Active transcription start sites of meningioma samples associated with risk of recurrence (2019)
- Authors:
- USP affiliated authors: CARLOTTI JUNIOR, CARLOS GILBERTO - FMRP ; NOUSHMEHR, HOUTAN - FMRP
- Unidade: FMRP
- DOI: 10.1093/neuonc/noz175.460
- Subjects: TRANSCRIÇÃO GÊNICA; MENINGIOMA; NEOPLASIAS CEREBRAIS; EPIGÊNESE GENÉTICA; METILAÇÃO DE DNA
- Language: Inglês
- Imprenta:
- Publisher: Society for Neuro-Oncology
- Publisher place: Oxford
- Date published: 2019
- Source:
- Título: Neuro-oncology
- ISSN: 1522-8517
- Volume/Número/Paginação/Ano: v. 21, suppl. 6, res. GENE-58, p. vi110, 2019
- Conference titles: Annual Scientific Meeting and Education Day of the Society for Neuro-Oncology
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
MALTA, Tathiane et al. Active transcription start sites of meningioma samples associated with risk of recurrence. Neuro-oncology. Oxford: Society for Neuro-Oncology. Disponível em: https://doi.org/10.1093/neuonc/noz175.460. Acesso em: 28 dez. 2025. , 2019 -
APA
Malta, T., Sabedot, T. S., Carlotti Júnior, C. G., & Noushmehr, H. (2019). Active transcription start sites of meningioma samples associated with risk of recurrence. Neuro-oncology. Oxford: Society for Neuro-Oncology. doi:10.1093/neuonc/noz175.460 -
NLM
Malta T, Sabedot TS, Carlotti Júnior CG, Noushmehr H. Active transcription start sites of meningioma samples associated with risk of recurrence [Internet]. Neuro-oncology. 2019 ; 21 vi110.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1093/neuonc/noz175.460 -
Vancouver
Malta T, Sabedot TS, Carlotti Júnior CG, Noushmehr H. Active transcription start sites of meningioma samples associated with risk of recurrence [Internet]. Neuro-oncology. 2019 ; 21 vi110.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1093/neuonc/noz175.460 - A distinct DNA methylation shift in a subset of glioma CpG island methylator phenotypes during tumor recurrence
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- Epigenomic (DNA methylation and expression) signatures define subsets of both IDHmut and IDHwt glioma with distinct clinical outcomes
- Molecular patterns at regulatory elements diferentially methylated in glioma
- Clinical data that matters: a distillation of neuro-oncology clinical trial inclusion criteria using machine learning
- Genomic landscape of idh-mutant primary glioblastomas shows distinct clinical and molecular features and that CDKN2A should be supplemented with MGMTp and G-CIMP for precise prognostication
- Tumor-myeloid cell interactions are dynamic and influence the evolutionary trajectory of adult diffuse glioma
Informações sobre o DOI: 10.1093/neuonc/noz175.460 (Fonte: oaDOI API)
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