Autophagy in hepatic steatosis: a structured review (2021)
- Authors:
- USP affiliated authors: KOWALTOWSKI, ALÍCIA JULIANA - IQ ; RAMOS, VITOR DE MIRANDA - IQ ; KAKIMOTO, PÂMELA AIAKO HYPÓLITO BRITO - IQ
- Unidade: IQ
- DOI: 10.3389/fcell.2021.657389
- Subjects: HEPATOPATIAS; METABOLISMO; LIPÍDEOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Cell and Developmental Biology
- ISSN: 2296-634X
- Volume/Número/Paginação/Ano: v. 9, p. 1-21 art. 657389, 2021
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
RAMOS, Vitor de Miranda e KOWALTOWSKI, Alicia Juliana e KAKIMOTO, Pâmela Aiako Hypólito Brito. Autophagy in hepatic steatosis: a structured review. Frontiers in Cell and Developmental Biology, v. 9, p. 1-21 art. 657389, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcell.2021.657389. Acesso em: 31 mar. 2026. -
APA
Ramos, V. de M., Kowaltowski, A. J., & Kakimoto, P. A. H. B. (2021). Autophagy in hepatic steatosis: a structured review. Frontiers in Cell and Developmental Biology, 9, 1-21 art. 657389. doi:10.3389/fcell.2021.657389 -
NLM
Ramos V de M, Kowaltowski AJ, Kakimoto PAHB. Autophagy in hepatic steatosis: a structured review [Internet]. Frontiers in Cell and Developmental Biology. 2021 ; 9 1-21 art. 657389.[citado 2026 mar. 31 ] Available from: https://doi.org/10.3389/fcell.2021.657389 -
Vancouver
Ramos V de M, Kowaltowski AJ, Kakimoto PAHB. Autophagy in hepatic steatosis: a structured review [Internet]. Frontiers in Cell and Developmental Biology. 2021 ; 9 1-21 art. 657389.[citado 2026 mar. 31 ] Available from: https://doi.org/10.3389/fcell.2021.657389 - Increased glycolysis is an early consequence of palmitate lipotoxicity mediated by redox signaling
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- Goldilocks calcium concentrations and the regulation of oxidative phosphorylation: too much, too little, or just right
- Bioenergetic and redox signaling in hepatic steatosis models: Integrative analysis of nutritional, hormonal, and inflammatory stimuli
- Mitochondrial Na+/Ca²+ exchanger (NCLX) in nutrient sensing pathways: regulation of autophagy and mTORC1 signaling
- Mitochondrial sodium/calcium exchanger (NCLX) regulates basal and starvation-induced autophagy through calcium signaling
- Changes in mitochondrial morphology modulate LPS-induced loss of calcium homeostasis in BV-2 microglial cells
- Resilient hepatic mitochondrial function and lack of iNOS dependence in diet-induced insulin resistance
- Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on Angiotensin II-Mediated increase in nox isoform expression and activity in vascular smooth muscle cells
- Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation
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