Using intermittent fasting as a non-pharmacological strategy to alleviate obesity-induced hypothalamic molecular pathway disruption (2022)
- Authors:
- USP affiliated authors: FREITAS, ELLEN CRISTINI DE - EEFERP ; SILVA, ADELINO SANCHEZ RAMOS DA - EEFERP ; OLIVEIRA, LUCIANA DA COSTA - FMRP ; MORAIS, GUSTAVO PAROSCHI - FMRP
- Unidades: EEFERP; FMRP
- DOI: 10.3389/fnut.2022.858320
- Subjects: HIPOTÁLAMO; INFLAMAÇÃO; JEJUM; OBESIDADE
- Keywords: Intermittent fasting (IF); Hypothalamus; Obesity; Hypothalamic inflammation; Non-pharmaceutical intervention
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Nutrition
- ISSN: 2296-861X
- Volume/Número/Paginação/Ano: v. 9, art. 858320, p. 1-15, 2022
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
OLIVEIRA, Luciana da Costa et al. Using intermittent fasting as a non-pharmacological strategy to alleviate obesity-induced hypothalamic molecular pathway disruption. Frontiers in Nutrition, v. 9, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.3389/fnut.2022.858320. Acesso em: 16 jul. 2025. -
APA
Oliveira, L. da C., Morais, G. P., Ropelle, E. R., Moura, L. P. de, Cintra, D. E., Pauli, J. R., et al. (2022). Using intermittent fasting as a non-pharmacological strategy to alleviate obesity-induced hypothalamic molecular pathway disruption. Frontiers in Nutrition, 9, 1-15. doi:10.3389/fnut.2022.858320 -
NLM
Oliveira L da C, Morais GP, Ropelle ER, Moura LP de, Cintra DE, Pauli JR, Freitas EC de, Rorato R, Silva ASR da. Using intermittent fasting as a non-pharmacological strategy to alleviate obesity-induced hypothalamic molecular pathway disruption [Internet]. Frontiers in Nutrition. 2022 ; 9 1-15.[citado 2025 jul. 16 ] Available from: https://doi.org/10.3389/fnut.2022.858320 -
Vancouver
Oliveira L da C, Morais GP, Ropelle ER, Moura LP de, Cintra DE, Pauli JR, Freitas EC de, Rorato R, Silva ASR da. Using intermittent fasting as a non-pharmacological strategy to alleviate obesity-induced hypothalamic molecular pathway disruption [Internet]. Frontiers in Nutrition. 2022 ; 9 1-15.[citado 2025 jul. 16 ] Available from: https://doi.org/10.3389/fnut.2022.858320 - Excessive training impairs the insulin signal transduction in mice skeletal muscles
- Eccentric exercise leads to performance decrease and insulin signaling impairment
- Excessive eccentric exercise-induced overtraining model leads to endoplasmic reticulum stress in mice skeletal muscles
- Uphill running excessive training increases gastrocnemius glycogen content in C57BL/6 mice
- Excessive training induces molecular signs of pathologic cardiac hypertrophy
- Excessive downhill training leads to early onset of knee osteoarthritis
- Excessive treadmill training enhances the insulin signaling pathway and glycogen deposition in mice hearts
- Excess of eccentric exercise, mechanical load on the knee joint and osteoarthritis in C57BL/6 mice
- Taurine supplementation can increase lipolysis and affect the contribution of energy systems during front crawl maximal effort
- Effects of the combination of intermittent fasting and physical exercise on autophagic and leptin pathway in the hypothalamus of obese mice
Informações sobre o DOI: 10.3389/fnut.2022.858320 (Fonte: oaDOI API)
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