High-Salt Intake Induces Cardiomyocyte Hypertrophy in Rats in Response to Local Angiotensin II Type 1 Receptor Activation1–3 (2014)
- Authors:
- USP affiliated authors: SHIMIZU, MARIA HELOISA MASSOLA - FM ; FURUKAWA, LUZIA NAOKO SHINOHARA - FM ; HEIMANN, JOEL CLAUDIO - FM
- Unidade: FM
- DOI: 10.3945/\200Bjn.114.192054
- Subjects: RATOS WISTAR; DIETA ANIMAL; FIBROSE CÍSTICA; FATORES DE RISCO; SAL (CONDIMENTO)
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2014
- Source:
- Título: The Journal Nutrition
- ISSN: 0022-3166
- Volume/Número/Paginação/Ano: v. 144, n. 10, p. 1571-1578, 2014
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
KATAYAMA, Isis A. et al. High-Salt Intake Induces Cardiomyocyte Hypertrophy in Rats in Response to Local Angiotensin II Type 1 Receptor Activation1–3. The Journal Nutrition, v. 144, n. 10, p. 1571-1578, 2014Tradução . . Disponível em: https://doi.org/10.3945/\200Bjn.114.192054. Acesso em: 11 fev. 2026. -
APA
Katayama, I. A., Pereira, R. C., Dopona, E. P. B., Shimizu, M. H. M., Furukawa, L. N. S., Oliveira, I. B., & Heimann, J. C. (2014). High-Salt Intake Induces Cardiomyocyte Hypertrophy in Rats in Response to Local Angiotensin II Type 1 Receptor Activation1–3. The Journal Nutrition, 144( 10), 1571-1578. doi:10.3945/\200Bjn.114.192054 -
NLM
Katayama IA, Pereira RC, Dopona EPB, Shimizu MHM, Furukawa LNS, Oliveira IB, Heimann JC. High-Salt Intake Induces Cardiomyocyte Hypertrophy in Rats in Response to Local Angiotensin II Type 1 Receptor Activation1–3 [Internet]. The Journal Nutrition. 2014 ; 144( 10): 1571-1578.[citado 2026 fev. 11 ] Available from: https://doi.org/10.3945/\200Bjn.114.192054 -
Vancouver
Katayama IA, Pereira RC, Dopona EPB, Shimizu MHM, Furukawa LNS, Oliveira IB, Heimann JC. High-Salt Intake Induces Cardiomyocyte Hypertrophy in Rats in Response to Local Angiotensin II Type 1 Receptor Activation1–3 [Internet]. The Journal Nutrition. 2014 ; 144( 10): 1571-1578.[citado 2026 fev. 11 ] Available from: https://doi.org/10.3945/\200Bjn.114.192054 - Low birth weight in response to salt restriction during pregnancy is not due to alterations in uterine-placental blood flow or the placental and peripheral renin-angiotensin system
- N-acetyl-L-cysteine exacerbates kidney dysfunction caused by a chronic high-sodium diet in renal ischemia and reperfusion rats
- Maternal renin-angiotensin system (RAS) blockade: Effect on adult offspring
- Sucrose overload during pregnancy and lactation influences blood presure, body weight, and adipose tissue mass in adult offspring
- Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist
- Body weight is influenced by the degree of salt intake and this association is due to changes in energy balance and hormonal profile
- Low salt intake modulates tissue insulin signaling, JNK activity and IRS-1ser307 phosphorylation
- Effect of salt intake on the placental angiotensin-converting enzyme activity (ACE) during pregnancy
- Differential sympathetic and angiotensinergic responses in rats submitted to low- or high-salt diet
- Salt overload during pregnancy alters uterine blood flow, placenta renin angiotensin system (RAS) and placenta weight
Informações sobre o DOI: 10.3945/\200Bjn.114.192054 (Fonte: oaDOI API)
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