Investigation of base excision repair gene variants in late-onset Alzheimer’s disease (2019)
- Authors:
- Autor USP: PINTO, NADJA CRISTHINA DE SOUZA - IQ
- Unidade: IQ
- DOI: 10.1371/journal.pone.0221362
- Subjects: DOENÇA DE ALZHEIMER; DANO AO DNA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2019
- Source:
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
ERTUZUN, Tugce et al. Investigation of base excision repair gene variants in late-onset Alzheimer’s disease. Plos one, v. 14, n. 8, p. 1-20 art. e0221362, 2019Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0221362. Acesso em: 20 fev. 2026. -
APA
Ertuzun, T., Semerci, A., Cakir, M. E., Ekmekcioglu, A., Gok, M. O., Soltys, D. T., et al. (2019). Investigation of base excision repair gene variants in late-onset Alzheimer’s disease. Plos one, 14( 8), 1-20 art. e0221362. doi:10.1371/journal.pone.0221362 -
NLM
Ertuzun T, Semerci A, Cakir ME, Ekmekcioglu A, Gok MO, Soltys DT, Souza-Pinto NC de, Sezerman U, Muftuoglu M. Investigation of base excision repair gene variants in late-onset Alzheimer’s disease [Internet]. Plos one. 2019 ; 14( 8): 1-20 art. e0221362.[citado 2026 fev. 20 ] Available from: https://doi.org/10.1371/journal.pone.0221362 -
Vancouver
Ertuzun T, Semerci A, Cakir ME, Ekmekcioglu A, Gok MO, Soltys DT, Souza-Pinto NC de, Sezerman U, Muftuoglu M. Investigation of base excision repair gene variants in late-onset Alzheimer’s disease [Internet]. Plos one. 2019 ; 14( 8): 1-20 art. e0221362.[citado 2026 fev. 20 ] Available from: https://doi.org/10.1371/journal.pone.0221362 - Is mouse mitochondrial DNA protected from alkylation damage by AAG?
- Removal of oxidative DNA damage via FEN1-dependent long-patch base excision repair in human cell mitochondria
- Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions
- PPAR coregulators are essential mediators of mitochondrial function and redox homeostasis in skeletal muscle cells
- ExoMeg1: a new exonuclease from metagenomic library
- Cockayne syndrome group B protein stimulates rapair of formamidopyrimidines by NEIL 1 DNA glycosylase
- The recombination protein RAD52 cooperates with the excision repair protein OGG1 for the repair of oxidative lesions in mammalian cells
- Molecular mechanisms for maintaining mitochondrial DNA stability in Alzheimer's disease
- Cell death induced by photoactive dyes depends on intracellular dye concentration and mitochondrial DNA damage
- Role of mTOR in the regulation of DNA repair activities in human mitochondria
Informações sobre o DOI: 10.1371/journal.pone.0221362 (Fonte: oaDOI API)
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