Mitochondrial ATP sensitive potassium channel opening cardiac hypertrophy by augmenting antioxidant defenses (2015)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; ESTRESSE OXIDATIVO; ANTIOXIDANTES
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
- Publisher place: São Paulo
- Date published: 2015
- Source:
- Título: Abstracts Book
- Conference titles: Congress of the International Union for Biochemistry and Molecular Biology - IUBMB
-
ABNT
CALDAS, Francisco Rodrigo Lemos et al. Mitochondrial ATP sensitive potassium channel opening cardiac hypertrophy by augmenting antioxidant defenses. 2015, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2015. . Acesso em: 04 out. 2024. -
APA
Caldas, F. R. L., Leite, I. M. R., Figueiras, A. B. T., Figueiredo Junior, I. L. de, Souza, T. A. G. M., Martins, P. R., et al. (2015). Mitochondrial ATP sensitive potassium channel opening cardiac hypertrophy by augmenting antioxidant defenses. In Abstracts Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). -
NLM
Caldas FRL, Leite IMR, Figueiras ABT, Figueiredo Junior IL de, Souza TAGM, Martins PR, Kowaltowski AJ, Facundo HDTF. Mitochondrial ATP sensitive potassium channel opening cardiac hypertrophy by augmenting antioxidant defenses. Abstracts Book. 2015 ;[citado 2024 out. 04 ] -
Vancouver
Caldas FRL, Leite IMR, Figueiras ABT, Figueiredo Junior IL de, Souza TAGM, Martins PR, Kowaltowski AJ, Facundo HDTF. Mitochondrial ATP sensitive potassium channel opening cardiac hypertrophy by augmenting antioxidant defenses. Abstracts Book. 2015 ;[citado 2024 out. 04 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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