Pharmacological beta-adrenergic receptor activation attenuates neutrophil recruitment by a mechanism dependent on nicotinic receptor and the spleen (2016)
- Authors:
- USP affiliated authors: FERREIRA, SERGIO HENRIQUE - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; CUNHA, THIAGO MATTAR - FMRP
- Unidade: FMRP
- DOI: 10.1007/s10753-016-0372-9
- Subjects: ANTAGONISMO DE DROGAS; LEUCÓCITOS; NEUTRÓFILOS; RECEPTORES ADRENÉRGICOS; BAÇO; NEUROIMUNOMODULAÇÃO
- Keywords: Isoproterenol; Neutrophil; Neuroimmunomodulation; beta-adrenergic receptor; Nicotinic receptor
- Language: Inglês
- Imprenta:
- Source:
- Título: Inflammation
- ISSN: 0360-3997
- Volume/Número/Paginação/Ano: v. 39, n. 4, p. 1405-1413, 2016
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
SILVA, Rangel L. et al. Pharmacological beta-adrenergic receptor activation attenuates neutrophil recruitment by a mechanism dependent on nicotinic receptor and the spleen. Inflammation, v. 39, n. 4, p. 1405-1413, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10753-016-0372-9. Acesso em: 19 fev. 2026. -
APA
Silva, R. L., Castanheira, F. V., Figueiredo, J. G., Bassi, G. S., Ferreira, S. H., Cunha, F. de Q., et al. (2016). Pharmacological beta-adrenergic receptor activation attenuates neutrophil recruitment by a mechanism dependent on nicotinic receptor and the spleen. Inflammation, 39( 4), 1405-1413. doi:10.1007/s10753-016-0372-9 -
NLM
Silva RL, Castanheira FV, Figueiredo JG, Bassi GS, Ferreira SH, Cunha F de Q, Cunha TM, Kanashiro A. Pharmacological beta-adrenergic receptor activation attenuates neutrophil recruitment by a mechanism dependent on nicotinic receptor and the spleen [Internet]. Inflammation. 2016 ; 39( 4): 1405-1413.[citado 2026 fev. 19 ] Available from: https://doi.org/10.1007/s10753-016-0372-9 -
Vancouver
Silva RL, Castanheira FV, Figueiredo JG, Bassi GS, Ferreira SH, Cunha F de Q, Cunha TM, Kanashiro A. Pharmacological beta-adrenergic receptor activation attenuates neutrophil recruitment by a mechanism dependent on nicotinic receptor and the spleen [Internet]. Inflammation. 2016 ; 39( 4): 1405-1413.[citado 2026 fev. 19 ] Available from: https://doi.org/10.1007/s10753-016-0372-9 - Granulocyte-colony stimulating factor (G-CSF)-induced mechanical hyperalgesia in mice: role for peripheral TNFα, IL-1β and IL-10
- Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice
- Kaurenoic acid from Sphagneticola trilobata inhibits inflammatory pain: effect on cytokine production and activation of the NO–Cyclic GMP–protein kinase G–ATP-sensitive potassium channel signaling pathway
- Role of CCR2 de novo expression in neutrophils in experimental arthritis
- Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception
- Investigation of the mechanisms involved in the analgesic effect of choline
- The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia
- The activation of peripheral kappa opioid receptors directly blocks inflammatory hyperalgesia through the stimulation of PI3Kgamma/AKT?NO signaling pathway
- IL-17 mediates articular hypernociception in antigen-induced arthritis in mice
- Granulocyte-colony stimulating factor (G-CSF) induces mechanical hyperalgesia via spinal activiation of MAP kinases and 'PL IND.3'K in mice
Informações sobre o DOI: 10.1007/s10753-016-0372-9 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas