Dietary restriction in cerebral bioenergetics and redox state (2014)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.redox.2013.12.021
- Subjects: MITOCÔNDRIAS; DOENÇAS NEUROMUSCULARES
- Language: Inglês
- Imprenta:
- Publisher place: Montevideo
- Date published: 2014
- Source:
- Título: Redox Biology
- ISSN: 2213-2317
- Volume/Número/Paginação/Ano: v. 2, p. 296-304, 2014
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc-nd
-
ABNT
AMIGO, Ignacio e KOWALTOWSKI, Alicia Juliana. Dietary restriction in cerebral bioenergetics and redox state. Redox Biology, v. 2, p. 296-304, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2013.12.021. Acesso em: 08 out. 2024. -
APA
Amigo, I., & Kowaltowski, A. J. (2014). Dietary restriction in cerebral bioenergetics and redox state. Redox Biology, 2, 296-304. doi:10.1016/j.redox.2013.12.021 -
NLM
Amigo I, Kowaltowski AJ. Dietary restriction in cerebral bioenergetics and redox state [Internet]. Redox Biology. 2014 ; 2 296-304.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.redox.2013.12.021 -
Vancouver
Amigo I, Kowaltowski AJ. Dietary restriction in cerebral bioenergetics and redox state [Internet]. Redox Biology. 2014 ; 2 296-304.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.redox.2013.12.021 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.redox.2013.12.021 (Fonte: oaDOI API)
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