Characterization of Cross Reactive Autoantigens in Rheumatic Heart Disease Using Phage Display Methodology (2006)
- Authors:
- Autor USP: KALIL FILHO, JORGE ELIAS - FM
- Unidade: FM
- DOI: 10.1016/j.clim.2006.04.136
- Subjects: PROTEÍNAS; MIOSINA; FEBRE REUMÁTICA; AUTOANTÍGENOS; CARDIOPATIA REUMÁTICA; AUTOIMUNIDADE; RESUMOS (EVENTOS)
- Language: Inglês
- Imprenta:
- Source:
- Título: Clinical immunology
- ISSN: 1521-6616
- Volume/Número/Paginação/Ano: v. 119, suppl., p. S84-S85, res. F.96, 2006
- Conference titles: FOCIS: Annual Meeting of the Federation of Clinical Immunology Societies
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
BESSA, Juliana et al. Characterization of Cross Reactive Autoantigens in Rheumatic Heart Disease Using Phage Display Methodology. Clinical immunology. Orlando: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.clim.2006.04.136. Acesso em: 19 out. 2024. , 2006 -
APA
Bessa, J., Giordano, R., Arap, W., Pasqualini, R., Kalil, J., & Guilherme, L. (2006). Characterization of Cross Reactive Autoantigens in Rheumatic Heart Disease Using Phage Display Methodology. Clinical immunology. Orlando: Faculdade de Medicina, Universidade de São Paulo. doi:10.1016/j.clim.2006.04.136 -
NLM
Bessa J, Giordano R, Arap W, Pasqualini R, Kalil J, Guilherme L. Characterization of Cross Reactive Autoantigens in Rheumatic Heart Disease Using Phage Display Methodology [Internet]. Clinical immunology. 2006 ; 119 S84-S85.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/j.clim.2006.04.136 -
Vancouver
Bessa J, Giordano R, Arap W, Pasqualini R, Kalil J, Guilherme L. Characterization of Cross Reactive Autoantigens in Rheumatic Heart Disease Using Phage Display Methodology [Internet]. Clinical immunology. 2006 ; 119 S84-S85.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/j.clim.2006.04.136 - Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target
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Informações sobre o DOI: 10.1016/j.clim.2006.04.136 (Fonte: oaDOI API)
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