Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target (2002)
- Authors:
- USP affiliated authors: KALIL FILHO, JORGE ELIAS - FM ; BENVENUTI, LUIZ ALBERTO - FM ; NORONHA, IRENE DE LOURDES - FM ; COELHO, VERÔNICA PORTO CARREIRO DE VASCONCELLOS - FM
- Unidade: FM
- DOI: 10.1016/s0966-3274(02)00032-1
- Subjects: PATOLOGIA; IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Elsevier
- Volume/Número/Paginação/Ano: n. 9, p. 101-110, 2002
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
SUÁREZ, Idania Marrero et al. Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target. Elsevier, n. 9, p. 101-110, 2002Tradução . . Disponível em: https://doi.org/10.1016/s0966-3274(02)00032-1. Acesso em: 11 fev. 2026. -
APA
Suárez, I. M., Noronha, I., Benvenutti, L. A., Kaer, L. V., Kalil, J., & Coelho, V. (2002). Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target. Elsevier, ( 9), 101-110. doi:10.1016/s0966-3274(02)00032-1 -
NLM
Suárez IM, Noronha I, Benvenutti LA, Kaer LV, Kalil J, Coelho V. Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target [Internet]. Elsevier. 2002 ;( 9): 101-110.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1016/s0966-3274(02)00032-1 -
Vancouver
Suárez IM, Noronha I, Benvenutti LA, Kaer LV, Kalil J, Coelho V. Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target [Internet]. Elsevier. 2002 ;( 9): 101-110.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1016/s0966-3274(02)00032-1 - Locally produced IL-15 may underlie the predominance of CD8+IL2RALPHA- T cells ar heart lesions of human Chagas Disease cardiomyopathy
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Informações sobre o DOI: 10.1016/s0966-3274(02)00032-1 (Fonte: oaDOI API)
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