CD36 shunts eicosanoid metabolism to repress CD14 licensed interleukin-1β release and inflammation (2018)
- Authors:
- USP affiliated authors: MEIRELLES, ALYNE FÁVERO GALVÃO - FCFRP ; BORDON, KARLA DE CASTRO FIGUEIREDO - FCFRP ; CUPO, PALMIRA - FMRP ; BOLLELA, VALDES ROBERTO - FMRP ; BRAGA, ELIANE CANDIANI ARANTES - FCFRP ; GUIMARÃES, FRANCISCO SILVEIRA - FMRP ; FACCIOLI, LUCIA HELENA - FCFRP ; SORGI, CARLOS ARTERIO - FCFRP
- Unidades: FCFRP; FMRP
- DOI: 10.3389/fimmu.2018.00890
- Subjects: RECEPTORES; INTERLEUCINAS; ADENOSINA TRIFOSFATO; LEUCOTRIENOS B; VENENOS
- Keywords: CD14 receptor; CD36 receptor; Cyclic adenosine monophosphate; Interleukin-1β; Leukotriene B4; Prostaglandin E2; Venom
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 9, art. 890, 2018
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
ZOCCAL, Karina F. et al. CD36 shunts eicosanoid metabolism to repress CD14 licensed interleukin-1β release and inflammation. Frontiers in Immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00890. Acesso em: 05 nov. 2024. -
APA
Zoccal, K. F., Gardinassi, L. G., Sorgi, C. A., Meirelles, A. F. G., Bordon, K. de C. F., Glezer, I., et al. (2018). CD36 shunts eicosanoid metabolism to repress CD14 licensed interleukin-1β release and inflammation. Frontiers in Immunology, 9. doi:10.3389/fimmu.2018.00890 -
NLM
Zoccal KF, Gardinassi LG, Sorgi CA, Meirelles AFG, Bordon K de CF, Glezer I, Cupo P, Matsuno AK, Bollela VR, Arantes EC, Guimarães FS, Faccioli LH. CD36 shunts eicosanoid metabolism to repress CD14 licensed interleukin-1β release and inflammation [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2018.00890 -
Vancouver
Zoccal KF, Gardinassi LG, Sorgi CA, Meirelles AFG, Bordon K de CF, Glezer I, Cupo P, Matsuno AK, Bollela VR, Arantes EC, Guimarães FS, Faccioli LH. CD36 shunts eicosanoid metabolism to repress CD14 licensed interleukin-1β release and inflammation [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2018.00890 - Partial purification and functional characterization of Ts19 Frag-I, a novel toxin from Tityus serrulatus scorpion venom
- Toxin Ts5 from Tityus serrulatus venom induces macrophage activation and cytokine production
- Ts6 and Ts2 from Tityus serrulatus venom induce inflammation by mechanisms dependent on lipid mediators and cytokine production
- Tityus serrulatus venom induces lipid body formation through TLR2 and TLR4
- Tityus serrulatus venom and toxins Ts1, Ts2 and Ts6 induce macrophage activation and production of immune mediators
- Toxins of Tityus serrulatus scorpion venom induce inflammatory mediators in vitro
- PPAR-'gama' activation by Tityus serrulatus venom regulates lipid body formation and lipid mediator production
- Electrophysiological characterization of the first Tityus serrulatus alpha-like toxin, Ts5: evidence of a pro-inflammatory toxin on macrophages
- Macrophage activation and production of immune mediators induced by Tityus serrulatus venom, Ts1 and Ts2
- CD14,TLR2 and TLR4 are essential to macrophages recognize Tityus serrulatus venom and its toxin 1 and to release lipid mediators, IL-6 and TNFA
Informações sobre o DOI: 10.3389/fimmu.2018.00890 (Fonte: oaDOI API)
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