Mitochondrial unfolded protein response (UPRmt) activation in cardiac diseases: opportunities and challenges (2019)
- Authors:
- USP affiliated authors: FERREIRA, JULIO CESAR BATISTA - ICB ; BOZI, LUIZ HENRIQUE MARCHESI - ICB ; CAMPOS, JULIANE CRUZ - ICB
- Unidade: ICB
- DOI: 10.1016/j.jacc.2019.05.066
- Subjects: ANATOMIA; DOENÇAS CARDIOVASCULARES; FISIOLOGIA CARDIOVASCULAR
- Language: Inglês
- Imprenta:
- Publisher place: North Carolina
- Date published: 2019
- Source:
- Título: Journal of the American College of Cardiology
- ISSN: 1558-3597
- Volume/Número/Paginação/Ano: v. 74, n. 7, p. 1011-1012, 2019
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
BOZI, Luiz Henrique Marchesi et al. Mitochondrial unfolded protein response (UPRmt) activation in cardiac diseases: opportunities and challenges. Journal of the American College of Cardiology. North Carolina: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.jacc.2019.05.066. Acesso em: 07 nov. 2024. , 2019 -
APA
Bozi, L. H. M., Campos, J. C., Gross, E. R., & Ferreira, J. C. B. (2019). Mitochondrial unfolded protein response (UPRmt) activation in cardiac diseases: opportunities and challenges. Journal of the American College of Cardiology. North Carolina: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.jacc.2019.05.066 -
NLM
Bozi LHM, Campos JC, Gross ER, Ferreira JCB. Mitochondrial unfolded protein response (UPRmt) activation in cardiac diseases: opportunities and challenges [Internet]. Journal of the American College of Cardiology. 2019 ; 74( 7): 1011-1012.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.jacc.2019.05.066 -
Vancouver
Bozi LHM, Campos JC, Gross ER, Ferreira JCB. Mitochondrial unfolded protein response (UPRmt) activation in cardiac diseases: opportunities and challenges [Internet]. Journal of the American College of Cardiology. 2019 ; 74( 7): 1011-1012.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.jacc.2019.05.066 - pH-Gated succinate secretion regulates muscle remodeling in response to exercise
- A selective inhibitor of Cardiac Troponin I Phosphorylation by Delta Protein Kinase C (δPKC) as a treatment for Ischemia-reperfusion injury
- Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling
- Comment on: “Aldehyde dehydrogenases contribute to skeletal muscle homeostasis in healthy, aging, and Duchenne muscular dystrophy patients” by Etienne et al.
- Mitophagy protects against statin-mediated skeletal muscle toxicity
- Mitochondrially-targeted treatment strategies
- β2‐adrenoceptor activation improves skeletal muscle autophagy in neurogenic myopathy
- Exercise preserves physical fitness during aging through AMPK and mitochondrial dynamics
- β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise
- Caracterização da disfunção cardíaca induzida pelo estresse do retículo endoplasmático: papel do treinamento físico aeróbico
Informações sobre o DOI: 10.1016/j.jacc.2019.05.066 (Fonte: oaDOI API)
Download do texto completo
Tipo | Nome | Link | |
---|---|---|---|
Mitochondrial Unfolded Pr... | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas