Role of mTOR in the regulation of DNA repair activities in human mitochondria (2016)
- Authors:
- Autor USP: PINTO, NADJA CRISTHINA DE SOUZA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; DANO AO DNA
- Language: Inglês
- Imprenta:
- Publisher: Associação Brasileira de Mutagênese e Genômica Ambiental
- Publisher place: Ribeirão Preto
- Date published: 2016
- Source:
- Título: Abstracts
- Conference titles: Congresso da MutaGen-Brasil
-
ABNT
FARIA, Caio Matheus Prates Batalha e SOUZA-PINTO, Nadja Cristhina de. Role of mTOR in the regulation of DNA repair activities in human mitochondria. 2016, Anais.. Ribeirão Preto: Associação Brasileira de Mutagênese e Genômica Ambiental, 2016. . Acesso em: 12 fev. 2026. -
APA
Faria, C. M. P. B., & Souza-Pinto, N. C. de. (2016). Role of mTOR in the regulation of DNA repair activities in human mitochondria. In Abstracts. Ribeirão Preto: Associação Brasileira de Mutagênese e Genômica Ambiental. -
NLM
Faria CMPB, Souza-Pinto NC de. Role of mTOR in the regulation of DNA repair activities in human mitochondria. Abstracts. 2016 ;[citado 2026 fev. 12 ] -
Vancouver
Faria CMPB, Souza-Pinto NC de. Role of mTOR in the regulation of DNA repair activities in human mitochondria. Abstracts. 2016 ;[citado 2026 fev. 12 ] - Is mouse mitochondrial DNA protected from alkylation damage by AAG?
- Removal of oxidative DNA damage via FEN1-dependent long-patch base excision repair in human cell mitochondria
- Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions
- PPAR coregulators are essential mediators of mitochondrial function and redox homeostasis in skeletal muscle cells
- ExoMeg1: a new exonuclease from metagenomic library
- Cockayne syndrome group B protein stimulates rapair of formamidopyrimidines by NEIL 1 DNA glycosylase
- The recombination protein RAD52 cooperates with the excision repair protein OGG1 for the repair of oxidative lesions in mammalian cells
- Molecular mechanisms for maintaining mitochondrial DNA stability in Alzheimer's disease
- Cell death induced by photoactive dyes depends on intracellular dye concentration and mitochondrial DNA damage
- Opposing action of NCoR1 and PGC-1 alpha in mitochondrial redox homeostasis
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