Using animal models for the studies of schizophrenia and depression: the value of translational models for treatment and prevention (2022)
- Authors:
- Autor USP: GOMES, FELIPE VILLELA - FMRP
- Unidade: FMRP
- DOI: 10.3389/fnbeh.2022.935320
- Subjects: DEPRESSÃO; NEUROTRANSMISSORES; PLASTICIDADE NEURONAL; CÓRTEX PRÉ-FRONTAL; ESQUIZOFRENIA
- Keywords: Depression; Dopamine; Bippocampus; Plasticity; Prefrontal cortex; Schizophrenia
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Behavioral Neuroscience
- ISSN: 1662-5153
- Volume/Número/Paginação/Ano: v. 16, art. 935320, p. 1-15, 2022
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
ULIANA, Daniela L. et al. Using animal models for the studies of schizophrenia and depression: the value of translational models for treatment and prevention. Frontiers in Behavioral Neuroscience, v. 16, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.3389/fnbeh.2022.935320. Acesso em: 02 out. 2024. -
APA
Uliana, D. L., Zhu, X., Gomes, F. V., & Grace, A. A. (2022). Using animal models for the studies of schizophrenia and depression: the value of translational models for treatment and prevention. Frontiers in Behavioral Neuroscience, 16, 1-15. doi:10.3389/fnbeh.2022.935320 -
NLM
Uliana DL, Zhu X, Gomes FV, Grace AA. Using animal models for the studies of schizophrenia and depression: the value of translational models for treatment and prevention [Internet]. Frontiers in Behavioral Neuroscience. 2022 ; 16 1-15.[citado 2024 out. 02 ] Available from: https://doi.org/10.3389/fnbeh.2022.935320 -
Vancouver
Uliana DL, Zhu X, Gomes FV, Grace AA. Using animal models for the studies of schizophrenia and depression: the value of translational models for treatment and prevention [Internet]. Frontiers in Behavioral Neuroscience. 2022 ; 16 1-15.[citado 2024 out. 02 ] Available from: https://doi.org/10.3389/fnbeh.2022.935320 - Tratamento repetido com canabidiol atenua alterações comportamentais e moleculares em um modelo de esquizofrenia baseado no antagonismo dos receptores NMDA
- Stress during critical periods of development and risk for schizophrenia
- Female rats are resistant to the long-lasting neurobehavioral changes induced by adolescent stress exposure
- Cortical dopamine dysregulation in schizophrenia and its link to stress. [Carta]
- Altered brain cannabinoid 1 receptor mRNA expression across postnatal development in the MAM model of schizophrenia
- Altered ventral striatum–hippocampus connectivity during reward processing as an endophenotype for psychosis
- The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulnerability
- Prelimbic medial prefrontal cortex disruption during adolescence increases susceptibility to helpless behavior in adult rats
- GABAA and NMDA receptor density alterations and their behavioral correlates in the gestational methylazoxymethanol acetate model for schizophrenia
- Nucleus reuniens inactivation reverses stress-induced hypodopaminergic state and altered hippocampal-accumbens synaptic plasticity
Informações sobre o DOI: 10.3389/fnbeh.2022.935320 (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
 | 003154608.pdf | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
