Inhalation of fine particulate matter during pregnancy increased IL-4 cytokine levels in the fetal portion of the placenta (2015)
- Authors:
- USP affiliated authors: SALDIVA, PAULO HILARIO NASCIMENTO - FM ; HEIMANN, JOEL CLAUDIO - FM ; FURUKAWA, LUZIA NAOKO SHINOHARA - FM
- Unidade: FM
- DOI: 10.1016/j.toxlet.2014.12.001
- Subjects: RATOS; PRENHEZ; INFLAMAÇÃO; POLUIÇÃO ATMOSFÉRICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Toxicology Letters
- ISSN: 0378-4274
- Volume/Número/Paginação/Ano: v. 232, n. 2, p. 475-480, 2015
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
MELO, Juliana Oliveira de et al. Inhalation of fine particulate matter during pregnancy increased IL-4 cytokine levels in the fetal portion of the placenta. Toxicology Letters, v. 232, n. 2, p. 475-480, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.toxlet.2014.12.001. Acesso em: 11 fev. 2026. -
APA
Melo, J. O. de, Soto, S. de F., Katayama, I. A., Wenceslau, C. F., Pires, A. G., Veras, M. M., et al. (2015). Inhalation of fine particulate matter during pregnancy increased IL-4 cytokine levels in the fetal portion of the placenta. Toxicology Letters, 232( 2), 475-480. doi:10.1016/j.toxlet.2014.12.001 -
NLM
Melo JO de, Soto S de F, Katayama IA, Wenceslau CF, Pires AG, Veras MM, Furukawa LNS, Castro I de, Saldiva PHN, Heimann JC. Inhalation of fine particulate matter during pregnancy increased IL-4 cytokine levels in the fetal portion of the placenta [Internet]. Toxicology Letters. 2015 ; 232( 2): 475-480.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1016/j.toxlet.2014.12.001 -
Vancouver
Melo JO de, Soto S de F, Katayama IA, Wenceslau CF, Pires AG, Veras MM, Furukawa LNS, Castro I de, Saldiva PHN, Heimann JC. Inhalation of fine particulate matter during pregnancy increased IL-4 cytokine levels in the fetal portion of the placenta [Internet]. Toxicology Letters. 2015 ; 232( 2): 475-480.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1016/j.toxlet.2014.12.001 - Maternal renin-angiotensin system (RAS) blockade: Effect on adult offspring
- Sucrose overload during pregnancy and lactation influences blood presure, body weight, and adipose tissue mass in adult offspring
- Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist
- Body weight is influenced by the degree of salt intake and this association is due to changes in energy balance and hormonal profile
- Low salt intake modulates tissue insulin signaling, JNK activity and IRS-1ser307 phosphorylation
- Effect of salt intake on the placental angiotensin-converting enzyme activity (ACE) during pregnancy
- Differential sympathetic and angiotensinergic responses in rats submitted to low- or high-salt diet
- Salt overload during pregnancy alters uterine blood flow, placenta renin angiotensin system (RAS) and placenta weight
- Sucrose overload during pregnancy and lactation influences blood pressure, body weight, and adipose tissue mass in adult offspring
- Salt intake and activated/inactivated renin angiotensin system (RAS) during pregnancy and lactation influences body weight and adipose tissue mass in adult offspring
Informações sobre o DOI: 10.1016/j.toxlet.2014.12.001 (Fonte: oaDOI API)
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