Synergic effects of R715, an antogonist of the bradykinin receptor subtype 1 (BK B1-R) which impaired angiogenesis, and dacarbazine on murine melanoma growth (2005)
- Authors:
- Autor USP: CHAMMAS, ROGER - FM
- Unidade: FM
- Subjects: MELANOMA; BRADICININA; QUIMIOTERÁPICOS; FÁRMACOS; RECEPTORES; RESUMOS (EVENTOS)
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2005
- Source:
- Título: Applied Cancer Research - Supplement
- ISSN: 1808-5539
- Volume/Número/Paginação/Ano: n. 2, p. 98-99, res. P-073, November, 2005
- Conference titles: São Paulo research conference Cancer Today From Molecular Biology to treatment
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ABNT
PINHEIRO, M. C et al. Synergic effects of R715, an antogonist of the bradykinin receptor subtype 1 (BK B1-R) which impaired angiogenesis, and dacarbazine on murine melanoma growth. Applied Cancer Research - Supplement. Ribeirão Preto: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 07 maio 2026. , 2005 -
APA
Pinheiro, M. C., Andrade, L. N. S., Nonogaki, S., Nantel, F., Battistini, B., Sirrois, P., & Chammas, R. (2005). Synergic effects of R715, an antogonist of the bradykinin receptor subtype 1 (BK B1-R) which impaired angiogenesis, and dacarbazine on murine melanoma growth. Applied Cancer Research - Supplement. Ribeirão Preto: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Pinheiro MC, Andrade LNS, Nonogaki S, Nantel F, Battistini B, Sirrois P, Chammas R. Synergic effects of R715, an antogonist of the bradykinin receptor subtype 1 (BK B1-R) which impaired angiogenesis, and dacarbazine on murine melanoma growth. Applied Cancer Research - Supplement. 2005 ;( 2): 98-99.[citado 2026 maio 07 ] -
Vancouver
Pinheiro MC, Andrade LNS, Nonogaki S, Nantel F, Battistini B, Sirrois P, Chammas R. Synergic effects of R715, an antogonist of the bradykinin receptor subtype 1 (BK B1-R) which impaired angiogenesis, and dacarbazine on murine melanoma growth. Applied Cancer Research - Supplement. 2005 ;( 2): 98-99.[citado 2026 maio 07 ] - Bradykinin-dependent pathways are activated within tumor microenvironments new targets for therapy
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