 883-889.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/S0100-879X2011007500094
    • Vancouver

      Moreira T dos S, Takakura AC, Damasceno R dos S, Falquetto B, Totola LT, Roberto Sobrinho C, Ragioto DT, Zolezi FP. Central chemoreceptors and neural mechanisms of cardiorespiratory control [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 883-889.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1590/S0100-879X2011007500094
  • Source: Clinical and experimental immunology. Unidades: FM, ICB

    Subjects: EXPRESSÃO GÊNICA, INFLAMAÇÃO, LINFÓCITOS, NEUTRÓFILOS, NUTRIÇÃO PARENTERAL

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      CURY-BOAVENTURA, M. F. et al. Effect of medium/ω-6 long chain triglyceride-based emulsion on leucocyte death and inflammatory gene expression. Clinical and experimental immunology, v. 165, n. 3, p. 383-392, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2249.2011.04432.x. Acesso em: 02 jul. 2024.
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      Cury-Boaventura, M. F., Gorjão, R., Lima, T. M. de, Fiamoncini, J., Godoy, A. B. P., Deschamphs, F. C., et al. (2011). Effect of medium/ω-6 long chain triglyceride-based emulsion on leucocyte death and inflammatory gene expression. Clinical and experimental immunology, 165( 3), 383-392. doi:10.1111/j.1365-2249.2011.04432.x
    • NLM

      Cury-Boaventura MF, Gorjão R, Lima TM de, Fiamoncini J, Godoy ABP, Deschamphs FC, Soriano FC, Curi R. Effect of medium/ω-6 long chain triglyceride-based emulsion on leucocyte death and inflammatory gene expression [Internet]. Clinical and experimental immunology. 2011 ; 165( 3): 383-392.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1111/j.1365-2249.2011.04432.x
    • Vancouver

      Cury-Boaventura MF, Gorjão R, Lima TM de, Fiamoncini J, Godoy ABP, Deschamphs FC, Soriano FC, Curi R. Effect of medium/ω-6 long chain triglyceride-based emulsion on leucocyte death and inflammatory gene expression [Internet]. Clinical and experimental immunology. 2011 ; 165( 3): 383-392.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1111/j.1365-2249.2011.04432.x
  • Source: Clinical and Vaccine Immunology. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      MATHIAS-SANTOS, Camila et al. Distinctive immunomodulatory and inflammatory properties of the Escherichia coli type II heat-labile enterotoxin LT-IIa and its B pentamer following intradermal administration. Clinical and Vaccine Immunology, v. 18, n. 8, p. 1243-1251, 2011Tradução . . Disponível em: https://doi.org/10.1128/CVI.00012-11. Acesso em: 02 jul. 2024.
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      Mathias-Santos, C., Rodrigues, J. F., Sbrogio-Almeida, M. E., Connell, T. D., & Ferreira, L. C. de S. (2011). Distinctive immunomodulatory and inflammatory properties of the Escherichia coli type II heat-labile enterotoxin LT-IIa and its B pentamer following intradermal administration. Clinical and Vaccine Immunology, 18( 8), 1243-1251. doi:10.1128/CVI.00012-11
    • NLM

      Mathias-Santos C, Rodrigues JF, Sbrogio-Almeida ME, Connell TD, Ferreira LC de S. Distinctive immunomodulatory and inflammatory properties of the Escherichia coli type II heat-labile enterotoxin LT-IIa and its B pentamer following intradermal administration [Internet]. Clinical and Vaccine Immunology. 2011 ; 18( 8): 1243-1251.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1128/CVI.00012-11
    • Vancouver

      Mathias-Santos C, Rodrigues JF, Sbrogio-Almeida ME, Connell TD, Ferreira LC de S. Distinctive immunomodulatory and inflammatory properties of the Escherichia coli type II heat-labile enterotoxin LT-IIa and its B pentamer following intradermal administration [Internet]. Clinical and Vaccine Immunology. 2011 ; 18( 8): 1243-1251.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1128/CVI.00012-11
  • Source: Peptides. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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      LOIOLA, Rodrigo Azevedo et al. Vascular mechanisms involved in angiotensin II-induced venoconstriction in hypertensive rats. Peptides, v. 32, n. 10, p. 2116-2121, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.peptides.2011.09.011. Acesso em: 02 jul. 2024.
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      Loiola, R. A., Fernandes, L., Eichler, R. A., Passaglia, R. de C. A. T., Fortes, Z. B., & Carvalho, M. H. C. de. (2011). Vascular mechanisms involved in angiotensin II-induced venoconstriction in hypertensive rats. Peptides, 32( 10), 2116-2121. doi:10.1016/j.peptides.2011.09.011
    • NLM

      Loiola RA, Fernandes L, Eichler RA, Passaglia R de CAT, Fortes ZB, Carvalho MHC de. Vascular mechanisms involved in angiotensin II-induced venoconstriction in hypertensive rats [Internet]. Peptides. 2011 ; 32( 10): 2116-2121.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.peptides.2011.09.011
    • Vancouver

      Loiola RA, Fernandes L, Eichler RA, Passaglia R de CAT, Fortes ZB, Carvalho MHC de. Vascular mechanisms involved in angiotensin II-induced venoconstriction in hypertensive rats [Internet]. Peptides. 2011 ; 32( 10): 2116-2121.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.peptides.2011.09.011
  • Source: Clinical Science. Unidade: ICB

    Assunto: FISIOLOGIA

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      ROSSONI, Luciana Venturini et al. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, v. 121, n. 10, p. 449-458, 2011Tradução . . Disponível em: https://doi.org/10.1042/CS20110259. Acesso em: 02 jul. 2024.
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      Rossoni, L. V., Wareing, M., Wenceslau, C. F., Al-Abri, M., Cobb, C., & Austin, C. (2011). Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, 121( 10), 449-458. doi:10.1042/CS20110259
    • NLM

      Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1042/CS20110259
    • Vancouver

      Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1042/CS20110259
  • Source: Journal of Biological Chemistry. Unidades: ICB, EERP, FMRP

    Subjects: IMUNOLOGIA, PROTEINAS, ADJUVANTES IMUNOLÓGICOS

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      OLIVEIRA, Carlo José Freire de et al. Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties. Journal of Biological Chemistry, v. 286, n. 13, p. 10960-10969, 2011Tradução . . Disponível em: https://doi.org/10.1074/jbc.M110.205047. Acesso em: 02 jul. 2024.
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      Oliveira, C. J. F. de, Nunes, A. de S., Francischetti, I. M. B., Carregaro, V., Anatriello, E., Silva, J. S. da, et al. (2011). Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties. Journal of Biological Chemistry, 286( 13), 10960-10969. doi:10.1074/jbc.M110.205047
    • NLM

      Oliveira CJF de, Nunes A de S, Francischetti IMB, Carregaro V, Anatriello E, Silva JS da, Santos IKF de M, Ribeiro JMC, Ferreira BR. Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties [Internet]. Journal of Biological Chemistry. 2011 ; 286( 13): 10960-10969.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1074/jbc.M110.205047
    • Vancouver

      Oliveira CJF de, Nunes A de S, Francischetti IMB, Carregaro V, Anatriello E, Silva JS da, Santos IKF de M, Ribeiro JMC, Ferreira BR. Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties [Internet]. Journal of Biological Chemistry. 2011 ; 286( 13): 10960-10969.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1074/jbc.M110.205047
  • Source: American Journal of Reproductive Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      OLIVEIRA, Leandro de et al. Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood. American Journal of Reproductive Immunology, v. 65, n. 1, p. 11-12, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1600-0897.2010.00915.x. Acesso em: 02 jul. 2024.
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      Oliveira, L. de, Larocca, R. A., Sass, N., & Câmara, N. O. S. (2011). Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood. American Journal of Reproductive Immunology, 65( 1), 11-12. doi:10.1111/j.1600-0897.2010.00915.x
    • NLM

      Oliveira L de, Larocca RA, Sass N, Câmara NOS. Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood [Internet]. American Journal of Reproductive Immunology. 2011 ; 65( 1): 11-12.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1111/j.1600-0897.2010.00915.x
    • Vancouver

      Oliveira L de, Larocca RA, Sass N, Câmara NOS. Proportion of invariant NKT Cells in normal pregnant women at term: an evaluation in peripheral blood, placenta and umbilical cord blood [Internet]. American Journal of Reproductive Immunology. 2011 ; 65( 1): 11-12.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1111/j.1600-0897.2010.00915.x
  • Source: Genes and Immunity. Unidades: FOB, FMRP, ICB

    Subjects: PERIODONTITE, ARTRITE REUMATOIDE, INFLAMAÇÃO, CITOCINAS

    Acesso à fonteDOIHow to cite
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      TROMBONE, A. P. et al. Periodontitis and arthritis interaction in mice involves a shared hyper-inflammatory genotype and functional immunological interferences. Genes and Immunity, v. 11, n. 6, p. 479-489, 2010Tradução . . Disponível em: https://doi.org/10.1038/gene.2010.13. Acesso em: 02 jul. 2024.
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      Trombone, A. P., Claudino, M., Colavite, P., Assis, G. F. de, Ávila-Campos, M. J., Silva, J. S. da, et al. (2010). Periodontitis and arthritis interaction in mice involves a shared hyper-inflammatory genotype and functional immunological interferences. Genes and Immunity, 11( 6), 479-489. doi:10.1038/gene.2010.13
    • NLM

      Trombone AP, Claudino M, Colavite P, Assis GF de, Ávila-Campos MJ, Silva JS da, Campanelli AP, Ibañez OM, De Franco M, Garlet GP. Periodontitis and arthritis interaction in mice involves a shared hyper-inflammatory genotype and functional immunological interferences [Internet]. Genes and Immunity. 2010 ; 11( 6): 479-489.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1038/gene.2010.13
    • Vancouver

      Trombone AP, Claudino M, Colavite P, Assis GF de, Ávila-Campos MJ, Silva JS da, Campanelli AP, Ibañez OM, De Franco M, Garlet GP. Periodontitis and arthritis interaction in mice involves a shared hyper-inflammatory genotype and functional immunological interferences [Internet]. Genes and Immunity. 2010 ; 11( 6): 479-489.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1038/gene.2010.13
  • Source: Experimental Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      NALIVAIKO, Eugene e ANTUNES, Vagner Roberto e PATON, Julian F. R. Control of cardiac contractility in the rat working heart–brainstem preparation. Experimental Physiology, v. 95, n. 1, p. 107-19, 2010Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2009.048710. Acesso em: 02 jul. 2024.
    • APA

      Nalivaiko, E., Antunes, V. R., & Paton, J. F. R. (2010). Control of cardiac contractility in the rat working heart–brainstem preparation. Experimental Physiology, 95( 1), 107-19. doi:10.1113/expphysiol.2009.048710
    • NLM

      Nalivaiko E, Antunes VR, Paton JFR. Control of cardiac contractility in the rat working heart–brainstem preparation [Internet]. Experimental Physiology. 2010 ; 95( 1): 107-19.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1113/expphysiol.2009.048710
    • Vancouver

      Nalivaiko E, Antunes VR, Paton JFR. Control of cardiac contractility in the rat working heart–brainstem preparation [Internet]. Experimental Physiology. 2010 ; 95( 1): 107-19.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1113/expphysiol.2009.048710
  • Source: Cell Biochemistry and Function. Unidade: ICB

    Assunto: FISIOLOGIA

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      POLETTO, Ana Cláudia et al. Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue. Cell Biochemistry and Function, v. 28, n. 2, p. 114-121, 2010Tradução . . Disponível em: https://doi.org/10.1002/cbf.1628. Acesso em: 02 jul. 2024.
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      Poletto, A. C., Anhê, G. F., Eichler, P., Takahashi, H., Furuya, D. T., Okamoto, M. M., et al. (2010). Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue. Cell Biochemistry and Function, 28( 2), 114-121. doi:10.1002/cbf.1628
    • NLM

      Poletto AC, Anhê GF, Eichler P, Takahashi H, Furuya DT, Okamoto MM, Curi R, Machado UF. Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue [Internet]. Cell Biochemistry and Function. 2010 ; 28( 2): 114-121.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1002/cbf.1628
    • Vancouver

      Poletto AC, Anhê GF, Eichler P, Takahashi H, Furuya DT, Okamoto MM, Curi R, Machado UF. Soybean and sunflower oil-induced insulin resistance correlates with impaired GLUT4 protein expression and translocation specifically in white adipose tissue [Internet]. Cell Biochemistry and Function. 2010 ; 28( 2): 114-121.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1002/cbf.1628
  • Source: Cancer Chemotherapy and Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      BASTOS-PEREIRA, Amanda Leite et al. Celecoxib prevents tumor growth in an animal model by a COX-2 independent mechanism. Cancer Chemotherapy and Pharmacology, v. 65, n. 2, p. 267-76, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00280-009-1031-8. Acesso em: 02 jul. 2024.
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      Bastos-Pereira, A. L., Lugarini, D., Oliveira-Christoff, A. de, Ávila, T. V., Teixeira, S., Pires, A. do R. A., et al. (2010). Celecoxib prevents tumor growth in an animal model by a COX-2 independent mechanism. Cancer Chemotherapy and Pharmacology, 65( 2), 267-76. doi:10.1007/s00280-009-1031-8
    • NLM

      Bastos-Pereira AL, Lugarini D, Oliveira-Christoff A de, Ávila TV, Teixeira S, Pires A do RA, Muscará MN, Cadena SMSC, Donatti L, da Silva de Assis HC da, Acco A. Celecoxib prevents tumor growth in an animal model by a COX-2 independent mechanism [Internet]. Cancer Chemotherapy and Pharmacology. 2010 ; 65( 2): 267-76.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s00280-009-1031-8
    • Vancouver

      Bastos-Pereira AL, Lugarini D, Oliveira-Christoff A de, Ávila TV, Teixeira S, Pires A do RA, Muscará MN, Cadena SMSC, Donatti L, da Silva de Assis HC da, Acco A. Celecoxib prevents tumor growth in an animal model by a COX-2 independent mechanism [Internet]. Cancer Chemotherapy and Pharmacology. 2010 ; 65( 2): 267-76.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s00280-009-1031-8

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