Abnormal sterol-induced cell wall glucan deficiency in yeast is due to impaired glucan synthase transport to the plasma membrane (2020)
- Authors:
- USP affiliated authors: SILBER, ARIEL MARIANO - ICB ; VÉLIZ, MAURO JAVIER CORTEZ - ICB
- Unidade: ICB
- DOI: 10.1042/BCJ20200663
- Subjects: PARASITOLOGIA; FUNGICIDAS; PAREDE CELULAR VEGETAL; LEVEDURAS; MEMBRANA PLASMÁTICA; ESTEROIDES; GLUCAGON; SACCHAROMYCES; AMINAS; POLÍMEROS SINTÉTICOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Biochemical Journal
- ISSN: 1470-8728
- Volume/Número/Paginação/Ano: art. BCJ20200663, [39 p.], 2020
- Status:
- Artigo aberto em periódico híbrido (Hybrid Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
ARMIJOS, Leydi Roxana Gutierrez et al. Abnormal sterol-induced cell wall glucan deficiency in yeast is due to impaired glucan synthase transport to the plasma membrane. Biochemical Journal, p. [39 ], 2020Tradução . . Disponível em: https://doi.org/10.1042/BCJ20200663. Acesso em: 01 abr. 2026. -
APA
Armijos, L. R. G., Sussmann, R. A. C., Silber, A. M., Cortez, M., & Lopez, A. H. (2020). Abnormal sterol-induced cell wall glucan deficiency in yeast is due to impaired glucan synthase transport to the plasma membrane. Biochemical Journal, [39 ]. doi:10.1042/BCJ20200663 -
NLM
Armijos LRG, Sussmann RAC, Silber AM, Cortez M, Lopez AH. Abnormal sterol-induced cell wall glucan deficiency in yeast is due to impaired glucan synthase transport to the plasma membrane [Internet]. Biochemical Journal. 2020 ; [39 ].[citado 2026 abr. 01 ] Available from: https://doi.org/10.1042/BCJ20200663 -
Vancouver
Armijos LRG, Sussmann RAC, Silber AM, Cortez M, Lopez AH. Abnormal sterol-induced cell wall glucan deficiency in yeast is due to impaired glucan synthase transport to the plasma membrane [Internet]. Biochemical Journal. 2020 ; [39 ].[citado 2026 abr. 01 ] Available from: https://doi.org/10.1042/BCJ20200663 - The glutamine synthetase of Trypanosoma cruzi is required for its resistance to ammonium accumulation and evasion of the parasitophorous vacuole during host-cell infection
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