Telomerase variant A279T induces telomere dysfunction and inhibits non-canonical telomerase activity in esophageal carcinomas (2014)
- Authors:
- Zhang, Yuwei
- Calado, Rodrigo do Tocantins
- Rao, Makadev
- Hong, Julie A.
- Meeker, Alan K.
- Dumitriu, Bogdan
- Atay, Scott
- McCormick, Peter J.
- Garfield, Susan H.
- Wangsa, Danny
- Padilla-Nash, Hesed M.
- Burkett, Sandra
- Zhang, Mary
- Kunst, Tricia F.
- Peterson, Nathan R.
- Xi, Sichuan
- Inchauste, Suzanne
- Altorki, Nasser K.
- Casson, Alan G.
- Beer, David G. - University of Michigan (UM)
- Harris, Curtis C.
- Ried, Thomas
- Young, Neal S.
- Schrump, David S.
- Autor USP: RODRIGUES, RODRIGO DO TOCANTINS CALADO DE SALOMA - FMRP
- Unidade: FMRP
- DOI: 10.1371/journal.pone.0101010
- Subjects: CARCINOMA (GENÉTICA); NEOPLASIAS GÁSTRICAS (GENÉTICA)
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2014
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
ZHANG, Yuwei et al. Telomerase variant A279T induces telomere dysfunction and inhibits non-canonical telomerase activity in esophageal carcinomas. PLOS ONE, v. 9, n. 7, p. e101010-1 - e101010-15, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0101010. Acesso em: 31 dez. 2025. -
APA
Zhang, Y., Calado, R. do T., Rao, M., Hong, J. A., Meeker, A. K., Dumitriu, B., et al. (2014). Telomerase variant A279T induces telomere dysfunction and inhibits non-canonical telomerase activity in esophageal carcinomas. PLOS ONE, 9( 7), e101010-1 - e101010-15. doi:10.1371/journal.pone.0101010 -
NLM
Zhang Y, Calado R do T, Rao M, Hong JA, Meeker AK, Dumitriu B, Atay S, McCormick PJ, Garfield SH, Wangsa D, Padilla-Nash HM, Burkett S, Zhang M, Kunst TF, Peterson NR, Xi S, Inchauste S, Altorki NK, Casson AG, Beer DG, Harris CC, Ried T, Young NS, Schrump DS. Telomerase variant A279T induces telomere dysfunction and inhibits non-canonical telomerase activity in esophageal carcinomas [Internet]. PLOS ONE. 2014 ; 9( 7): e101010-1 - e101010-15.[citado 2025 dez. 31 ] Available from: https://doi.org/10.1371/journal.pone.0101010 -
Vancouver
Zhang Y, Calado R do T, Rao M, Hong JA, Meeker AK, Dumitriu B, Atay S, McCormick PJ, Garfield SH, Wangsa D, Padilla-Nash HM, Burkett S, Zhang M, Kunst TF, Peterson NR, Xi S, Inchauste S, Altorki NK, Casson AG, Beer DG, Harris CC, Ried T, Young NS, Schrump DS. Telomerase variant A279T induces telomere dysfunction and inhibits non-canonical telomerase activity in esophageal carcinomas [Internet]. PLOS ONE. 2014 ; 9( 7): e101010-1 - e101010-15.[citado 2025 dez. 31 ] Available from: https://doi.org/10.1371/journal.pone.0101010 - Variações da qPCR e seus usos
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- Exome sequencing reveals a thrombopoietin ligand mutation in a Micronesian family with autosomal recessive aplastic anemia
- U2AF1 and other splicing factor gene mutations in telomere biology disorders are associated with hematologic neoplasia and worse overall survival
- The future of aplastic anemia treatment in Brazil: lessons learned for global hematology
- Androgen treatment mitigates hematopoietic cell telomere attrition in vivo
- Age-adjusted recipient pretransplantation telomere length and treatment-related mortality after hematopoietic stem cell transplantation
- Telomeres in disease
- Células reprogramadas. [Depoimento a Hérika Dias e Luis Ribeiro]
- Telomere shortening in hepatocyte proliferation and predisposition to alcohol-induced steatosis
Informações sobre o DOI: 10.1371/journal.pone.0101010 (Fonte: oaDOI API)
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