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Artigo de periodico
The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway (2020)
Leite, Jefferson Antonio; Pessenda, Gabriela ; Gomes, Isabel Cristina Guerra ; Santana, Alynne Karen Mendonça de; Pereira, Camila André; Costa, Frederico Ribeiro Campos; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Faria, Ana Maria Caetano ; Almeida, Danilo Candido de ; Câmara, Niels Olsen Saraiva ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Cells. Unidades: FMRP, ICB
Assunto: IMUNOLOGIA
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LEITE, Jefferson Antonio et al. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, v. 9, n. 4, p. 21 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9040959. Acesso em: 08 jul. 2024.
APA
Leite, J. A., Pessenda, G., Gomes, I. C. G., Santana, A. K. M. de, Pereira, C. A., Costa, F. R. C., et al. (2020). The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, 9( 4), 21 . doi:10.3390/cells9040959
NLM
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 jul. 08 ] Available from: https://doi.org/10.3390/cells9040959
Vancouver
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 jul. 08 ] Available from: https://doi.org/10.3390/cells9040959
Artigo de periodico
Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole (2020)
Silva, Maria Cláudia da; Carbone, Débora Cardozo Bonfim; Diniz, Pedro Fernando; Fernandes, Fabrício Freitas; Fuzo, Carlos Alessandro; Trindade, Carolina Santos Pereira Cardoso; Chang, Marilene Rodrigues; Silva, João Santana da
Source: Mycopathologia. Unidades: FMRP, FCFRP
Subjects: CANDIDA, EXPRESSÃO GÊNICA, RESISTÊNCIA MICROBIANA ÀS DROGAS, CANDIDOSE, ANTIFÚNGICOS
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SILVA, Maria Cláudia da et al. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole. Mycopathologia, v. 185, n. 4, p. 675-684, 2020Tradução . . Disponível em: https://doi.org/10.1007/s11046-020-00465-6. Acesso em: 08 jul. 2024.
APA
Silva, M. C. da, Carbone, D. C. B., Diniz, P. F., Fernandes, F. F., Fuzo, C. A., Trindade, C. S. P. C., et al. (2020). Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole. Mycopathologia, 185( 4), 675-684. doi:10.1007/s11046-020-00465-6
NLM
Silva MC da, Carbone DCB, Diniz PF, Fernandes FF, Fuzo CA, Trindade CSPC, Chang MR, Silva JS da. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole [Internet]. Mycopathologia. 2020 ; 185( 4): 675-684.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1007/s11046-020-00465-6
Vancouver
Silva MC da, Carbone DCB, Diniz PF, Fernandes FF, Fuzo CA, Trindade CSPC, Chang MR, Silva JS da. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole [Internet]. Mycopathologia. 2020 ; 185( 4): 675-684.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1007/s11046-020-00465-6
Artigo de periodico
Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment (2020)
Albuquerque, Sérgio de ; Cianni, Lorenzo ; Vita, Daniela de ; Lopes, Carla Duque ; Gomes, Ana S. M.; Gomes, Paula; Laughton, Charles; Leitão, Andrei ; Montanari, Carlos Alberto ; Montanari, Raphael ; Ribeiro, Jean Francisco Rosa ; Silva, João Santana da ; Teixeira, Cátia
Source: Chemical Biology & Drug Design. Unidades: FCFRP, IQSC, FMRP, EESC, ICMC
Subjects: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI
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ALBUQUERQUE, Sérgio de et al. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment. Chemical Biology & Drug Design, v. 96, p. 948–960, 2020Tradução . . Disponível em: https://doi.org/10.1111/cbdd.13663. Acesso em: 08 jul. 2024.
APA
Albuquerque, S. de, Cianni, L., Vita, D. de, Lopes, C. D., Gomes, A. S. M., Gomes, P., et al. (2020). Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment. Chemical Biology & Drug Design, 96, 948–960. doi:10.1111/cbdd.13663
NLM
Albuquerque S de, Cianni L, Vita D de, Lopes CD, Gomes ASM, Gomes P, Laughton C, Leitão A, Montanari CA, Montanari R, Ribeiro JFR, Silva JS da, Teixeira C. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment [Internet]. Chemical Biology & Drug Design. 2020 ; 96 948–960.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/cbdd.13663
Vancouver
Albuquerque S de, Cianni L, Vita D de, Lopes CD, Gomes ASM, Gomes P, Laughton C, Leitão A, Montanari CA, Montanari R, Ribeiro JFR, Silva JS da, Teixeira C. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment [Internet]. Chemical Biology & Drug Design. 2020 ; 96 948–960.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/cbdd.13663
Artigo de periodico
Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication (2020)
Mota, Caroline M.; Lima Júnior, Djalma de Souza; França, Flávia Batista Ferreira; Torres, Jhoan David Aguillón; Barros, Patrício da Silva Cardoso; Santiago, Fernanda Maria; Silva, João Santana da ; Mineo, José Roberto; Zamboni, Dario Simões; Mineo, Tiago Wilson Patriarca
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: INTERLEUCINA 1, NEOSPORA CANINUM, ESPÉCIES REATIVAS DE OXIGÊNIO
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MOTA, Caroline M. et al. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2020.00243. Acesso em: 08 jul. 2024.
APA
Mota, C. M., Lima Júnior, D. de S., França, F. B. F., Torres, J. D. A., Barros, P. da S. C., Santiago, F. M., et al. (2020). Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, 10. doi:10.3389/fcimb.2020.00243
NLM
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fcimb.2020.00243
Vancouver
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fcimb.2020.00243
Artigo de periodico
Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development (2020)
Sesti-Costa, Renata; Cervantes-Barragan, Luisa; Swiecki, Melissa K.; Fachi, José Luís; Cella, Marina; Gilfillan, Susan; Silva, João Santana; Colonna, Marco
Source: The Journal of Immunology. Unidade: FMRP
Subjects: ANIMAIS, INTERFERONS, RATOS, IMUNOLOGIA
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SESTI-COSTA, Renata et al. Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development. The Journal of Immunology, v. 204, n. 8, p. 2257-2268, 2020Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1900604. Acesso em: 08 jul. 2024.
APA
Sesti-Costa, R., Cervantes-Barragan, L., Swiecki, M. K., Fachi, J. L., Cella, M., Gilfillan, S., et al. (2020). Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development. The Journal of Immunology, 204( 8), 2257-2268. doi:10.4049/jimmunol.1900604
NLM
Sesti-Costa R, Cervantes-Barragan L, Swiecki MK, Fachi JL, Cella M, Gilfillan S, Silva JS, Colonna M. Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development [Internet]. The Journal of Immunology. 2020 ; 204( 8): 2257-2268.[citado 2024 jul. 08 ] Available from: https://doi.org/10.4049/jimmunol.1900604
Vancouver
Sesti-Costa R, Cervantes-Barragan L, Swiecki MK, Fachi JL, Cella M, Gilfillan S, Silva JS, Colonna M. Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development [Internet]. The Journal of Immunology. 2020 ; 204( 8): 2257-2268.[citado 2024 jul. 08 ] Available from: https://doi.org/10.4049/jimmunol.1900604
Artigo de periodico
TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection (2020)
Sacramento, Laís Amorim ; Benevides, Luciana; Maruyama, Sandra Regina Costa; Tavares, Lucas Alves ; Fukutani, Kiyoshi Ferreira; Françozo, Marcela; Sparwasser, Tim; Cunha, Fernando de Queiroz ; Almeida, Roque Pacheco de; Silva, João Santana da ; Carregaro, Vanessa
Source: PLOS Pathogens. Unidade: FMRP
Subjects: INFLAMAÇÃO, IMUNOLOGIA, INTERFERONS, LEISHMANIA INFANTUM, LEISHMANIOSE VISCERAL
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SACRAMENTO, Laís Amorim et al. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection. PLOS Pathogens, v. 16, n. 3, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1008435. Acesso em: 08 jul. 2024.
APA
Sacramento, L. A., Benevides, L., Maruyama, S. R. C., Tavares, L. A., Fukutani, K. F., Françozo, M., et al. (2020). TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection. PLOS Pathogens, 16( 3). doi:10.1371/journal.ppat.1008435
NLM
Sacramento LA, Benevides L, Maruyama SRC, Tavares LA, Fukutani KF, Françozo M, Sparwasser T, Cunha F de Q, Almeida RP de, Silva JS da, Carregaro V. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection [Internet]. PLOS Pathogens. 2020 ; 16( 3):[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.ppat.1008435
Vancouver
Sacramento LA, Benevides L, Maruyama SRC, Tavares LA, Fukutani KF, Françozo M, Sparwasser T, Cunha F de Q, Almeida RP de, Silva JS da, Carregaro V. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection [Internet]. PLOS Pathogens. 2020 ; 16( 3):[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.ppat.1008435
Artigo de periodico
Arginase and its mechanisms in Leishmania persistence (2020)
Pessenda, Gabriela; Silva, João Santana da
Source: Parasite Immunology. Unidade: FMRP
Subjects: LEISHMANIA, ÓXIDO NÍTRICO, MACRÓFAGOS, SISTEMA IMUNE, ARGININA
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PESSENDA, Gabriela e SILVA, João Santana da. Arginase and its mechanisms in Leishmania persistence. Parasite Immunology, v. 42, n. 7, p. 1-12, 2020Tradução . . Disponível em: https://doi.org/10.1111/pim.12722. Acesso em: 08 jul. 2024.
APA
Pessenda, G., & Silva, J. S. da. (2020). Arginase and its mechanisms in Leishmania persistence. Parasite Immunology, 42( 7), 1-12. doi:10.1111/pim.12722
NLM
Pessenda G, Silva JS da. Arginase and its mechanisms in Leishmania persistence [Internet]. Parasite Immunology. 2020 ; 42( 7): 1-12.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/pim.12722
Vancouver
Pessenda G, Silva JS da. Arginase and its mechanisms in Leishmania persistence [Internet]. Parasite Immunology. 2020 ; 42( 7): 1-12.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/pim.12722
Artigo de periodico
NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? (2020)
Oliveira, Jefferson Elias ; Leite, Jefferson Antônio; Pereira, Ítalo Sousa; Guimarães, Jhefferson Barbosa; Manso, Gabriel Martins da Costa; Silva, João Santana da ; Passaglia, Rita de Cassia Aleixo Tostes ; Sartori, Daniela Carlos
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: ENTEROPATIAS INFLAMATÓRIAS, DIABETES MELLITUS, HOMEOSTASE, INTESTINOS, BACTÉRIAS, RECEPTORES IMUNOLÓGICOS
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OLIVEIRA, Jefferson Elias et al. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers?. Frontiers in Immunology, v. 11, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01810. Acesso em: 08 jul. 2024.
APA
Oliveira, J. E., Leite, J. A., Pereira, Í. S., Guimarães, J. B., Manso, G. M. da C., Silva, J. S. da, et al. (2020). NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? Frontiers in Immunology, 11, 1-9. doi:10.3389/fimmu.2020.01810
NLM
Oliveira JE, Leite JA, Pereira ÍS, Guimarães JB, Manso GM da C, Silva JS da, Passaglia R de CAT, Sartori DC. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? [Internet]. Frontiers in Immunology. 2020 ; 11 1-9.[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fimmu.2020.01810
Vancouver
Oliveira JE, Leite JA, Pereira ÍS, Guimarães JB, Manso GM da C, Silva JS da, Passaglia R de CAT, Sartori DC. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? [Internet]. Frontiers in Immunology. 2020 ; 11 1-9.[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fimmu.2020.01810
Artigo de periodico
Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes (2020)
Pereira, Camila André; Carlos, Daniela; Ferreira, Nathanne dos Santos; Silva, Josiane F. da; Zanotto, Camila Ziliotto; Zamboni, Dario Simões ; Garcia, Valéria Duarte; Ventura, Dora Selma Fix ; Silva, João Santana da ; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidades: FMRP, IP
Subjects: INFLAMAÇÃO, DNA MITOCONDRIAL, ESPÉCIES REATIVAS DE OXIGÊNIO
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PEREIRA, Camila André et al. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes. Frontiers in Physiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.01557. Acesso em: 08 jul. 2024.
APA
Pereira, C. A., Carlos, D., Ferreira, N. dos S., Silva, J. F. da, Zanotto, C. Z., Zamboni, D. S., et al. (2020). Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes. Frontiers in Physiology, 10. doi:10.3389/fphys.2019.01557
NLM
Pereira CA, Carlos D, Ferreira N dos S, Silva JF da, Zanotto CZ, Zamboni DS, Garcia VD, Ventura DSF, Silva JS da, Tostes R de CA. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fphys.2019.01557
Vancouver
Pereira CA, Carlos D, Ferreira N dos S, Silva JF da, Zanotto CZ, Zamboni DS, Garcia VD, Ventura DSF, Silva JS da, Tostes R de CA. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fphys.2019.01557
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
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SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 08 jul. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Artigo de periodico
Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain (2020)
Oliveira, Maykon Tavares de; Sulleiro, Elena; Gimenez, Aroa Silgado; Lana, Marta de; Zingales, Bianca ; Silva, João Santana da ; Marin Neto, José Antonio; MolinaI, Israel
Source: PLOS Neglected Tropical Diseases. Unidades: IQ, FMRP
Subjects: TRYPANOSOMA CRUZI, IMIGRANTES, LATINO-AMERICANOS, REAÇÃO EM CADEIA POR POLIMERASE, DOENÇAS PARASITÁRIAS
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OLIVEIRA, Maykon Tavares de et al. Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain. PLOS Neglected Tropical Diseases, v. 14, n. 6, p. 1-14 art. e0008311, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0008311. Acesso em: 08 jul. 2024.
APA
Oliveira, M. T. de, Sulleiro, E., Gimenez, A. S., Lana, M. de, Zingales, B., Silva, J. S. da, et al. (2020). Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain. PLOS Neglected Tropical Diseases, 14( 6), 1-14 art. e0008311. doi:10.1371/journal.pntd.0008311
NLM
Oliveira MT de, Sulleiro E, Gimenez AS, Lana M de, Zingales B, Silva JS da, Marin Neto JA, MolinaI I. Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 6): 1-14 art. e0008311.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.pntd.0008311
Vancouver
Oliveira MT de, Sulleiro E, Gimenez AS, Lana M de, Zingales B, Silva JS da, Marin Neto JA, MolinaI I. Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 6): 1-14 art. e0008311.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.pntd.0008311
Artigo de periodico
Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease (2019)
Lopes, Carla Duque ; Possato, Bruna ; Gaspari, Ana Paula Segantin ; Oliveira, Ronaldo Junio ; Abram, Ulrich ; Almeida, José P. A; Rocho, Fernanda dos Reis ; Leitão, Andrei ; Montanari, Carlos Alberto ; Maia, Pedro Ivo da Silva ; Silva, João Santana da ; Albuquerque, Sérgio de ; Carneiro, Zumira Aparecida
Source: ACS Infectious Diseases. Unidades: IQSC, FMRP, FCFRP
Subjects: BIOQUÍMICA, DOENÇA DE CHAGAS
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LOPES, Carla Duque et al. Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease. ACS Infectious Diseases, v. 5, n. 10, p. 1698-1707, 2019Tradução . . Disponível em: https://doi.org/10.1021/acsinfecdis.8b00284. Acesso em: 08 jul. 2024.
APA
Lopes, C. D., Possato, B., Gaspari, A. P. S., Oliveira, R. J., Abram, U., Almeida, J. P. A., et al. (2019). Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease. ACS Infectious Diseases, 5( 10), 1698-1707. doi:10.1021/acsinfecdis.8b00284
NLM
Lopes CD, Possato B, Gaspari APS, Oliveira RJ, Abram U, Almeida JPA, Rocho F dos R, Leitão A, Montanari CA, Maia PI da S, Silva JS da, Albuquerque S de, Carneiro ZA. Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease [Internet]. ACS Infectious Diseases. 2019 ; 5( 10): 1698-1707.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1021/acsinfecdis.8b00284
Vancouver
Lopes CD, Possato B, Gaspari APS, Oliveira RJ, Abram U, Almeida JPA, Rocho F dos R, Leitão A, Montanari CA, Maia PI da S, Silva JS da, Albuquerque S de, Carneiro ZA. Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease [Internet]. ACS Infectious Diseases. 2019 ; 5( 10): 1698-1707.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1021/acsinfecdis.8b00284
Artigo de periodico
Programmed Death 1 (PD-1) and PD-1 Ligand (PD-L1) expression in chronic apical periodontitis (2019)
Delgado, Ronan Jacques Rezende; Pinheiro,Claudia Ramos; Gasparoto, Thaís Helena; Sipert, Carla Renata; Moraes, Ivaldo Gomes de; Garcia, Roberto Brandão; Bramante, Clóvis Monteiro; Bernardineli, Norberti; Nishiyama, Celso Kenji; Silva, João Santana da; Torres, Sérgio Aparecido; Garlet, Gustavo Pompermaier; Campanelli, Ana Paula
Source: European endodontic journal. Unidades: FOB, HRAC, FMRP
Subjects: PERIODONTITE PERIAPICAL, PERIODONTITE CRÔNICA, CITOCINAS, LINFÓCITOS T, RECEPTORES IMUNOLÓGICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DELGADO, Ronan Jacques Rezende et al. Programmed Death 1 (PD-1) and PD-1 Ligand (PD-L1) expression in chronic apical periodontitis. European endodontic journal, v. 4, n. 1, p. 3-8, 2019Tradução . . Disponível em: https://doi.org/10.14744/eej.2018.46330. Acesso em: 08 jul. 2024.
APA
Delgado, R. J. R., Pinheiro, C. R., Gasparoto, T. H., Sipert, C. R., Moraes, I. G. de, Garcia, R. B., et al. (2019). Programmed Death 1 (PD-1) and PD-1 Ligand (PD-L1) expression in chronic apical periodontitis. European endodontic journal, 4( 1), 3-8. doi:10.14744/eej.2018.46330
NLM
Delgado RJR, Pinheiro CR, Gasparoto TH, Sipert CR, Moraes IG de, Garcia RB, Bramante CM, Bernardineli N, Nishiyama CK, Silva JS da, Torres SA, Garlet GP, Campanelli AP. Programmed Death 1 (PD-1) and PD-1 Ligand (PD-L1) expression in chronic apical periodontitis [Internet]. European endodontic journal. 2019 ; 4( 1): 3-8.[citado 2024 jul. 08 ] Available from: https://doi.org/10.14744/eej.2018.46330
Vancouver
Delgado RJR, Pinheiro CR, Gasparoto TH, Sipert CR, Moraes IG de, Garcia RB, Bramante CM, Bernardineli N, Nishiyama CK, Silva JS da, Torres SA, Garlet GP, Campanelli AP. Programmed Death 1 (PD-1) and PD-1 Ligand (PD-L1) expression in chronic apical periodontitis [Internet]. European endodontic journal. 2019 ; 4( 1): 3-8.[citado 2024 jul. 08 ] Available from: https://doi.org/10.14744/eej.2018.46330
Artigo de periodico
The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy (2019)
Cardoso, Cristina Ribeiro de Barros ; Provinciatto, Pauline Rossetti; Godoi, Dannielle Fernandes; Fonseca, Monique Thaís Costa ; Ferreira, Beatriz Rossetti ; Teixeira, Gerlinde Agate Platais Brasil ; Cunha, Fernando de Queiroz ; Pinzan, Camila Figueiredo; Silva, João Santana da
Source: Allergologia et Immunopathologia. Unidades: FCFRP, EERP, FMRP
Subjects: HIPERSENSIBILIDADE ALIMENTAR, INFLAMAÇÃO, INTESTINOS, GASTROENTEROPATIAS
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ABNT
CARDOSO, Cristina Ribeiro de Barros et al. The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy. Allergologia et Immunopathologia, v. 47, n. 6, p. 535-543, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.aller.2019.02.006. Acesso em: 08 jul. 2024.
APA
Cardoso, C. R. de B., Provinciatto, P. R., Godoi, D. F., Fonseca, M. T. C., Ferreira, B. R., Teixeira, G. A. P. B., et al. (2019). The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy. Allergologia et Immunopathologia, 47( 6), 535-543. doi:10.1016/j.aller.2019.02.006
NLM
Cardoso CR de B, Provinciatto PR, Godoi DF, Fonseca MTC, Ferreira BR, Teixeira GAPB, Cunha F de Q, Pinzan CF, Silva JS da. The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy [Internet]. Allergologia et Immunopathologia. 2019 ; 47( 6): 535-543.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.aller.2019.02.006
Vancouver
Cardoso CR de B, Provinciatto PR, Godoi DF, Fonseca MTC, Ferreira BR, Teixeira GAPB, Cunha F de Q, Pinzan CF, Silva JS da. The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy [Internet]. Allergologia et Immunopathologia. 2019 ; 47( 6): 535-543.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.aller.2019.02.006
Artigo de periodico
Non-Leishmania parasite in fatal visceral Leishmaniasis-like disease, Brazil (2019)
Maruyama, Sandra R.; Santana, Alynne Karen Mendonça de; Takamiya, Nayore T.; Takahashi, Talita Y.; Rogerio, Luana A.; Oliveira, Caio A. B.; Milanezi, Cristiane Maria; Trombela, Viviane A.; Cruz, Angela Kaysel ; Jesus, Amélia R.; Barreto, Aline S.; Silva, Angela M. da; Almeida, Roque Pacheco de ; Ribeiro, José M. ; Silva, João Santana da
Source: Emerging Infectious Diseases. Unidade: FMRP
Subjects: DOENÇAS PARASITÁRIAS, LEISHMANIA
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ABNT
MARUYAMA, Sandra R. et al. Non-Leishmania parasite in fatal visceral Leishmaniasis-like disease, Brazil. Emerging Infectious Diseases, v. 25, n. 11, p. 2088-2092, 2019Tradução . . Disponível em: https://doi.org/10.3201/eid2511.181548. Acesso em: 08 jul. 2024.
APA
Maruyama, S. R., Santana, A. K. M. de, Takamiya, N. T., Takahashi, T. Y., Rogerio, L. A., Oliveira, C. A. B., et al. (2019). Non-Leishmania parasite in fatal visceral Leishmaniasis-like disease, Brazil. Emerging Infectious Diseases, 25( 11), 2088-2092. doi:10.3201/eid2511.181548
NLM
Maruyama SR, Santana AKM de, Takamiya NT, Takahashi TY, Rogerio LA, Oliveira CAB, Milanezi CM, Trombela VA, Cruz AK, Jesus AR, Barreto AS, Silva AM da, Almeida RP de, Ribeiro JM, Silva JS da. Non-Leishmania parasite in fatal visceral Leishmaniasis-like disease, Brazil [Internet]. Emerging Infectious Diseases. 2019 ; 25( 11): 2088-2092.[citado 2024 jul. 08 ] Available from: https://doi.org/10.3201/eid2511.181548
Vancouver
Maruyama SR, Santana AKM de, Takamiya NT, Takahashi TY, Rogerio LA, Oliveira CAB, Milanezi CM, Trombela VA, Cruz AK, Jesus AR, Barreto AS, Silva AM da, Almeida RP de, Ribeiro JM, Silva JS da. Non-Leishmania parasite in fatal visceral Leishmaniasis-like disease, Brazil [Internet]. Emerging Infectious Diseases. 2019 ; 25( 11): 2088-2092.[citado 2024 jul. 08 ] Available from: https://doi.org/10.3201/eid2511.181548
Artigo de periodico
Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis (2019)
Ketelut-Carneiro, Natália ; Souza, Camila de Oliveira Silva e ; Benevides, Luciana ; Gardinassi, Luiz Gustavo Araujo ; Silva, Maria Claúdia; Tavares, Lucas Alves ; Zamboni, Dario Simões ; Silva, João Santana da
Source: PLoS Pathogens. Unidades: FMRP, FCFRP
Subjects: IMUNIDADE, INTERLEUCINA 1, MACRÓFAGOS, PARACOCCIDIOIDOMICOSE
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ABNT
KETELUT-CARNEIRO, Natália et al. Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis. PLoS Pathogens, v. 15, n. 8, p. e1007990, 2019Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1007990. Acesso em: 08 jul. 2024.
APA
Ketelut-Carneiro, N., Souza, C. de O. S. e, Benevides, L., Gardinassi, L. G. A., Silva, M. C., Tavares, L. A., et al. (2019). Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis. PLoS Pathogens, 15( 8), e1007990. doi:10.1371/journal.ppat.1007990
NLM
Ketelut-Carneiro N, Souza C de OS e, Benevides L, Gardinassi LGA, Silva MC, Tavares LA, Zamboni DS, Silva JS da. Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis [Internet]. PLoS Pathogens. 2019 ; 15( 8): e1007990.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.ppat.1007990
Vancouver
Ketelut-Carneiro N, Souza C de OS e, Benevides L, Gardinassi LGA, Silva MC, Tavares LA, Zamboni DS, Silva JS da. Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis [Internet]. PLoS Pathogens. 2019 ; 15( 8): e1007990.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.ppat.1007990
Trabalho de evento-resumo
Deficiência do receptor de interleucina 1 causa agravamento da obesidade associado a redução do perfil Th17 intestinal em modelo experimental (2019)
Pucci, Taís Andrade; Lago, Silvia C.; Leite, Jefferson Antonio; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Resumos. Conference titles: Simpósio Internacional de Iniciação Científica e Tecnológica da USP (SIICUSP). Unidade: FMRP
Subjects: OBESIDADE, DIETOTERAPIA, ADIPOSIDADE
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ABNT
PUCCI, Taís Andrade et al. Deficiência do receptor de interleucina 1 causa agravamento da obesidade associado a redução do perfil Th17 intestinal em modelo experimental. 2019, Anais.. Ribeirão Preto: FMRP-USP, 2019. Disponível em: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210. Acesso em: 08 jul. 2024.
APA
Pucci, T. A., Lago, S. C., Leite, J. A., Silva, J. S. da, & Sartori, D. C. (2019). Deficiência do receptor de interleucina 1 causa agravamento da obesidade associado a redução do perfil Th17 intestinal em modelo experimental. In Resumos. Ribeirão Preto: FMRP-USP. Recuperado de https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
NLM
Pucci TA, Lago SC, Leite JA, Silva JS da, Sartori DC. Deficiência do receptor de interleucina 1 causa agravamento da obesidade associado a redução do perfil Th17 intestinal em modelo experimental [Internet]. Resumos. 2019 ;[citado 2024 jul. 08 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Vancouver
Pucci TA, Lago SC, Leite JA, Silva JS da, Sartori DC. Deficiência do receptor de interleucina 1 causa agravamento da obesidade associado a redução do perfil Th17 intestinal em modelo experimental [Internet]. Resumos. 2019 ;[citado 2024 jul. 08 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Artigo de periodico
A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis (2019)
Freitas Alves, V. B.; Sousa, B. Coutinho de; Fonseca, M. Thaís Costa; Ogata, H.; Caliari-Oliveira, Carolina; Yaochite, Juliana Navarro Ueda; Rodrigues-Junior, Valnês S. ; Chica, Javier Emilio Lazo; Silva, João Santana da ; Farias, Kelen Cristina Ribeiro Malmegrim de ; Pernomian, L.; Cardoso, C. Ribeiro
Source: Clinical and Experimental Immunology : The Journal of Translational Immunology. Unidades: FMRP, FCFRP
Subjects: ENTEROPATIAS INFLAMATÓRIAS, CÉLULAS ESTROMAIS, COLITE
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ABNT
FREITAS ALVES, V. B. et al. A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis. Clinical and Experimental Immunology : The Journal of Translational Immunology, v. 196, n. 2, p. 139-154, 2019Tradução . . Disponível em: https://doi.org/10.1111/cei.13262. Acesso em: 08 jul. 2024.
APA
Freitas Alves, V. B., Sousa, B. C. de, Fonseca, M. T. C., Ogata, H., Caliari-Oliveira, C., Yaochite, J. N. U., et al. (2019). A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis. Clinical and Experimental Immunology : The Journal of Translational Immunology, 196( 2), 139-154. doi:10.1111/cei.13262
NLM
Freitas Alves VB, Sousa BC de, Fonseca MTC, Ogata H, Caliari-Oliveira C, Yaochite JNU, Rodrigues-Junior VS, Chica JEL, Silva JS da, Farias KCRM de, Pernomian L, Cardoso CR. A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis [Internet]. Clinical and Experimental Immunology : The Journal of Translational Immunology. 2019 ; 196( 2): 139-154.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/cei.13262
Vancouver
Freitas Alves VB, Sousa BC de, Fonseca MTC, Ogata H, Caliari-Oliveira C, Yaochite JNU, Rodrigues-Junior VS, Chica JEL, Silva JS da, Farias KCRM de, Pernomian L, Cardoso CR. A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis [Internet]. Clinical and Experimental Immunology : The Journal of Translational Immunology. 2019 ; 196( 2): 139-154.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/cei.13262
Artigo de periodico
B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation (2019)
Benevides, Luciana ; Costa, Renata Sesti ; Tavares, Lucas Alves; Russo, Momtchilo ; Martins, Gislâine A.; Silva, Luis Lamberti Pinto da ; Arruda, Luisa Karla de Paula ; Cunha, Fernando de Queiroz ; Carregaro, Vanessa ; Silva, João Santana da
Source: Journal of Allergy and Clinical Immunology. Unidades: ICB, FMRP
Subjects: ALERGIA E IMUNOLOGIA, DIFERENCIAÇÃO CELULAR, LINFÓCITOS B, INFLAMAÇÃO, CITOMETRIA DE FLUXO, ELISA, REAÇÃO EM CADEIA POR POLIMERASE, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BENEVIDES, Luciana et al. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation. Journal of Allergy and Clinical Immunology, v. 143, n. 3, p. 1119-1130, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.jaci.2018.06.046. Acesso em: 08 jul. 2024.
APA
Benevides, L., Costa, R. S., Tavares, L. A., Russo, M., Martins, G. A., Silva, L. L. P. da, et al. (2019). B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation. Journal of Allergy and Clinical Immunology, 143( 3), 1119-1130. doi:10.1016/j.jaci.2018.06.046
NLM
Benevides L, Costa RS, Tavares LA, Russo M, Martins GA, Silva LLP da, Arruda LK de P, Cunha F de Q, Carregaro V, Silva JS da. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation [Internet]. Journal of Allergy and Clinical Immunology. 2019 ; 143( 3): 1119-1130.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.jaci.2018.06.046
Vancouver
Benevides L, Costa RS, Tavares LA, Russo M, Martins GA, Silva LLP da, Arruda LK de P, Cunha F de Q, Carregaro V, Silva JS da. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation [Internet]. Journal of Allergy and Clinical Immunology. 2019 ; 143( 3): 1119-1130.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.jaci.2018.06.046
Artigo de periodico
NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection (2019)
Benevides, Luciana ; Saltarelli, Verônica Marcela; Pioto, Franciele ; Sacramento, Laís Amorim ; Dias, Murilo Solano; Rodríguez, Gretel Rodríguez ; Viola, João Paulo de Biaso ; Carregaro, Vanessa ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ESALQ
Subjects: INFLAMAÇÃO, LINFÓCITOS T, MONÓCITOS, TOXOPLASMA GONDII, TOXOPLASMOSE
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ABNT
BENEVIDES, Luciana et al. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection. Frontiers in Immunology, v. 10, p. [10] , 2019Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.02105. Acesso em: 08 jul. 2024.
APA
Benevides, L., Saltarelli, V. M., Pioto, F., Sacramento, L. A., Dias, M. S., Rodríguez, G. R., et al. (2019). NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection. Frontiers in Immunology, 10, [10] . doi:10.3389/fimmu.2019.02105
NLM
Benevides L, Saltarelli VM, Pioto F, Sacramento LA, Dias MS, Rodríguez GR, Viola JP de B, Carregaro V, Silva JS da. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection [Internet]. Frontiers in Immunology. 2019 ; 10 [10] .[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fimmu.2019.02105
Vancouver
Benevides L, Saltarelli VM, Pioto F, Sacramento LA, Dias MS, Rodríguez GR, Viola JP de B, Carregaro V, Silva JS da. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection [Internet]. Frontiers in Immunology. 2019 ; 10 [10] .[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fimmu.2019.02105

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